neurology Flashcards

1
Q

aseptic mening

A

CHRONIC INFECTIONS
drugs alc spirochetes etc

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2
Q

spread of mening

A

hematogenous - crossesthe blood btrain barrier/ contiguous - from adjacent structures

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3
Q

mening clinical rep

A

fever headache vomm seizure photophobia confusion altered mental state red loc

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4
Q

acute mx of mening

A

treated symptomatically
ct, if no contra ind lp.
may recommend steroid except listeria

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5
Q

contra ind for lp

A

unexp seizures, cranial nerve pulse, dep loc, shunt present, systemic contraindications from skills stuff

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6
Q

monitor for complications

A

raised icp, cerebral infartion, brain abcess, hyponatremia, seizures, hydroceph, venous thromb

monitor for expected improvement

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7
Q

csf analysis inc what cell

A

viral - lymph gluc n and prot nto high - imp because doesnt need treament
bact - nuetro
fungal - lympho
tb - lympho

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8
Q

pathogen of pneuococ, manignococ, haemophilus

A
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9
Q

b lactamase

A

counteracts penicillin

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10
Q

hx >2wks of mening sym

A

usually tb or fungal

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11
Q

ires

A

immunresponse exacerabtion syndrome
find out the cause

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12
Q

how long does it take for organisms toreach the csf

A

2 weeks post exposure

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13
Q

nociceptors

A

periph sensory neurons that alert indiciduals to pain

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14
Q

mechanisms of pain

A

irritation/infkammation
traction
vascular pain

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15
Q

classific of headachr

A

prim- no underlying path cause, benign but often disabling
sec- underlying path casue
cranial neuralgias- nerve involement face pains

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16
Q

type sof prim headache

A

migraine
cluster
tension
trigenimal neuralgia

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17
Q

sec headachr causes

A

infection
vascilar eg subarach haem
malig htn
metab d/o
nonvasc cranial d/os- low ccsf pressure

18
Q

trigenimal neuralgia

A

compression of cnv pain radiating along cuorse of trigem nerve

19
Q

xanthochromia on lp

A

suggestive of sah
different from vascular bleed or thromnbosis as those will show cclots on lp

19
Q

snoop redf flags of sec headaches

A

systemic symptoms
onset of pain (acute/sudden eg thunderclap)
older
postural changes
(if precip with valsava, position, exertion)

20
Q

migraine attack phases

A

prodromal phase- activation of intracran center
aura phase- cortical spreading depression
headache phase- photophobia
postdrome- migraine hangover. fatigue

21
Q

ergotamines

A

migraine treatment but avoided because many cvs aes

21
Q

preventative migraine tx

A

antidep
beta bloc
caclium antag

nb theres different drug sets for the different tyoes of migraines

21
Q

triptans

A

most common treatment for migraines

22
headache worse with coughing/ straining/valsava
indicates inc icp which may be mass lessions
23
pit apoplexy
thunderclap headache between the eyes assoc visual disturbances
24
headache worse in morning in elderly
icp in spine and head equalises over night assoc swollen disc common
25
idiopathic intracran hypertension
lp req presents with optic compression- red visual field
26
low csf pressure headache
worse standing up better lying down (opp to high icp) lp req if csf opening pressure is low then it is diagnostic mri will find thickening of dural layers
27
pathophys of seizures
imbalance of excitabilty and inhibitory receptors in at a focal point that propagates the excitation to surrounding neurons via cc to other hemisphere
28
focal aware seizures
pt fully aware of events occuring during seizure
29
focal impaired awareness seizures
pt loses awareness of some events may be assoc with speech abn and memory loss
30
generalized seirzures
motor- tonic-clonic or other motor non motor- absence
31
epileptic diagnosis
two or more unprovoked seizures less than 24 hours apart. no metaimbalance because eg alcohol binges predisposes to seizures
32
myoclonic seizures
single jerk
33
epileptic spasms
most common in infants assoc trunkal spasms
34
absent seizures
no loss of tone, unaware, blank stare, may have multiple attacks presents in children previously petit mal
35
myoclonia
unable to open eyes during seizure
36
lennox gastaut synd
childhood phsychomotor retardation therapy difficult
37
febrile seizures
due to underlyling infection self limited does not devel seizures later on but any metab abn does inc risk of recurrent seizures