Physical Agents Flashcards
(98 cards)
Thermal physical agents:
- ultrasound
- hot pack
- ice pack
- diathermy
Mechanical physical agents:
- mechanical traction
- elastic bandage/stockings
- whirlpool
- ultrasound
electromagnetic physical agents
- ultraviolet
- laser
- TENs
absolute contraindications of general physical agent use:
- pregnancy
- malignancy
- pacemaker
- impaired sensation
- impaired mentation
general effects of physical agents:
- changes rates of circulation (chemical reactions)
- alter fluid flow
- alter cell function
- increase membrane permeability and transport
- helps move more quickly through inflammation stage
- increases enzyme activity rate
- promotes collagen deposition
purpose of inflammation:
- dilute/dissolve cells or agents
- can be harmful if directed at wrong tissue or overly exuberant
cardinal signs of inflammation
- heat (increased vascularity)
- redness (increased vascularity)
- swelling (blockage of lymphatic drainage)
- pain (pressure, chemical irritation, pain sensitive structures)
- loss of function (pain and swelling)
vascular response to inflammation
- transient constriction (NE-injured)
- non-injured vasodilation
- increased permeability (edema)
- changed physical attraction between vessel walls
- in vasoconstriction and increased permeability: slowing of flow and increased viscosity
- extravasation
- transudate (serum once outside the cell)
- exudate
- pus
cellular reaction to inflammation:
- platelets: (RBC losts outside in exudate, severe injury)
- neutrophils (WBC activated for chemotaxis and phagocytosis, 0-24 hrs)
- monocytes/macrophages (most important; vessel to interstitial)
- lymphocytes (immune response via antibodies in chronic inflammation)
- basophils (release histamine)
parts of proliferation phase
-epithelialization
-wound contraction
-neovascularization
fibroplasia
maturation phase
-balance of collagen synthesis and lysis (collagen conversion)
stages of scar formation
Stage 1: deposition of type III collagen; scar prone to rupture
Stage II: increased fibroplasias; covnersion type III to type I collagen; wond contraction begins, remodeling at peak
Stage III: scar consolidation; remodeling decreases
Stage IV: remodeling beginning to cease; least responsive to intervention
what stage of scar formation has the greatest responsiveness to intervention?
Stage II
old definition of pain:
unpleasant sensation to warn of tissue injury and with an emotional response
new definition of pain:
: conscious correlate of the implicit perception that tissue is in danger, where the quality and intensity of the pain depend on the degree of perceived threat
a-delta nerve fibers:
- fastest conducting, first component of pain, leads to reflexive withdrawal
- myelinated
- short duration
- sense mechanical and thermal sensations
- sharp, pricking, temp. changes
c fibers:
(polymodal nociceptors) slow conducting; second component of pain -non myelinated -long duration -dull, ache, burning sensation -not well tolerated
what type is the majority of nociceptors?
c fibers
80%
primary afferent neurons:
a delta fibers
c fibers
a-beta fibers
a-beta fibers
transmit non painful sensations related to vibration, stretching, mechanoreception, abnormal pain
- skin, bones, joints
- large myelinated axons
- conduct faster than a delta or c fibers
opiopeptin system of pain modulation with external opioids:
\:inhibitory action by binding to opioid receptors in the brain and peripherally -presynaptic: prevent influx of Ca -postsynaptic: activate outflux of K -activates endogenous opioids -
naloxone:
antagonist to opiates
-higher affinity for opioid receptors
endogenous opiates
- stimulated by noxious, stressful sensory inputs
- bind to same opioid receptors
what modality stimulates edogenous opiates:
- e stim, burst or low rate modes
- acupuncture and painful stimulation