O&T: Childhood Injuries Flashcards

1
Q

Childhood injury and Paediatric fractures

A

Childhood injuries:
- Common
- Related to socioeconomic factors:
1. Low family income
2. Poor education level
3. Big family structure
4. Poor accommodation

Paediatric fractures:
- 50% death in children <14 are related to trauma
- 15% childhood injuries involve musculoskeletal system
- most paediatric trauma are **single limb, **low energy injuries
- ***Upper limb fracture > Lower limb fracture
- basic principles of fracture treatment ~ adults
- differences in
1. Anatomy
2. Biomechanical properties of bone
3. Physiology of bone healing

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2
Q
  1. Anatomical difference
A
  1. **Bone less dense, more porotic, more vascular, thinner cortex, presence of physis
    - **
    less force required for fracture to occur
    - fracture more common than dislocation
    - fracture heals ***faster
  2. **Periosteum is thicker, stronger, more vascular (periosteum: contain osteoprogenitor cells —> osteoblasts for growth, remodeling, repairing of bone —> diameter growth of bone)
    - form **
    callus quicker
    - good soft tissue ***splintage
  3. Presence of **growth centres (physis + secondary ossification centres)
    - physeal injury —> growth arrest resulting in shortening / angular deformity
    - ossification centres in elbow (temporal appearance sequence: **
    CRITOE) (2, 4, 6, 8, 10, 12 (SpC C Fang Revision))
    —> Capitellum (1 yo) (2 yo)
    —> Radial head (3 yo) (4 yo)
    —> Internal (Medial) epicondyle (5 yo) (6 yo)
    —> Trochlea (7 yo) (8 yo)
    —> Olecranon (9 yo) (10 yo)
    —> External (Lateral) epicondyle (11 yo) (12 yo)
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3
Q
  1. Biomechanical differences
A
  1. **Weaker bone —> **Less energy needed to cause a fracture
  2. Bone more **elastic with thick periosteum resulting in
    - **
    Greenstick fracture (bone bends and cracks, instead of breaking completely into separate pieces)
    - **Plastic deformation (deform without fracture)
    - **
    Torus (Buckle) fracture (incomplete fractures of shaft of a long bone that is characterised by bulging of the cortex)
  3. Ligament injury less common
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4
Q
  1. Physiological differences
A
  1. More ***rapid healing of fractures
    - physis > metaphysis > diaphysis
  2. May result in **growth acceleration
    - more obvious in proximal tibia + femur
    - **
    shortening allowed during treatment of fractures to prevent overgrowth phenomenon
  3. Good **remodeling power according to the **plane of adjacent joint (e.g. flexion deformity in distal femur —> can be corrected, but valgus deformity cannot be corrected ∵ not in same plane as knee joint)
    - allow deformed bone to remodel itself into normal shape
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5
Q

Growth centres / plate

A
  • Special piece of cartilage
  • At 2 ends of bones
  • Appear at different time point of age
  • Each growth centres contribute to different degrees to bone growth
    —> e.g. 80% of humerus growth from proximal growth centre, 20% from distal growth centre
    —> e.g. 30% of femur growth from proximal growth centre, 70% from distal growth centre

4 zones:
1. Reserve / Resting zone
- cells remain dormant until they divide + proliferate
- tissue bank: growth plate retain ability to divide + grow
- damage —> non-functional growth plate —> ***grow arrest / deformity / leg-length discrepancy

  1. Proliferative zone (new cell production)
  2. Hypertrophic zone (chondrocytes enlargement in growth direction)
    - **most commonly injured (∵ **transition point between most mobile segment of growth plate and most rigid area of zone of endochondral ossification)
    —> but resting zone not damaged
    —> not affect bone growth
  3. Calcification zone (endochondral ossification)
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6
Q

Physeal injury: ***Salter-Harris classification

A
  • Resting zone located near to epiphysis than metaphysis

Type 1:
- Separation of epiphysis from metaphysis
- Mild displacement
- Intact periosteum
- ***Resting zone untouched —> Good prognosis

Type 2:
- Epiphysis-metaphysis separation
- Intact epiphysis, fragment of metaphysis attached
- Strain / Torn epiphyseal periosteum
- ***Resting zone untouched —> Good prognosis

Type 3:
- Physeal separation associated with epiphyseal fracture
- Intra-articular fracture
- Fracture through germinal layer (resting zone)
- ***Resting zone damaged —> Good prognosis with treatment

Type 4:
- Extended from metaphysis across growth plate into epiphysis
- ***Resting zone damaged —> Good prognosis with treatment

Type 5:
- Crush fracture of a part / whole physis from compression
- ***Resting zone damaged —> Poor prognosis

End result of physeal injury:
- Resting zone damaged (**Type 3-5: injury extending to epiphysis)
—> scarring in growth plate
—> formation of vertical septa
—> structural disorganisation
—> **
bone bridge / **bone bar formation (depend on location + size: **eccentric location + large size —> deformity)
—> growth arrest / deformity / leg-length discrepancy

  • Resting zone undamaged
    —> great remodeling potential
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7
Q

**Remodeling of bone + **Consideration of fracture management in children

A

Complex process involving balance between bone formation + resorption

Depends on:
1. Endochondral
2. Periosteal bone formation
3. Integrated resorption

Paediatric fractures have high remodeling ability
—> Acceptable limit of deformities is based on anticipated remodeling
1. **Child’s age / growth potential remaining
2. **
Distance of fracture from end of bone / physis —> closer to physis —> better remodeling
3. **Longitudinal growth potential of that physis (e.g. proximal humerus growth centre)
4. **
Angulation amount + direction

—> When closer to skeletal maturity —> require Better anatomical reduction + More stable fixation

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8
Q

Diagnosis of fracture in children

A
  • Difficult
  • X-ray require 2 views
  • may need MRI
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9
Q

Management of fracture in children

A

Difficult:
1. Nervous child
2. Unable to give good history
3. Uncooperative in P/E
4. Anxious parents

Initial assessment:
1. ABCDE (beware of differences in vitals in different age group: faster HR, lower BP, higher RR)
2. Investigations
3. Fracture assessment
- Adequate exposure of fracture site
- Look out for associated soft tissue injuries
—> Rule out **open fracture / open wound
—> Skin blistering / abrasions / necrosis
—> **
Skin impingement by bone ends
—> Subcutaneous swelling
—> **Neurovascular injury
—> **
Compartment syndrome

  1. Reduction if necessary (Pain control + Anaesthesia)
    - Open / Closed reduction
  2. Immobilisation if necessary
    - Cast / External / Internal fixation
  3. Rehabilitation always
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10
Q

X-ray examination

A
  1. Confirm fracture
  2. Configuration of fracture
    - Low energy trauma: Spiral / Oblique / Transverse
    - High energy trauma: Butterfly / Comminuted
    - Greenstick / Torus / Plastic deformation
  3. Alignment of fracture
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11
Q

Fixation methods in children

A

More conservative treatment ∵
- Good healing + remodeling potential
- Relatively less post-op contracture

Smaller deformities:
1. **Traction
2. **
Plaster

Larger deformities / Skeletal more mature:
3. K-wire fixation
4. Plate
5. External fixator
6. Intramedullary devices

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12
Q

Supracondylar fracture of humerus

A
  • Often 6-7 yo
  • Involves thin bone between **coronoid fossa and **olecranon fossa of distal humerus (Dumbbell shape shown in cross section)
    —> ***Dumbbell configuration make fracture unstable
    —> Very often have deformity in displaced fracture

Complications:
1. **Vascular injuries
2. **
Neurologic deficits (esp. **Anterior interosseous nerve)
3. **
Cubitus varus / Recurvatum
4. **Volkmann’s contracture
5. **
Compartment syndrome

Classification:
1. Extension (95%) (i.e. distal humerus向後彎)
- fall on outstretched hand —> ∵ laxity of elbow —> hyperextend
- associated with more complications (e.g. neurovascular injuries ∵ most are located **anterior to humerus —> impinged by fracture ends)
- most displaced fractures (Type 3) are extension type (97%)
- **
Gartland classification
—> **Undisplaced (type 1): Conservative with cast
—> **
Displaced with intact posterior cortex (Type 2): Surgery needed
—> ***Displaced with no cortical contact (Type 3): Surgery needed

  1. Flexion
    - fall on flaccid elbow

Assessing whether fracture is displace or not:
1. ***Baumann angle (~10o)
- angle made by capitellum and horizontal plane of distal humerus —> normal carrying angle of forearm

  1. ***Anterior humeral line
    - line along anterior cortex of humerus —> should intersect with capitellum
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13
Q

Neurological injury

A

E.g. assess by paper, scissors, stones —> assess flexors innervated by median nerve, extensors by radial nerve

  • MUST before operation
  • Difficult to examine
  • May not be able to identify
  • Detailed documentation
  • Majority due to ***neurapraxia (~ several weeks to recover)
  • Generally not considered an indication for open reduction / exploration
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14
Q

Deformities: Cubitus varus, Cubitus recurvatum, Volkmann’s contracture

A

Malalignment:
- Although rotational deformity can be compensated by shoulder
—> Remodeling is usually suboptimal
—> NOT acceptable

Deformity due to ***Poor remodeling power:
1. Physis in distal humerus only contribute 20% of growth
2. Hinge joint —> most Supracondylar fracture of humerus will lead to Cubitus varus —> direction of deformity not favour remodeling

Cubitus varus:
- Most common complication
- Resulted from:
1. Physeal damage
2. Malreduction —> Malunion
- Cosmetic deformity but little functional deficit

Cubitus recurvatum:
- Result from **residual dorsal angulation of fracture —> **Hyperextension deformity of elbow

Volkmann’s contracture:
- A form of Compartment syndrome
- Loss of motor / sensory function, claw hand deformity
Causes:
- **Brachial artery injury usually associated with supracondylar fracture of humerus
- **
Hyperflexion of cast —> now if need to hyperflex elbow for immobilisation —> use internal fixation instead of hyperflexion of cast

(Claw hand: (from web)
- Ischaemia + necrosis of forearm muscles —> contracture
—> Wrist flexed ∵ bulkier flexors —> more contracture
—> MCP extended ∵ Insertion of extensor tendons
—> IP joints flexed ∵ insertion of long flexor tendons)

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15
Q

P/E of Supracondylar fracture of humerus

A
  1. Swelling + Tenderness of forearm compartments (potential site of compartment syndrome)
  2. ***“Dimple sign”
    - occurs when a spike of bone penetrates brachialis muscle + anterior SC tissues —> catches on skin
  3. **Neurovascular injuries
    - need to rule out
    - check + check + check **
    radial pulse repeatedly —> Intimal arterial injury can occur slowly over several hours
  4. ***Compartment syndrome
    - need to rule out
  5. Associated injury
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16
Q

Investigations of Supracondylar fracture of humerus

A

X-ray:
1. ***Fat pad sign (swelling around fracture site)

(NB Type 1 supracondylar fracture of humerus can be missed)

17
Q

Treatment of Supracondylar fracture of humerus

A

***Restoration of alignment:
- Anterior humeral line
- Baumann angle (9-26o)
- Lateral + Medial cortices

  1. Reduction
    - Open / Closed reduction
  2. Immobilisation
    - Cast / External / Internal fixation
  3. Rehabilitation
18
Q

Open reduction

A

Indications:
1. **Vascular compromise (i.e. Torn / Impinged brachial artery)
2. **
Failure of reduction due to ***soft tissue interposition
- Buttonhole through the brachialis muscle

Complications:
- Elbow stiffness
- Myositis ossificans
- Neurovascular injury

19
Q

Post-op care

A
  1. Haemodynamic status
  2. Respiratory status
  3. ***Neurovascular monitoring
    - Skin colour
    - Temp
    - Capillary return
    - Pulse
    - Numbness
    - Weakness
    - Unremitting pain (compartment syndrome)
  4. ***Exclude compartment syndrome
  5. ***Swelling control
    - Limb elevation
    - Avoid local pressure that will impair circulation
    - Encourage active mobilisation of un-involved joints
  6. ***Avoid pressure / plaster sores
    - Prevent compartment syndrome
    - Frequent change of position
    - Bivalve POP to examine wound if suspected impingement / excessive swelling
    - Suitable mattress
  7. ***Pain control
    - Adequate analgesics
    - Tender loving care
    - Distraction e.g. video games
  8. Psychological support to parents
20
Q

(Extra:
- Hueter-Volkmann Law
- Wolff’s law)

A

Hueter-Volkmann Law:
- Growth is retarded by increased mechanical compression, and accelerated by reduced loading in comparison with normal values
—> More specifically applied to compression and tensile forces acting on physeal growth plates

Wolff’s law:
- Bone in a healthy animal will adapt to the loads under which it is placed
—> Loading on a particular bone increases, the bone will remodel itself over time to become stronger to resist that sort of loading