neurological gait Flashcards

1
Q

kinematics vs. kinetics

A
  1. kinetics: forces producing movement

2. kinematics: description of movement; velocity or distance or stride length (nothing to do with force)

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2
Q

ankle/ foot necessities for gait (3)

A
  1. heel first initial contact
  2. smooth transition to foot flat
  3. heel off -> toe off (push off)
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3
Q

what is the main deviation at ankle/ foot?
3 names
why do we care?

A
main deviation is diminished clearance in swing...
1. foot slap
2. foot drop
 these both can lead to 
3. foot drag

these create a huge falls risk

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4
Q

2 things to remember while testing gait

A
  1. worsens on uneven terrain
  2. may not be present inside or on shorter tests

these should be provacative tests

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5
Q

what causes the ankle/foot deviations?

and what are these secondary to? (3)

A
  1. ankle plantarflexion contracture/ tightness in gastroc or heel cord
  2. plantarflexion spasticity = UMN
  3. dorsiflexor weakness = can be LMN
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6
Q

**what contributes most to stability during gait?

A
  1. push off in stance phase** = comes from hip extension
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7
Q

foot drag compensations (6)

A
  1. circumduction (swinging foot to side)
  2. contralateral vaulting (tip-toes of other foot)
  3. hip-hiking (QL)
  4. contralateral trunk lean
  5. hip ER (pivoting on toe of uninvolved side)
  6. trunk extension
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8
Q

treatments for foots drag
what we do (2)
meds (2)
last resort

A
  1. stretch PF** a lot** 30-60s/hr
  2. strengthen DF
    all with task specificity
    meds:
    if due to spasticity- baclofen or tizanidine

bracing as last resort because immobilizes foot

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9
Q

knee necessities during gait (2)

A
  1. knee flexion at heel off

2. smooth transition of knee flexion -> extension during swing

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10
Q

2 gait deviations at knee and what they are secondary to

A
  1. absent or inadequate knee flexion at heel off
    • secondary to spasticity at quads or weakness of hamstrings
  2. extension thrust (as you travel thru stance, knee snaps back into ext ** not genu recuvatum)
    • secondary to PF spasticity or contractures
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11
Q

some treatment options for knee gait deviations (2)

A
  1. stretch quads

2. activate hamstrings

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12
Q

two reasons we don’t like orthodics (AFOs)

A
  1. can make contracture worse

2. cause weakness secondary to immobility

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13
Q

critical components of hip during gait (4)

A
  1. flexion during swing
  2. extension during stance* most important thing for stability during gait
  3. neutral aB/aDduction during stance
  4. ER during swing/ IR during stance
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14
Q

usual compensation for lack of hip flexion during swing

concomitant with…

A
  1. trunk extension during swing

2. concomitant with foot drop

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15
Q
trendelenberg
 def and 2 muscles that cause this
 unlikely but occasional cause
 compensation
 treatment
A
  1. def- stance phase weakness of hip aBductors (glut med or TFL)
  2. rarely from spasticity of aDductors
  3. compensation is ipsilateral trunk lean
  4. treatment- strengthen glut med in closed chain position
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16
Q

scissoring gait
what it looks like
secondary to (2)

A
  1. swing phase hip aDduction -> flexion
    secondary to:
  2. spasticity or (2) weakness of aBductors therefore can be UMN or LMN
17
Q

glut max lurch
what it looks like
secondary to (2)
compensation

A
  1. lurching gait because of inadequate mid -> late stance hip extension

secondary to:

  1. hip extensor weakness
  2. hip flexor contracture

compensation = trunk extension

18
Q

gait deviation for pelvis (1)
secondary to (2)
what these are caused by
treatment

A
  1. unilateral pelvic retraction
  2. secondary to weakness or atrophy in hip protractors/ and trunk extensors
  3. usually part of larger hemiplegic pattern so common in stroke during cortical shock
  4. treatment- PNF
19
Q

2 gait deviations for trunk and what they are secondary to

A
  1. excessive trunk flexion- b/c of weakness in extensors or flexion contracture from prolonged positioning
  2. diminished absent rotation- parkinsonism
20
Q

scapula gait deviation (1)
causes if unilateral vs. bilateral
treatment

A

deviation = protraction

unilateral= stroke/ hemiplegia
bilateral = postural or PD

treatment- stretch protractors or strengthen retractors

21
Q

arm gait deviation (2)
unilat vs. bilat
and other disease for excessive arm swing

A
deviation=  absent or diminished arm swing
unilateral = stroke/ hemiplegia or antalgic
bilateral = parkinsonism

or
deviation = excessive arm swing from huntingtons

22
Q

forward head posture causes (3)

A
  1. prolonged positioning
  2. afraid of falling (but this increases falls risk)
  3. PD
23
Q

parkinsonian gait (8)

A
  1. flexed trunk
  2. cervical hyperextension
  3. shuffling (bilat toe first)
  4. festination - shorter and shorter and faster and faster => falls
  5. absent arm swing
  6. absent trunk rotation
  7. tremor
  8. freezing (at doorways or threshold)
24
Q

cerebellar gait (2)

A
  1. wide BOS

2. ataxic (disordered)

25
hemiplegic gait (6)
1. unilateral 2. foot drag 3. possible extension thrust 4. possible trendelenberg 5. pelvic retraction 6. unequal step lengths and diminished time in stance on involved leg.
26
MS gait
1. anything is possible but worsens with fatigure- 6mwt- will slow down over time
27
tabes dorsalis
a.k.a. syphilis | stamping feet to increase sensory feedback due to dorsal column damage
28
3 biggest contributors to gait
1. Push off in stance 2. Heel strike 3. Med/ lat stability