Neuroradiology: Head and Brain Imagin Flashcards
(34 cards)
lacunar infarct (lacune)
- small cerebral infarcts.
- areas of hypoattenuation (low density), favors basal ganglia, pons, internal capsule
- common with HTN, atherosclerosis, DM
epidural hematoma
- hemorrhage between the dura and skull table
- lens shape, biconvex
- hyperattenuation
- midline shift common
- skull fractures common
- does not cross suture lines
- ARTERIAL
stroke - considerations
- initial study = CT noncon
- is it hemorrhagic or ischemic?
- <6hrs sxs, CT often falsely negative if ischemic
- MRI more sensitive early on if ischemic
- types of CVA: hemorrhagic, ischemic, lacunar infarcts
- if hemorrhagic, keep pt stable and call neurosurgery
- if ischemic, call neurologist and see if we can fix
Imaging for a suspected stroke
- if the stroke score is very high, you run noncon CT first and then you push contrast and get CTA (angiogram) of head and neck
- contrast makes everything look white and blood is white - you dont want to confuse ischemic from hemorrhagic - THIS IS WHY YOU GET NONCON FIRST
subdural hematoma
- Hemorrhage between dura and anarchnoid
- Bridging vein damage
- Crescent shaped, concave
- Acute: hyperattenuation
- Chronic: less dense, hypoattenuation
- Midline shift common
- Does not cross midline
- May cross suture lines
- VENOUS
CT Head/Brain
- axial cuts and bone windows are standard
- pathology: symmetry, densities, lucencies, blood (new, old, hemorrhage), ischemia, infarction, edema, tumors, mets, hydrocephalus, bony windows (skull fractures)
- it is standard and customary to only get axial slices of the brain
Special hydrocephalus
- normal pressure hydrocephalus (communicating type) - classic presentation = >50yo, gait disturbance, dementia, urinary incontinence
- dilated ventricles out of proportion to atrophy, sulci normal
-THIS CAN BE FIXED
subdural - avute vs. subacute
- acute blood is white
- subacute - after about 1 week - blood is more isodense
- chronic - after about 2 weeks - blood alppears hypodense
Approach to head CT interpretation
- check name, date, study, rotation (contrast?)
- check symmetry (midline shift, effacement)
- hyperattenuation (acute bleed, calcifications, FBs)
- hypoattenuation (edema, ischemia, old blood, tumor, air)
- cisterns, CSF spaces
- ventricle size, symmetry
- gyri, sulci symmetric, edema, atrophy
- soft tissue, sinuses, mastoids
- bone windows
- always interpret with attending physician, confirm with radiologist
- Blood Can Be Very Bad (blood, cisterns, brain, ventricles, bone)
Cerebral edema
- Effacement of gyri and sulci
- Loss of grey-white differentiation
- Ventricular compression
- Global or local response
- Herniation may result
- Increased intracraininal pressure:
- Headache, vomiting, papilledema, possibly ALOC
-Psuedotumor Cerebri (BIH), trauma, infection, tox, metabolic
CT white things with IV contrast
- abnormal = blood, tumor/mass/infection
- normal = vasculature, choroid plexus, pituitary
How CTs are taken
- 15-20 degree axial “cuts”
- 3mm-5mm wide
- skull base to vertes
- scout film
- grey matter appears grey
- white matter appears darker
Hydrocephalus
-dilated ventricles, temporal horns visible
Obstructive hydrocephalus: 2 types
- communicating - extraventricular cause (decreased reabsorption of CSF, acute/chronic, entire ventricular system, normal pressure hydrocephalus; 4TH VENTRICLE ENLARGED)
- non-communicating - intraventricular cause (obstruction of outflowof CSF - tumor, mass; narrow site - 3rd or 4th ventricle, foramena of monroe, aqueduct sylvius; 4TH VENTRICLE NORMAL SIZED, SULCI NORMAL)
-prominent temporal horns of the lateral ventricles is one of the first signs of hydrocephalus
Tumors, masses on CT
- tumors: intraaxial = w/in brain parenchyma (glioma, astrocytoma, etc.), extraaxial = outside of brain itself (meningioma, acoustic neuroma), metastases (round, multiple, enhance w/ contrast)
- masses (parasites, fungal)
indications for non-contrast head CT
- head trauma - clinically significant
- suspected acute CVA/TIA, focal neuro deficit
- HA - atypical worst of life
- delirium (if there is an obvious cause, CT may not be needed)
- HA + fever: meningitis/abscess/encephalitis
- seizure - first one
- cancer hx with new HA, ALOC, focal neuro finding
- consider in: vomiting w/o abdominal sxs, suspected child abuse
Cytotoxic cerebral edema
- hypoattenuation from encephalomalacia
- cell death after cerebral ischemia (infarct)
- possible midline shift, herniation
- affects both white and grey matter
Increased Intracranial Pressure
- Mass effect - midline shift; From blood or space occupying lesion
- Cerebral edema - Increase in brain volume (swelling) (May lose normal definition of gyri/sulci, Infection, reactive, malignancy, toxic, anoxic), Vasogenic, Cytotoxic
- Hydrocephalus - Increase in ventricle size
Vasogenic Cerebral edema
- hypoattenuation
- local edema around infection, malignancy
- vasogenic edema around acute hemorrhage
- possible midline shift, herniation
- predominantly affects white matter
- INTRACEREBRAL BLEED, not subdural, epidural, subarachnoid
subtle CVA signs
- hyperdense vessel sign
- loss of insular ribbon - grey matter stripe or interface with white matter
- lentiform nucleus and caudate nucleus not distinctly visible
- effacement of sulci
- dont need to recognize on CT, but know the list of signs
Plain x-rays - skull
- Rarely obtained - replaced by CT
- AP, Lateral(s), Towne views standard
- Indications: Child abuse survey, Foreign Body, Metastatic or Metabolic bone survey, If CT not available
indications for contrast CT
- with IV contrast = “contrast enhanced”
- CVA - non-contrast first in all, then if a significant deficit: CTA (CT angiogram of head and neck, CTA useful for intervention (stent))
- vascular lesions (AVM, aneurysm)
- tumors
- brain abscess, encephalitis
- MRI now often utilized in these conditions (except CVA)
CT white things without IV contrast
- abnormal = blood (acute), calcified masses (i.e. tumor)
- normal = bone, TYPICAL calcifications (pineal gland, choroid plexus, falx, basal ganglia)
hemorrhagic CVA
- acute: hyperattenuation: collections of blood
- favor basal ganglia, thalamus, pons, cerebellum
- local vasogenic edema
- effacement of gyri/sulci and midline shift common
- risks: HTN, coagulopathy, stimulants (cocaine, meth)
- less common than ischemic; more morbidity/mortality
- call a neurosurgeon
neurocysticercosis
- pork tapework parasite, common in mexico, latin americ
- often asymptomatic, often presents with first seizure