OCD Flashcards

1
Q

What is obsessive compulsive disorder?

A

• OCD - ANXIETY disorder (differs from Schizophrenia, which is a PSYCHOTIC disorder).
• OCD - characterized by OBSESSIONS (thoughts) and/or COMPULSIONS
(behaviours)

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2
Q

What are obsessions?

A

Persistent, unwanted, irrational thoughts.
Might include:
• Being contaminated or contaminating others,
• Fear of not having completed a ritual or specific act
• Hurting people
• Unwanted sexual thoughts, images, urges

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3
Q

What are compulsions?

A

Compulsions are the tasks that people do to relieve themselves of the obsessions. Compulsions can be mental or physical. Common compulsions include cleaning and/or washing;

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4
Q

Describe the OCD cycle

A

1) Obsession
Constant thoughts
2) Anxiety
Strong, uncomfortable feelings
3) Compulsion
Behaviour to relieve anxiety
4) Relief
From anxiety. Obsession response has been strengthened for future

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5
Q

Summaries the 5 diagnostic criteria for OCD listed by the DSM 5.

A

1) The obsessions cannot be ignored or suppressed and cause anxiety and/or distress.
2) The obsessions and/ or compulsions are time-consuming (take up more than one hour a day) and/ or cause clinically significant distress to daily life.
3) The symptoms cannot be explained by substance use or another medical condition or other mental disorders.
4) The clinician must determine if individual has fair insight the order or not and recognises that their beliefs are probably not true or if they have no inside and are definitely true.
5) Find out if the individual has a current or past history of attack disorder as this can affect the severity of the disorder

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6
Q

What is the onset of OCD when describing the features

A

For most people, the age of onset is late teens and early twenties, although it can start earlier or later - in 25% of male sufferers, before the age of 10. The symptoms usually develop
gradually, though occasionally there is a sudden, acute onset.

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7
Q

What is the prevalence of OCD when describing the features

A

Prevalence means how often or how widespread it occurs. The prevalence rate is between 1.1 and 1.8 percent of the population. In adults, OCD is more common in females than in males. However, in children, it is more common in males than females.

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8
Q

What is the prognosis of OCD when describing the features?

A

Prognosis is the likely course of the disorder. Although treatment can reduce symptoms, about 70% of people experience a chronic and lifelong course, with worsening and
improving symptoms.
The factors associated with a good prognosis include the following:
•Milder symptoms
•Brief duration of symptoms
•Good functioning before full onset

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9
Q

What is the risk factors of OCD when describing the features

A

• Family history. Having parents or other family members with the disorder can increase the risk of developing OCD.
• Stressful lite events People who have experienced traumatic or stressful events or who tend to react strongly to stress may have an increased risk of getting OCD.

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10
Q

What is the difference between symptoms features of a disorder

A

Symptoms of how a disorder shows itself in a patient. Features refer to the general information about the disorder

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11
Q

Describe the main features of OCD

A

The age of onset for OCD is late teens and early twenties, although it can start earlier or later - in 25% of male sufferers, before the age of 10.

Good prognosis for OCD is associated with milder and brief duration of symptoms and good functioning before onset.

The prevalence rate for OCD is between 1.1 and 1.8 percent of the population.

Risk factors for OCD include Family history. Having parents or other family members with the disorder can
increase the risk of developing OCD.
Stressful lite events People who have experienced traumatic or stressful events or who tend to react strongly to stress may have an increased risk of getting OCD.

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12
Q

Name one difference between OCD and schizophrenia

A

OCD is an ANXIETY disorder this is different to Schizophrenia, which is a PSYCHOTIC disorder

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13
Q

List the areas of the brain thought to be involved in OCD

A

•Thalamus
•Orbitofrontal Cortex
•Cingulate Gyrus
•Basal ganglia (which consists of the following two structures)
-Globus pallidus
-Caudate nucleus

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14
Q

Describe the role of the thalamus

A

The thalamus contains primitive checking and cleaning behaviours hardwired in the brain. If this is
overactive, it triggers a compulsion to engage in these behaviours.

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15
Q

Describe the role of the orbitofrontal cortex

A

The orbitofrontal cortex alerts the brain to potential worries in the environment. If overactive, the person would experience anxiety.

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16
Q

Describe the role of the cingulate gyrus

A

Connects orbitofrontal cortex to thalamus

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17
Q

Describe the role of the globus pallidus

A

• Part of BASAL GANGLIA
• Acts as a braking mechanism, controlling activity in thalamus
(“calming” Thalamus)

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18
Q

Describe the role of the caudate nucleus

A

• Part of Basal Ganglia
Inhibits action of globus pallidus fibres. If caudate nucleus is overactive, it over inhibits the globus pallidus fibres. This means the Globus pallidus can’t do its job of calming the Thalamus and it therefore becomes over reactive.

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19
Q

How can malfunction of different areas of the brain relate to OCD?

A

In OCD, the Basal Ganglia can malfunction. This means that the thalamus is overactive - therefore
cleaning and checking behaviours are more likely. These would be the compulsions found in OCD.
The overactive thalamus causes a cascade reaction in the orbitofrontal cortex, leading to the orbitofrontal cortex becoming overactive. The orbitofrontal cortex is hardwired to alert the brain when something seems odd, causing anxiety. This would lead the obsessions found in OCD.

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20
Q

Describe the feedback loop theory of OCD

A

An overactive caudate nucleus means that the globus pallidus does not perform its braking mechanism of inhibiting activity in the thalamus.
This means that the thalamus (which controls primitive checking and cleaning behaviours hardwired in the brain) is overactive. The overactive thalamus in turn causes over-activity in the orbitofrontal cortex. The overactive thalamus explains the compulsions found in OCD because it is responsible for primitive checking and cleaning behaviours. The overactive orbitofrontal cortex explains the obsessions found in OCD, because it is hardwired to alert the brain when something seem odd, causing anxiety.

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21
Q

strengths of a biological explanation for OCD (brain structure and functioning) evidence to support

A

There is a lot of supporting evidence for the idea that brain structure and functioning explains OCD.
•Brain scans show differences in brain structure between people who have OCD and healthy controls.
• Evidence for this: Menzies (2007) found people suffering from OCD had different amounts of grey matter in the orbitofrontal cortex.
• High in Scientific credibility - brain scans are objective and many studies have come up with similar results

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22
Q

strengths of a biological explanation for OCD (brain structure and functioning) applications

A

Surgical lesioning of the cingulate gyrus (the area that connects the thalamus with the orbitofrontal cortex has been used successfully as a treatment for OCD. This would suggest that the idea that OCD results from a faulty feedback loop between the thalamus and the orbitofrontal cortex is correct, since breaking the loop can cure OCD. However, Some people respond to surgery on the cingulate gyrus, some
don’t

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23
Q

Limitations of a biological explanation for OCD (brain structure and functioning)

A

• However, sometimes this surgery does not work; this might mean that OCD is more complex than this theory suggests. For example, Kireev et al (2013) suggest that the functions usually performed by the cingulate gyrus can be taken over by other areas of the brain. Therefore, a weakness of this explanation is that it is reductionist: it does not fully explain OCD.
• A further weakness is that is difficult to show cause and effect using this model. The differences
in brain activity in people with OCD may be a symptom of their OCD, rather than the cause.
• Brain activity & thoughts are related, so it is not easy to say whether the brain activity altered the thoughts, or the thoughts altered the brain activity, as they cannot be separated.

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24
Q

Describe one piece of supporting evidence for brain functioning as a biological explanation of OCD

A

When people with OCD are shown objects that bring on their symptoms, such as a dirty piece of clothing with someone who hates dirt & has a cleaning compulsion, there is an increase in the activity in the orbitofrontal cortex & the caudate nucleus
(McGuire et al. 1994)

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25
Q

Explain how surgical lesioning of the cingulate gyrus both
- Supports the theory
- Argues against the theory

A

•Supports: Surgical lesioning of the cingulate gyrus (the area that connects the thalamus with the orbitofrontal cortex has been used successfully as a treatment for OCD. This would suggest that the idea for OCD results from a faulty feedback loop between the thalamus & the orbitofrontal cortex is correct, since breaking the loop can cure OCD.
•However sometimes this surgery does NOT work; this might mean that OCD is more complex. KIREEV et al. (2013) suggest that the functions usually performed by the cingulate gyrus can be overtaken by other areas of the brain

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26
Q

According to the cognitive explanation what are the causes of obsessive thoughts in OCD

A

False beliefs learned in childhood
Memory problems
Hypervigilance (attentional problem)

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27
Q

Explain how false believes in the cognitive approach can explain OCD

A

People misinterpret their thoughts due to false beliefs (learned in earlier life)
Most people have intrusive thoughts but can shrug them off. People with OCD tend attach importance to them
Respond as if thought were a threat - leading to high anxiety and negative emotions (shame, guilt)
Tend to interpret thoughts as catastrophic, leading to anxiety and then the practicing of avoidance or ritual behaviours

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28
Q

List the five beliefs that researchers suggest may be important in the development and maintenance of obsessions.

A
  1. Exaggerated responsibility that one is responsible for preventing harm to others
  2. Belief - certain thoughts are important & should be controlled
  3. Somehow having a thought/ urge will increase the chances of it happening
  4. tendency to overestimate the likelihood of danger
  5. belief that one should always be perfect & mistakes are unacceptable
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29
Q

Explain how the faulty beliefs can lead to obsessive behaviours found in OCD

A

People who fear their own thoughts usually attempt to neutralize feelings that arise from their thoughts.
They might avoid situations which may spark such thoughts. Another way is by engaging in rituals, such as washing or praying. The anxiety produced by the thoughts lead to the compulsive behaviours seen in OCD.

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30
Q

Give a piece of research evidence that shows cognitive psychology can explain OCD as being due to memory problems.

A

Sher et al (1989) found that people with OCD had poor memories for their actions, for example, they really could not remember if they had turned the light off.

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31
Q

Describe hypervigilant explain OCD

A

Hypervigilance means that they
have an attentional bias which means that are overly sensitive to threat.
This is seen where they may use rapid eye movements to scan the environment, and they may attend selectively to threat related stimuli rather than neutral stimuli. This means that they feel very anxious. The threats that are perceived therefore become the basis of their obsessions. The compulsive behaviours are designed to reduce their anxiety.

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32
Q

Provide one argument AGAINST the cognitive explanation for OCD in terms of C&E

A

Cognitive theories do not really provide a cause & effect explanation of OCD. It does not show that faulty thinking is because of OCD; it could well be that faulty thinking is a symptom of OCD (with the underlying cause being something else e.g. brain structure/ functioning)

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33
Q

What is a biological treatment for OCD

A

Drug treatment

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34
Q

What are the three types of drug treatments used for OCD

A

1) antidepressants
2) anti-anxiety drugs
3) beta blockers

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35
Q

Describe how antidepressants work by treating OCD

A

Antidepressants such as sertraline are used to raise serotonin levels. They do this by blocking its reuptake from the pre-synaptic neuron. This means that more serotonin is available for a longer period.
dosage used to treat OCD is higher than that used to treat depression.
It may take up to 12 weeks for the drugs to take effect.
Some people do not respond to medication.

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36
Q

Describe how anti-anxiety drugs work by treating OCD

A

Anti-anxiety drugs called such as valium work by increasing the effectiveness of GABA in regulating anxiety. GABA is an amino acid that acts to lower physiological arousal and return the body to a resting state.

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37
Q

Describe how beta blockers work by treating OCD

A

work by blocking the stress hormones which are released into the blood stream by the adrenal glands. These stress hormones create symptoms of anxiety such as increased heart rate and respiration; beta blockers prevent this physiological response from occurring. Because the person feels less physiological stress, they may have fewer obsessional thoughts and therefore (because the obsessional thoughts lead to compulsive behavior) less compulsive behaviour.

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38
Q

Give a strength of drug treatments for OCD to do with alternative treatments

A

One strength of drug treatment is that is useful, because it can be used to treat cases of OCD which have not responded to cognitive behaviour therapy.

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39
Q

Give a strength of drug treatments for OCD to do with scientific credibility

A

There is empirical evidence to show that drug treatment can be effective in OCD. For example, Soomro et al (2007) found that antidepressants were more effective than placebo in reducing the symptoms of OCD. This means that the treatment has scientific credibility.

40
Q

Give a strength of drug treatments for OCD to do with effectiveness

A

•Drug treatment can be combined with CBT and has been shown to raise the effectiveness of CBT (POTS, 2004)
• Koran et al. (2002) found that anti-depressant medication did have long term effects compared to a placebo & was effective at preventing relapse over an 80 week trial.

41
Q

Give a strength of drug treatments for OCD to do with ethics

A

Modern drug therapy combined with psychological therapy has reduced the need to resort to neurosurgery, which had some serious ethical issues.

42
Q

Give a weakness of drug treatments for OCD to do with ethics

A

• One limitation of drug therapy is that it may cause side effects such as nausea and headaches. Although these do not last, they are not pleasant for the patient. The side effects may also limit the usefulness of drugs because people may not want to take them.
•In rare cases, use of medication may increase the patient’s anxiety rather than decreasing
• Patients are likely to relapse if drug treatment is stopped.

43
Q

Give a weakness of drug treatments for OCD to do with effectiveness

A

• Drug treatments alone cannot treat most people with OCD; they are most effective when combined with other forms of treatment such as CBT.
• There are individual differences in the way that people respond to drugs; for example Ravizza et al (1995) found that SSRis were not effective for 40% of people.

44
Q

What are the two techniques of CBT that focus on obsessions?

A

•Test beliefs
•Habituation training

45
Q

Cognitive therapy aims to prevent a common cognitive distortion of catastrophising. What is catastrophising?

A

In catastrophising, the person firstly predicts a negative outcome, and secondly, jumps to the conclusion that if the negative outcome did in fact happen, it would be a catastrophe.

46
Q

How does CBT use habituation training?

A

the client is asked to repeatedly think about their obsessive thoughts. The idea is that by deliberately thinking about obsessions, they will become less anxiety provoking, with the consequence that compulsive behaviour is not required to reduce high levels of anxiety.

47
Q

What is ERPT in CBT?

A

A behavioural therapy used as part of CBT for OCD is known as Exposure and
Response Prevention Therapy (ERPT).
ERPT focuses only on the compulsions found in OCD, so it complements the use of cognitive therapy which targets the obsessions
ERP deliberately exposes clients to objects or situations that cause anxiety and requires the client to resist performing the compulsive behaviour. The role of the therapist is to help the person develop ways in which they can resist performing the compulsive behaviours.

48
Q

List the steps in ERPT

A

•Inform the client about ERPT
•Construct an EXPOSURE HIERARCHY
•Repeated exposure to situations which cause anxiety
•Client must RESIST performing the compulsive behaviour

49
Q

Explain what is meant by catastrophising

A

Jumping to the worst possible outcome

50
Q

What is habituation?

A

Asking the client to think repeatedly about their obsessive thoughts

51
Q

What does ERPT stand for?

A

Exposure and Response Prevention Therapy

52
Q

Strengths of CBT as a treatment for obsessive-compulsive disorder to do with ethics

A

One strength of CBT is that it is said to be an ethical form of treatment. This is a strength as it means the treatment is approved by society and does not cause the patient anymore distress as it is ethical. For example CBT teaches the patient to expose themselves to feared situations even outside of the therapy session. This shows that therapy is ethical as in powers patient to use learned techniques on their own. This also helps with the issue and debate of social control as CBT empowers the patient which means and implies that they are less controlled by others in society.

53
Q

Strengths of CBT as a treatment for obsessive-compulsive disorder

A

•The use of CBT is supported by evidence which shows that it is effective in reducing the symptoms of OCD. For example, it is endorsed by the National Institute for Health and Clinical Excellence (NICE, 2006).
•CBT does not have any side effects (unlike medication for OCD).
•CBT has lower relapse rates than medication when it is discontinued.

54
Q

Limitations of CBT as a treatment for obsessive-compulsive disorder to do with why the treatment is limited

A

POTS 2004 found that CBT is an effective therapy for OCD when compared to a placebo group, they also found that sertraline (a drug therapy) was effective, and most effective was a combination of CBT and sertraline. This suggests that although CBT is effective, it is a limited treatment which should ideally be used in combination with other forms of therapy.
• It is difficult to disentangle which of the benefits from CBT are due to the cognitive component and which are due to the behavioral component.

55
Q

Limitations of CBT as a treatment for obsessive-compulsive disorder to do with individual differences

A

•Although about 70% of people respond well to CB, there are still many for whom it is ineffective. Individual dilferences mean that therapy needs to be tailored for the needs to the particular person. For example, Overbeek et al (2002) tound that a third of participants with OCD also had clinical symptoms of depression. This group showed less improvement
when treated with ERP and drug treatment combination compared with a group of participants with OCD only symptoms.
•ERPT - would not be effective for clients that do not have compulsions MASELLIS et al.
(2003) found that a substantial proportion of clients (up to 44%) only suffer from obsessions
They also found that up 75% had co-morbid depression, which lessens effects of ERPT.

56
Q

Describe one similarities between
CBT and drug treatment for OCD

A

There is evidence that both
treatments are effective for OCD.
For example, Soomro’s research
found that antidepressants were
more effective than placebo in
reducing symptoms of OCD.
Similarly, CBT is endorsed by NICE
(2206) who found that CBT is
effective in reducing symptoms of
OCD.

57
Q

Describe one difference between
CBT and drug treatment for OCD

A

Drug treatments for OCD may
have side effects, such as
nausea and headaches. This
may reduce the effectiveness
of drug treatments, as people
may not wish to take them.
In contrast to this, CBT for
OCD does not have any side
effects, which may make it
more likely for people to
continue with their therapy.

58
Q

What is your contemporary study for clinical psychology

A

Obsessive-Compulsive Disorder by POTS (2004)

59
Q

What was the aim of POTS (2004)

A

To compare three treatments:
1) CBT on its own
2) An SSRI (sertraline) on its own
3) CBT and sertraline combined
These were compared against a baseline placebo condition

60
Q

What was the procedure for POTS (2004)

A

• Drugs only and placebo, condition - required the children to fitters westay for first 6 weeks and then every other week (9
• CBT condition: 14 visits
• Combined condition - same treatment as CBT only condition
+ Sertraline
• Ps assessed at baseline, 4 weeks, 8 weeks, and 12 weeks

61
Q

What type of sample was used in POTS (2004)?

A

• A volunteer sample
the study ran across centres, in the USA
All diagnosed using the DSMIV
112 people (participant attrition= 97)
Ages 7-17 years

62
Q

How many participants were used in POTS (2004)?

A

• 112
• All American; 92% white

63
Q

What was the age range for participants in POTS’s study

A

7-17 years old

64
Q

How was the severity of the participants’ symptoms measured in POTS’s study?

A

•A standardised test - the
Children’s Yale-Brown Obsessive-
Compulsive Scale was used.
•Only those with a score of 16 or
above on this scale were included.

65
Q

Which children were
excluded from POTS’s
study, and why?

A

Children who were comorbid
with other disorders such as
Tourette’s s and major
depression
To avoid interaction effects.
(Comorbid refers to having an
additional disorder )
this controls for confounding variables;

66
Q

Why were children who had been diagnosed with ADHD required to be on stimulant medication and stable in POTS’s study?

A

To ensure that their
ADHD would not affect
the treatment for OCD.
•controlling
for the confounding
variable of ADHD;

67
Q

Why were none of the children on
any anti-obsessional medication at
the start of POTS study?

A

To ensure that any change in
the participants was due to the
treatments offered in the study
(CBT and/or sertraline)

68
Q

How were children allocated to the four conditions in POTS study?

A

Randomly, using
computerised system

69
Q

Name each of the four conditions in POTS study

A

• Drugs only
• Placebo pill only
• CBT only
• CBT and drug treatment

70
Q

How were the children assessed at the start of treatment in POTS study?

A

They were
interviewed
• Their baseline
measure was taken
using the CY-BOCS
using independent
evaluators.

71
Q

How long did POTS study last?

A

• 12 weeks
• This means it is a longitudinal
study
• Strength: can show change
and development in OCD
symptoms over time

72
Q

Why did each child have a specialist
psychiatrist assigned to them for the
duration of POTS study?

A

• To monitor their progress
• To provide them with support

73
Q

Describe the procedure for the drugs-only and placebo conditions.

A

• Children attended weekly sessions for the first six weeks
• Thereafter they attended every other week. (9 sessions in total).
•The dose would be established and changed during the clinical session.
•During the week their parents would monitor that the medication was being taken, bf they kept a medication diary.
• Any adverse reactions would result in the medication being changed or
stopped.

74
Q

How many sessions
did the CBT group
have in POTS study?

A

14

75
Q

What were the components of the CBT therapy in POTS study?

A

• Psychological education
•Cognitive training
•Mapping OCD target symptoms
•Exposure and response ritual prevention.

76
Q

What did each CBT session consist of in POTS study?

A

• Goal setting
• Review of the previous week
• Therapist assisted practice
• Homework
• Monitoring

77
Q

What was the combined
condition, and when did
the sessions take place in POTS study?

A

• Drugs and CBT in
conjunction
• Sessions were time
linked and provided
simultaneously.

78
Q

After the initial baseline
assessment, when were the
participants assessed again
during the course of POTS study?

A

They were assessed at weeks 4, 8,
and 12.

79
Q

Who assessed the participants in POTS study?

A

• Independent
evaluators trained to
a reliable standard.
• Controls for
researcher effects
• Increases validity of
the study

80
Q

How did the authors of POTS study try to ensure that the assessments were
reliable?

A

The evaluators were carefully trained
The evaluation process was strictly
supervised and reviewed.
Another strength of the study: high
interobserver reliability!

81
Q

How many of the original participants
completed POTS study?

A

• 97 out of 112.
Low participant attrition- high generalisability

82
Q

What is meant by remission?

A

Where a person has no signs and symptoms of the disorder

83
Q

How did the authors of the study define “entering remission” in POTS study?

A

• A drop below 10
on the CY-BOCS
• Objective; quantitative data

84
Q

Which condition shows the
smallest drop in symptoms
over the course of POTS study?

A

Placebo

85
Q

Which condition shows the
greatest drop in symptoms?

A

Combination (drug treatment + CBT)

86
Q

Rank the treatment in POTS study in terms of effectiveness

A

Best to worst:
•Combination treatment
•CBT alone
•Drug treatment alone
•Placebo

87
Q

What did POTS study conclude?

A

•CBT led to more improvement than
drugs; this suggested that the first line
of treatment should be CBT
•Minimal gain can be added by including the drugs where effective CBT is provided; but the drug may
compensate for less effective therapy.
•Drugs require careful monitoring if
used, as SSRIs have been linked to
suicidal ideation in young people.
•However, drug treatment was well
tolerated,
•Early intervention effective for children and young people with OCD

88
Q

List the strengths of generalisability in POTS study

A

/ Large sample -112 children
(relatively low drop out rate)
Representative of the target
population (children and
young people with OCD)
Analysis of sample showed
there was no difference in
groups (therefore any
difference in groups would
be down to IV)

89
Q

List the limitations of generalisability in POTS study

A

• Conducted in USA
may not generalise to
non-Western societies.
Other societies may
respond differently to
CBT and/or to
medication.
Excluded children who
were co-morbid

90
Q

List the strengths of reliability in POTS study

A

The evaluators who assessed the
children’s symptoms had been
trained to a reliable standard
The scale used to assess their
symptoms was a standardised
measure and thus reliable
All participating centres (three)
used the CBT manual in order to
maintain consistency in treatment

91
Q

List the weaknesses of reliability in POTS study

A

The study was conducted
over three centres BUT
One centre had better results
on the CBT-only condition.
This may be due to the
therapist effect, as one
therapist may have been
more proficient at CBT than
the others.

92
Q

List the applications of POTS study

A

•Useful, as it provides strong evidence about the most effective treatments for OCD in children and young people.
•Shows value of clinical treatments -cause & effect.
•Findings have shown effectiveness of CBT & sertraline combined (although individual differences mean that therapies still need to be tailored to the person - Although this study cannot show this).
•Shows usefulness of psychological
therapies
•SSRI’s can lead to suicidal ideation in some individuals - which needs to be taken into account

93
Q

List the strengths of validity

A

The assessment scale used (CY-BOCS) had been
validated as an accurate measurement of OCD
symptoms
The assessors were blind to the condition that the
participants were in so they could not be biased in their
evaluation of improvements.
The participants in the placebo condition were unaware
that the drug was not active, as was the therapist. This
controlled for demand characteristics and therefore
increased validity.
Participants were randomly allocated into conditions - can
infer cause & effect..

94
Q

List the strengths of ethics in POTS study

A

All participants gave full written consent
as did at least one of their parents.
Volunteer sample (so no one was coerced
into it/ did it against their will)
BUT… Is it always easy for a child to
decline if their parents want them to be a
part of study?
Each participant was assigned a
psychiatrist to monitor and support them.
Those in the drug treatment condition
were regularly checked and dosages
changed as necessary, thus protecting
them from harm.
Use of deception in the placebo
condition: but this was necessary in order
to ensure validity of the study. Everyone in
the placebo condition was offered the
therapy on completion of the study.

95
Q

How does POTS study link to the issue and debate of change over time?

A

• Practice changed as a result of POTS study,
Before 2004 CBT not routinely used for
children - this study has helped change this…
So that CBT is now the first line of treatment
for OCD. However it also shows that CBT and
drug treatment are very effective in
combination
(We are using less drastic measures like
drugs with side effects or brain surgery to
help relieve OCD)

96
Q

How does POTS study link to the issue and debate of practical issues?

A

Volunteer sample - therefore may not be
typical
- may be more motivated therefore
CBT results may be better than in the
general population
Sample from 3 academic centres - but only
in U.S.
- generalisability
Didn’t have co-morbid participants - don’t
know how effective for children with multiple
issues.
Longitudinal Study: Carried out over 12
weeks (time consuming & expensive)