NMx of Pregnancy at Risk: Pregnancy-Related Complications, Selected Health Conditions and Vulnerable Populations Flashcards

Chapter 19+20

1
Q

How does should a nurse manage spontaneous abortion?

Constant Monitoring, Support Interventions

A

Continue Monitoring
* Passage of products of conception
* Vaginal bleeding*
* Pad count
* Pain level
* Preparation for procedures
* Medications

Support
* Physical and emotional
* Stress that woman is not the cause of the loss
* Verbalization of feelings
* Grief support
* Referral to community support group

*gushing, blood clots; more remnants

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2
Q

Threatened Spontaneous Abortion

A

Assessment findings
* Slight vaginal bleeding present in early pregnancy
* No cervical dilation
* Mild abdominal cramping
* Closed cervical os
* No passage of fetal tissue

Diagnosis
- Vagina, ultrasound to confirm if sac is empty 
- Declining maternal serum, hCG and progesterone levels to provide additional information about viability of pregnancy

Therapeutic management
- Conservative, supportive treatment
- Possible reduction in activity in conjunction with nutritious diet and etiquette hydration 

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3
Q

Complete Spontaneous Abortion
(Passage of all products of conception)

A

Assessment findings:
- History of vagina bleeding in abdominal pain
- Passage of tissue with subsequent decrease in pain and significant decrease in vagina bleeding

Diagnosis
-Ultrasound demonstrating an empty uterus

Therapeutic management
- No medical or surgical intervention necessary
-Follow up appointment to discuss family planning 

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4
Q

Inevitable Spontaneous Abortion

A

Assessment findings
* Vaginal bleeding
* Rupture of membranes
* Cervical dilation
* Strong abdominal cramping
* Possible passage of products of conception

Diagnosis
- Ultrasound and the hCG Levels to indicate pregnancy loss

Therapeutic management
- Vacuum Curettage of products of conception are not passed to reduce risk of excessive bleeding and infection 
- Prostaglandin analogues, such as misoprostol to empty out uterus of retain tissue (Only used in fragments are not completely passed)

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5
Q

Missed Spontaneous Abortion

A

Assessment findings

* Absent uterine contractions
* Irregular spotting
* Possible progression to inevitable abortion

Diagnosis
- Ultrasounds identify projects of conception in Uterus

Therapeutic management
- Evaluation of uterus, but using suction Curettage during first trimester, dilation and evacuation during second trimester 

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6
Q

Incomplete Spontaeous Abortion

A

Assessment findings
* Heavy vaginal bleeding
* Intense abdominal cramping
* Cervical dilation

Diagnosis
* Ultrasound demonstrating their products of conception still in Uterus

Therapeutic management
- Client stabilization
- Evaluation of uterus via D&C or prostaglandin analogue 


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7
Q

What to assess for an spontaneous abortion?

A
  • Vaginal bleeding*
  • Cramping or contractions
  • Vital signs
  • Continuous monitoring
  • Pain level
  • Patient understanding
  • Provide support

*Is it a bright or dark red
Saturation of pad
How often must she change pad
saturated pad within 1 hour is SIGNIFICANT

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8
Q

What are the medications used for spontaneous abortion?

A
  • Misoprostol (cytotec)
  • Mifepristone (RU-486)
  • Dinoprostone (cervidil)
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9
Q

What is the hallmark sign of ectopic pregnancy?

Ectopic Preganancy: Eggs implants outside of the uteris.

A

Abdominal pain with spotting within 6 to 8 weeks after missed menses.

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10
Q

What are the laboratory and diagnostic findings of ectopic preganancy?

A

Transvaginal Ultrasound
Serum Beta hCG

Absence of hCG indicated no pregnancy.
Include additional testing to prevent rupture.

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11
Q

What is the therapeutic management of ectopic pregnancy?

Meds, Surgery

A

Medical: drug therapy (methotrexate*, prostaglandins, misoprostol, and actinomycin)

Surgery if rupture (salpingostomy)

Rh immunoglobulin

*methotrexate slows down the immune system and reduces inflammation

*Salpingostomy - removal of fallopian tube (unilateral or B/L depending on situation and maternal age)

*Rh immunoglobulin is administered if the women is Rh-

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12
Q

What is the therapeutic management of Gestational Throblastic Disease?

Gestational Thromoblastic Disease (GTD) is when the egg and sperm produces an empty embryo. Two types: Hydatidiform Mole (Placental development)  and Choriocarcinoma (trophoblast) . Cause is unknown. PREGNANCY IS NOT VIABLE

A

Dilatation & curretage (D&C)
* Immediate evacuation of uterine contents

Long term follow up and monitoring of hcG levels
* Check hcG levels every week for 3 consecutive weeks until hcG serum levels are undetectable, then monthly for 1 year
Chest radiograph every 6 months

Prophylactic chemotherapy

Choriocarcinoma

NO PREGNANCY FOR 1 YEAR
* Pregnancy within 1 → risk for hemorrhage
Use birth control for the duration of that year

*Intervention: D&C

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13
Q

Hydatiform Mole

GTD

A

Disorder of placental development

COMPLETE MOLE
* Benign neoplasmic tissue coming from gestation, not mother
* Mole contains no fetal tissue and develops from an “empty egg,” which is fertilized by normal sperm
* The embryo is not viable and dies, No circulation is established and no embryonic tissue is found.
* Associated with the development of trio carcinoma
Uterine enlargement, greater than expected for gestational dates, hyperemesis and preeclamptic symptoms

PARTIAL
* Triploid karyotype (69 chromosomes, Since to sperm cells, have double contribution by fertilizing the oven
* Women present clinical features of mist or incomplete abortion, including: Vagina bleeding, and a small or normal size for date uterus 

Clinical Manifestations
* Vaginal bleeding
* Anemia
* Excessively large uterus
* Preeclampsia
* Hyperemesis
* Amenorrhea
* Fluid retention and swelling
* Extremely high hcG levels
* Absence of FHR or fetal activity expulsion of grape-like vesicles

*vaginal bleeding and cramping or contractions

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14
Q

Choriocarcinoma

A

Results from chorionic malignancy from the trophoblastic tissue
Typically asymptomatic.

Indications:
* SOB (Indicates metastasis to lungs)
* Vaginal bleeding
* Small to normal sized uterus

Therapeutic management:

Chemotherapy 

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15
Q

Cervical Insufficiency

Clinical Manifestation & Diagnostic Tests

*Premature dilatation of cervix
Cause unknown; possibly due to cervical damage

A

Clinical Manifestation
* Pink tinged vaginal discharge
* Low pelvic pressure
* Cramping with abdominal bleeding
* Loss of amniotic fluid
* Cervical dilation

Diagnostic Tests
* Transvaginal ultrasound b/w 16-24 weeks
* Determines cervical length
* Evaluate for shortening
* Can predict early preterm labor

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16
Q

Cervical Insufficiency

Cervical Cerclage Management

Indications, Complication

  • Sewn cervix with ligature
A
  • Placed up to 28 weeks gestation, cannot be done after 28 weeks*
  • Indications of cerclage
    • History of second trimester pregnancy loss with PAINLESS DILATION
    • Prior cerclage placement for cervical insufficiency
    • Spontaneous preterm birth prior to 34 weeks gestation
    • Painless cervical dilatation on physical exam of 2nd trimester
  • Complications
    • Suture displacement
    • Rupture of membranes
    • Chorioamnionitis

  • by the 28th week, remove cerclage prior term and give betamethazone
17
Q

Therapetic Management for Cervical Insuffiency

A
  • Monitor closely for signs of preterm labor
    • Backache
    • Increase in vaginal discharge
    • Rupture of membrane
    • Uterine contractions
  • Bed rest
  • Pelvic rest
  • Avoidance of heavy lifting
  • Progesterone supplementation of women at risk for preterm birth
  • Placement of cervical pessary

*Pessary - round silicone device at mouth of cervix

18
Q

Placenta Previa

Clinical Manifestation, Therapeutic/ Nursing Management

  • Cause unknown; placental implants over cervical os
A

Clinical Manifestation
* R/F: previous delivery by caesarean section
* Vaginal bleeding (painless, bright red) in second or third trimester

  • spontaneous cessation then recurrence

Therapeutic management:
* dependent on bleeding
* amount of placenta over os
* fetal development and position
* maternal parity
* labor signs and symptoms

Nursing management
* Monitoring of maternal–fetal status
* Vaginal bleeding; pad count
* Avoidance of vaginal exams
* FHR
* Support and education: fetal movement counts, effects of prolonged bed rest (if necessary); signs and symptoms to report
* Preparation for possible cesarean birth

19
Q

What are the risk factors and clinical manifestations of Placental Abruption?

*Separation of placenta leading to compromised fetal blood supply
MEDICAL EMERGENCY
Etiology unknown

A

Risk factors
* smoking
* cocaine use during pregnancy
* maternal age over 35 years
* hypertension
* placental abruption in a prior pregnancy

Nursing Assessment
* Bleeding (dark red)
* Pain (knife-like), uterine tenderness, contractions
* Fetal movement and activity (decreased)
* Fetal heart rate
* Laboratory and diagnostic testing: CBC, fibrinogen levels, PT/aPTT, type and cross-match, nonstress test, biophysical profile

20
Q

What are the therapeutic and nursing managements of Placental Abruption?

*Separation of placenta leading to compromised fetal blood supply

A

Therapeutic management:
* assessment, control, and restoration of blood loss
* positive outcome
* prevention of DIC*

Nursing management
* Tissue perfusion: left lateral position, strict bed rest, oxygen therapy, vital signs, fundal height, continuous fetal monitoring
* Support and education: empathy, understanding, explanations, possible loss of fetus, reduction of recurrence

Interventions: blood tranfusions, EFM, two IVs, monitor fetus

*Disseminated Intavascular Coagulation: abnormal blood clots in blood vessels

21
Q

What are the risk factors and symptoms of hyperemesis  gravidarum?

A

Risk Factors:
* previous pregnancy with HG
* dizygotic birth
* pregnant for the first time

Symptom:
* persistent, uncontrollable N/V*
* Increased hCG

*begins before 9 weeks’ gestation and causes dehydration, nutritional deficiencies, ketosis, electrolyte imbalances, and weight loss of more than 5% of prepregnancy body weight.

22
Q

What are the therapeutic managements of Hyperemesis Gravidarum?

NonPharm/ Pharm Interventions

A

Therapeutic Management
* Consersative lifestyle and diet
* Hospitalization with I.V. therapy*

Medications:
* Doxylamine (Diclegis)
* Dimenhydrinate (Dramine)
* Promethazine (Phenergan)
* Diphenhydramine (Benadryl)
* Metoclopramide (Reglan)
* Ondansetron (Zofran)

  • Primarily Normal Saline; Lactate Ringers for patients going in labor; keep mother NPO for 24-36 hours
23
Q

When does chronic hypertenion develop into preeclampasia?

A

The pregant patient has a constant BP of more than 140/90 (>S/D) before pregnancy, before 20 weeks of gestation, and more than 12 weeks postpartum.

24
Q

What is gestational hypertension?

A

An onset of hypertension without proteinuria after 20 weeks of preganancy.

For a woman who previously had a normal blood pressure prior pregnancy.

25
Q

What are the risk factors and signs of preeclampsia?

Preeclampsia: HTN and proteinuria after 20 weeks or early postpartum.

A

Risk factors:
* family history
* nulliparity
* egg donation
* diabetes
* obesity

Classical signs:
* Dull headache
* Oliguria
* Blurred vision
* Proteinuria
* Epigastric pain
* Right upper quadrant pain
* Hyperactive deep tendon reflexes (DTRs)
* Progressive renal insufficiency

Nursing Assessment:
*BP, urinalysis, nutritional intake, weight, edema, proteinuria

26
Q

How to manage preeclampsia with severe features/ eclampsia?

A

Hospitalization (Pre-labor/labor)

NONPHARM
* seizure precations
* preconception counseling
* perinatal BP

PHARM
* Daily low dose aspirin (75 to 150mg)
* Betamethasone IM 12 mg 2x in 24 hours for preterm pre-eclamptic/eclamptic clients
* **Magnesium sulfate IV infusion 4 to 6 gm bolus given over 15 to 30 minutes. Maintenance dose of 1 to 2 gm/hr. **
* Hydralazine (Apresoline)- IV bolus 5 to 10 mg every 20 min as needed
* Labetalol (Normodyne)- IV dose 20 to 40 mg every 15 min
* Nifedipine (Procardia)- 10 to 20 mg PO for three doses and then every 4 to 8 hours.
* Furosemide (Lasix)- slow IV bolus of dose 10 to 40 mg over 1 to 2 minutes

*Hydralazine and Labetalol are IV push for HTN. If drops after first adminsitration, don’t give again.

*Furosemide (Lasix) is rarely used, unless patient has a coinciding cardiac issue.

27
Q

How to manage preeclampsia without severe features?

A

Management for mild symptoms:

Home
* Mild BP - place in bedrest or in side-lying postion
* monitor BP daily every 4-6 hrs/day, report for increased readings
* record daily fetal movement count, report for decrease in movement
* sodium restricted-diet
* encourage to drink six to eight 8-oz glasses of water daily
* go to the hospital if the home management fails to lower BP

Hospitalization
* monitor closely for s/s of severe preeclampsia or impending eclampsia
* give magnesium sulfate during labor

Management continues until the pregnancy reaches at least 37 weeks’ fetal gestation, fetal lung maturity is documented, or complications that immediate birth.

28
Q

How to manage eclamspia?

Eclampsia: onset seizure activity. MEDICAL EMERGENCY.

A

Management
* Airway, breathing and circulation
* Clear the airway, administer adequate oxygen
* Position woman on left side
* Suction readily available
* IV fluids to replace urine output
* FHR assessment
* Magnesium sulfate and antihypertensive drugs
* Uterine contraction monitoring
* Preparation for birth

29
Q

How to manage Magnesium Sulfate levels?

Magnesium Sulfate helps treat preeclampsia, provides neutral protection of fetus, and slows down labor.

A
  • Normal Magnesium Sulfate levels: 4-7
  • Monitor Deep Tendon Reflex (DTR)
  • Monitor signs of toxicity (mEq/L)*
    -10, decreased DTR
    -15, respiratory distress
    -25, cardiac arrest

*Antidote: Calciumgluconate (calcium chloride)

30
Q

How to assess for HELLP Syndrome?

Clinical Manifestations and Lab Results

Hemolysis, Elevated Liver enzymes, Low Platelets

A

Nurse Assessment
* *Monitor for complaints of nausea (with or without vomiting), malaise, epigastric or right upper quadrant pain, headache, and changes in vision.

LAB RESULTS
* Low hematocrit that is not explained by any blood loss
* Elevated LDH, AST, ALT, bilirubin level (liver impairment)
* Elevated BUN, uric acid, and creatinine levels (renal impairment)
* Low platelet count (less than 100,000 cells/mm3)

*Assessment and Management is same as preeclampsia

31
Q

How to manage HELLP Syndrome?

A

Hospitalization (Pre-labor/labor)

NONPHARM
* seizure precations
* preconception counseling
* perinatal BP

PHARM
* Daily low dose aspirin (75 to 150mg)
* Betamethasone IM 12 mg 2x in 24 hours for preterm pre-eclamptic/eclamptic clients
* **Magnesium sulfate IV infusion 4 to 6 gm bolus given over 15 to 30 minutes. Maintenance dose of 1 to 2 gm/hr. **
* Hydralazine (Apresoline)- IV bolus 5 to 10 mg every 20 min as needed
* Labetalol (Normodyne)- IV dose 20 to 40 mg every 15 min
* Nifedipine (Procardia)- 10 to 20 mg PO for three doses and then every 4 to 8 hours.
* Furosemide (Lasix)- slow IV bolus of dose 10 to 40 mg over 1 to 2 minutes

32
Q

Blood Incompatability

ABO vs Rh- Incompatibility, Nurse Assessment, and Nurse Management

A
  • ABO incompatibility: type O mothers and fetuses with type A or B blood (less severe than Rh incompatibility)
  • Rh incompatibility: exposure of Rh-negative mother to Rh-positive fetal blood; sensitization; antibody production; risk increases with each subsequent pregnancy and fetus with Rh-positive blood

Nursing assessment:
* maternal blood type and Rh status

Nursing management:
* RhoGAM at 28 weeks

33
Q

Polyhydramnios

Nurse Assessment, Therapeutic/Nursing Management

Aka Hydramnios
Amniotic fluid >2,000 mL

A

Nurse Assessment
* R/F:
* Therapeutic management: close monitoring; removal of fluid, indomethacin (decreases fluid by decreasing fetal urinary output)

Nursing management: ongoing assessment and monitoring; assisting with therapeutic amniocentesis

34
Q

Oligohydramnios

Amniotic fluid <500 mL

A

Nursing assessment: risk factors, fluid leaking from vagina

Therapeutic management: serial monitoring; amnioinfusion and birth for fetal compromise

Nursing management: continuous fetal surveillance; assistance with amnioinfusion, comfort measures, position changes

35
Q

Multiple Gestation

Assessment, Therapeutic/Nurse Management

Pregnancy with two or three fetuses.

A

Nursing assessment:
* uterus larger than expected for EDB
* ultrasound confirmation

Therapeutic management: serial ultrasounds, close monitoring during labor, operative delivery (common)

Nursing management: education and support antepartally; labor management with perinatal team on standby; postpartum assessment for possible hemorrhage

36
Q

Premature Rupture of Membranes

Prelabor ROM & PPROM

  • Prelabor Rupture of Membranes (PROM): Water breaks before labor
  • PPROM: Rupture before 37 weeks of gestation
A

Diagnostic Tests:

Treatment:
* dependent on gestational age
* no unsterile digital cervical exams until woman is in active labor expectant management if fetal lungs immature

Nursing management
* Infection prevention
* Identification of uterine contractions
* Education and support
* Discharge home (PPROM) if no labor within 48 hours

37
Q

Gestational Diabetes Mellitus (GDM)

Risk Factors and S/S of Hypo/Hyperglycemia

A

Risk Factors:
* Maternal Obesity (BMI >30)
* Maternal age older than 35 years or older
* Previous birth outcome often associated with GDM-macrosomia, maternal hypertension, fetal death or anomalies
* GDM or polyhydramnios in previous pregnancy
* History of abnormal glucose tolerance
* Family history of diabetes
* Multiple pregnancies
* Member of a high-risk ethnic group

S/S:
Hypoglycemia
* Tired, Irritatibility, Restless, Diaphoresis, Excessive Hunger

Hyperglycemia
* Flushed skin, dry mouth, cheynestokes respiration, fruity breath odor

38
Q

How to manage GDM?

A

Therapeutic Management
* Preconception counseling
* Blood glucose level control (HbA1c <7%)
* Glycemic control
* Nutritional management
* Hypoglycemic agents
* Close maternal and fetal surveillance
* Management during labor and birth

39
Q
A