Newborn Complications Flashcards

(35 cards)

1
Q

SGA causes

A

Small Gestational Age

causes:

  • maternal factors/disease/smoking
  • environmental, placental and fetal factors
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2
Q

Complications of SGA & IUGR

A
  • fetal hypoxia, asphyxia
  • aspiration syndrome
  • hypothermia
  • hypoglycemia
  • polycythemia
  • hyperbilirubinemia
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3
Q

Care for SGA is aimed…

A

at promoting growth (feeding and NTE) and caring for complications

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4
Q

IUGR

A

intrauterine growth restriction

  • advanced gestation
  • extremes of maternal age
  • lack of prenatal care
  • low socioeconomic status
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5
Q

IUGR associated with…

A
  • hypoglycemia (very common)
  • congenital malformations
  • intrauterine infections

future:

  • growth difficulties
  • cognitive delays
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6
Q

Differentiating SGA and IUGR

A

-not synonymous

SGA NB is constitutionally small but otherwise normal

IUGR is a fetus with delayed growth late in gestation. May not have a reduction in birth weight significant enough to be classified as SGA

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7
Q

Prognosis for SGA

A

-symmetric SGA most likely stay small

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8
Q

Prognosis for IUGR

A
  • often catch up by 2nd year
  • greater risk for impaired school performance, behavior problems, and poor fine motor control
  • as adults, IUGR infants are at a greater risk for obesity, type 2 DM and cardiovascular dz
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9
Q

Post-maturity syndrome

A

cause: unknown, more common in certain ethnicities (Australian, Greek and Italian)

complications:
- higher risk for morbidity
- hypoglycemia and cold stress
- meconium aspiration
- polycythemia
- seizure activity
- congenital anomalies

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10
Q

Post-term infant

A

Assessment:

  • dry, cracking skin
  • no vernex/lanugo
  • scalp hair profuse
  • body long, thin

nursing care:

  • most adapt well
  • monitor blood glucose frequently
  • assess respiratory status (risk - meconium aspiration)
  • neutral thermal environment - provide warmth
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11
Q

Pre-term infant

A
  • less than 37 weeks gestation
  • 12 percent of all US births, 17 percent of all AA
  • common in single mothers and adolescents
  • rise in multiple births from IVF
  • main problem is the variable immaturity of all body systems. Function is dependent on length of gestation
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12
Q

Thermoregulation

A
  • glycogen and brown fat not available
  • lose heat from blood vessels close to skin
  • high ratio of body surface area to body weight
  • extended position increases body surface area
  • decreased vasoconstriction ability of superficial blood vessels
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13
Q

Nursing Care of the Preemie

A
  • maintain respiratory function
  • cluster care to promote rest
  • maintain neutral environment
  • balance fluids and electrolytes
  • prevent infection
  • provide adequate nutrition - breast milk is best and may fortified and given as slow continuous feed via pump. Donor milk if needed
  • promote parent-infant attachment
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14
Q

Respiratory Distress Syndrome

A
  • inadequate surfactant production
  • pulmonary blood vessels aren’t fully developed
  • decreased pulmonary vascular resistance
  • left-to-right shunting through ductus arteriosus
  • increased blood flow back to lungs
  • surfactant required for alveolar stability
  • instability causes atelectasis
  • atelectasis causes hypoxemia and acidosis
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15
Q

Nursing care for the infant with RDS

A
  • before birth (prevent preterm birth, admin glucocorticoids)
  • after birth (surfactant replacement therapy, assess for signs of distress, cluster care)
  • monitoring blood gases and pulse oximetry
  • respiratory support includes nasal cannula, CPAP or intubation with ventilator
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16
Q

Feeding infants

A
  • bottle feeding/breast feeding
  • requires suck-swallow-breathing patterns
  • gavage feeding for infants
  • with poor suck-swallow-breathing patterns
  • on ventilators
  • who tire easily
  • losing weight
  • supplementation with fortifiers common
17
Q

Pre-term infant short term complications

A
  • apnea of prematurity
  • hypoglycemia
  • patent ductus arteriosus
  • respiratory distress syndrome
  • intraventricular hemorrhage
  • hyperbilirubinemia
  • necrotizing enterocolitis
  • sepsis
18
Q

Long term complications of the Preemie

A
  • higher rate of SIDS
  • retinopathy of prematurity
  • broncho-pulmonary dysplasia
  • speech defects
  • neurologic defects
  • auditory defects
  • abuse and neglect
19
Q

Asphyxia

A

-neonatal emergency can lead to hypoxia and possible brain damage or death if not correctly managed

20
Q

newborn asphyxia

A

defined as a failure to start regular respiration within a minute of birth

21
Q

perinatal asphyxia

A

leads to multi-organ system dysfunction

22
Q

CV

A

alterations in blood volume, redistribution of cardiac output and a syndrome of transient myocardial dysfunction

23
Q

Management of Asphyxia

A
  • adequate ventilation
  • perfusion and BP management
  • fluid management
  • avoid extreme glucoses
  • avoid hyperthermia
  • tx of seizures
  • hypothermia therapy followed by slow and controlled rewarming for infants with mod to severe HIE
24
Q

Meconium Aspiration Syndrome Nursing Care

A
  • tracheal suctioning
  • umbilical arterial line
  • umbilical venous catheter
  • high levels of O2
  • exogenous surfactant
  • prophylactic antibiotics
25
Meconium in the lungs...
- mechanical airway obstruction - chemical pneumonitis - vasoconstriction of pulmonary vessels - inactivation of surfactant
26
Meconium assessment
- apnea - pallor - bradycardia - barrel chest - distress yellow-green skin - nail discoloration
27
The key to meconium...
prevention -suction oropharynx and naspharynx before first breath. May need to intubate to aggressively suction -if aspiration is significant, will need ECMO
28
Pulmonary HTN
- Failure of the normal circulatory transition that occurs after birth - Breathing and increase PO2 is altered so that the normal mechanisms for transition are altered - Marked pulmonary hypertension that causes hypoxemia secondary to right to left shunting of blood
29
Symptoms of Pulmonary HTN
- tachypnea, respiratory distress - loud, single second heart sound (S2) - harsh systolic murmur (secondary to tricuspid regurgitation) - cyanosis - poor cardiac function and perfusion, hypotension
30
Management of Pulmonary HTN
- continuous monitoring of oxygenation, blood pressure, and perfusion - maintaining a normal body temp - correction of electrolytes/glucose abnormalities and metabolic acidosis - nutritional support - minimal stimulation/handling of the newborn - minimal use of invasive procedures
31
Physiologic Jaundice Review
-very common most resolve with fluids only - normal process due to: - infant's shortened RBC lifespan - slower uptake of bilirubin by liver - lack of intestinal bacteria - poorly established hydration especially if breast fed
32
Total bilirubin level peaks
4-5 days old
33
Physiologic Assessment
- Yellowish coloring of skin/sclera | - Deposits of bilirubin in tissues
34
Pathologic Hyper-bilirubinemia
- appears within first 24 hours - evidence of RBC destruction is increased retic count - serum bilirubin rises rapidly - conjugated bilirubin greater than 2mg - clinical jaundice persists in term infant
35
Causes/risk factors for Pathologic Hyper-bilirubinemia
- ABO incompatibility - Rh - Asphyxia - Neonatal/maternal drugs - hypothermia and hypoglycemia - prematurity