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Perio MT2 > Other periodontal Diseases and Conditions > Flashcards

Other periodontal Diseases and Conditions Flashcards

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1
Q

What are the 3 hallmarks of NUG and NUP?

A
  1. Punched out papilla
  2. Bleeding
  3. PAIN!!!
  4. NUP ONLY = attachment loss
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2
Q

What is the primary reason a person seeks treatment for NUG?

A

PAIN!

-patient cannot brush or eat

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3
Q

What are 2 “other” features that are most always seen with NUG and NUP?

A
  1. “pseudomembrane” (sloughed material)

2. Halitosis (terribly bad breath)

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4
Q

What separates NUG from NUP?

A

Attachment Loss!

NUP must include attachment loss, while NUP does not have AL

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5
Q

Name 5 characteristic that may be seen with NUP and NUG, besides 3 hallmarks, AL, halotosis and pseudomembrane (crappy question….I know)

A
  • Lymphadenopathy
  • Fever, malaise
  • Poor oral hygiene
  • Sequestrum formation
  • Recurrence/Chronicity
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6
Q

Describe the significance of recurrence/chronicity in NUG/NUP

A

Having one episode of NUG/NUP greatly predicts the likelihood of getting it again

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7
Q

What is NUS?

A

Necrotizing Ulcerative Stomatitis – when NUG/NUP moves beyond the mucogingival junction

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8
Q

What is the prevalence of NUG/NUP in young adults of industrialized countries?

A

2-7%

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9
Q

What is the prevalence of NUG/NUP in young adults in developing countries?

A

Much higher than 7%

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10
Q

What is the prevalence of NUG/NUP in immunocompromised individuals (HIV, etc)?

A

1-28% (HAHAHAHA! I swear its not a joke!)

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11
Q

When diagnosing, what is the clinical presentation of NUG that you look for?

A
  1. Ulceration and necrosis of interproximal papillae
  2. Painful gingiva
  3. Bleeding (spontaneous, on slight provocation)
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12
Q

What 2 diseases must be eliminated during differential diagnosis of NUG/NUP?

A
  1. Primary Herpetic Gingivostomatitis

2. Oral mucosal diseases

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13
Q

How does Primary Herpetic Ginigvostomatitis differ from NUG/NUP?

A
  1. HIGH FEVER IS HALLMARK!
  2. Affects tongue and lips
  3. Round lesions (herpes)
    (note, pain PLUS fever is especially seen in kids)
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14
Q

How does the etiology between NUG, PHG, and Desquamative differ?

A 
NUG = Bacteria
PHG = HSV (herpes simplex virus)
Desquam = Immunologic
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15
Q

How do the most affected age groups differ between NUG, PHG, and Desquamative?

A 
NUG = 15-30 (young adults)
PHG = Children 
Desquam = Adults
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16
Q

How does the most affected site differ between NUG, PHG, and Desquamative?

A 
NUG = Interdental papilla
PHG = Gingiva and Mucosa
Desquam = Gingiva and Mucosa
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17
Q

How do the signs/symptoms vary between NUG, PHG, and Desquamative?

A 
NUG = Ulceration, pain, bleeding
PHG = Vesicles, fever, foetor
Desquam = Desquamation, pain, burning sensation
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18
Q

T/F: neither NUG, PHG, nor Desquamative are contageous

A

False! PHG is contageous (duh, its herpes)….the other 2 are not.

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19
Q

What are the 3 common gram (-) bacteria seen in NUG/NUP?

A
  1. Spirochetes (Tremponema)
  2. Fusobacterium
  3. P. Intermedia
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20
Q

What is unique about the behavior of the spirochetes and rods associated with NUG/NUP?

A

Invasiveness – these microbes actually invade the tissues

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21
Q

What 2 viruses are commonly associated with NUG/NUP?

A
  1. Human cytomegalovirus

2. HIV

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22
Q

What are the 5 biggest Host Factors that influence pathogenicity of NUG/NUP?

A
  1. Immunosuppression (systemic disease or malnutrition)
  2. Pre-existing ginigivitis (poor oral hygiene)
  3. Previous history NPD
  4. Psychological stress (including lack of sleep)
  5. Smoking (VIP!!!)
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23
Q

What is the most severe NPD seen in kids due to severe malnutrition?

Study These Flashcards
A

Noma (don’t look it up, trust me its terrible)

24
Q

What are the goals of periodontal therapies to treat NPDs?

Study These Flashcards
A
  1. Eliminate disease activity

2. Avoid pain and general discomfort

25
What are the 3 common forms of acute treatment of NPDs?
1. Debridement 2. Oral rinses (H2O2-CHX) 3. Antibiotics (only if needed, not for pain)
26
What are some further treatment options beyond the acute therapies?
1. Further medical therapy 2. Surgical therapy (defect elimination) 3. Predisposing factor elimination
27
Define GINGIVAL abscess
- Localized, painful, rapidly expanding lesion in vital tissue - Usually of sudden onset - Generally limited to the marginal gingiva or interdental papillae (sulcus)
28
What is the usual etiology (cause) of gingival abscesses?
Irritation from foreign bodies forcefully embedded into previously healthy tissues
29
Define PERIODONTAL abscess
- Localized, purulent inflammation in the periodontal tissues - Acute or chronic, - Vital pulp - Extension from pocket to perio tissues (not restricted to gingiva)
30
Where does a periodontal abscess tend to localize upon extension from the pocket to periodontal tissues?
Along the lateral root surfaces
31
When does localization of a periodontal abscess tend to occur?
Localization occurs when drainage through the pocket is impaired
32
What is a common reason for impaired drainage leading to periodontal abscess?
Post-treatment shrinkage of tissue over incompletely removed calculus
33
What 2 events/procedures often are followed by a periodontal abscess?
1. Trauma (root fracture) | 2. Endodontic perforation
34
What are the 3 types of periodontitis-related abscesses?
1. Exacerbation of chronic lesion 2. Post-therapy abscess 3. Post-antibiotic abscess
35
When and to whom does a periodontitis-related abscess due to exacerbation of a chronic lesion occur?
- Occurs when chronic lesion becomes acute | - Seen in untreated patients and maintenance patients (recurrent infection)
36
When does a periodontitis-related Post-therapy abscess usually occur?
1. Following SRP (calculus) | 2. Following surgical therapy (calculus, foreign bodies)
37
Why might someone end up with a periodontitis-related post-antibiotic abscess?
- Taking antibiotics for OTHER reasons - 40% get abscess within 4 weeks - B/c MUST have mechanical debridement with antibiotics or get SUPER-INFECTION!!!!!
38
What are the 2 non-periodontitis-related abscesses?
1. Foreign body impaction | 2. Root morphology alterations
39
What are 2 common causes of non-periodontitis-related Foreign Body Impaction Abscesses?
1. Oral Hygiene devices | 2. Food Particles
40
What are 3 common causes of non-periodontitis-related Root morphology alteration abscesses?
1. External root resorption 2. Cemental (root) tears 3. Iatrogenic (endodontic perforation)
41
What percent of periodontal abscesses are emergency conditions?
8-14%
42
How common are periodontal abscesses among patients treated for periodontitis?
27%
43
How common are periodontal abscesses in untreated patients with periodontitis?
>50%
44
What is the most prevalent site for periodontal abscesses (which tooth) and why?
Molars, due to anatomical features such as multiple roots
45
What 2 bacteria tend to be found in periodontal abscesses?
1. Periodontitis flora | 2. P. gingivalis
46
What type of obstruction tends to prevent drainage and leads to abscess
Mechanical obstruction
47
What are the 4 MOST IMPORTANT features to look for when diagnosing as a periodontal abscess?
1. Swelling 2. Redness 3. Suppuration 4. Pain
48
What are ALL the features commonly associated with periodontal abscess for diagnosis
1. Swelling 2. Redness 3. Suppuration (spontaneous or elicited) 4. Pain 5. Gingival tenderness 6. Assoc. with deep pocket 7. BOP 8. mobility 9. Radiographic bone loss 10. Fever 11. Malaise 12. Lymphadenopathy
49
How does a periodontal abscess differ from a periapical abscess!!! KNOW! LIKELY TQ!
Peri-apical abscess = DEAD TOOTH | Periodontal abscess = tooth is usually vital
50
What are 2 important complications resulting from periodontal abscesses?
1. Tooth loss | 2. Systemic infection
51
What are the 2 mechanisms by which a periodontal abscess may become a systemic infection (travels to brain, blood, and/or vital organs)?
1. Dissemination thru tissues DURING THERAPY | 2. Dissemination thru bacteremia (untreated abscess) -- upon brushing or chewing
52
What is the #1 site in the mouth for metastatic tumors? TQ!!
Gingiva -- must biopsy if does not respond to tx within 2 weeks
53
What other diseases must you eliminate via differential diagnosis for a periodontal lesion?
1. Periapical (endodontic) abscess 2. Vertical root fractures 3. Endo-perio abscess 4. Osteomyelitis 5. Tumor
54
Describe therapy prescribed for periodontal abscesses
1. Acute lesion management: - incision and drainage - SRP - Antibiotics - Periodontal surgery 2. Treatment of original/residual lesion
55
When should you include antibiotics in acute treatment of a periodontal abscess?
If there is systemic involvement
56
When should you include periodontal surgery in acute treatment of a periodontal abscess?
If related to a POCKET

Perio MT2 (5 decks)

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