Muskuloskeletal Disorders Flashcards

(55 cards)

1
Q

-what is Developmental Dysplasia of Hip
- s/s in newborn vs older childres

A
  • hip instability after birth; usually associated with breech delivery
  • assymetry and unequal number of skin fold on posterior thighs/glutes
  • older child: limp, leg discrepancy
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2
Q

diagnostic evaluation for DDH

A

Ortolani and Barlow Test
ortolani- outward (abduction)
barlow- inward (adduction)

Ultrasound prior to 4 months of age
X ray after 4-6 months

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3
Q

populations at risk for Developmental Hip Dysplasia

A

female, history of DDH, oligohydramnios, high birth weight, firstborn

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4
Q

Pavlic Karness vs. casting for Dysplasia of hip

A

pavlic: prevents hip flextion and adduction
- skin care is important
- cothing and diaper should be under the straps

cast: for 12 weeks

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5
Q

what is the treatment for developmental dysplacia of hip in infants under 6 months

A

Pavlic Harness
- >6 months need surgery

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6
Q

how is Osteogenesis Imperfecta inherited

A

Autosomal dominant inherited disorder

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7
Q

what does osteogenesis imperfecta cause in the body
and what are s/s

A

extremely brittle bones, fractures, and bone deformities
s/s: BLUE sclerae, hearing loss, discolored teeth

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8
Q

how is osteogenesis imperfecta diagnosed and how is it treated

A

bone biopsy
-biphosphates, physical therapy, braces and splints,

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9
Q

what is congenital club foot

A

deformity of ankle and foot, involves bone and soft tissue contracture

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10
Q

how is congenital clubfoot diagnosed

A

diagnosed prenatally

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11
Q

symptoms of congenital clubfoot

A

affected foot is smaller,limb shorter, calf atrophy

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12
Q

what other disease can co-occur with congenital clubfoot

A

cerebral palsy, spina bifida, chromosomal defects

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13
Q

what populations is more likely to get congenital clubfoot

A

boys are affected 2x as often

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14
Q

how is congenital club foot diagnosed and treated

A

-physical exam
- serial casting until corrected
- if does not help by 3 months then surgery between 6-12 months of age
-abduction bracing at night for 3-5 years

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15
Q

an infant born with unilateral congenital clubfoot is at higher risk of having…..

A

chromosomal defects

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16
Q

scoliosis screening

A

Adam’s bend test: asymmetry of should height, scapular, or flank shape, or hip height
Standing radiographs to determine degree of curvature

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17
Q

therapeutic management for scoliosis

A

exercise, bracing, surgical intervention for severe curvature

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18
Q

what percent does a scoliosis curvature need to be to be considered severe and need surgical intervent.

A

over 25 degrees

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19
Q

what is Legg-Calve-Perthes

A

avascular necrosis of femoral head

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20
Q

clinical manifestations of Legg-Calve

A

delayed bone age, painless limp, stiffness of hip, decreased ROM, hip/thigh/knee pain, shortening ofleg affected

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21
Q

how do you test bone age

A

left wrist x-ray starting at age 3

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22
Q

diagnostic measure for Legg-Calve

A

XRAY, MRI

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23
Q

Treatment for Legg-Calve

A

initially-rest, limited weight bearing to reduce inflammation, traction to stretch the tight adductor muscles
meds- NSAIDS, PT, ROM exercises

24
Q

what population ismorelikely to be affected by Legg-Calve

25
what is Slipped Capital Femoral Epiphysis
spontaneous displacement of the proximal femoral epiphysis in a posterior and inferior direction ball of thight bone (femoral head) slips off the neck of thigh bonebecuase growth plate is damaged
26
clinical manifestations of Slipped Capital Femoral Epiphysis
limp, pain, episode of trauma with acute displacement, gradual displacement without definite injury
27
who is more at risk to get slipped capital femoral epiphysis
obesity, puberty hormone changes, bone changes
28
treatment for SCFE
goal: keep head of femur in acetabulum - rest - no weight bearing - sometimes surgery
29
What is Juvenile Idiopathic Arthritis
chronic childhood arthritis (last for 6 weeks or longer) chronic synovial inflammation with joint effusion, destruction, and destruction of cartilage
30
when is a child most likely to develop Juvenile Idiopathic Arthritis
Before 16 yrs Peaks- 1 to 3 years and 8 to 10 years
31
clinical manifestation of JIA
90% of children have negative rheumatoid factor stiffness, swelling, warm to touch, loss of mobility in affected joints, growth retardation
32
Meds and treatment for JIA
Meds: NSAIDS, antirheumatic drugs, tumor necrosis factor alpha inhibitors Tx: PT/OT to strengthen muscles, mobilize joints, splinting
33
4 criteria for diagnosis of JIA
1. age of onset is >16 yrs 2. One or more affected joints 3. Duration is >6 weeks 4. Exclusion of others forms of arthritis
34
diagnostit procedure for JIA
XRAY- rules out fractures
35
what is an EMG
diagnostic measure to assess the health of muscles and the nerve cells that control them (motor neurons)
36
What is a CPK test
creatine phosphokinase (CPK) - most specific test for muscular dystrophy - elevated CPK levels indicate muscle disease
37
If you have upper motor neuron lesions this is primarily going to be___
cerebral palsy
38
what is the most common permanent physical disability in childhood
cerebral palsy
39
etiology of Cerebral Palsy
any perinantal/ neonatal brain lesions prenatal infection, hypoxia, asphyxia, preterm birth
40
what is spastic cerebral palsy
most common type presents hypotonia
41
Quadriparesis Cerebral Palsy
4 extremities are involved - speech and swallowing difficulties - tongue protrusion - labile (eratic) emotions in some
42
developmental signs of CP
-poor head control after age 3 months - stiff/rigid limbs - unable to sit without support at age 8 months - no smiling by age 3 months - feeding difficulties
43
management of CP
mobilizations, meds, seizure control
44
what percent of CP patients have a normal IQ
50-60%
45
therapy goals for CP
- establish locomotion, communication, and self-help - gain optimum integration of motor function - correct defects are early and affectively aspossible
46
meds to treat spasticity in CP
diazepam and baclofen -muscle relaxant valium- pain med botox
47
signs of CP freqently present:
-delayed developmental milestones - hyper or hypotonicity - arching (feet, position) - feeding difficulty
48
muscular dystrophies all have a genetic origin with
gradual degeneration of muscle fibers, progressive weakness, and wasting of skeletal muscles
49
genetic inheritance of Duchene Muscualar Dystr
x-linked (males are most exclusively affected)
50
cause of Duchene MD
dystrophin protein is absent
51
characteristic of Duchene MD
delayed motor development esp walking - muscle weakness begins 3-5 yrs old -progressive muscle weakness -calf muscle hypertrophy
52
labs and diagnostic test for Duchene MD
Labs:Serum CPK and AST high in first 2 years of life Test:confirmed by EMG, muscle biopsy,
53
what condition presents with Lordosis
Duchene MD - accentuated lower back curve, belly pushing out
54
management of Duchene MD
no curative treatment - maintain function in unaffected muscles as long as possible - so do ROM, bracing, perform ADLs - meds:corticosteroids like prednisone to increase muscle strength
55
in what conditions do children tend to be overweight or obese
Duchene MD and Legge-Calve