NURS 444 week 10 Flashcards

1
Q

Cervical injury

A
  • anterior cord syndrome
  • posterior cord lesion
  • brown- sequard syndrome
  • central cord syndrome

*the higher the injury, the most severe the consequence

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2
Q

Initial Assessment

A
  • assessment of respiratory pattern and ensuring adequate airway
  • assess for indications of intraabdominal hemorrhage or hemorrhage around fracture site
  • assess GCS
  • establishment of level of injury; tetraplegia, quadraplegia, quadriparesis, paraplegia, paraparesis
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3
Q

Spinal Shock

A

> flaccid paralysis
loss of reflex activity below the level of lesion
bradycardia
paralytic ileus
hypotension

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4
Q

Cardiovascular assessment

A
  • due to disruption of the ANS
  • bradycardia, hypotension, and hypothermia result from a loss of sympathetic input.
    Can lead to dysrhythmias
  • systolic bp <90. needs tx because loss of perfusion to spinal cord makes it worse
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5
Q

Autonomic dysreflexia

A

stimulates SNS, life-threatening
- commonly in upper spinal cord injuries
- severe hypertension
- bradycardia
- severe headache
- nasal stuffiness
- flushing
- treatment
- blurred vision

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6
Q

assessment for spinal cord injury

A

> resp assessment
GI and GU
musculoskeletal
psychosocial assessment
labs
CT or x-ray

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7
Q

Interventions for autonomic dysreflexia

A

SIT PATIENT UP! elevate HOB to 40 degrees

find underlying cause like bladder distention

notify

loosen clothing

give nitro or hydralazine (antihypertensive)

maintain bowel function- dis-impact if necessary

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8
Q

ineffective tissue perfusion interventions

A

> reduction and immobilization of the fracture to prevent further damage to spinal cord from bone fragments

> traction and external fixation but surgery may be necessary

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9
Q

immobilization for Cervical injuries

A
  • fixed skeletal traction to realign, facilitate bone healing and prevent further injury
  • halo fixation and cervical tongs
  • stryker frame, totational bed, kinetic tx table
  • pin site care and monitoring of ropes
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10
Q

immobilization of thoracic and lumbosacral injuries

A

^ for thoracic injuries: bedrest and possible immobilization with a fiberglass or plastic body cast

^ lumbar and sacral injuries: immobilization of spine with a brace or corset worn when the client is out of bed; custom-fit thoracic lumbar sacral orthoses preferred

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11
Q

Drug therapy for spinal injuries

A

+ dantrolene
+ 4-AP potassium channel b.
+ baclofen
+ etidronate disodium
+ dextran
+ atropine sulfate
+ dopamine hydrochloride
+ naloxone and TRH
+ sygen
+ methylprednisone (contraversial)

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12
Q

Airway/ Breathing interventions for spinal cord injuries

A
  • Airway management is priority.
  • injuries at or above t-6 are at high risk for resp. complications
  • assisted coughing, quad cough (help bring up secretions), cough assist
  • use of incentive spirometer
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13
Q

Interventions: Impaired physical mobility; Self-care deficit

A

< spinal cord injuries: monitor for risk of pressure ulcers, contractures, and DVT/ pulm. emboli
< proper positioning, skin inspection, ROM exercises, heparin, and graduated compression stockings
< prevent orthostatic hypotension
< promote self-care

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14
Q

Interventions; Impaired Urinary Elimination: Constipation

A

~ a bladder retraining problem
~ spastic bladder: manipulating external area
~ flaccid bladder: valsava maneuver
~ encourage consuption of 2000- 2500 mL of fluid daily to prevent UTI
~ long-term renal comp.
~ s&s of UTI not perceived by client

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15
Q

Establishing a Bowel retraining program

A
  • consistent time w/ bowel elimination
  • high fluid intake: at least 2000mL/day
  • high fiber diet
  • rectal stimulation
  • stool softeners PRN
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16
Q

Interventions: impaired adjustments

A

> invite clients to ask questions about life changes: reply openly and honestly
encourage clients their perceptions and coping strategies options
education to clarify misconceptions

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17
Q

Myasthenia Gravis

A

^ autoimmune of neuromuscular junction
^ characterized by fluctuating weakness
^ 14 cases per 100,000 in US
^ most common- ages 10 and 65, peak between 20-30
^ 3x more common in women
^ weakness primarily in muscles innervated by cranial nerves, skeletal, resp.
^ Thynoma- encapsulated thymus gland
^ antibodies that attack ACh receptors found in blood 80-90% of clients. excessive thyroid hormone also found
^ progressive and partially resolved by rest

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18
Q

Most common MG symptoms

A
  • involvement of eye muscles; ocular palsies, ptosis, diplopia, weak or incomplete eye closure
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19
Q

Myasthenia Crisis

A

exacerbation of symptoms caused by undermedication with anticholinesterase

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20
Q

Cholinergic Crisis (in MG)

A

acute exacerbation of muscle weakness caused by overmedication of cholinergic (anticholinesterase) drugs

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21
Q

Myasthenia Gravis Crisis s&s

A

!! resp. distress
!! increased pulse and BP
!! poor cough
!! secretion aspiration
!! dysphagia
!! weakness
!! improve w/ edrophonium (tensilon test)

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22
Q

Cholinergic crisis s&s

A

!! ABD. CRAMPS
!! DIARRHEA
!! N&V
!! EXCESSIVE SECRETIONS
!! MIOSIS
!! fasciculations
!! weakness
!! worse w/ edrophonium (tensilon test)

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23
Q

First interventions in MG crisis

A
  • Tension test
  • PRIORITY: maintain adequate resp. function
  • cholinesterase inhibiting drugs are withheld because they increase resp. secretions and are usually ineffective for the first few days after the crisis begins.
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24
Q

Tensilon Testing

A

within 30 to 60 sec. after injection of Tensilon, most myasthenic clients show marked improvement in muscle tone that lasts 4 to 5 minutes.

  • prostaglandin is also used
  • cholinergic crisis is due to overmedication
  • MG crisis is due to undermedication
  • atropine sulfate if the antidote for Tensilon complications
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25
Q

Tensilon

A

cholinesterase inhibitor to allow more acetylcholine to reach receptor sites

26
Q

Cholinergic Emergency Crisis: interventions

A

+ anticholinergic drugs are withheld while the client is maintained on a vent.
+ atropine may be given and repeated, if necessary
+ observe for thickened secretions due to drugs
+ improvement is usually rapid after appropriate drugs

27
Q

Atropine

A

given if patient worsens with Tensilon test

28
Q

Cholinergic crisis management

A
  • immunosuppression
  • plasmapheresis
  • resp. support
  • promoting self-care guidelines
  • assisting w/ communication
  • nutritional support
  • eye protection: inability to close eyes, fake tears in day and lub at night
  • surgical management (thymectomy)
  • small frequent high-calorie meals
29
Q

Plasmapheresis

A

removes circulating antibodies.

  • plasma selectively separated from whole blood; blood cells are returned to the client without plasma
  • plasma usually replaces itself. If not, then client is transfused w/ albumin
30
Q

Education for Myasthenia G.

A

< exacerbations include; stress, infection, surgery, hard physical exercise, sedatives, enemas, and strong cathartics (laxative)
< avoid overheating, crowds, overeating, erratic changes in sleeping patterns, emotional extremes
< teach warning signs
< importance of compliance

31
Q

MS

A
  • autoimmune
  • Destruction of myelin sheath of CNS
  • between 20-40
  • > 2x more likely in whites
  • women 2x more than men
32
Q

Major type of MS

A
  • relapsing-remitting
  • progressive- relapsing
  • primary progressive
  • secondary progressive
33
Q

Common physical assessment findings

A

> flexor spasms at night
intention tremor
dysmetria
blurred vision, diplopia, decreased visual acuity, scotomas, nystagmus
hypalgesia, numbness, tingling or burning
bowel and bladder dysfunction

34
Q

Drug Therapy MS

A

+ biological response modifiers
+ immunosuppressives
+ steroids
+ antispasmodic drugs
+ adjunctive

35
Q

MS management

A

promoting mobility and self-care

managing cognitive probs

adapting to changes in sexual functioning

managing bladder & bowel

treating visual disturbances

complementary and alt. therapies

36
Q

causes of seizures

A

_ drugs/ toxins
_ trauma
_ infection
_ lesions
_ fever
_ metabolic alterations
_ extra-cranial disorders
_ genetics
_ idiopathic (75% of seizures)

37
Q

three major classes of seizure onset

A
  1. generalized onset
  2. focal onset
  3. unknown onset
38
Q

Seizure phases

A

Prodromal phase- can occur a week before. Trigger

Aural phase- sensory warning

Ictal phase- onset of symptom to end

Postictal phase- recovery phase

39
Q

Generalized onset seizure

A

both hemispheres involved
Motor;
- tonic: sudden stiffness arms, trunk or legs
- clonic: repeated jerking of arms, legs
- tonic-clonic
- myoclonic: jerking of muscle or muscle group
- atonic: muscles become limp

40
Q

Focal Onset

A

1 hemisphere

-awareness
-impaired awareness
-motor
-non-motor

41
Q

non-motor seizure

A

Absence:

typical

atypical

myoclonic

42
Q

non-motor: Absence

A

Typical absence:
usually in children, brief starry spell that lasts <10 sec

Atypical absence seizures:
starring spells w other manifestations
- eye blinking
- jerking movements of lips
- lasts >10 sec
- usually continue into adulthood.

***more dangerous because not aware of them, can occur 100x/ day and glucose consumption is greater&raquo_space;> hypoxemia, hypoglycemia

43
Q

Atonic & myoclonic

A

Atonic (motor)
- “drop” attack
- typically < 15 sec

Myoclonic (motor/ non)
- rhythmic arm abduction (3 ratcheting movements per sec.) leading to progressive arm elevation

  • usually 10-60 sec
  • eyelid myoclonia- jerking of eyelids
44
Q

Focal-onset seizures

A

Described by level of awareness

  • focal awareness seizure: conscious and alert
  • focal impaired awareness seizures: loss of consciousness or awareness: eyes open but no interaction
45
Q

Status Epilepticus

A
  • emergency
  • can occur with any type of seizure
  • neuron can seize to function due to exhaustion > permanent brain damage risk
  • continuous seizure or recurrence of seizures w/o return to consciousness
46
Q

Convulsive status epilepticus

A

*most common form

  • can lead to fatal resp. insufficiency, hypoxemia, dysrhythmias, hyperthermia, systemic acidosis (not breathing)
47
Q

nonconvulsive status epilepticus

A

long or repeated focal impaired awareness seizures

48
Q

Refractory status epilepticus (RSE)

A

continuous seizure activity despite administration of first and second line therapy

49
Q

Sudden unexplained death in epilepsy (SUDEP)

A

higher in men and people without medication therapy with seizure disorder

50
Q

Nurse’s role in status epilepticus

A
  • protect head
  • ease patient to the floor
  • maintain patent airway
  • turn to the side
  • loosen constrictive clothing
  • initially; rapid-acting lorazepam (Ativan), diazepam (Valium)
  • assess and record details
  • give oxygen, glucose, D50
51
Q

Psychosocial complications in seizure disorders

A

** effects on lifestyle is most common

** suicide risk higher (r/t tx drugs?)

52
Q

Diagnostics for seizure disorders

A

+ accurate health hx
+ blood work (liver&kidney, CBC, serum chems), urine: to rule out metabolic
+ EEG
+ CT or MRI
+ cerebral angiography, SPECT, MRS, MRA, and PET (certain situations)
+

53
Q

Goal for seizure disorder caare

A

minimize seizures with little or no drug effects

54
Q

Drug Therapy in seizures

A

Aimed at prevention
- stabilize nerve cell membranes and prevent spread
- 70% patients controlled with meds.
- monitor drug serum levels

55
Q

Seizure drug SE

A

many have long half-life: may be given 1-2 x/day
!! idiplopia
!! drowsiness
!! ataxia
!! mental slowness

56
Q

What a nurse should assess regarding anti-seizure drugs

A

Teach not to discontinue abruptly
- a simplified drug regimen helps with compliance

assess:
- nystagmus
- hand & gait coordination
- cog. functioning
- mental awareness

57
Q

unusual SE for anti-seizure drugs

A
  • rashes
  • gingival hyperplasia (Dilantin)
  • blood dyscrasias
  • hisituism (women grow hair in unusual areas)
  • renal/ hepatic toxicity
58
Q

3 requirements for surgery (seizures)

A

1) confirmed seizure disorder diagnosis
2) trial and failure of drug therapy
3) defined electroclinical syndrome

59
Q

Surgery (seizures)

A

+ anterior temporal lobe resection
+ may remove cortex
+ separate two hemispheres (corpus colostomy)

-about 80% seizure-free in 5 years
- about 72% seizure-free in 10 yrs
- not all seizure types benefit

60
Q

Vagal Nerve Stimulation
(seizure interventions)

A

other adjunct to drugs when foci is not accessible
- may increase blood flow
- may raise levels of neurotransmitters

61
Q

Responsive stimulation
(seizure interventions)

A

continually monitors EEG to detect abnormalities, then responds to seizure activity by delivering electrical stimulation to exact point

  • similar to cardiac pacemaker
  • neuropace RNS system
62
Q

Nursing Assessment: Objective

A

> Tonic-clonic;
` loss of consciousness, muscle tightening, then jerking
dilated pupils, hyperventilation then apnea postictal somnolence

> Focal (aware);
` aura
`sensory, motor, cognitive phenomena

> Focal: impaired awareness;
` altered consciousness w/ inappropriate behaviors, automatism, amnesia of event