Ophthalmic Exam Flashcards
(85 cards)
define triage timelines
- Emergent – Requires care within 1 hour
- Urgent – Requires care within 4 to 6 hours
- Semi-urgent – Requires care within 24 hours
give examples of emergent eye conditions
Chemical Splash/ Burn
Painless Unilateral Vision Loss:
- Central Retinal Artery Occlusion
- Arteritic Anterior Ischemic Optic Neuropathy secondary to GCA
Acute Ocular Trauma
- blow out fx
- Enophthalmos and/or vertical displacement of eye
Penetrating Injury
Severe Unilat Pain w/Vision Loss
- Acute Angle Closure Glaucoma
Cellulitis: periorbital

give examples of urgent eye conditions
FB
Corneal Abrasion
Sudden Onset Diplopia:
- oculomotor palsy III
- Trochlear palsy IV
- abudcens palsy VI
Acute Onset Flashes and Floaters
- Posterior Vitreal Detachment (PVD)
- Retinal Detachment (RD)
Sudden Onset Red Eye
- Subconjunctival Hemorrhage
- Conjunctivitis - viral, bacterial, Epidemic Keratoconjunctivitis, allergic
- Keratitis – Microbial Keratitis aka Corneal Ulcer, Herpes Simplex
- Uveitis (Iritis) - Acute Anterior Uveitis

give examples of semi-urgent eye conditions
Chalazion
Hordeolum (stye)
Cellulitis –
- Preseptal Cellulitis:
- Orbital Cellulitis: EMERGENCY STAT opth consult, hospitalzation

Tx of chemical burns/ splashes
IRRIGATE min 30 mins - normal saline is best, use tap water if thats all you have
determine ofending chemical and pH - normal 7.0-7.3
STAT ophthal consult through ED once pH is normalized
when dealing w/ a Penetrating Injury is it important to ALWAYS -
Cover eye with shield and send for STAT ophthalmology consult through ED
emergent causes of Painless Unilateral Vision Loss
diagnostic characteristic?
Central Retinal Artery Occlusion
- VA in range of counts fingers to NLP
- Usually patient with Hx of HTN, carotid occlusive disease, cardiac valve disease – typically secondary to arterial embolus to eye via Ophthalmic Artery
- Will see diffuse whitening of retina with “cherry red spot” in macula
Nonarteritic Anterior Ischemic Optic Neuropathy si/sx
EMERGENT Painless Unilateral Vision Loss
If loss reported upon awakening
note inferior hemisphere field loss +/- good to fair VA
Secondary to transient non/hypo-perfusion of Short Posterior Ciliary Arteries surrounding/nourishing the optic nerve
Will see optic nerve head edema +/- splinter hemorrhages
Nonarteritic Anterior Ischemic Optic Neuropathy
In patient presenting w/ Painless Unilateral Vision Loss
Consider Arteritic Anterior Ischemic Optic Neuropathy secondary to Giant Cell Arteritis if:
- Patient >55yo
- Temporal scalp tenderness +/- jaw claudication
emergent Severe Unilat Pain w/Vision Loss is likely ??
Acute Angle Closure Glaucoma
Si/Sx Acute Angle Closure Glaucoma
Severe Unilat Pain w/Vision Loss
nausea/vomiting,
reporting rainbow or halos around lights
Vision reduced secondary to hazy, edematous cornea
IOP >45mmHg
Tx Acute Angle Closure Glaucoma
RAPIDLY ↓IOP
- topical BB - (timolol)
- CAI (dorzolamide, brinzolamide)
- alpha agonist (apraclonidine)
- PO acetazolamide or methazolamide
Requires STAT referral to ophthalmology for LPI once edema clears
Tx FB
Superficial - irrigate out
Cotton tipped swab, sponge spear or 25G or smaller needle - oblique approach with bevel up tangent to surface
Rx broad-spectrum antibiotic QID – tobramycin
NEVER Rx anesthetic bc??
melt the cornea
Tx corneal abrasion
topical broad-spectrum AB QID – tobramycin
Possibly bandage CL for comfort if defect extensive
Homatropine cycloplegia to relax Ciliary Body and decrease pain
PO analgesics as required
proper technique to remove corneal abrasion
- Remove gently from edge of defect in toward center of defect with spud or other fine instrument
- Attempting to remove in opposite direction may result in healthy tissue being removed
Sudden Onset Diplopia is caused by?
nerve palsy
III – Oculomotor:
IV – Trochlear
VI – Abducens -
CN III Palsy – Oculomotor is an emergency IF??
•aneurysm -> pupil fixed and dilated or minimally reactive
muscle innervation
III – Oculomotor:
IV – Trochlear
VI – Abducens
III – Oculomotor: Superior, Inferior, Medial Rectus, Inferior Oblique, Levator Palpebrae
- Anyueisms
- Microvascular
IV – Trochlear –> Superior Oblique
VI – Abducens –> Lateral Rectus
- In adults, due to:
- Microvascular disease
- Lesion in Cavernous Sinus
- Trauma
Etiology & Si/Sx of CN III Palsy
CN III Palsy – Oculomotor
Si/Sx
Eye is down and out
upper lid ptosis
pupil dilated (microvasc vs aneurysm)
- aneurysm - pupil fixed and dilated or minimally reactive
- Microvascular - pupil normally reactive secondary to DM or other systemic disease
differentiating b/w causes of CN III palsy
- Aneurisms
- Microvascular
look at pupil!!
aneurysm - pupil fixed and dilated or minimally reactive
•Microvascular - _pupil normally reactiv_e secondary to DM or other systemic disease
Si/Sx of CN IV palsy
trochlear
Vertical/tilted diplopia with compensatory head tilt to opposite side,
•Diplopia worse in downgaze (SO - non-emergent)
Si/sx CN VI palsy
causes in adults?
abducens
Horizontal diplopia with affected eye turned in (LR)
•In adults, due to:
- Microvascular disease
- Lesion in Cavernous Sinus
- Trauma

