Phys Dx Heart Flashcards
(49 cards)
S1
During Systole, ventricles contract and aortic and pulmonic valves open and MITRAL AND TRICUSPID CLOSE
S2
During Diastole, ventricles relax and blood fills as mitral and tricuspid open. PULMONIC AND AORTIC CLOSE.
S3
Sound created during early diastole AS BLOOD RAPIDLY FILLS THE VENTRICLE. RAPID DISTENSION OF THE WALL CAUSES VIBRATION
S4
Sound created during late diastole AS ATRIA CONTRACTS AND FORCES LAST BIT OF BLOOD TO VENTRICLE. VIBRATION OF VALVES, PAPILLARY MUSCLES, AND VENTRICULAR WALL.
Systolic Murmur
Sound when blood rushes through a narrow leaky valve or wall during systole
Diastolic Murmur
sound blood makes when rushing through a narrow, leaky valve or wall during diastole
Thrill
What heart conditions cause it?
Palpable Murmur - vibrations caused by a loud murmur
- can be caused by vigorous flow through narrowed opening - AS, VSD
Lift/Heave
Cardiac Impulse feels more vigorous than normal and can be felt through chest wall
- ventricular hypertrophy or hyperdynamic ventricular activity
Diaphragm vs. Bell
Diaphragm Best for high pitched sounds S1 and S2
Bell best for low pitched sounds S3 and S4
Auscultation Area Aortic
2nd ICS at RSB
Auscultation Area Pulmonic
2nd ICS at LSB
Auscultation Area 2nd Pulmonic
3rd ICS at LSB
Auscultation Area Tricuspid
4th and 5th ICS, LSB
Auscultation Area Mitral
5th ICS, MCL
Bisferiens Pulse
A bisferiens pulse is an increased arterial pulse with a double systolic peak. Causes include pure aortic regurgitation, combined aortic stenosis and regurgitation, and, though less commonly palpable, hypertrophic cardiomyopathy.
Pulsus Alternans
The pulse alternates in amplitude from beat to beat even though the rhythm is basically regular (and must be for you to make this judgment). When the difference between stronger and weaker beats is slight, it can be detected only by sphygmomanometry. Pulsus alternans indicates left ventricular failure and is usually accompanied by a left-sided S3.
Paradoxical Pulse
Rising pressure with expiration and Systolic pressure decreases during inspiration
common with asthma and COPD
Abnormal S1
Normally: S1>S2 at apex and S1<S2 at base:
S1 softer: weaker contractions, emphysematous lungs
S1 louder: diseased AV valve or more forceful closure of AV valve - mitral stenosis, fever, tachycardia
Splitting of S2: Physiological vs. Pathological vs. Fixed Splitting
Best heard over Pulmonic Area:
Physiological Splitting of S2: during inspiration, Aortic closure (A2) then Pulmonic Closure (P2)
Pathologic Splitting of S2: during expiration suggestive of Heart disease - can be early aortic closure (mitral regurg) or late pulmonic closure (pulmonic valve stenosis, RBBB)
Fixed Splitting - doesn’t vary with inspiration or expiration (ASD, RV failure)
Paradoxical Splitting - Expiration but not during inspiration and A2 after P2 (LBBB)
Ventricular Gallup Rhythm
S1+S2+S3
S3 Gallop
When is it physiologic and when is it pathologic?
Physiologic in kids, young adults, and in pregnancy
Pathogic S3: over 40, heart failure, anemia, volume overload, decreased myocardioal contractility
Atrial Gallop Rhythm
S1+S2+S4 (S4 aka atrial kick or atrial gallop)
S4
when is it pathological?
Uncommon in healthy adults but normal in athletes
Patholig due to resistance in filling ventricles, stiffness of the heart muscle
- HBP, CAD, AS, cardiomyopathy
Cardiac Conduction
electrical current that stimulates myocardial contractions
SA node > AV Node > Atrial Septum > Bundle of His> Purkinjie Fibers in the ventricles