pain science Flashcards

1
Q

types of pain

A

acute, referred, and chronic

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2
Q

acute pain

A
  • symptom resulting form an injury or disease
  • less than 12 wks
  • well localized/defeined
  • example: c spine pain after mva
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3
Q

referred pain

A
  • pain that occurs at a side different from the source of disease or injury
  • convergence of cutaneous, visceral and skeletal muscle nociceptions at nerve root of the spinal cord
  • example - left shoulder, UE, JW , chest pain with MI
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4
Q

chronic ic pain

A
  • recurring pain existing for 3-6 months beyond normal time expected for healing
  • can follow acute pain
  • associated with structural and functional changes in CNS
  • example: fibromyalgia
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5
Q

purpose of acute pain

A

to warn of danger and to provoke a withdrawal response

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6
Q

purpose of persistent pain

A

no longer severs as a protective mechanism

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7
Q

how is pain communicated to the brain

A

1- sensory receptor
2 - first order neuron
3- second order neuron
4- thalamus
5 - third order neuron
6 - sensory cortex
output

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8
Q

Aa fibers characteristics

A
  • myelinated yes
  • large diameter (fast)
  • sensation - proprioception
  • receptor type (GTO, muscle spindle)
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9
Q

AB fibers

A
  • myelinated
    large fast
    proprioception, light touch, pressure, vibration, hair movement
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10
Q

A delta fibers

A
  • myelinated
  • small slow
  • extrem temp/nocicpetion
  • precise location , withdraw reflexes
  • free nerve endings
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11
Q

C fibers

A
  • no myelinated
  • smallest slow
  • extreme touch and temp/nociceptin
  • slow, second sensation, prevents further tissue damage
  • free nerve endings
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12
Q

facilitators

A

substance P, glutamate

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13
Q

inhibitory

A

enkephalins, GABA, B endorphin

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14
Q

three main pain control theories

A
  • gate control theory
  • descending pain control theory
  • endogenous opioid theory
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15
Q

Gate control theory

A

stimulation of AB fibers transmit information fastest, and therefore first, up tot the SC, therefore closing the gate and preventing nociceptive transmission from delta and C fibers

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16
Q

descending pain control theory

A

ADelta and C fiber transmission to the thalamus, activated efferent nerves to activated enkephalin interneurons located at dorsal horn, releasing serotonin, which suppresses substance P, inhibits transmission

17
Q

endogenous opioids theory

A

stimulation of Adelta and C fibers at hypothalamus causes release of endogenous opioids called endorphins

18
Q

is there pain center in the brain

A

no

19
Q

pain is an output of the brain 100% of the time (3)

A
  • tissue input
  • environment
  • other beliefs
20
Q

pain

A

is a multiple system output activated by an individuals specific pain neuromatrix

21
Q

nociceptive pain

A

is the normal pain from tissue damage

21
Q

neuropathic pain

A

is parian from nerve damage or dysfunction

22
Q

nociplastic pain

A

is pain that doesn’t have a clear physical cause and may be influenced by various factors

22
Q

periphery nerve level

A

ion channels creating more sensitive nerve fibers that fire more easily, so more frequent firing

23
Q

SC level

A

less interneurons present to inhibit, substance P increases, which can amplify transmission

24
Q

brain level

A