Passmed - Resp Flashcards
(35 cards)
Causes of upper zone fibrosis?
C - Coal worker’s pneumoconiosis
H - Histiocytosis/ hypersensitivity pneumonitis
A - Ankylosing spondylitis
R - Radiation
T - Tuberculosis
S - Silicosis/sarcoidosis
Causes of lower zone fibrosis?
idiopathic pulmonary fibrosis
most connective tissue disorders (except ankylosing spondylitis) e.g. SLE
drug-induced: amiodarone, bleomycin, methotrexate
asbestosis
CO2 retention in COPD
Bad part of the lung (emphysema, COPD, alveoli broken down) -> lung constricts blood flow to that part of lung and perfused other part (ventilation-perfusion mismatch) = physiological shunt (blood diverted to good part of lung).
If put on high flow oxygen, mismatch is off and oxygen goes to bad parts of lungs so blood goes to bad parts of lung so is physiologically bad to give O2
ROME ABG
Respiratory = Opposite
low pH + high PaCO2 i.e. acidosis, or
high pH + low PaCO2 i.e. alkalosis
Metabolic = Equal
low pH + low bicarbonate i.e. acidosis, or
high pH + high bicarbonate i.e. akalosis
Moderate asthma?
PEFR 50-75% best or predicted
Speech normal
RR < 25 / min
Pulse < 110 bpm
Severe asthma?
PEFR 33 - 50% best or predicted
Can’t complete sentences
RR > 25/min
Pulse > 110 bpm
Life-threatening asthma?
PEFR < 33% best or predicted
Oxygen sats < 92%
Silent chest, cyanosis or feeble respiratory effort
Bradycardia, dysrhythmia or hypotension
Exhaustion, confusion or coma
Obstructive sleep apnoea management
Following weight loss, CPAP is the first-line treatment for moderate/severe obstructive sleep apnoea
Obstructive sleep apnoea predisposing factors
obesity
macroglossia: acromegaly, hypothyroidism, amyloidosis
large tonsils
Marfan’s syndrome
Obstructive sleep apnoea sleep assessment and diagnosis?
Assessment of sleepiness
Epworth Sleepiness Scale - questionnaire completed by patient +/- partner
Multiple Sleep Latency Test (MSLT) - measures the time to fall asleep in a dark room (using EEG criteria)
Diagnostic tests
sleep studies (polysomnography) - from monitoring of pulse oximetry at night to full polysomnography, measure EEG, respiratory airflow, thoraco-abdominal movement, snoring and pulse oximetry
first-line pharmacological treatment of COPD
A SABA or SAMA
Risk factors for the development of aspiration pneumonia include:
Poor dental hygiene
Swallowing difficulties
Prolonged hospitalization or surgical procedures
Impaired consciousness
Impaired mucociliary clearance
The features of acute severe asthma
PEFR 33-50% best or predicted, inability to complete full sentences, RR >25/min and pulse >110 bpm
Tracheal deviation, resonant to percussion and absent breath sounds in the context of respiratory distress and shock
think a tension pneumothorax
most common cause of occupational asthma
Isocyanates
Hypercalcaemia + bilateral hilar lymphadenopathy → ?
sarcoidosis
Tuberculosis typically causes x zone pulmonary fibrosis
upper
x is recommended for 5 days in acute exacerbations of COPD
Oral prednisolone
Following weight loss, x is the first-line treatment for moderate/severe obstructive sleep apnoea
CPAP
Pulmonary hypertension is a cause of a (heart sound)
loud S2 (due to a loud P2)
Patients diagnosed with pneumonia who have COPD should be given x even if no evidence of the COPD being exacerbated
corticosteroids
Symptom control in non-CF bronchiectasis -
inspiratory muscle training + postural drainage
Persistent productive cough +/- haemoptysis in a young person with a history of respiratory problems →
?bronchiectasis
x prophylaxis is recommended in COPD patients who meet certain criteria and who continue to have exacerbations
Azithromycin