OMM Exam 2 Flashcards

1
Q

Respiratory System:

Autonomic Innervation of lung, Lymphatics of lung, Anatomic Structures of the lung, chapman’s points of the lung

A

Autonomic Innervation: Sympathetic is T1-T6 bilaterally. Broncho-dilation, epithelial hyperplasia with thickened secretions, VC with local hypo-perfusion.

Vagus is parasympathetic. thin secretions, bronchoconstriction.

Right lung and most of the left lung is drained by right lymphatic duct.

Anatomic: vertebrae and ribs.

Muscle: diaphragm and other respiratory muscles.

Chapman’s Points: Between 3rd and 4th rib for upper lung, between 4th and 5th rib for lower lung.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is pathogenesis of the lung infectionS

What are Viscerosomatic components of lung function?

A

Infection –> irritation –> loss of surfactant–> exudate formation –> congestion, edema–> reduced pulmonary function.

They basically are the C2-C3, and T1-T6.

There are palpable muscle and joint changes.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What is the diaphragm stressed by?

A

Ribs and spine via viscerosomatic reflexes, tissue resistance due to congestion.

This stress produces strain of the lower 6 ribs and thoracolumbar junction–> leads to increased lumbar lordosis and flattening of the diaphragm –> decreased pressure gradient between the thoracic and abdominal cavities –> results in decreased lymph flow and increased tissue congestion.

T1-T6 paraspinal muscles will have increased tone.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

OMT in pneumonia describe the goals/ techniques.benefits.

A

Goal: decrease the congestion by using lymphatic drainage.

Pump is CI when patient is febrile. You have to open the thoracic inlet first.

Decreasing SNS hyperactivity by treating VSR first –> rib raising, para-spinal inhibition, chapman’s points.

Normalize parasympathetic tone–> OA decompression,

Decrease mechanical impediments to thoracic cage motion

Improve Diaphragm function –> doming of the diaphragm, treating C3-C5.

OMT benefits removes waste, reduces vascular constriction, synergistic with medical therapy, improves respiration.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

MOSPE Trial

A

Significant reductions in length of stay, duration of IV antibiotics, and respiratory failure and death when OPP is implemented

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

OMT and COPD

A

Improves RV, TLC, and pCO2 and O2 saturation.

OMT is used to improve diaphragmatic function, thoracic drainage, and chest cage motion.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

OMT and Asthma:

A

OMT is used to treat asthma.

During attacks–> increase sympathetic tone during attacks. T1-T6 rib raising –> bronchodilation.

Between attacks: improve thoracic, sternal, costal motion

Shown to reduce length of stay, episode frequency and severity, improve peak flow.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Scoliosis definition, types, naming, severity

A

Lateral curvature of spine.

Types: Dextro –> rotated right, sidebent left.

Levo –>rotated left, sidebent right

Structural: does not correct with side-bending.

Functional :corrects with sidebendng

Severity:
Mild: 5-15 degrees
Moderate: 20-45 degress
Severe: > 50 degrees. >50 degrees compromises respiratory function, >75 degrees compromises cardiovascular symptoms.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Osteopathic Considerations

A

Body develops compensatory curves to maintain balance.

Rotation occurs into convexity. Dextro–> rotated right.

On convex side, the ribs are posterior and separate. On Concave side, ribs are closer and anterior

Disc space compression on concave side. Structural changes and growth retardation on concave side.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

Scoliosis Screening

A

Look/feel for scioliosis.

Look for rib hump when patient bends forward.

Check for short leg.

Imaging to determine severity.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Cobb angle

A

<10 degrees is reevaluate in 6 - 12 months

> 10 follow up every 4- 6 months.

Treat patients whose curves progress more than 5 degrees.

Curves <20 in adult will not usually progress

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are treatment goals?

A

Goals: improve balance and flexibility, address the primary cause, and prevent progression and complication (fusion)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What are modalities in OMT, braces, surgery

A

Increase muscle balance, optimize function, treat any SD, exercise to reduce lumbosacral angle and strengthen Psoas and abdominal muscles.

Braces, Surgery if curve is >45 degrees

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

Short Leg Syndrome

A

Unlevel sacral base –> develop spinal curvature (rotoscoliosis), innominate rotation, and side shift.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

When to suspect short leg syndrome

A

When structural asymmetry, recurrent SD, tissue texture changes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

For short leg syndrome, what horizontal planes do you assess?

A

Mastoid process/occipital base

AC joints

Inferior angle of scapulae

Iliac crests

Greater trochanter

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Short Leg Syndrome Early Compensation:

A

C shaped curvature is the compensation. The cephalad planes is depressed opposite to the pelvic horizontal plane.

Ex: left sacral base lowering causes there to be left sided scoliosis.

Right sides pelvic lowering, left cephalad landmrks lowering.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Late compensation

A

S shaped curve basically. The shoulder and greater trochanteric planes are on the same side.

Ex: left sacral base lowering causes left pelvic and left shoulder depression.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Describe the the movements of the legs, the effect on spine, effect on leg, and pain components of progressive spinal disease

A

Example: left sacral base turning. Left short leg, right long leg.

Left sided scoliosis. Pelvis shifts and rotates away from side of sacral base declination.

On short leg-> anterior innominate rotation.

On long leg–> posterior innominate rotation.

Long leg–> there is IR and pronation

Increase in lumbosacral angle by 2-3 degrees.

Vertebrae in most caudal scoliotic curve SB away and rotate towards the base declination.

Degenerative arthritis of the long leg.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Short leg syndrome osteopathic findings:

A

Somatic dysfunction.

Soft tissue involvement: tissues on the concave side shorten, while tissue on the convex side lengthen.

Tight abductors on side, tight adductor on the other side.

Ilio-lumbar ligament basically stressed on the side of the convexity–> referred pain down the testicles, labia, and upper medial thigh.

Sacroiliac ligament is stressed on the side of the convecxity, pain referral down lateral leg.

long leg–> unilateral sciatica and hip pain.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Short leg syndrome OMT Diagnosis

A

Look for any signs of somatic dysfunction.

Iliac crest heights, femoral head heights, sacral base unleveling, and scoliotic compensation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

What is treatment for the short leg syndrome OMT wise?

A

<5mm not treated

If functional–> OMT

If anatomic –> do tissue realignment –> heel lift. In heel lift what you do is you basically in fragile persons life no more than 1.5 mm/2 week, while in flexible spine patients you do 1.5/week or 3mm/week.

If sudden loss of leg length–> you lift fully.

Proper lifting has been done there is no SD, negative standing flexion test, and repeat X-ray.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

What is compartment syndrome basics?

A

Compartment is basically a group of muscles held together by fascia

In compartment syndrome, there is elevated pressure in the compartment, compromising circulation and function of tissue in the compartment

Compromised circulation causes ischemia and irreversible neuromuscular deficits.

24
Q

Pathogenesis of compartment syndrome

A

It is when there is basically an increase in pressure in fixed compartment. This causes decrease in AV pressure gradient –> Arteriolar pressure is unable to overcome pressure in compartment –> blood is shunted away from compartment –> capillary collapse–> decreased O2 to muscles –> hypoxia induced production of vasoactive substances. This causes more fluid release into the tight space.

25
Q

Ischemia causes what? :

A

Nerve damage

1 hour–> normal conduction

1-4 hours–> reversible damage

8 hours–> irreversible damage

Muscle damage

4 hours–> reversible damage

4-8 hours–> variable damage

8 hours–> irreversible damage

26
Q

What is etiology, MCC location, Symptoms, Physical exam in diagnosing compartment syndrome?

A

Etiology is that there is trauma/ fracture in the long bones of the fore arm or leg

MC is the anterior compartment of the lower leg

Symptoms:

pain out of proportion (deep, bruning, tightness)

Paresthesia (nerve ischemia)

5 P’s –> pain, pallor, paresthesia, pulselessness, paresthesias

Physical Exam: Pain on passive stretching (early

Decreased light touch and 2 point discrimination

weakness

paralysis

27
Q

What are diagnostic measures for compartment syndrome ?

A

Measure compartment pressure –> normal are 0-4 mm

Stryker device

28
Q

What are indications for fasciotomy?

A

Absolute pressure theory: compartment pressure is between 30 -45 mmhg –> you need fasciotomy

Pressure Gradient theory: compartment pressure is within 20 mmhg of diastolic BP

29
Q

Treatment for compartment syndrome includes:

A

removal of any restrictive coverings, do not raise lower extremity (maintain at heart level)

maintain hydration and urinary output

Pain management, O2

serial exams

Fasciotomy

30
Q

What are possible cases of fibromyalgia?

A

Increases in substance P and glutamate

low blood flow to thalamus,HPA Axis dysfunction, low serotonin and tryptophan
abnormal cytokine function, genetics –> COMT gene polymorphisms and 5-HTP2A receptor polymorphisms

31
Q

Clinical

A

MCC of chronic widespread MSK pain ,

bilateral, chronic, relapsing, diffuse, aching

Hyperesthesia, allodynia
Key ares: neck, shoulders, second ribs, elbows, hips, buttocks, and knees.

32
Q

What are the ACR Criteria for the fibromyalgia? 1990

A

They are basically widespread pain in all 4 body quadrants, minimum of 3 months, tenderness in 11/18 standard tenderpoints.

33
Q

What is the ACR diagnosis criteria for the firbomyalgia 2010?

A

Widespread pain index (WPI) –> how many pain areas in last week (0-19)

Symtom severity scale ( what is level of severity in last week) 0-3

(12 points max)

WPI> 7 AND SS> 5

0R

wpi 3-6 AND ss SCORE >9

IS FIBRO

34
Q

What are differentials for Fibromyalgia?

A

Trigger points producing localized pain: Incidence is male = female.

Fibro: multiple tenderpoints in predictable, bilateral locations, Chronic widespread pain.

Female> male

Chronic Fatigue Syndrome: typically after viral illness. Fatigue is associated with atleast 4 of the following:

decreased mem/concentration, sore throat, tender cervical/axillary lymph nodes, pain in multiple joints, new-onset headache, post-exertion malaise, non-restorative sleep, muscle pain/

35
Q

Additional symptoms

A

Fatigue–> starts and is worse in the morning. Sleep disturbance –> alpha delta sleep.

No long period of stage 4 sleep.

Constant sleep interruptions with awake like brain activity.

Stiffness: beings in morning and persists throughout the day–> limits activity

Psychosocial factors: helpless, persistent stress, anxiety, poor coping mechanisms.

36
Q

Aggravating factors of fibromyalgia:

A

cold/humid weather, non restorative sleep, fatigue, excess physical activity anxiety stress

37
Q

Low back pain basics:

A

Second MC reason for visit. Don’t seek care after 3 months and have pain 1+ year.

Poor outcome predictors –> 1+ years

38
Q

Poor outcome predictors:

A

Maladaptive behaviors, coping behaviors, functional impairment, poor general health status, presence of psychiatric comorbiditie

39
Q

Risk factors:

A

smoking, obesity, older age, female, psychological factors

40
Q

Thought process, simplified

A

Dysfunction: what we doin lab
Derangement: disc herniation
Degeneration: arthritis, osteoarthritis, psoriatic, infectious, gouty/pseudogouty, ankylosing spondylitis

41
Q

Differential diagnosis in the thoracic region:

A

Rule out fracture, malignancy/metastasis, multiple myeloma, infection,

Consider osteoporotic or traumatic compression fracture and cause of osteoporosis

Visceral causes: aortic dissection, MI, pneumonia, pneumothorax, pericarditis, hepato-biliary disease referral pattern, nephro-lithiasis or renal disease

42
Q

What is DISH?

A

Diffuse idiopathic skeletal hyperkeratosis. Calcification of vertebral ligaments –> bony formation. No HVLA techniques.

43
Q

Neural foramen encroachment

A

It is due to bone spurs and disc degeneration. May lead to radiculopathy.

44
Q

What is vertical disc herniation?

A

When the disc protrudes vertically into center. Shmorl nodes. Figure 8 on imaging

45
Q

Sacroiliitis

A

Pain/inflammation of one or both SI joints. Lateralized lumbosacral pain.

Caused by backwards sacral torsions, unilateral sacral dysfunctions, Beding while twisting with pre-existing tension through posterior myofascial pain

46
Q

What are the dirty half dozen of LBP?

A

Non-neutral dysfunction–> frs
Dysfunciton of pubic symphysis

Restriction of anterior nutated movement of sacral base, either a posterior torsion or posteriorly nutated sacrum.

short leg, pelvic tilt syndrome

muscle imbalance

47
Q

What is the management of LBP?

A

OMT: consider imaging first, use comfortable techniques, address dysfunctions related to spine

Interventional Approach: Prolotherapy –> inject irritant–> stimulate healing response. Epidural injections, facet and nerve blocks.

Additional Methods: Excercise, PT, lifestyle, diet, ect.

McKenzie Protocol: reducible derangement:

One direction centralizes/decreases symptoms –> preferred direction

One direction produces/increases symptoms.

Treatment: exam. patient and response to movement –> treat base on direction of preference.

48
Q

PAIR are:

A

Seronegative arthritis

autoimmune, inflammatory arthritis

Psoriatic arthritis, ankylosing spondylitis, IBF, reactive arthritis

49
Q

Describe ankylosing Spondylitis

A

At risk are young men (20- 30 yo), HLA-B27 genetic mutation is common, family history.

Clinically: SI joint fusion, LBP that is worse at night and better in the morning. Stiffness with inactivity. Extra articular–> iritis, aortitis, and pulmonary fibrosis.

50
Q

What happens in thoracic curve in ankylosing spondylitis?

A

It becomes flattened

51
Q

Who does Shuerman’s Disease affect:

A

It is increased thoracic curvature

13-1 6 y/o children. There is uneven growth ofvertebral bodies –> wedging/sharp angle change–> T7-T10.

52
Q

Osteoporosis affects whom? What is it?

A

It is loss of bone matrix strength. Affects post-menopausal women.

Associated with wedge fractures.

MC at T12.

53
Q

What is psoas spasm?

A

Non-neutral dysfunction at L1 and L2 `

54
Q

What are the stages ?

A

Bilateral spasm

Unilateral spasm (dominant)

Sacral torsion ( forward)

Pelvic side shift to opposite side

Contralateral piriformis sapsm

Contralateral sciatic nerve irritation

55
Q

Increase AP lumbar curvature

A

It is pregnancy.

Spondylolisthesis –> forward slipping of one vertebrae to the next due to pars intercuralis fracture.

Spondylolysis: fracture with no slipping.

Step sign, worsens with extension.

grading
1, 2, 3,4, 5