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ESA 5 > Neuro extra stuff > Flashcards

Neuro extra stuff Flashcards

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1
Q

Describe the layers of the cerebellum

A 
VI layers then White matter.
VI to thalamus
V to subcortical
I-IV to cortex
Thalamus to IV
BS to all
Cortex to I-IV
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2
Q

Parietal dominant function?

A

Speech (Brocas) and language
Logic - calculation
Problem solving
Motor skills

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3
Q

Lesion in anterior frontal results in?

A 
Loss of personality
Apathy
Asociality
Loss of social rules/ inhibition
Euphoria without feeling so
amoral
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4
Q

Temporal lesion

A

Memory e.g. people/ agnosias/ recognition

Speech

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5
Q

Parietal non dominant function

A 
Emotion of language
Music/art
Visiospatial
Body awareness
Whole pucture
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6
Q

How are the left and right sides of the brain connected?

A

Corpus callosum and anterior and posterior commissures

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7
Q

Where does the interpretation of written and spoken word occur?

A

Wernicke’s area temporal lobe

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8
Q

Function of the angular gyrus?

A

Written word interpretation from occipital to Wernicke’s via angular gyrus

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9
Q

How does information pass from Wernicke’s area to Brocs’s?

A

Via the arcuate fasciculus

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10
Q

What are the types of memories and where are they stored? What feature of neurones allows this to happen?

A

Declarative- cortex, hippocampus and other cortical regions
Procedural - Cerebellum, basal ganglia, pre-motor cortex
neuronal plasticity

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11
Q

Storage of memory?

A

Intermediate = seconds allowing sense of present
Short = seconds to minutes = working memory
Transfers to long spontaneously or based on emotion, rehersal association.
Consolidation occurs via long term potentiation

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12
Q

How does memory formation occur? how do we forget?

A

Sensory info to sensory cortex. Then forms a loops with amygdala/ hippo/ thalamus/ basal forebrain/ pre frontal cortex and back.
Long term potentiation with NDMA glutamate receptors]

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13
Q

Result of the destruction of the hippocampus?

A

Antereograde amnesia

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14
Q

What is amnesia? Causes

A

Vascular interrution
Trauma
Infections, Vit B def
Retrograde in alzheimers

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15
Q

What is dementia

A

An aquires loss of global brain function that is significant enough to effect dailey function and quality of life
Loss includes intellect reason and personalit without loss in consciousness (with delerium there is)

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16
Q

Symptoms of dementia

A 
Loss of progressive:
Memory
intellect
personality
behaviour
speech
movement (bradykinesia)
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17
Q

Causes of dementia briefly

A 
Vascular (20%)
ALzheimers
Lewi bodies
Fronto-temporal dementia
Drugs
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18
Q

Protein and the deal with Alx

A

B amyoid deposition
Neurofibillary tangles
Prognosis 5 years
AGe and female = RF

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19
Q

Protein and the deal with lewi bodies

A 
alpha synuclein
prognosis 8 years
REM bleep behaviour disorder fighting in sleep
Delusions 
paranoia
Later it resembled alzheimers dementia
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20
Q

Protein and the deal with fronto-temporal dementia

A 
T Tau
Prog = 8 years
Behaviour and personality loss
Semantic dementia (loss of meaning from language/ words)
Progressive non-fluent aphasia
Disinhibitions
lOss motivation
Lack of empathy
Repetitive
Convulsive behaviours
Craving of sweet, fatty, alcohol, ciggs
Loss of table etiquette
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21
Q

Describe early dementia

A

Loss of memory for recent events
Global distruption of personality
Gradual change in behaviour

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22
Q

describe intermediate dementia

A

Loss of intellect
Mood changes
Cognitive failure to learn

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23
Q

Describe late dementia

A

Reduction in self care
REstless wandering
Incontinence

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24
Q

Differentials for dementia

A

Delerium

Depression

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25
What is normal pressure hydrocephalus and when does it occur
Symptoms = Dementia, incontinence and gait disturbance Ventriculomegally (caused byu build up of CSF?) Rare
26
Which layers does meningitis affect?
Leptomeninges = Pia and arachnoid
27
Causative organisms of meningitis
``` Neonates = Listeria Monocytogenes, E coli. 1-5 = H influenzae B (less now due to vaccine) 5-30 = N meningitidis >30 = S pneumoniae ```
28
What is encephalitis
Viral infection of brain parachyma.
29
Where does herpres virus often infect? result?
Temporal lobes | Epilepsy
30
Where does polio and rabies often occur?
``` Rabies = BS Polio = Spinal cord ```
31
What is perivascular cuffing
Aggregation of lymphocytes perivascularly in encephalitis
32
What is tomoplasmosis?
Toxoplasma gondii causes Primary = flue, muscle aches, lymph nodes. Later eye problems, seizures, coordination
33
How do Prions form and cause damage
Mutated proteins Ingested proteins that then affect own proteins (post translational conformational change) May be famililal or sporadic mutations. Form very stable structures and aggregate and cause neuronal death- spongiform encephalopathies
34
Pressure in brain and coughing? How long can compensation occur?
0-10mmhg coughing + 20 up to 60
35
Describe subfalcine herniation
Cingulate gyrus through falx cerebri. Ischemia of frontal and pariatal and corpus callosum
36
Describe tentorial herniatation
Uncus and parahippocampus through tentorial notch. Damage to CN3 and occlusion of PCA and superior cerebellar arteries. Can cause Duret haemorrhage in midbrain/pons
37
Describe tonsilar herniation
Cerebellar tonsils through foramen magnum compessing the brainstem - CVS apnoea
38
What are prodramal symptoms of raised ICP?
Intitial Headache VOmiting Papilloedema
39
Acute phase of raised ICP
Pupil dilation Coma (BS) Possiibly hemiparesis from cerebellum LAter = BS
40
Give examples of brain tumours
Meningioma bengin Malignant = astrocytoma Lymphoma Most are mets (lung)
41
Difference between focal damage and diffuse damage in trauma
``` Focal = bruising and laceration- tearing of BV and contsions Diffuse = tearing of axons as they go between tissues of varying densiities e.g. white nad gray, pit stalk (DAI) leads to gliotic scarring and persistent vegitative state ```
42
Types of infarct in brain?
Regional e.g. named vesssel | Lacuna - less than 1cm affected with hypertension often in basal ganglia./
43
Difference between falx cerbri and falx cerebelli?
Falx cerebelli posterior | Falx cerebri superior
44
What makes MRI better than CT?
Less radiation Multiple planes Free from artifacts Higher defintion
45
Describe PET scans and their use
``` Maps Biochemistry Positron emission tomography Uses positron emitting pharamceuticles e.g. L dopa Hypometabolic areas e.g. siezures Dementia ```
46
Decribe EEG uses
Electroencephalogram Diagnose brain death Epilepsy to select appropriate anticonvulsants
47
Describe electrophysiological-evoked potentials and types
Stimulation of nerve pathways and monitored and compared to background EEG VER (visual evoked response) - optic neuropathy Auditory Somatosensory (SER) - peripheral nerve stim (can be used in neurosurgery)
48
What is electromyography and its uses
Needle into muscle to record electrical activity e.g. fibrillation potentials = hyperexcitability following dennervation or fasciculation potentials
49
T1 and T2 MRIs and water/ fat
WW2 Water white T2 T1 = fat white
50
How is taste different from flavour?
Flavour includes smell | Taste for pattern of stimulation of different neurones
51
How does tastes vary on tongue
All areas detect all types of taste: Sweet sour, bitter, salty and umami Deficient sweet and salt at tip Deficient sour and bitter at back
52
Emotiona components and outputs
Fornix, mammary body, anterior nuclei of thalamus, hippocampus. Outputs: Autonomic RF Cortex Fronto-sensory cortex (emotional behaviour)
53
What is dioptic strength and what does it depend on?
1/focal length(m) Measures strength of lens Length changed by intraocular pressure (300-325mmHg)
54
Production and drainae of intraocular fluid? What happens if it goes wrong?
Produced by ciliary body and absorbed by Schlemm (venous sinuses)
55
Corrections for short and long sighted
``` Short = myopia = concave Long = hyperopia - convex ```
56
What is presbyopia?
long sighted with age | Progressive
57
Does accommodation in eye?
Ciliary muscle contracts to Increase convexity and allow nearer objects to be seen
58
What is the near point?
Closest point at which an object can be seen
59
What is visual acuity and what determines?
2 point discrim | Density and receptive fields
60
What is a pathological blind spot called?
Scotomata
61
How is colour vision tested?
Ishihara chart
62
Definition of trichonmats and dichromats
3 vs 2 cones
63
Colour deficit tyoes in trichromatis
Protanomaly red Deutanolaly green Tritanomaly blue
64
Loss of cones in dichromats
Protanopia red Deutanopia green tritanopia blue
65
Why do after images occur
Sensitises to opposite colour | See complementary colour
66
Descrube le fort fractures
1 = maxillary horizontal above teeth 2 = horizontal at nose and verical in miaxillar at orbit = separation of maxilla 3 Horizontal nose sphenoid and frontozygomatic arches/ suture

ESA 5 (4 decks)

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