Oxygenation Flashcards
Passive process of breathing
Regulated by O2, CO2, and pH of blood
When CO2 increases [hypercarbia], body knows to increase rate and depth of breathing
Ventilation
movement gas in and out of lungs
Diffusion
oxygen and carbon dioxide exchange (alveoli & red blood cells)
Perfusion
distribution of red blood cells
Increased rate depth
increased ventilator effort, removes co2, (hypercarbia)
Chronic lung diseases
respond to hypoxemia instead of hypercarbia (receptors in carotid arteries and margarita
Purpose of lungs/alveoli is to promote
GAS EXCHANGE
GAS EXCHANGE
Occurs at the alveolar capillary membrane
capillary membrane
membrane thickness affects pulmonary edema, exudate, infiltrates effusions –> slow diffusion
Lung volumes
age gender and height
Tidal volume
amount of air exhaled following normal inspiration
Health status, activity, pregnancy, exercise, obesity, obstructive/restrictive lung diseases all impact TV
Alveoli function
expand during inhalation, taking in oxygen, and shrink during exhalation, expelling carbon dioxide
Ventilation = Respirations
12-20 breaths per minute
Rate/depth/rhythm
RR above 27 linked with increased risk of cardiac arrest
Age- kids, babies breathe much faster
Males & children use more abdominal muscles. Women thoracic muscles
Pain- shallow, increased, may split chest wall
Anxiety- shallow increased
Medications- narcotics, anxiolytics, amphetamines, cocaine
Illnesses
Hemoglobin function– less hemoglobin, altitudes lowers amount, abnormal cell function (think sickle cell), anemia– loss RBCS to carry
Exercise
Breath Sounds -
Expected (Normal) - Bronchial, Bronchovesicular, Vesicular
Adventitious (Abnormal) - Crackles/rales- fine to coarse bubbly sounds, associated with air passing through fluid or collapsed small airways
Wheezes- high pitched whistling, narrow obstructed airways
Rhonchi- loud low pitched rumbling, fluid or mucus in airways, can resolve with coughing
Stridor- choking, children
Pleural friction rub
Hyperpnea
Respirations are labored, increased in depth, and increased in rate (greater than 20 breaths/min) (occurs normally during exercise).
Hyperventilation
Rate and depth of respirations increase. Hypocarbia sometimes occurs.
Hypoventilation
Respiratory rate is abnormally low, and depth of ventilation is depressed. Hypercarbia sometimes occurs.
Diffusion/perfusion
oxygen saturation
SpO2
95% to 100%
Interference with light transmission –> patient motion, jaundice, intravascular dyes, dark nail polish
Interference with arterial pulsations –> PVD, hypothermia, pharmacologic vasoconstrictors, decreased cardiac output, edema, tight probes
Also affected by the things that affect respirations
When patient shallow and tachypnea, pulse ox can decrease
Inspiration and expiration
Expiration passive process
To be done well- need elastic recoil of lung tissue, surfactant helps keep surface tension of alveoli and keep them open
Compliance- ability of lungs to distend/expand, relies on intrathoracic pressure changes
Airway resistance- bronchoconstriction
factors affecting oxygenation
Decreased oxygen-carrying capacity
Hemoglobin levels, carbon monoxide
Hypovolemia
Decreased inspired oxygen concentration
Altitude, hypoventilation increased metabolic demand
Chest wall movement
Pregnancy, obesity, musculoskeletal diseases, trauma, neuromuscular diseases, central nervous system (CNS) alterations
factors affecting oxygenation
Morbidly obese- reduced lung volumes, heavy lower throax abdomen cant lay down flat or in recumbent postion
Spinal abnormalities
Flail chest– rib fractures= instability; abdominal surgeries!, or any surgery
Guillain barre, myasthenia gravis, ALS
C3-C5 results in paralysis of phrenic nerve= phrenic nerve control diaphragm, below C5– watch out for intercostal nerve damage which doesn’t allow for accessory muscle help and prevents anterposterior chest expansion
Damage to medulla oblongata neural regulation of respiration, abnormal breathing patterns develop
Alterations is respiratory functions
Goal of ventilation= normal arterial carbon dioxide tension and normal arterial oxygenation tension
Labs:
PaO2= 80-100
PaCO2= 35-45
Oxygenation saturation (SpO2) = greater than 95%
EtCo2= 35-45
Hypoventilation
Inadequate alveolar ventilation to meet demand
Not enough oxygen, and/or too much carbon dioxide
Causes:
Medications, alveolar collapse=atelectasis (lung diseases)
S/S:
Mental status changes, dysrhythmias
Can lead to cardiac arrest, convulsions, unconsciousness, death