perio exam 2 Flashcards
(48 cards)
is bop sensitive?
no, b/c trauma could also cause bleeding. it is specific-if no bleeding then there is a great chance pt is healthy
gingivitis ( dental pq only) …
can have local contributing factors
ging w/ systemic disease modification
endocrine association or blood discrasias (ex. leukemia associated)
(which drug from pharm causes that?)
ging w/ systemic disease modification:
endocrine association ex’s:
puberty associated, menstrual cycle, pregnancy, diabetes
ging modified by meds
ging enlargements, oral contraceptives
ging from malnutrition
vit c deficiency or lack of vit A, B2, and B12 complex
summary slide: pq should be there for ging
unless pregnant or medicated
ging around implants
peri-mucositis
6 characteristics to all ging diseases:
- signs/symptoms limited to ging
- precursor-pq
- clinical signs of infmm
- no att loss or on a stable but reduced periodontium
- reversibility of the disease by removing the etiology
- possible role as a precursor of att loss
11 characterists of pq induced ging
- pq @ margin
- margin-start of disease
change in:
3.color 4. contour 5. sucular temp - incresed gingival exudate
- absence of att loss
- absence of bone loss/9.bone loss
9.histological changes
10reversible w/pq removal - bop
color: normal ging v. inflammed v. severly infmm
coral pink, tissue’s vascularity and overlying eli intact v. red and not intact v. red and CYANOTIC; vascular proflieration, decreased keratin and venous stasis
where do the color changes start?
g. margin and interdental papilla then spread to the att g
g bleeding: w/ inc infmm…
chronic/recurrent bleeding
when is there spontaneous bleeding?
inc infmm.. engorged caps, thinning/ulceration of the sulcular epi.
chronic/recurrent bleeding from trauma
spontaneous bleeding=acute/severe cases-maybe related to systemic disease
healthy adjectives
health=firm/resilliant w/dull surface texture (stippling maybe) and scalloped ging fills interdental spaces (papilla there)
what can lead to leathery, firm ging consistency?
chronic g can indue fibrosis and eli proliferation
infmm v. severe infmm descriptions
infmm=extracellular fl and exudate, degeneration of ct & epi, engorged ct/thin epi, and knife edged ging w/loose margins
sometimes clefts (Stillman’s) or festoons (McCalls) may develop
infmm w/ exudate=smooth and shiney,
infmm w/fibrotic changes=nodular and firm
swollen, soft, friable
severe: sloughing w/grayish flake-like debris (necrosis)
mild gingivitis
PD-? mm Plaque ?% BOP ? %
PD-1.5-3 mm Plaque 50% BOP 15 %
moderate gingivitis
PD-? mm Plaque ?% BOP ? %
PD-3-4 mm Plaque 50% BOP 30%
severe gingivitis
PD-? mm Plaque ?% BOP ? %
PD-pseudopockets to 5 mm Plaque 50% BOP 80 %
(no att loss still!)
all had same pq comp-BOP %’s changed
ging enlargement-3 descriptions
- chronic infmm response characteristic w/exudate and proliferative features
- clinically deep red lesions w/soft, friable, smooth, shiny surface and bleeding tendency
- relatively firm resilient and pink lesions w/ greater fibrotic component, abundant fibroblasts and collagen fibers
(3 said “also”, I hope as in “could be this instead” vs. 2 and 3 together….)
What is/are the primary etiological factor(s) for ging?
bacterial plaque
What is/are the secondary etiological factor(s) for ging?
local factors such as 1. calculus 2.marginal deficiencies 3.malocclusion 4.tooth/root anomalies (enamel pearls/cemental tears) 5. BW 6. roots fractures 7. cervical toot reabs
BW=?
histo dimensions of the dento-gingivl junction health .69-sulcus .97=epi attachment 1.07-ct total 2.73
bw = a min of ?mm coronal to alveolar crest?
3 mm
