Neurogenic Communication Disorders Flashcards

(31 cards)

1
Q

neurogenic etiologies

A

usually a disease or trauma

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2
Q

progressive neurological disorders

A
  • parkinson’s
  • multiple sclerosis
  • myasthenis Gravis
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3
Q

parkinson’s

A

-deterioration of the substantia nigra in the basil ganglia and areas of the brainstem

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4
Q

what neurotransmitter is lacking in the motor system in parkinson’s

A

dopamine

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5
Q

what is the cause of parkinson’s?

A

unknown

some forms are genetically inherited

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6
Q

symptoms of parkinson’s

A
  • resting tremor, slow movement, rigidity,festination

- varying rates of deterioration

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7
Q

speech symptoms of parkinson’s

A

low vocal volume, vocal tremor, imprecise and slow articulation

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8
Q

how is the disease treated?

A

no cure, but is managed with medication that supply part of the dopamine molecule to the brain

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9
Q

what is speech therapy for parkinson’s usually focused on?

A
  • help people maintain skills
  • slow down rate of decline in speech
  • focus on techniques to maintain adequate vocal volume, which shows improvement in voice and artic
  • oromotor exercises and speech over artic strategies also help
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10
Q

parkinsonism

A

a cluster of symptoms similar to those of parkinsons although secondary to another eitiology such as alzheimer’s or multiple strokes

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11
Q

what is Ms?

A

a disease in which the body’s immune system attacks the myelin surrounding neuronal axons

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12
Q

what is myelin?

A

the fatty substance that is contained in some specialized glial cells (support cells) in the central nervous system that wrap around the axons of neurons
-myelinated neurons can conduct neural signals at a much faster rate than unmyelinated neurons

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13
Q

how does the lack of myelin affect Ms?

A

nerual signals cannot travel as fast as they ordinarily would and as a result, motor movement is slow and imprecise

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14
Q

MS medical treatment?

A

corticosteroids

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15
Q

speech symptoms of MS

A
  • imprecise articulation
  • slow and labored speech
  • hoarse and breathy vocal quality
  • difficult maintaining volume
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16
Q

what does MS speech therapy focus on?

A

-compensatory strategies to maximize speech production

17
Q

what is myasthenia gravis?

A
  • problem occurs are neuromuscular junction
  • autoimmune disease
  • body attacks the muscular receptors for acetylcholine, an important neurotransmitter
  • deterioration occurs quickly and regains function after a period of rest
18
Q

MG treatment

A

-managed with drugs (mixed effects)

19
Q

MG speech therapy

A

slp can give short term strategies for maximizing speech intelligibility
-augmentive communication strategies

20
Q

embolic stroke

A

artery is blocked by an embolism, usually arterial plaque, but also possibly a blood clot
(not the worst kind of stroke)

21
Q

hemorrhagic stroke

A

-a bleed in a blood vessel, often caused by hypertension

doesn’t respond to drugs, more serious

22
Q

arteriovenous malformation

A

congenital defect in the vascular supply to the brain, AVMs are prone to hemmorhage

23
Q

risk factors for cva

A
  • hypertension (high blood pressure loosens the plaque which makes it liable to clot-embolism)
  • diabetes (weakens blood vessels-hemorrhage)
  • irregular heartbeat
24
Q

prognosis depends on

A

-age, premorbid language function, location of lesion

25
verbal apraxia
praxis with speaking
26
oral apraxia
praxis with all voluntary movements ex) can't round lips OR produce /w/ sound
27
treatment for apraxia
- teaching placement | - giving feedback (Sensory)
28
white matter tracts
bundles of neurons the allow the brain to communicate with the body
29
upper motor neurons
connect cortex with brainstem and spinal cord
30
stroke affecting white matter symptoms
``` speech is slow and labored -imprecise -tongue is often bunched in back of mouth -harsh vocal quality -mandible doesn't open freely result=spastic dysarthria ```
31
white matter therapy focusses on...
- massaging muscles - heat reduces muscle tensions - reducing muscle tone - improving range of motion