NRSRGRY - CVD Flashcards

(40 cards)

1
Q

What is the most frequent cause of new, rapid onset, non-traumatic neurologic deficit

A

CVD

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2
Q

Risk factors of CVD (4)

A
DHHS
Diabetes
High Cholesterol
High BP
Smoking
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3
Q

Mechanisms of CVD (3)

A

Atheroma
Endothelial damage
Weakening of vessel wall

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4
Q

Hypodensity on CT

a. Ischemic
b. Hemorrhagic

A

Ischemic

Hemorrhagic is hyperdensity

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5
Q

Clot forms in the vessel wall and occludes a vessel. What type of CVD is this?

A

Thrombotic Ischemic disease

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6
Q

Clot is elsewhere, thrown distally and lodges into vessel and occludes. What type of CVD?

A

Embolic Ischemic disease

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7
Q

Hemorrhagic diseases (4)

A

CHAA

Cerebral aneurysm
Hypertension
Amyloid Angiopathy
Ateriovenous malformation

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8
Q

Ischemic disease accounts for what percentage of acute CVD?

A

85%

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9
Q

T about stroke EXCEPT

a. symptoms will vary based on the functions of the neural tissues supplied by the occluded vessel and the presence of collateral circulation
b. occlusion proximal to the circle of willis will generally result in a stroke.
c. Neurologic deficit is always permanent
d. A and B
e. B and C

A

E

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10
Q

ACA stroke lobe involvement

A

medial frontal and parietal lobes

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11
Q

ACA stroke neuro deficits

A

contralateral leg weakness

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12
Q

MCA lobe involvement

A

lateral, frontal, temporal and parietal lobes

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13
Q

MCA stroke neuro deficits

A

contralateral face and arm weakness, language deficits

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14
Q

Other effects of proximal MCA stroke (aside from neurodeficits)

A

intracranial mass effect and midline shift

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15
Q

PCA stroke lobe involvement

A

occipital lobe

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16
Q

Effect of R PCA stroke

A

L homonymous hemianopia

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17
Q

PICA stroke lobe involvement

A

lateral medulla, inferior half of the cerebellar hemispheres

18
Q

Effect of PICA stroke

A
nausea
vomiting
nystagmus
dysphagia
ipsilateral Horner's syndrome
Ipsilateral limb ataxia
19
Q

Horner’s syndrome

A

Miosis
Ptosis
Anhidrosis

20
Q

infarction, or stroke, occurs in the lateral medulla.
Symptoms:

hoarseness
nausea
vomiting
hiccups
rapid eye movements, or nystagmus
a decrease in sweating
problems with body temperature sensation
dizziness
difficulty walking
difficulty maintaining balance
A

Wallenburg syndrome

21
Q

Management of embolic disease: reopening of occluded vessel via what drug?

22
Q

Life-saving surgery for patients with embolic disease who had stroke?

A

Decompressive hemicraniectomy or suboccipital hemicraniectomy

23
Q

L hemiplegia, what artery affected?

24
Q

Causes of hemorrhagic CVD

A
Hypertension
Amyloid angiopathy
AVM
ANeurysm
Venous thrombosis
Hemorrhagic conversion of ischemic infarct
fungal infections
25
Where does hemorrhagic stroke typically occur?
Basal ganglia or cerebellum
26
Most hypertensive hemorrhages are medically managed EXCEPT for
patients with clot <1cm from the cortical surface
27
R sided weakness, headache, vomiting a few hours PTA,
L Basal ganglia hemorrhage
28
amyloid deposition in the media of small cortical vessels.
Amyloid angiopathy
29
Focal dilatation of the vessel wall (balloon-like outpouching/fusiform)
Cerebral aneurysm
30
Where does cerebral aneurysm usually occur
branch points of major bessels
31
Thin-walled rupture of cerebral aneurysm cause
subarachnoid hemorrhage
32
Describe the headache of cerebral aneurysm
Thunderclap headache, worst headache of my life
33
Management of Cerebral aneurysm
``` Cranial CT Four-vessel cerebral angiography Early aneurysm occlusion Craniotomy, clipping Endovascular, coiling ```
34
Approximate mortality rate of cerebral aneurysm
50% in the first month
35
Approximately ___ of survivors return to pre-SAH function
1/3
36
____ of survivors have moderate to severe disability
2/3
37
Abnormal dilated arteries and veins without an intervening capillary bed
AVM
38
AVM presents with (3)
SAH intraparenchymal hemorrhage seizures
39
Hemorrhage rate of AVM
2-4% in a year
40
treatment of AVM
``` microsurgical excision endovascular embolization stereotactic radiosurgery (<3cm diameter) ```