Pancreatitis Flashcards

(29 cards)

1
Q

Acute Pancreatitis

causes women and men?

A
  • gallbladder disease “cholelithisis” (WOMEN)

- chronic alcohol (MEN)

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2
Q

Acute Pancreatitis Abdominal pain signs

A
  • Radiates to back
  • Sudden onset
  • Deep, piercing, continuous, or steady
  • worse when eats
  • Starts when recumbent
  • Not relieved with vomiting
  • Generally, is unrelieved by antacids

Abdominal tenderness with muscle guarding

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3
Q

Other signs of acute pancreatitis

A
Cyanosis 🔵
Dyspnea
N/V 🤮
Low grade fever 🥵 
Leukocytosis
Hypotension/ tachycardia 
Jaundiced 🟡
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4
Q

Pseudocyst

What is it?
Signs?
Treatment?

A
  • accumulation of fluid, pancreatic enzymes, debris and exudate surrounded by wall
  • SIGNS: abdominal pain, palpable mass, N/V, anorexia
  • detected w/ image
  • treat w/ surgical percutaneous or endoscopic drain
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5
Q

Pancreatic abscess

What is it and signs?

A
  • infected pseudocyst
  • result from extensive necrosis
  • may rupture

SIGNS: upper abd pain, palpable mass, high fever 🤒 , leukocytes

Need print surgical drainage

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6
Q

Systemic complications

A
🫁
•Pleural effusion, pulmonary embolism
•Atelectasis
•Pneumonia
•ARDS
❤️/🩸
•Hypotension
•Thrombi, DIC
•Hypocalcemia: tetany
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7
Q

Laboratory tests

A

** Serum amylase and lipase level- elevated within 24 hours of onset of symptoms

  • Liver enzymes-elevated
  • Triglycerides-elevated
  • Glucose level-can be elevated
  • Bilirubin level-can be elevated
  • Serum calcium level-decreased
  • WBC-can be elevated
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8
Q

Diagnostic Studies

A

•Abdominal ultrasound
•X-ray
CT scan is the best test for pancreatitis and related complications
•Endoscopic retrograde cholangiopancreatography (ERCP)*

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9
Q

Acute pancreatitis GOALS include

A
  • Relief of pain
  • Prevention or alleviation of shock
  • Decrease pancreatic secretions
  • Correction of fluid/electrolyte imbalances
  • Prevention/treatment of infections
  • Removal of precipitating cause
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10
Q

Pain relief

A
  • IV Opioids (MSO4)
  • Antispasmodics like Bentyl
  • Carbonic anhydrase inhibitor (Acetazolamide)
  • PPIs (Prilosec)
  • NPO status
  • NGT
  • Position changes
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11
Q

Prevention or alleviation of shock

A
  • IVF and nutrient replacement
  • Blood volume replacements
  • Central Venous Pressure (CVP) Monitoring
  • Vasoactive drugs: Dopamine
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12
Q

Decrease pancreatic secretions

A

•Gut rest
- suppression of enzymes
- NPO status
- NGT
•Antiacids
•If unable to resume eating after treatment may need enteral feeds
•If unable to tolerate enteral feeds, then parental nutrition (TPN/IL)

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13
Q

Correction of fluid/electrolyte imbalances

A
  • Aggressive IVF; nutrient replacement
  • CVP
  • Blood volume replacements
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14
Q

Prevention/treatment of infections

A
  • Enteral feedings
  • Abx
  • Endoscopic gram stain and gx
  • Ultrasound/CT/physical exams for pseudocyst/abcess
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15
Q

Removal of precipitating cause

A

1) Stop ETOH
2) Remove gallstones
- ERCP plus endoscopic sphincterotomy
- Laparoscopic cholecystectomy

Drainage of necrotic fluid collection

•NOTE: no specific drug cures Pancreatitis; symptom management only

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16
Q

Nutritional Therapy

A
  • NPO status initially
  • Enteral versus parenteral nutrition (NG vs IV) prn
  • Small, frequent feedings when able
  • High-carbohydrate
  • NO alcohol
  • Supplemental fat-soluble vitamins
17
Q

Acute care

What to monitor ?

A

1) Monitoring
- Vitals (Hypotension, fever, tachypnea)
- response to IV fluids
- fluid and electrolyte balance
- serum glucose

2) Assess respiratory function
3) Observation for paralytic ileus, renal failure, mental changes

18
Q

Dietary teaching

A

•Low-fat diet
•high-carbohydrate
-less stimulating to pancreas
•No crash or binge diets as they precipitate attacks

19
Q

Chronic Pancreatitis causes

A
  • continuous prolonged inflammatory and fibrous process

- alcohol, gallstones, tumor, pseudocyst, trauma

20
Q

Chronic Pancreatitis

A
  • Inflammatory disorder with progressive destruction of the pancreas.
  • Cells are replaced with fibrous tissue with repeated attacks.
  • Results in obstruction of the pancreatic and common bile duct and duodenum.
  • Atrophy of the epithelium of the ducts, inflammation and destruction of the secreting cells of the pancreas.
  • Excessive and prolonged consumption of alcohol major cause
  • Long-term consumption of alcohol damages the cells of the pancreas, causes hypersecretion of protein in pancreatic secretions which results in protein plugs and calculi in the pancreatic ducts.
21
Q

Chronic OBSTRUCTIVE pancreatitis causes

A

1) Inflammation of sphincter of Oddi

2) Cancer of ampulla of Vater, duodenum, or pancreas

22
Q

Chronic NON-OBSTRUCTIVE pancreatitis

What is the most common cause ?

A
  • Inflammation and sclerosis in head of pancreas and around duct
  • Most common cause is alcohol abuse
23
Q

Chronic signs

A
  • Abdominal pain
    • same area as Acute
    • heavy, gnawing feeling
    • more freq
- malabsorption/ weight loss
💩constipation
🟨mild jaundice/ dark urine
💩steatorehea- foul smelling fatty stool
- diabetes
24
Q

Chronic Pancreatitis labs

A
•Amylase and lipase may be slightly increased or normal
⬆️ bilirubin level
⬆️alkaline phosphatase level
****Mild leukocytosis
⬆️sedimentation rate
25
Chronic diagnostic studies
CT MRI ultrasound can show enlargement, ductal dilation, and pseudocysts
26
Chronic care What kind of thing does the patient need to take ?
1) pancreatic enezyme replacement - pancrelipase (take w meals) 2) bile salts to help fat soluble vit 3) insulin if diabetic 4) acid neutralizing agent for gastric activity 5) antidepressant to reduce neuropathic pain
27
Chronic surgery Indicated for obstruction or pseudocyst present
1) Endoscopic procedures: - Pancreatic drainage-to divert bile flow or relive obstruction - ERCP with sphincterotomy and/or stent placement
28
Chroincic pancreatitis other signs
* Decreased or absent bowel sound * 🟩🟨skin discoloration * Grey Turner’s spots or sign-bluish flank discoloration * Cullen’s sign-bluish periumbilical discoloration * Shock-may occur from hemorrhage into the pancreas
29
Pain assessment and management
* Opioids * Position changes * Frequent oral/nasal care esp. with NGT * Proper administration of antacids to neutralize gastric secretions * Hold oral feeds