pain diagnoses Flashcards
(33 cards)
pulpitis
signs and symptoms
treatment
short sharp pain (seconds)
triggered by stimuli
analgesics relieve pain
no TTP
give LA, provide temporary dressing (GIC) + definitive restoration later
pt presents with short sharp pain, lasts a few seconds. triggered by cold drinks. analgesics relieve pain and there is no TTP. what is the diagnosis?
reversible pulpitis
irreversible pulpitis
signs and symptoms
management
pain of long duration (hours)
dull and throbbing ache, pulsatile
spontaneous and may disturb sleep
analgesics don’t relieve pain effectively
may be worsened by heat or lying down
in later stages, cold might actually relieve symptoms
may be TTP
exaggerated or reduced response to EPT
*pain remains after removal of the stimulus
localisation difficult initially but as inflammation spreads to periapical tissues it will become more sensitive to pressure
management:
XLA or RCT
access and pulp extirpation
if pt experiences pain after goof anaesthesia, access and dress it with a corticosteroid + antibiotic paste, e.g. ledermix and seal with cotton wool and gic
contraindicated in under 12 due to tetracycline component can stain teeth
odontopaste alternative as doesn’t contain tetracycline
don’t prescribe antibiotics
what is a characteristic feature of pulpal pain ?
unable to localise the affected tooth but the pain does not cross the midline as the pulp doesn’t contain any proprioceptive nerve endings
how to come to a pulpal diagnosis
vitality tests help to some extent but
more defined by patient symptoms and history
why should you be careful when sensibility testing and using these results?
these test the nerve supply- not the blood supply
blood supply is the indicator of vitality
reversible pulpitis
radiographic signs
primary/secondary caries
no PA pathology
irreversible pulpitis
meaning
severe inflammation of the pulp (the nerve and blood vessels inside a tooth) from which the pulp is unlikely to recover
will eventually progress to necrosis
irreversible pulpitis
radiographic signs
deep caries/large restoration/crown
may see early PA changes, e.g. widening of the PDL
cracked tooth syndrome
signs and symptoms
special tests
management
sharp pain on biting- clasically on release of bite
short duration
special tests: transillumination and tooth sleuth detects pain on release on biting
positive response to vitality testing
may be associated with bruxism
tx: requires coronal protection: onlay/crown
RCT may be required if crack extends into pulp
XLA as last resort
placement of orthodontic band around tooth can relieve symptoms and help with diagnosis
characteristic patient sign of periapical periodontitis
PDL contains proprioceptive nerve endings, pt can identify the tooth causing pain + is localised
pulp polyp
hyperplastic pulpitis
occurs as a result of proliferation of chronically inflamed young pulp tissue when exposed to the oral cavity
pulp calcification
results in eventual occlusion of the pulp chamber by either physiological secondary dentine or tertiary dentine which is laid down in response to environmental stimuli as reactionary or reparative dentine
reactionary vs reparative dentine
a response to a mild noxious stimulus whereas reparative dentine is deposited directly beneath the path of injured dentinal tubules as a response to strong noxious stimuli
pt presents with:
pain on biting
very tender to put teeth together
disturbs sleep
TTP
dull ache exacerbated by biting down
deep carious crowned tooth
well localised
what is your diagnosis?
acute symptomatic periapical periodontitis
periapical disease:
pathology
inflammation of the tissues surrounding the root caused by infection of the root canal system
endodontic biofilm is made primarily of gram negative bacteria. these organisms release virulence factors such as lipopolysaccharides inducing a host response which leads to inflammation, resorption of mineralised tissues surrounding the root,
acute apical periodontitis
signs and symptoms
management
pain on biting
very tender to put teeth together
disturbs sleep
TTP
dull ache exacerbated by biting down
deep carious crowned tooth
well localised
usually no response to vitality testing unless multi rooted tooth one canal may be vital
may be a granuloma or cyst: periapical radiolucency, widening of PDL or loss of lamina dura
tx: RCT/XLA
asymtomatic periapical periodotnitis
(chronic apical periodontitis)
does not give any positive signs of disease following examination apart from evidence of periapical inflammation
acute periapical abscess
pathology
inflammatory response to microorgansims or their irritants have leached out into the periapical tissues
- initial infection and inflammation of wbcs at site of infection
- pulpal necrosis is a breeding ground for bacteira
- infection spreads to periapical area and root apex
- The inflammatory process in the periapical tissues causes cell death (necrosis) and a liquefaction process where dead tissue is broken down and converted into pus
- The accumulated pus, along with bacteria, debris, and inflammatory cells, forms an abscess, which is a localised collection of pus within a tissue.
acute periapical periodontitis:
pathology
chronic inflammation at the tip of a tooth root (periapical region) due to bacterial infection of the root canal system. This inflammation, a form of periapical periodontitis, can lead to liquefaction necrosis and eventually the formation of a granuloma. The process involves an initial inflammatory response, where cells like PMNs (polymorphonuclear leukocytes) infiltrate the area and cause tissue breakdown (liquefaction necrosis). If the inflammation persists, the body attempts to wall off the infected area, leading to the formation of a granuloma, which is a mass of inflammatory cells and surrounding fibrous tissue.
acute apical abscess
signs and symptoms
radiographic signs
acute periapical abscess
management
when a patient who is immunocompromised and has a dental infection, what is important to consider?
infection may cause systemic sepsis
signs of sepsis
altered mental state
rapid breathing and heart rate
failure to pass urine
low systolic blood pressure
non-blanching rash
cyanosis of the skin
temp of less than 36 degrees