Pain, neuromuscular control, and biofeedback Flashcards
(36 cards)
What are the categories of pain?
• Acute pain vs Chronic pain – Don’t forget to look for the yellow flags • Nociceptive – Somatic or visceral • Neuropathic – Peripheral or Central • Psychogenic • Carcinogenic
What is Kubler-Ross’s death and dying model?
– 5 stages of response to terminal illness
– Denial, anger, bargaining, depression,
acceptance
T/F
Kuber-Ross’s death and dying model is applicable to athletic injury
False
What are cognitive-appraisal models of injury
• Models state that response to injury
depends on understanding of the injury
• Response to injury is not neatly divided
into stages in particular order
• Response to injury can be influenced by
the actions and message of the doctor!!!
T/F
When assessing a patient you need to be blunt with what you say to the patient, telling them exactly how it is regardless of what the patient might insinuate. Injuries are just physical anyways.
False
Patients often hear different things that what you say, must be aware of this.
Must also be aware of the psychological/emotional aspect to injuries.
Examples of telling patients of their injuries the wrong way:
“Your meniscus is shot”
“You blew out your knee”
“Severe degeneration with disc disease”
“You have the bones of 60 year old man”`
What is FAKTR?
Functional and kinetic treatment with rehab
What are some of the primary characteristics of A-beta fibers?
Touch, pressure, hair deflection
Myelinated
Large diameter
Low threshold
What are the basic characteristics of A-delta fibers
– Warm and cold receptors, hair follicles, free
nerve endings
• Touch, pressure, temperature and pain
• Free nerve endings respond to noxious stimuli
such as pricking, pinching and crushing
– Myelinated
– Smaller diameter than A-beta (1-6 micrometers)
» Slower conduction velocity
What are the basic characteristics of C fibers?
– Pain, touch, pressure, temperature
• Include efferent postganglionic fibers of
sympathetic nervous system, mechanoreceptors,
nociceptors and thermoreceptors
• Smallest peripheral nerves associated with pain
– Unmeylinated
– Small diameter (less then one micrometer)
» Slow conduction velocit
What are the most important parts of the thalamus for pain reception?
VPL (ascending fibers from the head synapse) and VPM (fibers from the head and face synapse)
After VPL/VPM where does the pain signal go?
Somatosensory cortex then limbic system
What is modulation phase?
• Any activity after the cortex has received input
• Have an excitatory or inhibitory role on new
impulses
• Hypothalamus
In peripheral pain modulation what does ice do?
– Decrease the effects of chemical mediators
– Decrease speed of pain transmission
What is gate theory?
– Non-painful stimulus can block the transmission of
noxious stimulus
– Substantia Gelatinosa in dorsal horn of spinal cord
acts as a “switch operator”.
– Interneuron that utilizes enkephalin is present in
substantia gelatinosa
– Inhibits pain transmission within the dorsal horn
What is motor TENS?
– Low frequency, high intensity stimulation of
peripheral nerves
Causes activation of Reticular Formation and
pituitary gland
DEOS
Descending endogenous opiate system
What are the components of noxious pain modulation?
– Electrical stimulation of C fibers in the injury
area (Noxious TENS)
– Activates the Periaqueductal Gray (PAG) and
the Raphe nucleus
– Serotonin neurons in the dorsal horn inhibit
the second order neuron either directly or
through an interneuron
– Also with ice stimulation of C fibers during
burning and aching sensation
What is nerve block pain modulation?
• When stimulation encroaches on the
refractory period of the sensory nerve and
causes inhibition
What is nerve block pain modulation called and what are it’s results?
Called Wedenski’s inhibition or action
potential failure and results in anesthesia
between the electrodes
What 2 EPA’s are best at achieving nerve block pain modulation?
Russian current, Interferential current
What is EIH
Exercise induced hypalgesia. Decreased pain sensation during activity because of the activation of DEOS and catecholamines (must consider this if treating an athlete who just came off the field, may not feel the extent of the injury ‘cause all jacked up).
Neuromuscular control consists of 3 components, all of which must be addressed in a rehabilitation plan, what are they?
– Consciously controlled muscle contraction (for loss of contraction ability due to injury)
– Reflex responses (e.g. ankle reflex to resist eversion)
– Complex movement patterns (injury leads to loss of these unconscious learned patterns)
• or complex functional movements
What does a complex movement pattern refer to?
Unconscious movement patterns that are “second nature” after much practice.