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Other Important Topics Flashcards

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1
Q

Timelines of PTSD and stress disorders

A
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2
Q

What is an acute stress disorder? How long must symptoms last for in order to warrant diagnosis?

A

Stress reaction that occurs in the first 4 weeks following a traumatic event. Symptoms must last for >3 days to warrant diagnosis

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3
Q

What is PTSD?

A

Stress reaction occurring for over 4 weeks following exposure to traumatic event

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4
Q

When does acute stress disorder become PTSD?

A

After 4 weeks

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5
Q

When does PTSD become chronic PTSD?

A

After a year

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6
Q

Core symptoms of PTSD

A

THINK: RAH(e) you have PTSD

Reexperiencing – flashbacks, nightmares, repetitive intrusive images

Avoidance – avoiding people/places/situations/associations with event

Hyperarousal – hypervigilance for threat, exaggerated startle response, sleep problems, difficulty concentrating

Emotional Numbing

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7
Q

What additional symptoms point towards a diagnosis of complex PTSD?

A

Emotion Dysregulation (hyperactivation, de-activation)
Negative Self-Concept (feeling diminished, defeated, worthless, guilt, despair)
Disturbed Relationships (difficulties in feeling close, little interest in relationships or social engagement)

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7
Q

What is required before a formal diagnosis of PTSD?

A

routine referral to the community health team

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7
Q

Give 3 differences between PTSD and acute stress disorder

A

PTSD defined by symptoms in clusters not necessarily totality

PTSD has dissociative subtype but depersonalisation/derealisation rare in ASD

PTSD feature non-fear based sx (e.g. risky behaviour) whereas ASD doesnt

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8
Q

Investigations for PTSD

A

Full and thorough history
TSQ – Trauma Screening Quesitonaire (10 qs)
CRIES8 – Child Revised Impacts of Events Scale; used for children
If ?PTSD – routine referral to CMHT for formal diagnosis
Rule out organic causes for depression

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9
Q

PACES: What screening tools can be used for PTSD? What’s the difference between them?

A

TSQ – Trauma Screening Quesitonaire (10 qs)
CRIES8 – Child Revised Impacts of Events Scale; used for children

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10
Q

What is the management of acute stress disorder?

A

Conservative management

NOTE: Arrange follow up after a month

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11
Q

Management of PTSD

A

Trauma-Focused CBT
Offered to all patients with PTSD symptoms lasting > 1 month

Eye Movement Desensitisation and Reprocessing (EMDR)
Offer to adults with a diagnosis of PTSD or clinical important symptoms who have presented > 3 months after non-combat related trauma

Group Therapy: meeting and speaking with other people with similar experiences.

Pharmacological Treatment
Consider SSRI (e.g. paroxetine and sertraline (licensed)) or venlafaxine for adults with PTSD. Consider antipsychotics (e.g. risperidone) in addition to psychological therapies if they have failed to respond to other drug treatment or have disabling symptoms/behaviours (e.g. hyperarousal)

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12
Q

Who is Trauma focussed CBT offered to? When?

A

Offered to all patients with PTSD symptoms lasting > 1 month

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13
Q

Who is Eye movement desensitisation and reprocessing offer to? When?

A

Offer to adults with a diagnosis of PTSD or clinical important symptoms who have presented > 3 months after non-combat related trauma

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14
Q

What pharmacological treatment is used in PTSD?

A

Consider SSRI (e.g. paroxetine and sertraline (licensed)) or venlafaxine for adults with PTSD.

NOTE: Consider antipsychotics (e.g. risperidone) in addition to psychological therapies if they have failed to respond to other drug treatment or have disabling symptoms/behaviours (e.g. hyperarousal)

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15
Q

What are 1st line medications in PTSD?

A

SSRI (e.g. paroxetine and sertraline (licensed)) or venlafaxine for adults with PTSD

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16
Q

When should antipsychotics be considered in patients with PTSD?

A

Consider antipsychotics (e.g. risperidone) in addition to psychological therapies if they have failed to respond to other drug treatment or have disabling symptoms/behaviours (e.g. hyperarousal)

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17
Q

Difference between personality traits and disorders

A

Patient have no insight in a personality disorder, and they cause significant distress and/or impaired function

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18
Q

Examples of personality traits

A

Positive traits: Kind, Confident, Generous
Negative traits: Lazy, Rude, Violent

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19
Q

3 clusters of personality disorders

A

Cluster A - Weird
Cluster B - Wild
Cluster C - Wacky

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20
Q

What personality disorders fall within cluster A (Weird)?

A

Paranoid
Schizoid
Schizotypal

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21
Q

Features of paranoid disorder

A

Deep-seated distrust of others, including friends and family
Guarded, suspicious, struggles to build close relationships
Hallmark is Projection – immature defence mechanism aimed at attributing unacceptable thoughts to others

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22
Q

Hallmark of paranoid disorder

A

Projection – immature defence mechanism aimed at attributing unacceptable thoughts to others

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23
Example of paranoid disorder
Patient accuses doctor of being judgemental and suspicious
24
guarded, suspicious, struggles to build close relationships
Paranoid disorder
25
Features of schizoid personality disorder
Likes to be alone Voluntary social isolation/withdrawal More comfortable in that way Does not enjoy close relationships Little/no interest in sexual experiences Few/no pleasure activities (hobbies) Lacks close friends, detachment, limited emotional expression NOTE: Think of school: we all knew kids who were reclusive and did not spend time with friends
26
What personality disorder should schizoid NOT be confused with? How to differentiate?
DO NOT CONFUSE WITH AVOIDANT --> want to make relationships, but afraid of doing so as afraid of rejection
27
Features of schizotypal
Fear of social interactions and few close friends Classic feature: odd beliefs and/or magical thinking e.g. superstitious, believes in telepathy, sixth sense etc. Not psychotic and no hallucinations, just weird and strange beliefs Ideas of reference: often believe that events happening are somehow related to them No delusions: a key feature is that they are open to challenges to their beliefs, they may reconsider their superstitions when confronted
28
Classic feature of schizotypal
odd beliefs and/or magical thinking e.g. superstitious, believes in telepathy, sixth sense etc.
29
Difference between schizotypal and psychosis
No delusions: a key feature is that they are open to challenges to their beliefs, they may reconsider their superstitions when confronted NOTE: In schizotypal they are open to confrontation/challenges about their thoughts
30
What personality disorders are in cluster B (Wild)?
Antisocial Borderline Histrionic Narcissistic
31
Features of Antisocial personality disorder
Aka Sociopathy Lots of the people in jail More common in men Disregard for rights of others, often breaks the law Impulsive, lacks remorse If it occurs in a child < 18yo, named conduct disorder (CD)
32
What is antisocial personality disorder called if it occurs in a child?
If it occurs in a child < 18yo, named conduct disorder (CD)
33
What does diagnosis of antisocial personality disorder require?
Diagnosis requires to be at least 18yo and have evidence of CD before the age of 15 - not necessarily diagnosed, a hx is sufficient e.g. violence, killing animals, cruelty
34
Difference between schizoid, antisocial and avoidant personality disorder
Schizotypal  no interest in friends Antisocial  no friends due to their actions being so antisocial e.g. violent Avoidant  trying to make friends, but no success due to deep rooted fear
35
Borderline personality disorder AKA
Emotionally unstable personality disorder (EUPD)
36
Features of EUPD
Unstable personal relationships They have great friends and the second day, they hate them – “All people are either very good or very bad” Stormy relationships, common to them in on/off patterns Fear of abandonment Impulsivity: spending sprees, sex with strangers, reckless driving Self-mutilation: cutting, burning Suicide gestures/attempts: rarely follow through Splitting is a major ego defence mechanism -black-and-white thinking, cannot hold opposing view
37
How is EUPD treated?
Dialectical Behaviour Therapy (form of CBT) designed to treat chronic suicidality is now the gold standard
38
Which personality disorder is the only one with treatment? What is it?
Borderline, with DBT
39
Features of histrionic personality disorder
Wants to be the centre of attention The kind of people you see at a party who talk loudly, tell wild stores, use grandiose hand gestures  anything to get everyone to look at them More common in women Inappropriate sexually provocative behaviour Often wears provocative clothing, touching others frequently Concerned with physical appearance to draw attention – exotic outfits, hats, shoes etc. NOTE: Think centre of attention
40
Features of narcissistic personality disorder
Grandiosity, inflated sense of self Brags and thinks everything they do is great Lacks empathy and views other people as competitors – does not point out when someone else is doing something good Wants to hear that they are great all the time Overreacts to criticism with anger/rage NOTE: Usually lack empathy, sleep with best mates girlfriend and don’t understand what they did wrong
41
What personality disorders are in cluster C (Wacky)?
Avoidant Obsessive Compulsive Dependent
42
Features of avoidant personality disorder
Avoids social interactions and “displays social inhibition” Reason is that they feel inadequate – hypersensitive to rejection Afraid people won’t like them and of embarrassment Struggles with intimate relationships
43
Features of obsessive compulsive personality disorder
Preoccupied with order and control Loves ”to-do” lists, always needs a plan Inflexible at work or in relationships because they want things done a certain way The behaviours they engage in help them achieve their goals – in contrast to OCD where the behaviours are barriers
44
How does OCD differ from OCPD?
In OCPD the behaviours they engage in help them achieve their goals (ego syntonic) In contrast to OCD where the behaviours are barriers (ego dystonic)
45
Features of dependent personality disorder
Clingy, low self-confidence Struggle to care for themselves Depend on others excessively Rarely alone, often in a relationship Hard to make decisions on their own, need someone to tell them what to do Difficulty expressing opinions Often involved in abusive relationships because they’re afraid to walk away
46
What are people with depdendent personality disorder at risk of?
Abusive relationships
47
What is Neuroleptic Malignant Syndrome? When does it occur?
Rare life-threatening psychiatric emergency Occurs if suddenly stopping or changing dose of antipsychotic or dopaminergic drugs e.g. Levodopa (Parkinson’s Disease)
48
What is Seretonin Syndrome? When does it occur?
Overstimulation of serotonin receptors More commonly occurs from a drug combinations but can occur singularly:
49
What drugs can cause seretonin syndrome?
SSRIs MOA TCAs Amphetamines+ Ecstasy
50
How does the onset of neuroleptic malignant syndrome and seretonin syndrome vary?
NMS - hours to days Seretonin syndrome - <12 hours, usually in a few hours
51
Signs and symptoms that appear in both of NMS/Seretonin syndrome
Autonomic dysfunction: High fever (> 40°C), shivering, excessive sweating (diaphoresis), tachycardia, palpitations Headache Abdominal pain Nausea, vomiting, diarrhoea Altered mental state Coma 🡪 NMS stupor
52
Specific signs and symptoms for NMS
Muscle deactivation – stiffness, rigidity, hyporeflexia Dysphagia Incontinence
53
Specific signs and symptoms for seretonin syndrome
Muscle activation - tremor, myoclonus, clonus, hyperreflexia Oculogyric crisis: EOM spasms leading up upward gaze Dilated pupils
53
Management of NMS and seretonin syndrome
Conservative Stop causative drug: resolves once medicines have been discontinued Stabilise e.g. IV crystalloid fluids – prevents renal failure esp. in NMS Medication Muscle relaxants: benzodiazepines & bromocriptine 🡪 IV dantrolene (after senior review SS: IM chlorpromazine (antipyretic + sedative). IV Paracetamol
54
Investigations for NMS and seretonin syndrome
Vital signs: either hypertensive crisis or shock ECG: 12 lead syndrome Bloods: FBC / CRP / blood cultures: rule out sepsis U&Es: AKI (NMS) and rule out electrolyte imbalances CK: hyperthermia causes rhabdomyolysis (NMS) LFTs ABG (metabolic acidosis) Clotting screen TFTs: rule out thyrotoxicosis Urinalysis: drug screen (rule out overdose), myoglobulin CT head: rule out CNS infections, brain tumours (malignant hyperthermia), normal pressue hydrocephalus Consider Lumbar Puncture to rule out meningitis and encephalitis
55
What medication may be used in the management of NMS or seretonin syndrome?
Muscle relaxants: benzodiazepines & bromocriptine 🡪 IV dantrolene (after senior review) IV Paracetemol
56
What specific medication may be used for seretonin syndrome?
IM chlorpromazine (antipyretic + sedative).
57
What is gender dysphoria defined as?
incongruence between a person’s experienced and expressed gender, and their primary and secondary sexual characteristics.
58
How is gender dysphoria managed conservatively?
supporting a person to live the way that they want to, in their preferred gender identity or as non-binary and treatment of any concurrent MH disorders
59
When to consider medical or surgical options in gender dysphoria?
After a year
60
Medical options for gender dysphoria
GnRH analogues to block puberty Hormonal treatment Oestrogen +/- androgen suppression Androgens
61
Surgical options for gender dysphoria
Re-assignment surgery Mammoplasty Hair removal
62
PACES: What information to be given to people over 16 who want gender-affirming hormones?
side-effects include infertility, blood clots and weight gain.
63
How many dopamine pathways are there in the brain? What are they?
Mesolimibic pathway (positive symptoms) -limbs – fast/positive Mesocortical (negative symptoms) – cortical – clever - depressed Nigrostriatal (Extrapyramidal side effects) – striatal – striving for (extrapyramidal) side effects Tuberoinfundibular (hyperprolactinaemia) – tubero – sounds like hyperpro -… lactinaema
64
What dopaminergic pathway is involved in positive symptoms?
Mesolimibic pathway (positive symptoms) -limbs – fast/positive
65
What dopaminergic pathway is involved in negative symptoms?
Mesocortical (negative symptoms) – cortical – clever - depressed
66
What dopaminergic pathway is involved in extrapyramidal side effects?
Nigrostriatal (Extrapyramidal side effects) – striatal – striving for (extrapyramidal) side effects
67
What dopaminergic pathway is involved in hyperprolactinaemia?
Tuberoinfundibular (hyperprolactinaemia) – tubero – sounds like hyperpro -… lactinaema
68
What do typical antipsychotics act on? What are they AKA? Give some examples
Typical antipsychotics –widely acting on D2 dopamine receptors Also known as First Generation Antipsychotics (FGAs) Examples – Chlorpromazine, Haloperidol
69
What do atypical antipsychotics act on? What is it AKA? Give examples
less affinity for dopamine, more on serotonin and 5-HT2A Also known as Second Generation Antipsychotics (SGAs) Examples – Clozapine, Olanzapine, Quetiapine, Risperidone
70
Example of a partial agonist
Apiprazole
71
Difference between typical and atypical antipsychotics
Newer atypicals are associated with a lower risk of extrapyramidal side effects Atypicals all act as antagonists at the 5HT-2A serotonin receptor
72
Gold standard drug for psychosis that is used in treatment resistant schizophrenia
Clozapine
73
Clozapine specific side effects
Neutropenia/leucopenia/agranulocytosis – requires regular FBC blood test monitoring
74
What do patients on Clozapine require regular monitoring for? Why?
Patients require regular FBC monitoring due to the risk of neutropenia/leucopenia/agranulocytosis (approximately 1% of patients)
75
Rules of dosage for clozapine
Clozapie must be started at a low dose and titrated slowly due to the risk of cardiovascular instability Clozapine must be re-titrated if stopped for more than 48 hours
76
What type of medication might be beneficial for patients with poor oral compliance?
Depot injections
77
Management of hyperprolactinaemia as a SE of anti-psychotics
Switch to a prolactin sparing agent ( quetiapine, and ziprasidone) add in aripiprazole (partial dopamine agonist), dopamine agonists are generally avoided (e.g. carbergoline, bromocriptine)
78
Symptoms of hyperprolactinaemia
Women – Reduced libido, amenorrhoea, galactorrhoea, osteoporosis ?increased risk of breast cancer Men – Reduced libido, erectile dysfunction, gynaecomastia, galactorrhoea
79
What dopaminergic pathway is suppressed by hyperprolactinaemia?
Release is supressed by dopamine as part of the tuberoinfundibular pathway
80
What is the classic parkinsonism symptom tetrad that may be seen in drug induced parkinsonism?
Core features - Bradykinesia, rigidity, pill rolling tremor (4-6 Hz), postural instability Additional features – Hypomimia (mask face), festinating gait with reduced arm swing
81
How does drug induced parkinsonism present differently to parkinson's?
Generally is bilateral, which is in contrast to Parkinson’s Disease which typically starts asymmetrically
82
Management of drug induced parkinsonism
Management – Switch medication, add in a regular antimuscarinic (e.g. procyclidine 5mg)
83
What is tardive dyskinesia? How to manage?
An involuntary orofacial dyskinesia associated with long term antipsychotic use Typically seen as continuous mouth and tongue movements, classic “lip smacking” presentation. May also involve eye closing, jaw clenching and may affect the trunk/extremities Develops after months or years of treatment Management – May be permanent even if medication is stopped. Stop causative agent. Consider Tetrabenazine 25mg
84
What is dystonia? How to manage?
Involuntary muscle spasm/contractions Develops acutely, within hours of starting antipsychotics Can affect any part of the body. Specific names are given to certain forms Oculogyric crisis (eyes rolling upward) Torticollis (head and neck twisted to one side) Laryngeal dystonia (can compromise airway and be life threatening) Management – Anticholingerics (e.g. procyclidine 5-10mg PO or IM)
85
What is akathisia? When does it occur? How is it managed?
A sense of internal restlessness. The patient feels compelled to constantly move, rock, fidget or pace around. Very unpleasant! And associated with an increased risk of suicide. Occurs within days to weeks of starting/increasing an antipsychotic Diphenhydramine (sedating antihistamine), Propranolol, low dose benzodiazepines
86
What is metabolic syndrome? How to manage?
Triad of weight gain, dyslipidaemia and insulin insensitivity Monitor weight, blood pressure, lipid profile and HbA1C Treat any complications on their merits (e.g. statins, anti-glycaemics, anti-hypertensives) Promote healthy eating and exercise

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