Overview Flashcards

1
Q

What is dysarthria?

A

A group of neurogenic speech disorders that reflect abnormalities in various parameters of the speech mechanism.

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2
Q

What is apraxia of speech?

A

A neurologic speech disorder that reflects an impaired capacity to plan or program sensorimotor commands for speech.

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3
Q

Motor speech disorders are NOT

A
cognitive linguistic impairments
sensory deficits
musculoskeletal defects
voice disorders
normal changes in speech
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4
Q

What is the site of lesion for flaccid dysarthria?

A

brainstem motor neurons or nerves to muscles

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5
Q

What is the site of lesion for ataxic dysarthria?

A

Cerebellum, or tracts leading to and from cerebellum

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6
Q

What is the site of lesion for hypokinetic dysarthria?

A

Basal ganglia (specifically substantia nigra)

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7
Q

What is the site of lesion for hyperkinetic dysarthria?

A

basal ganglia

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8
Q

What is the site of lesion for spastic dysarthria?

A

fiber tracts connecting cortex with brainstem motor neurons

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9
Q

What are the cortical production components of speech, from first to last?

A

Plan, program, control/monitor, execute

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10
Q

Corticospinal tracts are responsible for what?

A

connecting cortex to spinal nerves

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11
Q

Corticobulbar tracts are responsible for what?

A

connecting cortex to cranial nerves

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12
Q

Corticoreticular tracts do what?

A

indirect pathways, responsible for reflexes, posture, and tone. subconscious

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13
Q

What are the effects of damage to the FCP?

A

weakness, paralysis (if all LMN damaged), fasciculations, fibrillation

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14
Q

What is a neuromuscular juncture?

A

synapse in the PNS; axon terminals, motor endplates, shwann cell sheathes

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15
Q

What is a motor end plate?

A

specialized post synaptic area in neuromuscular junction of PNS

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16
Q

What is the function of groups of fibers in the CNS?

A

tracts; designed to synapse with other neurons

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17
Q

What is the function of groups of fibers in the PNS?

A

nerves; transmitting primarily to muscle or from sensory end organs

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18
Q

What is a disease of the cell body associated with dysarthria?

A

ALS (CNS and/or PNS)

19
Q

What are two diseases of the axons associated with dysarthria?

A
MS (CNS)
Guillain Barre (PNS)
20
Q

What is a disease of the synapse between nerve and muscle (PNS) (Neuromuscular junction) associated with dysarthria?

A

Myasthenia gravis

21
Q

Which cortical motor areas are associated with dysarthria?

A

primary motor cortex
premotor area
supplementary motor area

22
Q

Which cortical motor areas are associated with stuttering?

A

right frontal operculum (broca’s)

right insula

23
Q

Which cortical motor areas are associated with apraxia of speech?

A

Broca’s area

left insula

24
Q

What are are the cortical sensory areas we need to know/

A

primary sensory cortex (temperature, pain, pressure)
primary visual cortex
primary auditory cortex

25
What are the functions of the cerebellum?
muscle tone movement sequencing and scaling (timing and force) motor learning or repeated adjustments in force and movement sequencing role in attention and language
26
What are the effects of cerebellar damage (associated with dysarthria)?
``` hypotonia intention tremor dysmetria decomposition of movement ataxia ```
27
What are the functions of the basal ganglia?
integrator of motor information for complex movements postural support needed for skilled, voluntary movements regulates amplitude, velocity, initiation of movements
28
What are the effects of damage of the BG?
``` Hypokinesia (PD) muscle rigidity resting tremor slowness of movement (bradykinesia) reduced movement amplitude (hypokinesia) ?? ```
29
What is the function of the thalamus?
relay all sensory info but smell | relay motor signals from BG and cerebellum
30
What is voluntary motor control?
Ability to perform an action on command | depends on control signals originating in motor cortical areas that are sent to PNS
31
What is the pyramidal system?
direct pathways -- skilled movement, input to alpha motor neurons
32
What is the extrapyramidal system?
indirect pathways -- support skilled movement, input to gamma motor neurons
33
What are the symptoms of UMN damage?
spasticity weakness or paresis (limited atrophy) hyper-reflexia
34
What are the symptoms of LMN damage?
``` hypotonicity atrophy hyporeflexia weakness muscle fasciculations in acute state ```
35
What is unusual about CN VII in terms of innervation?
upper face receives bilateral UMN/corticobulbar tract input | lower face muscles receive contralateral UMN input
36
What is unusual about the innervation of CN XII?
receives primarily contralateral corticobulbar input
37
What are the parts of the pyramidal system?
corticobulbar, corticospinal, corticopontine tracts
38
What are the parts of the extrapyramidal system?
rubrospinal, reticulospinal, olivospinal, vestibulo spinal tracts
39
What is an AMR? SMR?
both part of DDK AMR: repetition of single syllable SMR: repetition of syllable sequence
40
is AMR or SMR better in people with AOS?
AMR
41
Can we use DDK rate to make inferences about speed or regularity of tongue movement for connected speech?
NO
42
What are impairments of respiratory-laryngeal integrity that could be present in dysarthria?
decreased respiratory support decreased respiratory-phonatory coordination/control reduced phonatory function (hypoadduction, hyperadduction)
43
What are some indications of impairments of respiratory-laryngeal integrity?
``` tired/fatigued when talking and at rest run out of breath when talking have to push words out (effortful talking) poor loudness control weak cough ```
44
What are some ways of evaluating respiratory-laryngeal functions?
straw thing cough grunt/glottal coup