Ophthalmology Flashcards

(61 cards)

1
Q

Chalazion

A

Enlargement of the meibomian gland due to a blockage of
its duct and inspissation of its secretory products.

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2
Q

Hordeolum

A

– purulent bacterial
infection of the meibomian
gland

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3
Q

• Meibomianitis

A

involvement of
multiple glands

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4
Q

Cherry eye

A

Prolapsed gland of the third eyelid

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5
Q

The only indication for removal of the gland of the third eyelid or
entire third eyelid

A

Neoplasia

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6
Q

Two common techniques for the treatment of cherry eye

A
  1. Morgan pocket technique
  2. Orbital tacking
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7
Q

What complication can occur with improper morgan pocket techniqu?

A

cyst formation if tears are not
allowed to drain from either end of
the two incisions

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8
Q

Goblet cells

A

produce the mucinous layer of the tear film normally
and respond to inflammation by increasing production of mucus.

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9
Q

Lymphoid follicles – Presence anywhere but the bulbar surface of the third
eyelid indicates _________________.

A

nonspecific antigenic stimulation

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10
Q

Thin vessels under the epithelium

A

‘Conjunctival vessels’

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11
Q

Larger, deeper vessels

A

‘Episceral vessels’

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12
Q

Conjunctival hyperemia

A

congestion of superficial, or conjunctival
vessels.

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13
Q

Episcleral injection

A

Congestion of deeper and larger episcleral vessels.
Identifiable vessels that course perpendicular to the limbus

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14
Q

What is used to differ episcleral injection from hyperemia?

A

phenylephrine

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15
Q

Strongest topical antiinflammatory steroid

A

Neomycin-Polymycin-Dexamethasone

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16
Q

Topical NSAID

A

Diclofenac

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17
Q

Topical T-cell inhibitor antiinflammatory

A

Optimmune (cyclosporin) 0.2%

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18
Q

Feline eosinophilic keratoconjunctivitis is a secondary disorder to what?

A

Feline herpesvirus conjunctivitis

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19
Q

Most common cause of feline conjunctivitis and feline keratitis

A

FHV-1

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20
Q

Epithelium containing tissues that herpes loves

A

mucoepithelial cells of tonsils, conjunctiva, nasal mucosa,
and cornea

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21
Q

Latent FHV-1 virus becomes reactivated mainly due to what?

A

environmental stress

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22
Q

Symblepharon

A

permanent adhesion between the conjunctiva and the cornea

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23
Q

Dendritic ulceration is pathgnomonic for

A

FHV-1

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24
Q

“ghost vessels” and
very early sequestrum
from previous corneal
ulceration can be seen in

A

FHV-1

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25
Treatment of ocular FHV-1
Antivirals: Topical Cidofovir 0.5% Oral Famciclovir Supportive care: Topical Erythromycin
26
Autoimmune and may be related to FHV-1 infection
Feline eosinophilic keratoconjunctivitis
27
Raised white or yellow corneal and/or conjunctival infiltrates specific for
Feline eosinophilic keratoconjunctivitis
28
Second most common cause of conjunctivitis behind FHV-1
Chlamydial conjunctivitis
29
Chemosis is specific for \_\_\_\_\_\_\_
Chlamydia
30
Diagnosis of chlamydial conjunctivitis
cytology - intracytoplasmic inclusion bodies
31
Diagnosis of mycoplasma
Cytology – Numerous coccoid membrane associated bacterial inclusions
32
Oral ulceration pathognomonic for which disease?
Calicivirus
33
3 layers of the tear film
Lipid – outermost Aqueous – middle Mucinous – innermost
34
3 Functions of the tear film
Nourish • Cleanse • Protect
35
What produces the aqueous layer of the tear film?
Lacrimal gland and gland of 3rd eyelid
36
Goes to the veterinarian for eye discharge. Receives topical antibiotic and the discharge clears. Antibiotic is stopped and the discharge returns. What diagnostic test should be performed in these cases?
Schermer tear test
37
Diagnosis of aqueous layer disorders
1. Superficial corneal and conjunctival inflammation = keratoconjunctivitis + 2. Schirmer Tear Test I
38
Causes of Quantitative KCS
* Immune mediated destruction of lacrimal tissue * Excision of the third eyelid gland * parasympathetic CN VII lesions * Canine Distemper Virus * Drugs
39
Quantitative KCS diagnosis
Quantitative KCS = Aqueous deficiency Schirmer Tear Test \<15mm/min + clinical signs
40
In the majority of KCS cases, topical ______________ is the mainstay of therapy and twice daily treatment controls clinical signs if it is given for life
cyclosporine
41
Treatment of KCS
Cyclosporine A - If no response - Tacrolimus
42
four objectives of KCS treatment
Replace tears -lube Stimulate more tears and reduce inflammation - cyclosporin Temporary Antibiotic
43
Pathophysiology of Neurogenic KCS
Loss of parasympathetic innervation to the lacrimal gland and ipsilateral nostril
44
Indication for Pilocarpine. What type of drug is it?
neurogenic KCS parasympathomemetic
45
Pathophys and presentation of K9 distemper KCS
Pathophysiology: Viral destruction of lacrimal glandular epithelium Clinical presentation: young dogs without proper vaccination. Acute and Severe.
46
WHat t do when medical therapy for KCS fails?
Parotid duct transposition
47
What produces the lipid layer of the tear film?
Meibomian glands
48
What stabilizes the aqueous layer of the tear film and prevents evaporation?
Lipid layer
49
superficial keratoconjunctivitis with a normal STT I and without any other identifiable cause
qualitative keratoconjunctivitis sicca
50
What produces the mucinous tear film layer and what is its function?
Conjunctival goblet cells Creates surface tension that anchors the aqueous and lipid layer to the cornea
51
Diagnosis of mucin layer disorders
qualitative keratoconjunctivitis sicca Tear Film Breakup Time (TFBUT)
52
Corneal fibrosis
disorganized epithelium and stroma create a whitish hazy appearance
53
Treatment of Qualitative keratoconjunctivitis sicca
Lube and immunomodulating
54
Epiphora
an overflow of tears on the face
55
Imperforate Punctum
aplasia of the punctum
56
Uncomplicated/Simple Ulcer
A superficial corneal ulcer that heals in 7 days or less
57
Characteristic of an indolent ulcer
lack of epithelial adherence to stroma
58
Collagenolysis
enzymatic destruction of corneal collagen
59
– Keratomalacia
Softening or “melting” of the corneal stroma due to collagenolysis • From bacteria/fungi and neutrophil enzymes
60
Most common bacteria assocciated with a melting ulcer
Pseudomonas aeruginosa
61