orthopaedics Flashcards

1
Q

perthes disease what is it

A

avascualr necrosis of femoral head for know known reason

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2
Q

perthes disease presentation

A

limp
pain in hip
limited abduction and internal rotation

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3
Q

perthes disease investigations and findings

A

x ray - joint space widening due to lack f blood supply

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4
Q

perthes disease management

A

younger <6 the better prognosis - can remodel
bed rest and NSAIDS <1/2 femoral head effected (herring A/B)
surgery if older >1/2 femoral head effected (herring BC/C)

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5
Q

slipped upper femoral epiphysis what is it

A

10-16yr obese males

displacement through growth plate with epiphysis slipping down and back

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6
Q

slipped upper femoral epiphysis presentation

A

limp
flexion, abduction, medial rotation limited
risk early OA if missed
symptoms usually present for weeks to several months before diagnosis

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7
Q

slipped upper femoral epiphysis management

A

surgical

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8
Q

septic arthritis cause

A

s.aureus (most common)
h.influenza (if unvaccinated)
assume in all limping children

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9
Q

septic arthritis presentation

A

spetic signs - low BP, high HR/RR/lactate
warm tender joint
reduced ROM
usually one joint infected

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10
Q

septic arthritis investigations

A

joint aspiration

mc+s

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11
Q

kocher criteria for septic arthritis

A

WCC >12,000
Inability to bear weight
Fever >38.5
ESR >40

all 4 99% septic arthritis

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12
Q

hip septic arthritis causes

A

Group B streptococcus – neonates
Staph aureus – common in children > 2 years of age
Neisseria gonorhoeae – most common in adolescents

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13
Q

transient synovitis of the hip cause

A

most common casue of hip pain in children

diagnosis is by exclusion of others

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14
Q

transient synovitis of the hip aetiology

A

viral illness preceded by recent viral URTI

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15
Q

transient synovitis of the hip management

A

NSAIDS

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16
Q

development dysplasia of the hip risk factors

A

female
breech
family Hx
first born

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17
Q

development dysplasia of the hip investigations

A

barlow and ortolani manoeuves

observe asymmetry, leg length, skin folds

18
Q

development dysplasia of the hip diagnosis

A

US

important to diagnose early as risk OA

19
Q

development dysplasia of the hip management

A

<6 months - long term splinting, pavlik harness
6-18 months - immobalise, examine under anaesthetic
>18 months - surgical correction

20
Q

osteogenesis imperfecta what is it

A

inherited disorder of type 1 collagen

joint laxity, fragile low density bones

21
Q

osteogenesis imperfecta presentation Sillence classification (4 types)

A

type 1 - mild, most common. many fractures, blue sclera, hearing loss
type 2 - lethal perinatal. many fractures, blue sclera, dwarfism
type 3 - severe. many fractures, spine limb deformity, transparent teeth
type 4 - moderate

22
Q

osteogenesis imperfecta diagnosis

A

x ray - low bone density, bowing of longer bones

23
Q

osteogenesis imperfecta management

A

bisphosphonates - alendronate

always think of non accidental injury

24
Q

rickets what is it

A

low bone mineral content

25
Q
rickets presentation
(presentation of symptomatic vitamin D deficiency)
A

growth retardation, hypotonia, bow legged, hypocalcaemic convultions, fractures

26
Q

ricketts causes

A

vit d deficiency - malabsorption, poor diet
drug induced - anticonvulsants
renal osteodystrophy

27
Q

ricketts investigations

A
low PO43-
raised ALP
raised PTH
low active (25-hydroxy) vitamin D
28
Q

ricketts management

A

vit d and calcium

29
Q

osteomyelitis common organisms

A

staph aureus
pseudomonas
e.coli
h.influenza

30
Q

osteomyelitis presentation

A

gradual onset
unwillingness to move
tender, warm, erythema

31
Q

osteomyelitis management

A

vancomycin and cefotaxime until organism known

32
Q

osteomyelitis investigations

A

raised esr,crp,wcc
mc+s
bone biopsy

33
Q

juvenile idiopathic arthritis diagnosis

A

normocytic anaemia
raised platelets WBC ESR/CRP
ANA RF HLA B27

34
Q

juvenile idiopathic arthritis managment

A

paracetamol and NSAIDS
methotrexate (dmard)
tocilizumab - il6 receptor blocker decrease inflammatory cytokine production

35
Q

juvenile SLE features SOAP BRAIN MD

A

mainly adolescent females. relapsing and remitting

serositis
oral ulcers
arthritis
photosensitivity

bloods - anaemia, thrombocytopenia, leucopenia, low c3/c4
renal - proteinuria
ANA
Immune - double stranded DNA
neurological - seizures, psychosis

malar rash
discoid rash

36
Q

juvenile SLE management

A

hydroxychloroquine and NSAIDS

37
Q

juvenile idiopathic arthritis presentation

A
fever spikes with salmon pink rash and joint Pain
Swelling - persistent
Joint stiffness (morning)
Loss of range of movement
Joint deformity
Warmth 
Colour change
38
Q

juvenile idiopathic arthritis - oligoarticular

A

less than 4 joints

knee and ankle are common

39
Q

juvenile idiopathic arthritis - polyarticular

A

more than 4 joints

40
Q

septic arthritis management

A

staph areus - IV Cefuroxime