Ophthalmology Flashcards

(32 cards)

1
Q

What are the causes of sudden painful visual loss?

How do you differentiate them clinically

A

Closed angle glaucoma: Red eye, hazy cornea, dilated pupil

Anterior uveitis: flush, iris pus, fixed oval pupil

Optic neuritis: central loss, colour loss, RAPD, worse on movement

Giant cell arteritis: Painful jaw/ scalp

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2
Q

What are causes of painless sudden visual loss?

How do you distinguish them?

A

Amaurosis fugax: ‘curtain coming down’,

Central retinal artery occlusion: RAPD, ‘cherry red’ spot on pale retina

Central retinal vein occlusion: retinal haemorrhages

Vitreous haemorrhage: Dark spots, diabetics, anticoagulants

Posterior vitreous detachment: Flashes and floaters

Retinal detachment: Dense shadows peripheral to central; curtain over visual field

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3
Q

Name and distinguish the causes of gradual visual loss?

A

Cataracts: ‘starbursts’ at night, red reflex loss

ARMD: central field loss, wavy appearance to straight lines

Diabetic retinopathy: blurred, blotched vision, ‘cotton wool’ spots

Chronic open angle glaucoma: peripheral loss, halos, can be painful

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4
Q

How do you manage acute angle closure glaucoma?

A

Urgent referral to ophtho

Improve flow: pilocarpine and apraclonidine

Reduce secretions: B-blocker IV acetazolamide

*Blockers/inhibitors block production, agonists improve flow, a–agonists do both)

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5
Q

How do you manage anterior uveitis

A

Urgent Ophtho review

Dilate eye with atropine, cyclopentate

Steroid eye drops for inflammation

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6
Q

How do you manage optic neuritis

A

Give high dose steroids

MRI for white matter lesions (>3 measns 50% MS risk in 5 yrs)

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7
Q

How do you treat giant cell arteritis?

A

Give high dose glucocorticoids (IV methylprednisilone if evolving changes prior to pred)

Optho review same day

Artery biopsy

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8
Q

How do you treat amaurosis fugax?

A

Aspirin 300mg as per stroke

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9
Q

How do you manage

CRVO

A

CRVO

Macular oedema: Anti-VEGF

Neovascularisation: Lasering

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10
Q

How are vitreous haemorrahge and retinal detachment managed?

A

Urgent Ophtho review

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11
Q

How are cataracts managed?

A

If visual impairment, QOL and patient choice are fitting, lens replacement

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12
Q

How is age related macular degeneration investigated and managed?

A

Investigations

1st: Slit lamp

+ fluoreiscin angiography if neovascular suspected

Management

vitamins A, C, E

VEGF if wet/neovascular

+ laser photocoagulation to slow progression

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13
Q

How is diabetic retinopathy categorised?

A

Non-proliferative

Mild: >=1 microaneurysm

Moderate: Cotton wool spots, hard exudates

Severe: Blot haemorrhages in 4 quadrants, venous bleeds in 2

Proliferative

neovascularisation

Maculopathy

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14
Q

How do you manage diabetic retinopathy?

A

All: Optimise cardiac risk factors, regular review

Non-proliferative: Observe, laser photocoag if severe

Proliferative: laser coagulation

Maculopathy: VEGF if visual acuity change

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15
Q

How do you investigate primary open angle glaucoma?

A

Fundoscopy: cup/disc 0.7 pallor and bayonetting of vessels

Slit lamp, tonometry to confirm

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16
Q

How do you tell the difference between keratitis and conjunctivitis?

A

Keratitis // Conjuncitvitis

hypopyon, contact lense // discharge if infective, seasonal, atopic history if allergic

17
Q

How do you manage keratitis?

A

Refer to Ophtho same day for slit lamp

Stop contact lenses

Quinilone antibiotics

Cycloplegics for pain

18
Q

How do you investigate and treat herpes simplex keratitis?

A

Immediate referral to Ophtho

Fluoreiscin staining shows corneal ulcer

give oral aciclovir

19
Q

What organism is responsible for keratitis that is

Bacterial

Fungal

Viral

A

Bacterial: S. aureus, P. aeringuosa (contacts)

Fungal: Acanthomoebic (soil, contaminated water)

Viral: Herpes simplex

20
Q

How do viral, bacterial and allergic conjunctivitis differ

Demographic

Symptoms

Treatment

Pathogen

A

Viral // Bacterial // allergic

Adults // Children // equal

Watery discharge, URTI // Thick discharge // watery discharge, itch

Self-limiting 1-2w // if 5-7d, chloramphenicol // antihistamine –> mast cell stabilisers

adeno, HSV // S. aureus, chlamydia, gonorrhea // NA

21
Q

What is the difference between esophoric and exophoric ambylopia?

How are they managed

A

Eso: Inward, Exo: Outwards

Covering good eye causes other to move opposite way

Refer for Ophtho

Specs/patch good eye/corrective surgery

*tropia if all the time, phoria if sometimes*

22
Q

Which ophtho meds cause

brown pigmentation, eyelash growing

Hyperaemia, adversity in MAOIs or TCAs

Headaches, blurred vision

A

PGE analogues (lantaprost)

sympathomimetics (brimonidine)

pilocarpine

23
Q

What nerve damage causes the following

Down + out eye, dilated eye

Up and outward rotation, vertical diplopia

Eye cannot abduct

A

3rd nerve (oculomotor)

4th nerve (superior oblique)

6th nerve (abducens)

24
Q

What is the difference between a Holmes-Adie pupil and Argyll-Robertson?

A

HA: Unilateral dilation, slow dilation following constriction; will get smaller over time. +/- ankle and knee reflex loss

AR: Constricted pupil that accomodates but does not react. Neurosyphilis.

25
Name the location of the lesion and the associated pathologies
1. **Optic nerve:** ipsilateral loss (neuritis, ischaemia, trauma) 2. **Optic chiasm central:** bitemporal hemianopia (adenoma, suprasellar aneurysm) 3. **Optic chiasm lateral:** ipsilateral monocular hemianopia (3rd ventricle distension, IC/PCA atheroma) **4. Optic tract:** contralateral homonymous hemianopia (MCA stroke and tumours) **5. Occipital cortex:** contralateral homonymous hemianopia with macular sparing (PCA stroke, trauma)
26
Contralateral homonymous quandrantopia is associated with
MCA stroke, tumour and trauma
27
What's this?
Normal retina
28
Whats this?
Central retinal artery occlusion
29
Whats this
Central retinal vein occlusion
30
Whats this?
Maculopathy Advanced diabetic retinopathy
31
Whats this?
ARMD
32