OMM 1 Flashcards
(135 cards)
1
Q
A
2
Q
TMJ dysfunction symptoms
A
pain, clicking when mouth is open or closed, HA, sinus congestion, dizziness, facial pain, tinnitus, hoarseness, ear pain
3
Q
Digastric and suprahyoid
A
depresses mandible
4
Q
Depression/opening of the jaw ->
A
depresses mandible
5
Q
Unilateral contraction of pterygoids
A
contralateral deviation and anterior
6
Q
Contralateral pterygoids
A
draw articular disc anteriorly to facilitate opening
7
Q
Direct blow to a closed mouth or WHIPLASH
A
posterior capsule injury
8
Q
Malocclusion Class 2
A
overbite
9
Q
Malocclusion Class 3
A
underbite
10
Q
Upper molar extraction
A
temporal bone compressed into occiput
11
Q
Lower molar extraction
A
temporal bone compressed into TMJ -> strained sphenomandibular joint -> sphenoid pulled inferiorly and contralaterally
12
Q
Internal rotation of temporal bone
A
anterior lateral movement of mandible
13
Q
External rotation of temporal bone
A
posterior and medial movement of mandible
14
Q
Mandible deviates away from _______ rotated mandible
A
internally
15
Q
Mandible deviates toward ______ rotated mandible
A
externally
16
Q
Stylomandibular ligament
A
attaches temporal to mandible
17
Q
Short leg ->
A
unleveling of occiput -> temporal and TMJ dysfunction
18
Q
Flexion head
A
external rotation of paired bones (wide, flat)
19
Q
Extension head
A
internal rotation of paired bones (long, narrow)
20
Q
CNV1 may be affected by dysfunction of
A
Dysfunction of temporal bone
21
Q
CNV2 may be affected by dysfunction of
A
temporals, sphenoid, maxillae and mandible
22
Q
CNV3 may be affected by dysfunction of
A
sphenoid bone
23
Q
CNV1 dysfunction may affect what
A
ethmoid sinus
24
Q
Tic doulourex
A
CNV2 dysfunction
25
Trigeminal neuralgia
CNV3 dysfunction
26
External carotid may be affected by dysfunction of
temporal, occipital, sphenoid
27
Internal carotid may be affected by dysfunction of
cervical dysfunction C6-C2
28
Internal jugular may be affected by dysfunction of
temporal
29
Lymphatics to head may be affected by dysfunction of
upper thoracic spine, upper ribs (1-4), and clavicle
30
External carotid dysfunction may cause
weakness and altered sensation on opposite side
31
Internal carotid dysfunction may cause
vision abnormalities, and dizziness
32
Internal jugular dysfunction may cause
head congestion
33
Which n passes through the pterygopalatine fossa
CNV2
34
Abducens n lies under the
petrosphenoidal ligament
35
Entrapment of CN VI can lead to
strabismus and diplopia or sixth nerve palsy
36
Abducens n may be affected by dysfunction of
Sphenoid or temporal dysfunction
37
Bell's Palsy
dysfunction of temporal bone impinging on facial n
38
parasympathetics to the eye are carried by
CNIII
39
parasympathetics to the lacrimal gland and nasopharyngeal mucosa are carried by
CNVII
40
CNVII parasympathetic hyperactivity -\>
thin, watery secretions, excessive tear production
41
Somatic dysfunction at T1-T4 may cause
photophobia, tinnitus and unsteadiness, vasoconstriction
42
Hearing decrease and vertigo may result from dysfunction of __________ impinging on CN8
sphenoid, occiput and temporal bones
43
HEENT Chapman’s Reflex Points - anterior
clavicle to 2nd rib
44
HEENT Chapman’s Reflex Points - posterior
suboccipital musculature, intertransverse spaces C1-C2
45
All facial bones except mandible are driven by
sphenoid
46
Referred sinus pain follows
trigeminal
47
CNV1 innervates all sinuses except
Maxillary (CNV2)
48
Otitis Media may be caused by ______________ of the temporal bone
internal rotation
49
secretory to submandibular, sublingual and lacrimals
CNVII
50
Parasympathetic n carried by CNVII
Greater Petrosal
51
Parasympathetics carried by glossopharyngeal synapse ____________ and innervate
otic ganglion; parotid gland
52
Sympathetics to the head are carried via
T1-T4 -\> sphenopalatine ganglion
53
Vertigo may result from dysfunction of
temporal bone
54
Internal rotation of the temporal bone
closes off the ET; high pitched ringing
55
Treating sinusitis with OMT
inhibitory trigeminal stimulation over CNV1 and CNV2
56
relaxing medial pterygoid muscle, allows
tensor veli palatine to open ET
57
out pouching of respiratory diverticulum
d22
58
bifurcation into R/L bronchial buds
d27
59
branching into lobes, 3 on R, 2 on L
5w
60
branching into tertiary buds, 16w (terminal bronchial buds) -\> 300-700 million sacs in mature lung, 20-70 million sacs
16w
61
Rib lesions are common with
extended Type II dysfunctions
62
Diaphragm and quadratus lumborum attach to which ribs
11-12
63
Inhalation ribs
up in front, down in back
64
Structural Rib Dysfunction
Not related to breathing, rather a disturbance due to rib motion and biomechanical restrictions of the thoracic spine
65
key ribs are often
structural ribs
66
Functional Thoracic Inlet
vertebral units of T1-4, ribs 1 and 2 plus their costo-cartilage, and the manubrium
67
Anatomic Thoracic Inlet
manubrium, rib 1, body of T1
68
Chronic Obstructive Lung Disease
chronically contracted diaphragm, overuse syndrome of accessory muscles
69
Accessory Muscles: Forced inhalation
scalenes, SCM, serratus anterior, external intercostals
70
Accessory Muscles: Forced exhalation
rectus abdominus and internal intercostals
71
asthma reflex
T2 left
72
linking of airway
Normal epithelium is ciliated columnar with mucus secreting goblet cells
73
\_\_\_\_\_\_\_\_\_\_\_ influence is dominant in normal functioning lung
parasympathetic (watery mucus)
74
While asthma is “obstructive”, there may be a component of
restrictive lung disease
75
Restrictive lung disease limits
the amount of air that can get in and out of the lung
76
Obstructive lung disease limits
the rate of airflow out of the lung
77
COPD
chronic bronchitis AND/OR emphysema + airway hyperreactivity
78
antitrypsin
inhibits lung elastase and prevents lung destruction and emphysema/COPD
79
COPD findings
hyperinflation/barrel chest, hyperresonance, sd in right upper thoracics
80
FEV1 \>80% FEV1 50-80% FEV1 30-50% FEV1
stage 1 stage 2 stage 3 stage4
81
Chronic bronchitis
long-term exposure to irritant (smoking), productive cough
82
Emphysema
abnormal permanent entrapment of air - enlarged alveolar sacs
83
Obstructive Lung Disease
air is trapped in lung, maximum inhalation (barrel chest), forced expiration (no IRV, FVC
84
blue bloater
COPD - Chronic hypoxemia leads to erythrocytosis, pulmonary hypertension, and eventually right ventricular failure
85
restrictive lung disease
A stiff thoracic cage results, increased respiratory effort
86
Order of treatment of thoracic cage
Treat the spinal segment first, then any structural rib, then any respiratory dysfunction.
87
key area of lymphatic obstruction
fascia
88
Anterior and middle scalenes attach to
rib 1
89
Posterior scalene attaches to
rib 2
90
Diaphragm: 3 apertures levels
vena cava (T8), esophagus (T10), aorta (T 12)
91
lRT Parasympathetics
OA, AA, C2, suboccipital
92
Earl Miller
1923 - developed lymphatic pump technique
93
Thoracic pump is more effective if the chest cage is
compliant
94
A.T. Still Research Institute established
1913
95
Louisa Burns D.O., a pioneer in osteopathic research, joined AT Still research institute
1914
96
research grants from the AOA to Osteopathic institutions
1939
97
Frank Chapman
Neurolymphatic reflexes in 1920
98
“Lymphatic Reflexes: A Specific Method of Osteopathic Diagnosis and Treatment” published in
1929 by Chapman
99
Charles Owens, D.O.
pelvic-thyroid-adrenal syndrome” (PTA
100
Paul Kimberly, D.O.
FAAO re-introduced Chapman’s reflexes in the KCOM curriculum in the late 70’s
101
Ward E. Perrin
1943 grad from CCOM, CCOM faculty
102
first recipient of the AOA’s Bureau of Research Gutensohn/Denslow Award in 1984
Dr. Kelso
103
William Garner Sutherland, D.O
1939 Cranial
104
Floyd Peckham, DO
1921 grad CCOM - helped keep CCOM afloat financially
105
Denslow
1941-1943: spinal reflex research
106
HEENT parasympathetics (VS)
occiput, C1, C2
107
HEENT sympathetics (VS)
T1-T5
108
The input from both visceral and somatic structures end on common
interneurons
109
Lawrence Jones
1955 tender points
110
Upper cervical left side dysfunction may cause
AV node effects (PNS) --\> impaired conduction and dropped ventricular contractions
111
Upper cervical right side dysfunction may cause
SA node effects (PNS) --\> Bradycardia
112
Upper thoracic right side dysfunction may cause
SA node effects (SNS) --\> tachycardia
113
Upper thoracic left side dysfunction may cause
AV node effects (SNS) --\> premature ventricular contractions
114
Cardiac Rhythm
T1-T2
115
Myocardium
T1-T5 (L \> R)
116
Posterior wall MI
T5
117
Bronchomotor
T1-T3 (b/l or ipsilateral)
118
Lung
T1-T4 (b/l or ipsilateral)
119
Esophagus
T2-T6 R
120
Gastric
T4-T10 L
121
Upper RT
T1-T5
122
Oropharynx
T1-T2
123
Thyroid
C4-C6, T2
124
Ventricular involvement
C8-T3L
125
Atrial involvement
T4-T6L
126
Anterior infarct
T2-T3L
127
Inferior infarct
T3-T5L, C2
128
HTN Linkage Pattern
C6, T2ESR-R & Left Inhalation Rib 2 T6FSR-L & Right Exhalation Rib 6
129
Underlying CAD
Type I curve convex Right
130
Anterior Adrenal CP
2-2.5” above & 1” lateral to umbilicus
131
Posterior Adrenal CP
b/w T11-T12 b/w SP and TP
132
Anterior Kidney CP
1” above & 1” lateral to umbilicus
133
Posterior Kidney CP
b/w T12-L1 b/w SP and TP
134
MI Anterior CP
2nd ICS near sternum
135
MI Posterior CP
b/w T2-T3 b/w SP and TP
