Obstetrics Flashcards

(34 cards)

1
Q

Well managed epidural analgesia benefits

A

Reduced material and feral acidosis
Increased uteroplacental flow
Reduction in uterine activity
Reduction in incoordinate uterine activity

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2
Q

1st stage labour blockade requires which nerves to be blocked

A

T10-L1

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3
Q

Second stage labour requires what nerves to be blocked

A

S234

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4
Q

What is paracervical block and why shouldn’t it be used in labour

A

Paracervical block is local to the lateral fornix Vagina , blocks paracervical ganglion and provides analgesia to cervix and uterus

Used in gynae procedures

Not used labour as no benefit second stage and can cause profound fatal bradycardia, LAST, Infectuon, and neuropathy

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5
Q

Causes of maternal death 2020

A

VTE commonest
Cardiac remains high as indirect cause
SUDEP worryingly high - refer anyone with nighttime or uncontrolled seizures

Other causes, sepsis, mental health, haemorrhage

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6
Q

Amniotic fluid embolism presents with

A
Refecatory hypoxaemia (cyanosis) 
Pulmonary hypertension 
Systemic hypotension 
Petechiae
Seizures 
Foetal distress
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7
Q

PDPH when should follow up happen and for how long

A

OOA guidance is reviewed within 24 hours then daily, and continue until headache resolves

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8
Q

PDPH conservative treatment

A

Prolonged bed rest not recommended
Hydration
IV fluids only if oral not possible

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9
Q

PDPH pharmacological management

A

Simple analgesia
Short term opiods
Caffeine

No evidence for theophylline acth steroids triptans gabapentinoids

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10
Q

PDPH invasive treatments

A

No evidence for acupuncture occipital nerve blocks etc

Epidural saline may transiently improve ymsoykms

Epidural blood patch - when conservative therapy ineffective ans woman experiences difficulty performing activities of daily life and caring for her baby

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11
Q

EBP timing and steps prior

A

Less than 48 hours reduction in efficacy
No investigations needed prior but if stays after two or is evolving or neurology need scan
Written consent

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12
Q

Risks of ebp

A

Repeat Dural puncture
Back pain
Neurological complications

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13
Q

Risks of not performing EBP

A

Insufficient evidence but suggestion is reduction headache prevention haematoma

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14
Q

What level should EBP be done

A

Same level or one space lower
20ml
Review within 4 hours procedure
Verbal and written advice and write to GP and midwife

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15
Q

Pathophyisology of PDPH

A

CSF leak
Reduction in ICO and downward traction on pain sensitive intracranial structures
Cerebral vascular venodilation

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16
Q

Differential diagnosis of PDPH

A
Migraine 
CVT 
SUBDURAL 
SAH 
SOL 
Meninigitis 
Dehydration 
Caffeine withdrawal 
PET 
Tension headache 
Lactation headache
17
Q

Features PDPH

A

Frontonocciputal
Better supine
Pressure over abdomen temporarily relieves pain in some

Other features include 
CN palsies 
Visual disturbabce 
Neck stiffness 
Photophobia
18
Q

Prevention of PDPH

A

Spinal needle selection - smaller gauge and pencil point

Loss of resistance to saline

Replace stylet
? Bevel parrelel to fibres

Senior

19
Q

Define pre eclampsia

A

New hypertension more than 140/90
Over 20 weeks pregnant
Evidence of end organ damage ie proteinuria

20
Q

Initial treatment pre eclampsia

A

Labetalol 200mg oral or IV (incremental 50mg)

Nifedipine 10mg

21
Q

Management of pre eclampsia on labour ward

A
Antihypertensives 
Limit fluids 1ml kg hr
Foetal assessment 
Inform neonatal team 
Discuss with haematology 
Thromboprophylaxis
22
Q

Ga changes for pre eclampsia

A
Blunt laryngoscope response with ie opiates 
Avoid ergomettine 
Low volume syntocin infusion 
Senior presence 
Hdu post operatively 
Avoid nsaids
23
Q

Management of seizure in pre eclampsia

A
100% o2 left lateral 
Magnesium 4 gram over 5 minutes 
1 gram per hour 
Can have further magnesium if needed 
Monitor for OHS and deep tension reflexes
24
Q

Maternal risk factors amniotic fluid embolism

A

Strong uterine contractions
Uterine rupture
Multiparty

25
Foetal risk factors for amniotic fluid embolism
Male fetus Polyhydraminos Multiple pregnancy Assisted delivery
26
Immune theory afe
Phase 1 foetal cells in maternal blood stream, increased levels pulmonary vasoconstrictors leading to pulmonary hypertension and RV failure Phase 2 LV failure pulmonary oedema DIC atone haemorrhage Other theory is mechanical
27
Maternal comorbidities associated with obesity
``` Gestational diabetes VTE Cardiomyopathy Preeclampsia OSA ```
28
Obstetric complications increased in obesity
``` PPH Preterm labour Instrumental delivery Induction of labour Wound infections ```
29
Patient factors contributing to difficult airway in pregnancy
Difficult face mask ventilation (increased fat stores) Difficult laryngoscopy (oedema) Increased reflux ( reduced lower oesophageal tone and angle of his) Reduced FRC
30
Anaesthetic factors contributing to difficult airway obs
Time pressured Altered drug dosing Reduced exposure to obs GA Isolated site
31
Obs difficult airway algorithm
1. Pre induction planning and prep RSI and consider 20cm h20 ventilation Laryngoscopy 2+1 2. Declare failed intubation Call for help Maintain oxygenation SGA x2 or FMV 3. Declare CICO 100% oxygen Exclude laryngospasm FONA
32
Measures to minimise airway issues obs GA
``` Identify at risk patients Plan Position Antacids prophylaxis Video laryngoscopy Adequate paralysis Senior anaesthetist Safe extubation plan ```
33
Risk factors pre eclampsia
``` Nulliparity Multiple pregnancy Previous Age over 40 Bmi over 35 Fix Diabetes Hypertension Renal disease Inter pregnancy gap more than ten years ```
34
Iron deficiency in pregnancy
Who says 110 Iron def in obs increases maternal deaths Low birth weight Preterm labour