Parasites (Yr3) Flashcards

1
Q

what are nematodes also known as?

A

roundworms

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2
Q

what are platyhelminths also known as?

A

cestodes and trematodes

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3
Q

what are trematodes also known as?

A

fluke

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4
Q

what are cestodes also known as?

A

tapeworm

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5
Q

what is the feeding stage of protozoa called?

A

trophozoite

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6
Q

what does Ostertagia ostertagi cause in cattle?

A

PGE

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7
Q

where is Ostertagia ostertagi found in cattle?

A

abomasum

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8
Q

what type of lifecycle does Ostertagia ostertagi have?

A

typical trichostrongyle (trichostrongyle eggs)

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9
Q

what happens to Ostertagia ostertagi once L3 is ingested?

A

reaches abomasum and burrows into gastric gland where it develops to L5 and emerges into the lumen and mature to lay eggs

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10
Q

what is the pathogenesis of Ostertagia ostertagi?

A

L5 damages gastric glands (parietal cells replaced by epithelial) leading to loss of acid production
this reduces the bacteriostatic effect and less pepsinogen is converted to pepsin making the mucosa more permeable

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11
Q

what animals is type I ostertagiosis seen in?

A

dairy replacement calves towards the end of the first grazing season (July-September), most of the group will be effected

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12
Q

what causes type I ostertagiosis?

A

ingestion of large numbers of L3 in July (green watery diarrhoea)

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13
Q

what causes type II ostertagiosis?

A

mass emergence from hypobiosed larvae from the gastric glands in late winter/early spring

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14
Q

when is type II ostertagiosis seen?

A

late winter/early spring in yearling calves (mass emergence of hypobiosed larvae)

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15
Q

what is the main pathogen causing PGE in cattle?

A

Ostertagia ostertagi

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16
Q

what nematodes are found in the abomasum of cattle?

A

Haemonchus contortus
Ostertagia ostertagi
Trichostrongylus axei

17
Q

what nematodes are found in the small intestine of cattle?

A

Nematodirus spp.
Trichostrongylus spp.
Cooperia spp.

18
Q

what factors affect the development of the pre-parasitic stages of Ostertagia ostertagi?

A

temperature (>10°C)
humidity
dispersal from faecal pat (rainfall)

19
Q

what factors effect the survival of Ostertagia ostertagi L3?

A

ensheathed (wears L2 cuticle)
temperature (hates heat)
moisture (desiccation is lethal)
limited food reserve = limited survival

20
Q

what stage of the lifecycle undergoes hypobiosis?

A

L4 (in the gastric gland)

21
Q

where will the L3 causing type I ostertagiosis come from?

A

L3 overwintering on pasture used the previous year

22
Q

when does the amount of Ostertagia ostertagi eggs peak on pasture?

A

July - meaning outbreaks will occur around three weeks after this due to the pre-patent period

23
Q

why are beef calves less likely to get ostertagiosis?

A

grazed with immune mothers (autumn born calves may encounter it in the spring when turned out)

24
Q

what can be used for definitive diagnosis of Ostertagia ostertagi?

A

raised plasma pepsinogen levels
(clinical signs, grazing history, season)

25
Q

what is clean pasture?

A

pasture not grazed by cattle for last 12 months

26
Q

what is safe pasture?

A

pasture used previous year but safe by June

27
Q

what is the typical treatment strategy for Ostertagia ostertagi?

A

treat first season grazing calves 3 weeks post-turnout with ivermectin to prevent egg shedding
treat again 5 weeks later (due to ivermectins 2 week residual activity)
overwintered larvae should have died off by then so no more treatment required

28
Q

if using a strategic worming control strategy for Ostertagia ostertagi, when should you treat?

A

when FEC >200e/g

29
Q

do spring born suckler calves need treating for Ostertagia ostertagi?

A

mothers will pick up majority if worm burden when grazing but treat at housing to prevent type II

30
Q

does Dictyocaulus viviparous have an intermediate host?

A

no, it has a direct lifecycle

31
Q

what is the lifecycle of Dictyocaulus viviparous?

A

L1 coughed up, swallowed and excreted in faeces
L1 moults to L3 which is ingested
lympho-tracheal migration moulting to L4 in lymph nodes (penetration phase)
L4 moults to L5 in lungs and migrates to trachea/bronchus (pre-patent phase)
adults lay eggs that hatch and to L1 (patent phase)
adults are expelled and protective immunity acquired (post-patent phase)

32
Q

what are the most pathogenic stages of Dictyocaulus viviparous?

A

pre-patent and patent

33
Q

what pathology can be seen in the post-patent phase?

A

lung tissue can epithelialise and scar which may never recover

34
Q

what factors effect the pre-parasitic stages of Dictyocaulus viviparous development?

A

temperature
moisture
dispersal (pilobolus fungi)

35
Q

can Dictyocaulus viviparous overwinter?

A

yes

36
Q

when is disease caused by Dictyocaulus viviparous seen?

A

August/September in dairy replacement calves

37
Q

how can Dictyocaulus viviparous be diagnosed?

A

baermann (L1 in faeces)
antibody ELISA
clinical signs/season

38
Q

can animals become immune to Dictyocaulus viviparous?

A

no, immunity is short lived

39
Q

what is the vaccination protocol for Dictyocaulus viviparous?

A

two doses, four weeks apart before turnout