Ophthalmology Flashcards

(42 cards)

1
Q

Features of acute angle closure glaucoma

A

Severe pain (ocular or headache)
Decreased visual acuity
Symptoms worse with mydriasis
Hard, red eye
Haloes around lights
Semi dilated non reacting pupil
Corneal oedema results in dull or hazy cornea

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2
Q

Acute angle closure glaucoma investigations

A

Tonometry for IOP
Gonioscopy (special slit lamp for looking at angle)

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3
Q

Management of acute angle closure glaucoma

A

Pilocarpine
Timolol
Apraclonidine
IV acetazolamide

Definitive management is laser peripheral iridotomy

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4
Q

Risk factors for ARMD

A

Age
Smoking
Family history

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5
Q

Causes of sudden visual loss

A

Ischaemic/vascular
Vitreous haemorrhage
Retinal detachment
Retinal migraine

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6
Q

Retinal vein occlusion causes

A

Glaucoma
Polycythaemia
Hypertension

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7
Q

Central retinal artery occlusion features

A

Afferent pupillary defect
Cherry red spot on a pale retina

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8
Q

Causes of vitreous haemorrhage

A

Diabetes
Bleeding disorders
Anticoagulants

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9
Q

Vitreous haemorrhage features

A

Sudden visual loss
Dark spots

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10
Q

Posterior vitreous detachment features

A

Flashes of light in periphery
Floaters in temporal central vision

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11
Q

Retinal detachment features

A

Dense shadow that starts peripherally then spreads to central vision
Veil/curtain over vision
Straight lines appear curved

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12
Q

Anginoid retinal streaks appearance

A

Irregular dark red streaks radiating from the optic nerve head

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13
Q

Causes of anginoid retinal streaks

A

Pseudoxanthoma elasticum
Ehler-Danlos
Paget’s
Sickle cell
Acromegaly

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14
Q

Classification of dry ARMD

A

90% of cases
Drusen - yellow round spots

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15
Q

Classification of wet ARMD

A

Choroidal neovascularisation
Leakage of serous fluid
Worst prognosis

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16
Q

Symptoms of ARMD

A

Reduction in visual acuity
Difficulties in dark adaptation
Photopsia (flickering/flashing light)
Hallucinations leading to Charles-Bonnet

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17
Q

Signs of ARMD

A

Distortion of light perception
Drusen on fundoscopy
Well demarcated red patches in wet

18
Q

Management of ARMD

A

Zinc with vit A C & E
VEGF - randizumab, bevacizumab
Laser photocoagulation

19
Q

What is a relative afferent pupillary defect

A

When the affected and normal eye appear to dilate when light is shone in the affected eye

20
Q

Causes of relative afferent pupillary defect

A

Retina - detachment
Optic nerve - optic neuritis (MS)

21
Q

Causes of optic neuritis

A

MS
Diabetes
Syphilis

22
Q

Features of optic neuritis

A

Unilateral decrease in visual acuity over days
Poor discrimination of colours
Pain worse on eye movement
Relative afferent pupillary defect
Central scotoma

23
Q

Optic neuritis investigation

A

MRI of brain and orbits with gadolinium contrast

24
Q

Management of optic neuritis

A

High dose steroids

25
Signs of orbital cellulitis vs pre-septal cellulitis
Painful eye movement Restricted eye movement Reduction in colour vision Abnormal pupillary responses to light Reduced visual acuity Reduced visual fields Chemosis
26
Features of retinitis pigmentosa
Night blindness Tunnel vision Fundoscopy - black bone scipule-shaped pigmentation in the peripheral retina, mottling of retinal pigment epithelium
27
Diseases associated with retinitis pigmentosa
Refsum Ushers Alports Kearns-Sayre
28
Features of Horner’s syndrome
Miosis Ptosis Enophthalmos Anhidrosis
29
Horner’s of face arm and trunk causes
Stroke Syringomelia MS Tumour
30
Horners of face only causes
Pancoasts Tumour Thyroidectomy Trauma
31
Horners with no anhidrosis causes
Carotid artery dissection Carotid aneurysm Cavernous sinus thrombosis Cluster headache
32
Hypertensive retinopathy stage 1 features
Arteriolar rowing and tortuosity Increased light reflex Silver wiring
33
Hypertensive retinopathy stage 2 features
Arteriovenous nipping
34
Hypertensive retinopathy stage 3 features
Cotton wool exudates Flame and blot haemorrhages
35
Hypertensive retinopathy stage 4 features
Papilloedema
36
Holmes-Adie pupil features
Women Unilateral Dilated pupil Slow to accommodation Absent ankle/knee reflex
37
Dacryocystitis features
Watering eye Swelling and erythema to inner canthus
38
Dacryocystitis management
Systemic antibiotics
39
Causes of mydriasis
Third nerve palsy Holmes-Adie Traumatic iridoplegia Phaeochromocytoma Congenital Atropine Cocaine/amphetamines TCA’s
40
When do you get bilateral occipital lobe infarcts and what signs do you get.
Period of hypotension - cardiac arrest Cortical blindness with preservation of pupils reaction to light.
41
Lesions at the chiasm have what signs?
Bitemporal hemianopia If they spread up from below the defect is worse in the upper fields (pituitary tumours) If they spread down from above the defect is worse in the lower fields (craniopharyngioma)
42
Lesions before the chiasm produce what defect
Visual defect in the ipsilateral eye Optic nerve damage - central, asymmetrical and unilateral Acuity often affected Causes - optic neuritis, optic atrophy, glaucoma, trauma