OSCE 1 Flashcards

(56 cards)

1
Q

1.1 a Check the breathing system on machine circle absorber

A
  1. Visual inspection
  2. Ensure soda lime present not used up
  3. Identify blockages
  4. Leak test
    - Flow 0, Close APL, Occlude Y, pressure bag to 30cmh20 w/ flush, ensure remains fixed >10s, open apl - ensure decreased
  5. Leaks?
  6. 1 way valves - connecting bags

second bag - sim lung on Y, fill with flush + manually ventilate - ensure inflate and deflation lung connected y - watch movement unidrectional

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2
Q

1.1 b What are the benefits of the system circle absorber

A
  1. Economy - FGF reduced - <1lmin, reduced consumption volatiles
  2. Humidification - inspired gas saturated w/ water vapour from expire gas
  3. Reduce heat loss - conservation of heat - exothermic reaction co2 absorption assists maint body temp
  4. Reduced pollution - using low FGF - escaped volatile minimised
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3
Q

1.1 c What happens if a unidirectional valve malfunctions

A

Mixing of inspired gas with expired gas - Co2 = hypercapnia

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4
Q

1.1 d What is the mesh size of granules
what’s that in mm

What do you understand by x-y mesh

A

4-8 mesh or 3-4 mm spheres

Strainers w/ 4-8 mesh - 4 equal strands per linear inch both vertical + horizontal axes
8 mesh = 8 = strands per linear inch
granules of 4-8 mesh size pass thru strainers w/ 4-8 mesh

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5
Q

1.1 e What constitutes soda lime

What are contaminants produced w/ soda ime

A

NaOH, CaOH, KOH, Water, Silica

Compound A - sevo
CO - des iso enflurane

methane + acetone also produced

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6
Q

1.1 f Chemical reactions soda lime

A

Co2 reacts w/ water = carbonic acid - reacts cal hydrox - calc carb and water

co2 also reacts w/ naoh -> sodium bic - reacts calcium hydroxide to regenerate sodium hydroxide

CO2 + H20 -> H2C03 & H+ & HCO3-

CaOH2 & ++ & HCO3- -> CaCO3 + 2 H2O

CO2 + 2 NaOH -> Na2CO3 & H2O & Heat

Na2CO3 & CaOH2 -> 2NaOH & CaCO3

water required for absorption
moisture already present
more added patient

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7
Q

1.1 g Unidirectional valve - how does it function

A

Inspiration -
FG & CO2 free gas from reservoir bag passes thru inspiratory unidirectional vavle & inspiratory limb to patient

During expiration
inspiratory undirect valve closed and expire gas from exp limb pass thru expiratory unidirectional valve to soda lime canister

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8
Q

1.2 ECG

A

Trop T - greatest sensitivity and specificity detecting Acute Mi
norally not serum

Aspirin decrease mortaility

Delta wave - WPW

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9
Q

1.3 Haemodynamics

Cardiac index calculation
Normal ranges

SVR calc
normal range
SVRI

DO2
CaO2

PVR

A

CI = CO / BSA
2.8-3.5

Stroke index = SV/BSA

SVR = MAP - CVP / CO x 80
900-1200
SVRI = SVR/CI -

Fluid guidance - BP, CVP, Wedge

DO2 = CaO2 x 10 x CO
CaO2 Hb x SaO2 x 1.34/100
1
PVR PAP - PAWP / CP x 80

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10
Q

1.4 Stats

Correlation coefficient is denoted by
what is it

Regression involve

R ranges from

Independent variable

Independent variable plot on

complete absence

A

Correlation coefficient is denoted by letter R
denotes association between 2 quant variables

Regression involves estimating best straight line to summarise association

R ranges from 1 -> -1

When 1 vary increase w/ other - positive

Independent variable plot on X axis

Assoc strength
0.2 very weak .4 weak .6 mod .8 strong 1 strong

complete absence represented by 0

correlation =/= causation

Significance of data tested using t-test for parametric data or a non paratmetic test - spearman rank correlation

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11
Q

1.5 Anatomy IJV

label pg 19

Describe course

Tributaries

A
Originates @ jugular foramen
-continues w. sigmoid sinus
runs down neck terminate 
between sternoclavicular joint
join subclavian vein form brachiocephalic

Common facial Vein

Lingual Vein

Superior and middle thyroid veins

Pharyngeal venous plexus

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12
Q

1.5 Anatomy IJV

Relations

ant x3

post x 3

med x3

A

Anterior
1 ICA

2 Vagus between V+A

3 SCM lower part b

Posterior

1 Symp Chain

2 Dome pleura

3 Thoracic duct

medial

1 Carotid Artery

2 CN IX-XII

3 Deep cervical LN close to vein

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13
Q

1.5 b Describe CVC insertion

A

1 Sterile + head down

2 LA to skin

3 US + use

4 approiate skin punc + direction needle

5 aspiration blood + insert guidewire

6 Passes dilo and railroad cvc catheter

7 Aspirates lumens + suture

8 CXR

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14
Q

1.5 c CVC complications

group

A

A Mechanical

Needling / Introduction catheter

1 Haemorrhage
2 PTX
3 Haemothroax
4 Air embolism
5 Nerve damge
6 exvasc catheter placement
7 chylothorax

B Infective
C Thrombotic

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15
Q

1.5 d Veins of head and neck on page 19

A
1 Supficial temporal vein
2 facial vein
3 retromandibular v
4 IJV
5 Ant jug v
6 post auricular V
7 EJV
8 Vertebral
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16
Q

1.5 e

Tributaries of IJV

A
1 Inferior petrosal vein
2 Pharyngeal veins
3 Facial vein
4 Superior thyroid vein 
5 Middle thyroid vein
6 EJV
7 IJV
8 Ant jug v
9 Brachiocephalic V
10 Subclavian V
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17
Q

1.6 F

Major veins of head and neck describe

A

Ijv contin of sigmoid sinus

Runs down neck vertrical
lateral to interal carotid artery
then lateral to CCA

Glossopharyngeal + hypogloassal N
-forward between IJ and CCA
Vagus descend between and behind v + a in same sehath

Many tributaries within neck
Inferior petrosal sinus, common facial, lungual pharygneal super midldle thyroid veins
sometimes occip

EJV commences sub of partoid gland @ level mandilbe
passes down to midpoint of clav and enter subclav vein
crosses SCM

RIJ is straighter - more common for canullation
NICE - US

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18
Q

1.7 ATI
a
Awake tracheal intubation explain to pt
14 points on marking sheet

A

1 Introduce

2 Understanding previous problem

3 Explain why intubate

4 Normally done

5 Why different

6 Other methods - VL after induction

7 Disadvantage
failure intubvate
further danger d/t difficulty BMV

8 Adv / benef
Maint airway = safer

9 May use sedation

10 monitoring

11 LA tech

12 Compare to camera test

13 simple terms

14 clarity

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19
Q
1.7 ATI
b
indication
C/I
Procedure

how

why nose

A

Indi - known / suspected difficult airway
aspiration risk + difficult intubate antic
cervical cord instabiltiy

c/i  rel
upper airway bleeding
bleeding tendency
stridor
uncoop patient

full monit
iv acces

supplement o2 thr
sedation + reversal

head end / operator in front

Nasal - Alignment / uncomfortable

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20
Q

1.7 ATI
C
Techniques airway anaesthesia

Nose

Oropharynx

Lower airway

A

1 Nose
Cocaine 1.5mg kg or
lidocaine 5% w/ phenyl 0.5%

Oropharynx Lido 4%
gargle 4-5ml solution
3-4 spray of 10% lidocaine (spray 10mg)

Neb also a technique

Lower airway
Spray as you go
4% lido - thru scope w/ epidural catheter down scope
direct visualisation

scope adv to base 2ml 4% sprayed
another 1-2,ml sprayed glottis and vocal cord
scope advance & 1ml thru vocal cords

Total 3-6ml 4% required

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21
Q

1.7 ATI

Internal laryngeal N

A

Int laryngeal nerve
branch superior laryngeal
block internal / external approach

hyoid bone located directly above thyroid
greater horn - located lateral most part none
SLN block walking 25g needle off greater horn inject 2ml 2%
accident art inject into CCA possible complication

internal laryngeal run under muc membrane covering piriform fossa

Trans laryngeal 
skin cricothyroid infiltrate w/ LA
needle / cannula attach to syringe - n saline thru cric
direct bac and caudal avoid vc trauma
aspiration air
4ml 4% lido inject end inspiration
cough - spread above below
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22
Q

1.7 Surgical cricothyroidotomy ? NOT RECOMMENDED PRACTICE NO MORE? - LOOK UP

A

FONA das algorithim

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23
Q

1.7
b
Cricothyroid membrane

A

subcutaenous midline structure

between strap muscles of neck

8mm deep below skin
rang 3-14mm
adults 9mm high 22-30mm wide
wargest tube <9mm outer diam

Identify
feel thyroid notch & follow down
identify stabilise catilage thumb and middle using index locate membrane in midline
not pro pal trachea and follow up

24
Q

1.8 Trauma patient assessment

A
A
B
C
D
Rapid prim survey
resus vital fxnions
detailed assessment and initiation dfinitice care
25
1.8 b Indication for CTB in head injury x7 RFactors to warrant further look x 3
1 GCS < 13 on initial assessment · 2 GCS < 15 at 2 hours after injury on assessment in the emergency department · 3 Suspected open or depressed skull fracture · 4 Any sign of basal skull fracture · 5 Post-traumatic seizure · 6 Focal neurological deficit · 7 More than one episode of vomiting since the head injury risk factors 1. Age > 65 years · 2. A history of bleeding or clotting disorder · Dangerous mechanism of injury (a pedestrian or cyclist struck by a motor vehicle, an occupant ejected from a motor vehicle or a fall from height of > than 1 metre or 5 stairs) · 3.More than 30 minutes’ retrograde amnesia of events immediately before the head injury On assessment can the patient actively rotate the neck to 45 degrees to the left and right? if Y - no need for further
26
1.8 c | Neurosurgical r/f
1. Persisting coma GCS 8< after initial resus 2. Confusion >h 3 Deterioration in LOC after admin 4 Progressive FND
27
1.9 PA Catheter dNormal PCWP where does prox lumen open where from tip how much volume in baloon draw traces wheres the thermistor 4 uses 4 complications measured values derived values
4-12 Prox lumen - open 25cm tip in RA measure cvp 1.5ml Draw traces as inserted and wedges thru diagram page 35 scaler of 40mmhg, RA 0-4, RV 25 -2 PA Resembles trace CVP in RA Thermistor situated? 4cm prox tip 1 assessment of volume status CVP unreliable 2 Sampling mixed venous blood calc shunt fraction 3 Measure CO using thermodilution 4 Derivation of CV indices such PVR DO2 and uptake Arrhythmia on insert knotting in rv balloon rupture Pulmonary infarction Measured CVP PAP PCWP CO SVO2 Derived CI SV SVI SVR SVRI PVR PVRI
28
1.9 b PAC other
PAFC swan and ganz in 970 7 or 7.5G and 110cm long Balloon tip capacity 1.5ml Before insert - traducer and zero - wave up down appear monitor - ensure corrected to correct advamced 20cm before inflate baloon RA - CVP mon rv systol 25
29
1.10 Paeds resus Compressions Doses
5 rescue breaths Compressions 15:2 Doses Adren 10mcg kg .1ml/kg of 1:10000 IO dose 10ug Amiodarone 5mg / kg Atropine 20mcg / kg Dose DCCV 4 J Kg Weight 2x age + 4 Fluids 20ml kg IO after 3 attempts
30
1.10 b Hs Ts
Hypoxia hypovolaemia hypo / hyper kalaemia hyothermia Tension PTX Tamponade Toxins Thromboembolism
31
1.13 a Anaphylaxis Sim station 10 points
1. Checks vitals + calls critical incident 2. call 4 help 3. 100% O2 4. Check ETT 5. Auscultate 6. Ask for adrenaline 7. Check drip give floods 8. look cutaneous signs 9. Admin Anaphylaxis adjuncts 10. Reassess vitals + repeat adren prn
32
1.13 b recognising anaphlylaxis 6 features
1 Tachycardia 2 Hypotension 3 Severe bronchospasm 4 Low sats - im gas exchange & reduced peripheral perfusion 5 low etco2 reduced pumonary perfusion 6 Unresponsiveness to ephedrine metaraminol
33
1.13 Anaphylaxis QRH steps
❶ Call for help. Note the time. Stop or do not start non-essential surgery. ❷ Call for cardiac arrest trolley, anaphylaxis treatment pack and investigation pack. ❸ Remove all potential causative agents and maintain anaesthesia. • Important culprits: antibiotics, neuromuscular blocking agents, patent blue. • Consider chlorhexidine as cause (impregnated catheters, lubricants, cleansing agents). • Consider i.v. colloids as a possible cause. • Change to inhalational anaesthetic agent (if not already). ❹ Give 100% oxygen and ensure adequate ventilation: • Maintain the airway and, if necessary, secure it with tracheal tube. ❺ Elevate patient’s legs if there is hypotension. ❻ If systolic blood pressure < 50 mmHg or cardiac arrest, start CPR immediately. ❼ Give drugs to treat hypotension (Box A): • Hypotension may be resistant and may require prolonged treatment. • Give adrenaline bolus and repeat as necessary. • Consider starting an adrenaline infusion after three boluses. • If hypotension resistant, give alternate vasopressor (e.g. metaraminol, noradrenaline infusion +/- vasopressin) • Give glucagon in ß-blocked patient unresponsive to adrenaline. ❽ Give rapid i.v. crystalloid: 20 ml.kg-1 initial bolus, repeated until hypotension resolved. ❾ Give hydrocortisone as part of resuscitation (Box B). ❿ If bronchospasm is persistent, consider → 3-4 ⓫ Take 5-10 ml clotted blood sample for serum tryptase as soon as patient is stable. • Plan for repeat sample at 1-2 hours and >24 hours. ⓬ Give chlorphenamine when feasible (Box B). ⓭ Plan transfer of the patient to an appropriate critical care area. Note tasks in Box D. ⓮ Prevent re-administration of possible trigger agents (allergy band, annotate notes/drug chart)
34
Drugs as per QRH for hypotension
Box A: DRUGS TO TREAT HYPOTENSION IF CARDIAC ARREST → 2-1 • Adult adrenaline: i.v. 50 μg (= 0.5 ml of 1:10 000) i.m. 0.5 mg (= 0.5 ml of 1:1000) if i.v. not possible • Paediatric adrenaline: i.v. 1.0 μg.kg-1 (0.1 ml.kg-1 of 1:100 000) [1:100 000 solution made by diluting 1 ml of 1:10 000 up to 10 ml] • If no i.v. access, intraosseous adrenaline dose same as i.v. • Suggested adrenaline infusion regimes (adult): 5 mg in 500 mL dextrose = 1:100 000, titrate to effect 3 mg in 50 mL saline. Start at 3 ml.h-1 (= 3 μg.min-1), titrate to maximum 40 ml.h-1 (= 40 μg.min-1) * Glucagon (adult): 1 mg, repeat as necessary * Vasopressin (adult): 2 units, repeat necessary (consider infusion)
35
1.13 Anaphylaxis | C Other drugs
``` Box B: OTHER DRUGS • Hydrocortisone i.v. doses: • Adult: 200 mg • Child 6-12 years: 100 mg • Child 6 months-6 years: 50 mg • Child <6 months: 25 mg • Chlorphenamine i.v. doses: • Adult: 10 mg • Child 6-12 years: 5 mg • Child 6 months-6 years: 2.5 mg • Child <6 months: 250 μg.kg ```
36
1.13 Anaphlyaxis | D Dont forgets from QRH
Box D: DON’T FORGET • Repeat testing for serum tryptase at 1-2 hours and >24 hours. Store at 4 degrees • Liaise with hospital laboratory about analysis of samples. * Liaise with department anaphylaxis lead regarding referral to a specialist allergy or immunology centre to identify the causative agent * Inform the patient, surgeon and general practitioner. Report Inform patient
37
1.13 e Physiology of anaphylaxis from Kerry B Diff Anaphylactic vs anaphlyactoid reaction
Anaphylactic - acute hypersensitivity rxn - previously sensiteised to antigen Immune med Ige mast cell med His + LK released mast cell Not relate to dose type 1 or immed hypersensitivity Anaphylactoid - no prior exposure, can occur on 1st exposure, not IgE med, less severe (can be fatal) related to dose of agent Similar symptoms = sim causative agents rel from mast cell difficult different clinc
38
1.13 f Physiology of anaphylaxis Acute reaction
1st exposure no symp ige atnibod develop over 14 days attached to mast cell mast cell may have 500000 IgE attach membrane second and subseq exposure - severe reaction combines with cell bound IgE mast cell release contents severeal mediator rel - symp can produce his alone symptoms vary mild to severe depending amt histamine rel anaphylaxis - without protection - old theory 1st exposure used body protection / defences against toxic agent body without defences for subsequent exposures.
39
1.14 Monitoring - capnography a ID it Main stream what is advantage Factors affecting response time
Main stream Cuvette Co2 sesnor - between ett and system No need sampling vapour condense on sensor - can result fals high sense heated 39C heavier and cumbersome Adv No delay, no loss in gass no mixing sample w/ inspired
40
1.14 b side stream
Side stream Sensor located main unit gas aspirated - small pump via sampling tube at rate of 50-150ml min gas also contain anaes - should be scavenged / return system Factors affecting response time 1 Response time depend on transit + rise time 2 Transit time depends on length of sample tube 3 rise time depends on optimum flow and size of chamber - very low flow and large chamber increase rise time
41
1.14 C | Capnography phases
1 Inspiratory baseline - should be at 0 as elevated means rebreathing 2 Expiratory upslope shallow - obstruction 3 Plateau representing mixing of alveolar gas if sloped not flat - uneven mixing - COPD 4 Inspiration Fall to 0 downstroke
42
1.14 Patterns of capnography on page 57
page 57
43
1.14 E Measurement of CO2 Capnometry vs capnography plotted how
Capnometry measure of Co2 conc during resp cycle Capnography graphic representation CO2 conc - waveform plot as EtCO2 vs time CO2 plot y Time - X
44
1.14 F | How does it work
Capnography uses IR absorption Co2 gases molecules 2/= diff atoms absorb rad in IR spec Co2 strong absorption at 4.26 Spec scope page 58 Amt IR absorb proport to CO2 conc Electrical output photodetector present partial pressure Co2 in sample chamber
45
1.14 G Changes in trace sudden decrease to 0 sudden decrease low exponential drop gradual decrease
Sudden decrease in etco2 to near 0 = airway disconnection totally obstructed airway or vent malfunction Sudden decrease to low level reduced CO or leak Exponential drop due to reduced pulmonary perfusion - low CO / PE Gradual decrease hypervent / hypothermia gradual increase - hypervent or increase body temp
46
1.15 a 1 Diathermy uses 2 Physical principle 3 current 4 Why plate large area 5 What happens if plate disconnected + diathermy activated 6 Other problems with diathermy 7 Mono vs Bi
1 High freq A/C cut or coag tissue during surgery cutting - sine wave coag - damped or pulse sine wave 2 High frequency current (min fx on cardiac and skel muscle) localised high density current (current per unit area) High at forceps at low at earth plates depending equipment - accetpable level leakage determ 3 0.5- 1Mhz 4 Large plate reduce current density 5 Current flow thru patient and earther thru any metal - attach patient 6 Fire and explosion pacemaker dysfxn 7 monopolar needs neutral plate bipolar forceps travels one limb and leaves other other bipolar - lower power
47
1.15 b Safety features Precautions prevent hazards to patient
1 Outer case is earthed 2 Isolating capacitor - high impedance to low frequency current 3 Floating circuit active electrode and neutral electrodes are isolated from earth connection 'earth free circuit' 1 Good connection to neutral plate to patient 2 not activating diathermy until active electrode contact with tissues 3 Regular servicing
48
1.15 C | Pictures
CF - leakage current <10amperes BF Higher protection vs shcok than provided w/ B LIKE cf - FLOATING RESPECT TO EARTH B type = non cardiac grounded applied parts come into contact with patient C type = heart f floating - isolating CF is for cardiac application - higher degree protection vs shock than BF
49
1.16 A Radiology CXR CT ratio And heart borders in page 66
Normal cardiothoracic ratio
50
1.18 Awarenss communication station a marking sheet 15 points
1 introduce 2 Discuss in presence of witness 3 confirms facts listens to hix find out what does recollect 4 apology 5 advise precaution measure taken 6 unstable - stabilised careful drug - possible during that had awareness 7 any pain during it 8 rare, complcation with trauma patients 9 interview senior consultant 10 Follow up 11 Psychological support 12 Ressures future ga safe 13 advise documented and write gp 14 want to know anything more 15 polite rapport
51
1.18 b | Memory
Explicit memory - concsiou awarenss w/ recall +/- pain implicit - perception during anaes w/out recall or concious awareness poss confused ketamine high obs and trauma tiva human error judgement or faults
52
1.18 C | Key points
See patient early ward nurse / reposible apology not guilt - helps patient get facts right find out what recall explanation and conduct and safety special interest boss reassure
53
1.19 a Anatomy - intercostal nerve block Anatomy of chest on page 72
page 72 1-8
54
1.19 b 3 structures passing thru IC space how aranged i 3 strctures how arranged ii how many veins in each space iii Indications iv which 2 muscles are IC nerves and vessels found V Complications vi what type of nerve is IC
i V A N above down ii 1 posterior 2 anterior iii ``` Thoracic surgery breast upper abdo procedures - chole rib # IC drain ``` iv NV bundle between internal IC + Inner IC muscle ``` v PTX intravasc injection Bleeding nerve damage LAST ``` vi Mixed spinal
55
1.19 c Intercostal nerves emmerge where divsions? what they do connect what where does ant division pass Where doe t1 t2 etc provide Prob with block?
t1-t12 Spinal nerves emerging intervertebral foramina divide into anterior post div post - sens and motor to parvert region connect symp chain anterolat to vert body and comm thru rami communicantes ``` ant division ic space - ICN runs costal groove inner surf inf border of body rib = protection ``` T1 - UL T2 Intercostbrachail T3-6 thorax t7 down - abdominal wall t11 t12 - subcostal Block - no viscera cover D/T vascularity - high propensity for LAST Higher peak plasma vs other blocks
56
Cranial Nerves and how to test
1 Olfactory - smell 2 Optic ``` Pocket acuity chart - seperately visual fields Diam pupils Direct + Consensual Accom reflex ``` ``` 3 4 6 Oculomotor Trochlear Abducens Ex ox muscle SO4 LR6 3 - levetor palpebrae - eye elevator - PS fibre w/ 3rd - a/w disrupt ps activit - pupil dilate ``` 5 trigeminal - mastication + sensory innervation feel masseter bulk - clench teeth ``` sense Opthalmic max mandib pin prick fine touch cold + warm ``` corneal reflex V + VII Cotton bud touching cornea - observe reflex blinking 7 Facial look creases raise eyebrows, shut eyes tight, show teeth bloow air out lesion peripheral - entire half face central - upper half spare - dual innervation ``` 8 Vestbicoc hearing and balance - hearing tested bilat connect precise diagnosis difficult whisper webber rinne audiogram ``` ``` 9 + 10 Glossopharyngeal + Vagus Test together hoarseness swall dific aa look palate - uvula reamains midline soft palate rises symett unilat - uvula drawn to normal side gag sens ix motor x - stim back throat ``` 11 Accessory trapezius + SCM wwasting muscle shrug shoulder and turn head 12 hypogloassoal tongue muscle - ask prtude - wasting fasic deviation unilat lesion - towards affect side