Orthopaedic Hip Conditions Flashcards

1
Q

Which group of muscles attach onto the greater trochanter of the femur? The lesser trochanter?

A

Greater trochanter: Abductors and rotators of the hip

Lesser trochanter: Iliopsoas

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2
Q

What is the labrum of the hip and where does it lie?

A

Lies around the edge of the acetabulum

It’s a fibrocartilaginous structure that adds depth and stability to the hip joint, as well as assisting in the distribution of forces

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3
Q

Describe the blood supply to the hip

A

Profunda femoris artery gives off two branches at the hip:

  • MFCA (medial femoral circumflex artery)
  • LFCA (lateral femoral circumflex artery)
  • Main blood supply to hip from the ascending head of the MFCA
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4
Q

What effect do intracapsular and extracapsular fractures have on the blood supply to the hip joint?

A

Extracapsular: blood supply may be maintained

Intracapsular: blood supply often disrupted

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5
Q

What is osteoarthritis of the hip joint? What bone changes are seen?

A

Progressive degeneration of AC around the hip joint, followed by secondary bone / joint changes

  • Decreased joint space
  • Roughening of bone (osteophytes etc.)
  • Sclerosis of joint (whitening on X-Ray)
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6
Q

What are the symptoms of osteoarthritis affecting the hip joint?

A
  • Pain (in groin / thigh / trochanteric region)

- Stiffness of hip joint (decreased ROM)

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7
Q

What is trochanteric bursitis? Where is the trochanteric bursa?

A
  • Bursa is between the greater trochanter and iliotibial band

Trochanteric bursitis is inflammation & swelling of the trochanteric bursa, often due to friction between the sac and the trochanter & band

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8
Q

Symptoms / signs of trochanteric bursitis?

A
  • Pain lying on side
  • pain worsens on walking (+/- certain other movements)
  • Tenderness over greater trochanter on palpation
  • Pain on active abduction
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9
Q

What are some causes of trochanteric bursitis? Which groups are more likely to get it?

A
  • More common in females
  • Can be precipitated by trauma
  • Often associated with overuse (athletes etc)
  • Abnormal anatomy (scoliosis / hip muscle wasting / osteoarthritis)
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10
Q

Investigations for trochanteric bursitis?

A

X-Ray* (often to exclude osteoarthritis / may see trochanter abnormality)

  • MRI
  • USS (can also be therapeutic - guided steroid injection)
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11
Q

Management of trochanteric bursitis?

A
  • NSAIDs (topical / oral)
  • Physiotherapy
  • Steroid injection*
  • Surgery (bursectomy - not usually required)
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12
Q

What is avascular necrosis? Who tends to be affected? Which hip is more commonly affected?

A
  • Death of the bone due to loss of blood supply
  • More common in men & people aged 35-50
  • 80% of cases tend to be bilateral, may be offset in time though
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13
Q

What are some risk factors for avascular necrosis?

A

Trauma:
- irradiation / fracture / dislocation

Systemic: (majority)

  • Idiopathic
  • Alcoholism
  • Hypercoaguable states
  • Steroids
  • Haematological (sickle cell / lymphoma / leukemia)
  • Caisson’s disease (divers / mine workers)
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14
Q

Describe the pathophysiology of avascular necrosis

A
  • Coagulation of intraosseous microcirculation
  • Venous thrombosis
  • Retrograde arterial occlusion
  • Intraosseous hypertension
  • Reduced blood flow to femoral head
  • Cell death
  • Chondral fracture and collapse
  • Head will often regenerate, but may have an irregular shape causing pain
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15
Q

Symptoms / signs of avascular necrosis?

A
  • Insidious onset groin pain
  • Pain worse with exertion (stairs / hills)
  • Arthritis like examination: reduced ROM / stiffness
  • Pain often seems disproportionate to examination findings
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16
Q

Investigations of Avascular Necrosis?

A

X-Ray: changes may only be visible once stage of bone collapse is reached

MRI*** (identifies earliest changes)

17
Q

Radiological signs of avascular necrosis?

A
  • Blackening of bone due to necrosis
  • Crescent sign: flattening of femoral head + lytic linear region below chondral surface
  • Osteophytes / cysts (later change)
18
Q

Non-surgical management of avascular necrosis?

A
  • Reduce weight bearing (crutches) + analgesics & NSAIDs
  • Bisphosphonates
  • Anticoagulants
  • Physiotherapy
19
Q

Surgical management of avascular necrosis?

A

Restore blood supply

  • Core decompression to reduce venous hypertension
  • Core decompression + Vascularized graft
  • Rotational osteotomy: remove lesion from weight bearing area
  • Hip replacement / fusion
20
Q

What is femoroacetabular impingement? What can it lead to?

A

Impingement (rubbing against) of femoral neck against the anterior edge of the acetabulum

Can cause:
- Early onset secondary osteoarthritis
- Labral degeneration and tears
-

21
Q

What are the two types of femoroacetabular impingement? Brief description of each

A

CAM lesion
- Deformity (lump) of neck of femur causes femur to impinge on edge of acetabulum

PINCER lesions:
- Anterosuperior acetabular rim overhang impinges on neck of femur

22
Q

Presentation of femoroacetabular impingement?

A
  • Groin pain, worse with flexion
  • Limited ROM (reduced flexion & internal rotation)
  • Pain on FADIR test: Flex, ADduct and internally rotate hip joint
23
Q

Investigations of femoroacetabular impingement?

A

X-Ray

MRI

CT

24
Q

Management of femoroacetabular impingement?

A
  • Activity modification / Physiotherapy / NSAIDs
  • Arthroscopy (shave down deformity)
  • Open surgery: Resection / osteotomy
  • Arthroplasty (resurfacing)
25
Q

What type of labral tear is most common at the hip joint (direction)? What are some risk factors?

A

Anterosuperior tears

  • femoroacetabular impingement
  • Trauma
  • Dysplasia
  • Osteoarthritis
  • Collagen disease (Ehlers Danlos)
26
Q

Presentation of hip labral tears?

A
  • Groin / hip pain. usually worse on specific movements, not present at all times
  • Snapping / jamming sensation in hip
  • Positive FABER test: flexion, ABduction, External Rotation of hip causes pain
27
Q

Investigations of labrum tears of the hip?

A

MRI arthrogram* +/- contrast

X-Rays

28
Q

Management of labrum tears of the hip?

A
  • Activity modification / NSAIDs / Physio
  • Steroid injection
  • Arthroscopy (often in younger patients - repair / resect the labrum)