Neuro Radiology Flashcards

(69 cards)

1
Q

What is going on in this patient’s noncontrast CT?

A

This is also subarachnoid hemorrhage, it is just at a lower level!

Don’t let them trick you by giving you an out-of-frame view of a subarachnoid hemorrhage

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2
Q

What is going on in this patient’s noncontrast CT?

A

To be honest this is tricky, it looks a lot like a single calcified mass.

But this particular patient has an intraventricular hemorrhage. These occur due to tearing of subependymal veins.

The best place to look for intraventricular hemorrhage is in the occipital horns of the lateral ventricles. Commonly associated with injury to the corpus callosum. It should be noted that the choroid plexus often calcifies and thus can look quite dense, and should not be mistaken for intraventricular hemorrhage.

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3
Q

What is going on in this patient’s noncontrast CT?

A

This patient has intraparenchymal hemorrhage

It can be due to trauma as well as other causes such as hypertensive hemorrhage, a hemorrhagic stroke, a ruptured AVM, or a hemorrhagic neoplasm.

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4
Q

Major causes of the five main types of head bleed (epidural, subdural, subarachnoid, intraventricular, intraparenchymal)

A
  • Epidural: Middle meningeal artery tear
  • Subdural: Bridging vein tear
  • Subarachnoid: Ruptured aneurysm
  • Intraventruclar: Subependymal vein tear
  • Intraparenchymal: No specific vessel, often in setting of trauma

Note that of all of these, only epidural and subarachnoid are arterial bleeds

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5
Q

What is going on in this C-spine CT?

A

Spinous process facture, aka Clay Shoveler’s fracture

An avulsion of the spinous process due to pulling from the trapezius muscle. Treatment consists of pain medication and physical therapy.

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6
Q

Five low-risk criteria for C-spine imaging

A
  • C-spine imaging is required in all blunt trauma cases unless the patient meets all of these criteria:
    • Absence of posterior midline cervical-spine tenderness
    • No evidence of intoxication
    • Normal level of alertness
    • Absence of focal neurological deficit
    • No clinically apparent painful injuries that might distract from pain of a cervical spine injury
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7
Q

Why is it subarachnoid hemorrhage specifically that can cause vasospasm?

A

Because subarachnoid hemorrhage surrounds cerebral blood vessels.

This is usually seen in the setting of diffuse subarachnoid hemorrhage secondary to ruptured aneurysm rather than trauma, and typically manifests several days after the subarachnoid hemorrhage has occurred

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8
Q

What is going on in this MRI?

A

Diffuse axonal injury with blurring of the gray-white junction and small foci of petechial hemorrhage in white matter tracts

CT is not very sensitive for detecting DAI – MRI is required for diagnosis.

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9
Q

What is going on in this noncontrast CT?

A

Acute subdural hemorrhage

You can tell it is acute because the blood is bright

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10
Q

What is going on in this noncontrast CT?

A

Chronic subdural hemorrhage

You can tell by the color of the coagulated and fibrosing blood. This bleed has been maturing for at least 3 weeks to obtain this apperance.

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11
Q

What is going on in this noncontrast CT?

A

This one is really tough. Based on the ventricles, you can tell that there is a mass lesion, but it is almost impossible to differentiate from the gray matter of the cortex.

This is a subacute subdural hemorrhage. In the process of maturation, from ~3 days - 3 weeks, this is how a subdural hemorrhage will appear – isodense with gray matter.

Often your clinical suspicion for subacute SDH should be triggered when there is evidence of mass effect and altered mental status, but the CT generally looks normal except that the gray matter looks too thick.

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12
Q

Major brain fissures and sulci

A
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13
Q

Normal saggital T1 weighted midline brain MRI

A

For orientation purposes, anterior is to your left (A). The corpus callosum (CC) is located superiorly. The pituitary gland (P) sits in the sella turcica and connects to the hypothalamus via the pituitary stalk or infundibulum ( dotted white arrow ). The mammillary bodies (M) are located anterior to the brainstem. The cerebral aqueduct ( solid white arrow ) is superior to the midbrain. The brainstem is composed of the midbrain (Mi), the pons (Po), and the medulla (Me). The fourth ventricle (4) communicates with the cerebral aqueduct and lies between the cerebellum (Ce) and the brainstem.

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14
Q

Axial MRI T1/T2 of deep gray structures in the brain

A

Axial T1-weighted (A) and T2-weighted (B) images of the brain demonstrate that CSF in the lateral ventricles is dark on T1 and bright on T2 ( solid black arrows ).

Gray matter, which contains the neuronal cell bodies, is actually gray on T1-weighted images ( open white arrow ) and white matter, which contains myelinated axon tracts, is whiter ( white circle ). The caudate nuclei ( solid white arrows ) and lentiform nucleus ( dotted white arrows ) together form the basal ganglia. The thalamus ( dotted black arrows ) is located posterior to the basal ganglia.

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15
Q

Lentiform nucleus

A

The lentiform nucleus is composed of the putamen (laterally) and globus pallidus (medially).

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16
Q

Finding the caudate, thalamus, and lentiform nucleus

A
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17
Q

What is going on in this saggital MRI?

A

A pineal gland tumor is seen compressing the midbrain and other surrounding structures

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18
Q

Finding the pineal gland

A

It is easiest to find on saggital view, but can be found axially by finding the top of the cerebellum and looking just anteriorly

Occasionally there will be benign pineal calcifications

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19
Q

You’re not going to see a skull fracture unless you. . .

A

. . . put on the bone window

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20
Q

Blow-out fracture

A

Most common fracture of the orbit. Caused by direct eye trauma.

It is a fracture of the inferior orbital floor leading into the maxillary sinus, or the medial wall of the orbit leading into the ethmoid sinus. Sometimes this fracture produces inferior rectus entrapment syndrome (restriction of upward gaze, diplopia).

Helpful signs to suggest this fracture may include: orbital emphysema, apparent soft tissue/fat entrapment in the superior maxillary sinus, blood in the maxillary sinus.

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21
Q

Tripod fracture

A

Another common eye fracture. Usually the result of blunt force to the cheek

Involves separation of the zygoma from the remainder of the facial bones by separation of the frontozygomatic suture, fracture of the floor of the orbit, and fracture of the lateral wall of the ipsilateral maxillary sinus

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22
Q

Almost all epidural hematomas (>95%) have. . .

A

. . . an associated skull fracture

Often in the temporal bone

In contrast, subdural hematomas usually are not.

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23
Q

Vasogenic edema affects ___.

Cytotoxic edema affects ___.

A

Vasogenic edema affects primarily white matter. White-gray differentiation is preserved.

Cytotoxic edema affects white matter and gray matter. White-gray differentiation is often lost.

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24
Q

60% of hypertensive hemorrhagic strokes occur in . . .

A

. . . the basal ganglia

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25
In the setting of SAH, how can you tell where the likely aneurysm was?
Where there is the most blood!
26
Beam hardening artifact
Bright/dark bands due to a hard surface
27
Partial volume averaging
Don't be fooled into thinking these are bleeds! They will occur near the skull base Happens when the computer averages bright bone with dull cortex.
28
Brain cisterns
Suprasellar cistern Sylvian cisterns
29
How can you tell if there is herniation through the foramen magnum?
The basal cistern is lost! You won't be able to find it
30
What comes out of the suprasellar cistern laterally?
The MCA! This is where you look for them If hyper dense, "dense MCA sign", this suggests a clot is present in the artery
31
What is going on in this noncontrast head CT?
**Brain abscess** Note the **vasogenic edema** with clear gray-white differentiation The fibrous walls of the abscess are the same density as the gray matter. They may be quite thin or quite thick, so don't let their density fool you.
32
Intra-axial = \_\_ Extra-axial = \_\_
Intra-axial = Intraparenchymal Extra-axial = Extraparenchymal (includes subarachnoid, subdural, epidural, etc)
33
What is going on in this noncontrast CT?
Epidural hematoma with associated subgaleal hematoma on the skin side The subgaleal can be really helpful in helping you localize!
34
How can you tell if something is a fracture, or a suture?
Symmetry! If there is one on the other side, it is more than likely a suture.
35
What is going on in this noncontrast head CT?
Subdural hematoma *just around the posterior falx* Remember that the subdural space continues along the falx Falx asymmetry is a way to tell
36
#1 and #2 causes of SAH
#1: Trauma #2: Aneurysm rupture
37
Pseudosubarachnoid hemorrhage
Diffuse edema of the brain where the swollen brain tissue pushes up against the MCA can give the appearance of the subarachnoid hemorrhage "star"
38
With intra-axial bleeds, you will NOT see . . .
. . . **mass effect** Not typically. So, you will see a large area of white-out brain with minimal edema and no shift. If it was a tumor, you would see *significant* edema and shift.
39
Hydrocephalus vs atrophy on head imaging
Atrophy: Proportional increase in sulcal and ventricular size Hydrocephalus: Enlargement of ventricles with disappearance of sulci
40
Tonsilar herniation on neuro imaging
Loss of CSF in the foramen magnum
41
What to consider when mastoid air cells are filled with fluid
* Infection/inflammation * Truama (usually linear)
42
Aneurysm size vs risk of rupture
* \< 10 mm - 0.05% / year * 10 mm - 1% / year * 25 mm 6% / year
43
MR spec in assessment of CNS pathology
* **Malignancy** * Increase in choline/creatinine ratio * Decrease in N-acetyl-aspartate/creatinine ratio * **Anaerobic metabolism** (infection or necrotic tumor) * Presence of a lipid/lactate peak
44
Normal MR spec
45
Do you need contrast for an MR angiogram?
NO! You can just use time-of-flight sequencing, you do NOT need contrast for MR brain. You do for CT angio, and you do for MRA neck. But NOT MR brain.
46
How many ACOMs are there? How many PCOMs are there?
There is only *one* ACOM that links the left and right ACAs There are *two* PCOMs that link the PCAs to the MCAs
47
What four vessels come off of the tip of the basilar artery?
Both PCAs and both superior cerebellar arteries
48
Internal carotid artery branches
49
Venous cerebral infarct
Most commonly due to venous sinus thrombosis Reduces perfusion pressure of that region. Often progresses to hemorrhage due to increased pressure.
50
Strokes may not appear on CT for up to. . .
. . . **6 hours** Meanwhile, MRI with DWI can detect strokes within *minutes*, but the MRI takes a while to perform.
51
Insular ribbon sign
Can help diagnose stroke on CT prior to typical stroke changes Loss of the ipsilateral insular ribbon due to swelling from the stroke
52
Vanishing basal ganglia sign
CT sign that suggests acute stroke Asymmetry in the hyperintensity of the basal ganglia on one side
53
Dense MCA sign
You can *see* the thrombus inside the MCA
54
Differentiating MRI sequences
55
Main MRI sequences for neuroimaging
56
"T1 is the default for neuroimaging"
The white matter is white, The gray matter is gray.
57
What is T2 FLAIR really good for?
Vasogenic edema T1 doesn't show much. T2 shows the lesion *and* associated edema. T2 FLAIR shows you what is lesion and what is edema.
58
How can you tell if an MRI is pre or post contrast?
The nose knows. Check the nasal mucosa.
59
How should you think of something that appears on DWI, but *not* on ADC
If something is bright on DWI *and* ADC, this is a **"T2 shine phenomenon"** It is some T2 bright feature, like *vasogenic* edema. (rather than cytotoxic edema)
60
Why is there diffusion restriction in cytotoxic edema, but not vasogenic edema?
Because the Na-K ATPase does not work in cytotoxic edema, so cells will swell! More swelling, less free interstitial space, less diffusion
61
GRE is good for. . .
. . . **small hemorrhages or small calcifications** Particularly for lacunar infarcts with small hemorrhage.
62
External carotid artery branches
* Mnemonic from inferior to superior, alternating anterior and posterior: **S**ome **a**natomists **l**ike **f**reaking **o**ut **p**oor **m**edical **s**tudents 1. **S**uperior thyroid a. 2. **A**scending pharyngeal artery 3. **L**ingual a. 4. **F**acial a. 5. **O**ccipital a. 6. **P**osterior auricular a. 7. **M**axillary a. 8. **S**uperficial temporal a.
63
Stroke findings on CT will not appear for . . .
. . . **the first 6 hours (with the exception of hyperdense MCA sign)** This will probably be on the exam
64
What is the most common cause of intraprenchymal hemorrhage (particularly in older patients)
**1. Hypertension** Close second: Cerebral amyloid angiopathy Central/peripheral?
65
What type of hemorrhage can track along the falx and thicken it?
Subdural hemorrhage Because the subdural space runs along the falx, but the epidural does not.
66
Diffuse edema vs hydrocephalus on neuro imaging
Diffuse edema: All fluid spaces are decreased, *including* the ventricles Hydrocephalus: The ventricles are enlarged, all other fluid spaces are decreased
67
Lenticulostriate territories
68
Pseudosubarachnoid sign
Looks like a subarachnoid sign, but is in fact the normal arteries against a hypointense background This may seem tricky, but in reality you will see it in the context of diffuse cerebral edema, so you just need to know to ignore it in that case.
69
Patient with no PMH who has 2 weeks of low back pain without trauma What imaging is indicated?
NONE No red flags (so long as there are no neurologic signs)