Optimising Function - CARDIORESP Flashcards

1
Q

What is the ABCDE approach?

A

A - airway: head tilt chin life, suctioning, recovery position, oxygen, help
B - breathing: position, drugs, support ventilation (bag-valve-mask) (good lung down)
C - circulation: capillary refil, HR, BP, pulse palpation, urine output. Consider hypovolaemia (low fluid): large bore cannulae, bloods, fluid replacement, ECG.
D - disability: reversible causes, pupil size and reactivity, ACVPU or GCS, blood glucose: correct abnormal blood sugar levels, lateral position, consider airway support.
E - exposure: head to toe examination.

Then complete full history/review to handover.

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2
Q

What is sepsis?

A

Arises when the body’s response to an infection injures its own tissues and organs.

Organ dysfunction due to a dysregulated host response to infection.

Septic shock is when circulatory, cellular and metabolic abnormalities increase mortality.

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3
Q

What are the symptoms of sepsis?

A

S lured speech or confusion
E xtreme shivering or muscle pain
P assing no urine (in a day)
S ever breathlessness
I t feels like you are going to die
S kin mottled or discoloured

Will begin to be catabolic (lose muscle mass)

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4
Q

What are the common causes of sepsis?

A

Meningitis
Skin or soft tissue infection
Catheter related infection
Urinalysis tract infection
PNEUMONIA
Bloodstream infection
Abdominal infections (appendicitis, infectious diarrhoea, gallbladder infection).

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5
Q

What is the treatment of sepsis?

A

Fluids
Antibiotics
Vasopressors
Enteral feeding
Insulin therapy
Lung protective ventilation
Urinary catheter

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6
Q

What are the quality checks for a chest x-ray?

A

Inclusion - whole chest is in view
Projection - PA or AP (department vs mobile)
Rotation - clavicle equidistant from spinous processes
Exposure - should identify inter vertebral spaces
Adequacy of inspiration - taken full breath in so diaphragm is down - count 6 ribs anteriorly
Artefact -

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7
Q

What is the A to E assessment for analysing x wrays?

A

Airway
Bones and soft tissues - fractures, density, symmetry
Cardiac shadow - lingula
Diaphragm - cardiofrenic and costofrenic angles
Eventually - the lung fields. Upper, middle and lower zones (not lobes)

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8
Q

What is the hilar point in the lungs?

A

Enlarged could suggest oedema

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9
Q

What is consolidation on lung x rays?

A

Presents as white and patchy
Consistent with pneumonia
Often confined to segment or lobe
No shift in structures
May be air bronchogram (patent airway)

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10
Q

What is Atelectasis? And what does it look like on a lung x ray?

A

Lung collapse where small airways start to close
Presents as a more uniform white/opaque area
May be lobar or whole lung

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11
Q

What does a pneumothorax look like on a lung x ray?

A

Between visceral and parietal pleura is an air leak

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12
Q

What is pleural effusion? What does it look like on an x ray?

A

Fluid in the lungs
Dense white opaque area
Best seen in upright film
A meniscus of air/fluid level may be present
A large effusion may push structures over to the opposite side of the

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13
Q

What does hyperinflation look like on a lung x ray?

A

Dark lung fields
Flattened diaphragm
Elongated heart
Presents in obstructive lung disease eg ``COPD, `cg and Bronchiecstasis.

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14
Q

What does pulmonary oedema look like on a chest x ray?

A

Caused by fluid linking from the interstitial tissues into the alveolar and small airways, and manifests as consolidation
In acute pulmonary oedema, alveolar oedema radiates symmetrically from the hilar regions in a bats wing distribution of airspace shadowing.

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15
Q

What does gas transport and exchange require?

A

Ventilation (airflow, chest wall expansion, lung compliance)
Perfusion
Ventilation/perfusion matching
O2 delivery to the tissues (heart, vascular system, blood)
O2 extraction at the level of the tissue (perfusion)

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16
Q

What is oxygen saturation?

A

Equates to the proportion of the oxygen molecules bound to the Hb.
97% is carried by red blood cells/bound to haemoglobin
3% dissolved in plasma

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17
Q

What is the oxygen dissociation curve?

A

Demonstrates the % saturation of O2 at various partial pressures of O2
Eg shows the equilibrium of oxyheamoglobin and non-bonded haemoglobin at various partial pressues.

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18
Q

What do different shifts to the O2 dissociation curve mean?

A

Right shift - reduced affinity. Increased temperature
Left shift - decreased temperature

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19
Q

How can CO2 levels in the blood alter?

A

Increased metabolic rate - increased co2 production
Impaired gas exchange - decreased co2 elimination
Hyperventilation - increased co2 elimination
Hypoventilation - decreased co2 elimination

High co2, increase H+ ions, decreased pH = acidic
Changes in CO2 levels are termed RESPIRATORY
EG
Hyperventilation - increased CO2 elimination = RESPIRATORY ALKALOSIS
Hypoventilation - decreased CO2 elimination - RESPIRATORY acidosis.

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20
Q

What are the three main buffer systems in the body?

A

Bicarbonate
Phosphate
Protein

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21
Q

How does HCO3 respond to pH?

A

The kidneys excrete HCO3 molecules into the urine if the blood is more alkaline than normal
Kidneys retain more HCO3 if the blood is more acidic than normal

Termed METABOLIC

High HCO3 - decreased hydrogen ions - increase PH = ALKALOSIS
Low HCO3 - increase hydrogen ions - decrease pH = ACIDOSIS

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22
Q

What are the classifications of acidosis?

A

Decrease pH
Increase CO2 - respiratory acidosis
Decrease HCO3 - metabolic acidosis

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23
Q

What are the classifications of alkalosis?

A

Increased pH
Decreased CO2 - respiratory alkalosis
Increased HCO3 - metabolic alkalosis

24
Q

What are respiratory and metabolic compensation?

A

The lungs can respond to altered levels of pH in the blood by changing the levels of CO2 - respiratory compensation

The kidneys can respond to altered levels of pH in the blood by changing the levels of bicarbonate - metabolic compensation

25
Describe respiratory compensation
In metabolic ACIDOSIS - hyperventilation occurs to ‘blow off’ CO2 and reduce hydrogen ions returning pH back towards normal In metabolic ALKALOSIS - Hypoventilation occurs to retain CO2 and increase hydrogen ions returning pH back towards normal.
26
Describe metabolic compensation
In respiratory ACIDOSIS - the kidneys excrete less HCO3 into the urine, allowing more hydrogen ions to be ‘mopped up’, and returning the pH In respiratory ALKALOSIS - the kidneys excrete more HCO3 into the urine, reducing circulating levels in the blood.
27
What is the oxygen dissociation curve?
The % saturation of O2 at various partial pressures of oxygen.
28
What is peripheral artery disease?
A term used to describe a chronic condition where atherosclerotic obstruction (narrowing or occlusion) of the peripheral arteries affects the blood supply to the lower limbs.
29
What causes peripheral arterial diseases?
Limitation of blood flow to the affected limb Less common causes: inflammatory disorders (eg vasculitis) and non-inflammatory arteriopathies (eg fibromuscular dysplasia).
30
What are some examples of chronic limb-threatening ischaemia?
Mostly caused by atherosclerosis Peripheral arterial disease Non inflammatory arteriopathies Adventital cystic disease Buergers disease Thromboembolism Popliteal entrapment syndrome Arterial dissection
31
What are some examples of acute limb-threatening ischaemia?
Aortic dissection or embolisation Hypercoagulable states Graft thrombosis Iotragenic complications of vascular interventions Cardiac ambolisation
32
What are some risk factors for peripheral artery disease?
Smoking Diabetes mellitus Hypertension Hypercholesterolaemia
33
What are some complications of peripheral artery disease?
Impairment of quality of life due to mobility limitations Severe pain Psychological complications such as depression Tissue loss (ulceration and gangrene) Risk of amputation Infection Procedural complications in people undergoing invasive treatments for peripheral arterial disease
34
What are typical features of acute limb ischaemia?
The 6 Ps: Pain - constantly present and persistent Pallor - or cyanosis or mottling Pulselessness - ankle pulses are always absent Poikilothermia - perishing with cold Paresthesia - or reduced sensation or numbness Paralysis - or power loss
35
What are the differences between ischaemia due to an embolus and thrombosis?
Embolus: onset is acute, limb appears mottled, vascular examination of the other leg is usually normal. Thrombosis: onset is more gradual (due to increased collateral circulation with pre-existing peripheral artery disease). The leg may be bluish, and there is no distinct demarcation. Presentation is usually with worsening claudication. Pulses in the other leg may be abnormal.
36
Why might a patient need a tracheostomy?
Has an obstructed upper airway Likely to need prolonged artificial ventilation Patient is unable to mainta8in an airway independently The p[atients bronchial secretions cannot be cleared normally The patient is having surgery to the orafacial region and might affect the upper airway (eg due to swelling).
37
Why perform a tracheostomy?
Avoids damage to the larynx and pharynx, mouth care is improved More comfortable than a ET tube (endotracheal tube), less sedation required The patient can eat and drink if they can swallow Facilitates ventilator weaning by reducing anatomical dead space by 25-30%. Reduction in resistance to airflow.
38
What are the different types of tracheostomy tubes?
Single and dual cannula tubes - single often used with children, but a double lumen is safer in case of blockage (outer and inner cannula). Fenestrated tubes - little holes in the tube, so the patient also aids the breathing with their upper airway. Placed in in advance but with a non fenstrated inner tube, so reduce changing the tubes often. Good for weaning. Final stage of weaning would be using the decanulation cap, with a DOUBLE FENESTRATED tube so the patient uses their upper airways. Cuffed tubes - needed when ventilating patients, a closed system. Uncuffled tubed - allows the patients to breath some air in and out of their upper airway, but there is resistance. Long length/adjustable tubes - used if the patient has a large neck, eg obese or burns patients with swelling. Tube can be adjusted as swelling goes down, without changing the whole tube. Subglotic line - you can aspirate using this, putting a syringe into the line to remove the secretions that are sitting on top of the cuff. This allows you to deflate the cuff, without letting the secretions drop into the lungs causing the patient to struggle.
39
What are considerations for basic care of patients with tracheostomy’s fitted?
Humidification - tracheostomy bypasses the natural de-humidification processes in the upper airway. Needed to prevent secretions and the bronchitracheal tree from drying. Use a BUCANNON BIB (long term use) or a SWEDISH NOSE. Checking cuff pressure - cuff remains inflated when patient requires long term ventilation, patient has reduced conscious level, patient has excessive oral secretions. Patients can develop tracheal stenosis after the tracheostomy has been removed as the trachea gets subject to high pressures from the cuff. Complications can occur from an under or over inflated cuff. Should be between 15-25 cmH2O. If pt can make a sound then cuff is not inflated enough. Suctioning and tube hygiene - need to use a non-fenestrated inner tube when suctioning. Communication needs to be- cannot talk with an inflated cuff. Might use alphabet or picture boards, lip reading, iPads, notepads. Always have a call bell. Involve speech and language therapists. Ensuring safety - signs by the bed side “This patient has a tracheostomy”.
40
What signs would you look for that a patient with a tracheostomy is struggling to breathe?
Recruitment of accessory muscles, paradoxical breathing Cyanosis Oxygen stats low, respiratory rate high Bleeding, dislodged tracheostomy.
41
What criteria would you use to decide whether someone was ready to wean off of a tracheostomy?
- the patient is able to maintain adequate gas exchange self-ventilating positive and negative supplemental oxygen (not more the 40% supplemental oxygen) - there are no signs of deteriorating broncho pulmonary infection or excessive pulmonary secretions (requiring lots of suction). - the patient has a stable lung status with oxygen therapy less than 40% - the initial reason for the insertion of the tracheostomy has been resolved and/or been considered - the patient is cardiovascularly stable.
42
What are the four stages of weaning?
Cuff deflation - double fenestration Gloved finger occlusion - you yourself cover the airway and check for stridorous noises (if they struggle, remove finger and they are not ready). One way speaking valve - opens as pt breathes in, closes and they breathe out. Gradually build up time, always start in the morning. Decannulation cap. DOUBLE FENESTRATION WHEN WEANING.
43
When can decannulation be considered?
Able to obey commands Adequate cough and ability to clear secretions effectively and independently. Cardiovascularly stable No new lung infiltrates on x ray. Tolerates cuff deflation for 24 hours. Tolerates speaking valve 12 hours or more (usually during day time) or decannulation cap for up to 4 hours. MDT agreement for decannulation.
44
What is the tracheostomy emergency algorithm?
- Call for airway expert expert help - Look, listen and feel at the mouth and tracheostomy - Use waveform capnography (if available) - Apply oxygen to the tracheostomy - Apply oxygen to the face - Remove speaking valve or cap - Remove inner tube - Reassess breathing - Attempt to pass a suction catheter - If the tube as a cuff, deflate this now - Reassess the patient - If the patient is still deteriorating then remove the tracheostomy tube - Reassess again - If the patient is still not breathing ventilated by upper airways - Cover the stomach to stop gas escaping - If unsuccessful, attempt ventilation via the stoma - Use a paediatric face mask or a supra-glottis airway (such as a laryngeal mask). If no improvement secondary emergency oxygenation options include: - Oral intubation - Intubation of the stomach with a 6.0mm tracheostomy or tracheal tube - Use of bronchoscope/Aintree catheter/Bougie/Airway Exchange Catheter.
45
Describe manual hyperinflation and closed suction
Indications are for secretion clearance and lung atelectysis (part of the lung collapses). Contraindications - untrained tension pneumothorax Precautions - Bullae, surgical emphysema, recent lung surgery, rib fractures and flail segments, frank haemoptysis, raises intercranial pressure, cardiovascular instability, high PEEP settings.
46
What are some classical features of intermittent claudication?
Claudication is pain caused by too little blood flow to muscles during exercise. Most often this pain occurs in the legs after walking at a certain pace and for a certain amount of time - Fatigue, discomfort or pain in the muscles of lower extremities after walking, that is relived by rest and reproduced by walking the same distance again. - May be described as aching or burning - Symptoms usually occur in the distal proximity before the proximal extremity, the calves are usually affected than the thigh, buttocks and hips. - Symptoms may be unilateral or bilateral. - Some people present with atypical symptoms, and may use terminology such as ‘tired’, ‘giving way’, ‘sore’ and ‘hurts’, rather than describing cramp.
47
What are some features of chronic limb threatening ischaemia?
- chronic rest pain may be worsening at night because of the decrease in blood pressure when asleep and the loss of beneficial gravitational effects on lower limb circulation - many people do not have a history of intermittent claudication (may not be apparent in people with limited mobility, or who don’t walk far enough to cause symptoms). - dependent rubor, pallor on elevation of the extremity. - skin changes including non-healing foot wounds, ischaemic ulcers and tissue loss (usually over pressure areas), and gangrene (usually of the toes). - absent foot pulses.
48
What are the steps for testing peripheral arterial disease of lower limbs?
Skin observation and palpation Pulse palpation (femoral pulses, popliteal pulses, dorsalis pedis pulse, posterior tibial pulses). Plantar flexion test 6 minute walking test Ankle - brachial index Blood pressure in toe Toe - brachial index
49
What should you consider when palpating a pulse?
Bounding or weak, fast or slow, irregular or regular, equal or unequal bilaterally. 1. Intensity graded 1 - 4. 1 = barely detectable, 2 = slightly diminished, 3 = normal pulse, 4 = bounding (stronger than normal). 2. Rhythm, to be certain that variation in rhythm is not due to minor fluctuations that occur in the respiratory cycle. 3. Rate, counting the total number of palpable beats that occur during a minimum of 15 seconds. 4. Can ascultate the heart whilst palpating to ascertain is every pulse gets transmitted as a palpable beat.
50
How is the plantar flexion test used for peripheral artery disease?
Pt is asked to perform 50 sequential, symptom-limited calf raises. Pts with PAD frequently report pain that does not allow them to continue after a low number of repetitions.
51
What is the ankle - brachial index?
Non-invasive test for assessing a peripheral vascular disease. The ABI assesses the severity of arterial insufficiency. A - ultrasound device amplifies the sound of arterial blood flow B - systolic pressure recorded in the brachial artery of the arm C - sound of arterial blood flow located in the ankle D - systolic pressure sequentially recorded in the arteries of the ankle after each arterial flow is located.
52
What are breathing pattern disorders?
An alteration in the normal biomechanical patterns of breathing that result in intermittent or chronic symptoms that may be respiratory and/or non-respiratory. 3 drivers: physiological, psychological and biomechanical
53
What should you look for when assessing breathing pattern disorders?
Observation of posture and breathing pattern: - mouth or nose breathing - forward head position - audible breath sounds - pattern of breathing (apical, breath stacking) - abnormal inspiration-to-expiration ratio - increased minute volume (rate or volume increases) - frequent sighs, yawns or coughs? - altered speech pattern
54
What are some tools that you can use to assess breathing pattern disorders?
Nijumen questionnaire Breath hold test Ventilators parameters BPAT
55
What are some management strategies for breathing pattern disorders?
Education: - rationale for symptoms - reassurance - stress management - total body relaxation - rescue treatment Breathing re-education: - awareness of faulty breathing patterns - relaxation of jaw, upper chest, shoulders and accessory muscles - abdominal breathing pattern re-education - awareness of ‘normal breathing’ rates and rhythms (rest, speech, activity).