pneumonia Flashcards

(37 cards)

1
Q

pneumonia

A

any type of infection of the lower respiratory system

cause: viral, fungal, protozoa, or parasitic

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2
Q

what does PNA cause the body?

A

inflammation of the lung tissues, alveolar air spaces filled with purulent fluid, inflammatory cells, fibrin

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3
Q

how is PNA transmitted

A

inhaled infectious droplets

droplet precautions –> mask, gloves, eye shield, gown

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4
Q

risk factors for PNA

A

male, winter time
age extremes (>80)
compromised immunity
underlying lung disease
alcoholism
altered LOC
impaired swallowing = aspiration
nursing home resident
hospitalization
influenza

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5
Q

CAP vs. HAP

A

community acquired vs hospital acquired

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6
Q

CAP

A

one of the most common reasons for hospitalizations
easier to treat

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7
Q

HAP

A

PNA developed within 48 hrs after admission
*worse outcomes than CAP - more deadly, contagious, strong
*ICU care

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8
Q

VAP and HCAP

A

ventilator associated pna
*associated with endotracheal intubation
*VAP bundle to prevent

healthcare associated pna

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9
Q

patho of PNA

A
  1. aspiration of oropharyngeal secretions [MOST COMMON] OR inhalation of droplets containing bacteria/pathogens

once in lower airway,
2. inflammatory rxn stimulated in lungs –> vasodilation & infection begins to spread into the respiratory tract and alveoli
3. goblet cells are stimulated and mucus is excrete –> mucus accumulates between the alveoli and capillaries (can’t have gas exchange)

  1. alveoli attempt to open and close against the purulent exudate, but most can’t –> gas exchange not ideal, difficult to breathe
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10
Q

failure of cough reflex

A

mucociliary defense mechanism
failure allows exudate to invade the alveoli

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11
Q

mucociliary defense mechanism is ineffective in ___________ because…

A

ineffective in smokers because impairs ciliary fxn
*smoking is a risk factor for all resp diseases

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12
Q

clinical manifestations of pna

A

usually happens after an URI
fever, chills, productive OR dry cough, malaise, pleural pain, dyspnea, hemoptysis (coughing up blood)

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13
Q

productive cough

A

bacterial
purulent, sputum may be green, rusty color, red currant jelly
usually gram negative in HAP

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14
Q

nonproductive/scanty cough

A

viral
often the cause of CAP

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15
Q

severe manifestations of PNA

A

tachypnea, signs of resp distress/failure

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16
Q

respiratory distress

A

maintain O2 only by increasing WOB

tachypnea, tachycardia, nasal flaring, pursed lips, stridor/wheezing, agitation, delayed cap refill, pale

17
Q

respiratory failure

A

can’t compensate for inadequate O2 despite extra respiratory effort and rate
circulatory and resp system collapse

RR >60
grunting, retractions, mottling, head bobbing, severe air hunger, bradycardia, hypotension

18
Q

respiratory attack

A

bradypnea, inefficient respirations, cyanosis/gray, no air movement

19
Q

how do you diagnose PNA

A

-s/s from physical assessment
-lung exam: dullness on percussion, inspiratory crackles, increased tactile fremitus, egophony (“e” to “a”)
-diagnostic tests (CXR, CBC, sputum C&S)

20
Q

diagnostic tests for pna

A

CXR: infiltrates
CBC: determines if bacterial (WBC increase/leukocytosis)
+ sputum for C&S: identifies specific bacteria and ATB that will kill it

21
Q

bacterial pna

A

gram positive: staphylococcus aureus, streptococcus pneumoniae
gram negative: pseudomonas auruginosa, aceintobacter, klebsiella pneumoniae

22
Q

staphylococcus aureus

A

gram positive bacteria, HAP

enters through bloodstream (IV) –> to lungs

common cause: MRSA

23
Q

streptococcus pneumoniae

A

CAP
pneumococcal pna
sputum usually brown or rusty color tinge

24
Q

gram negative organisms

A

gram - infections make you SICKER and more DIFFICULT TO TREAT

25
aspiration pna
aspirated material from GI tract stimulates inflammatory rxn *severity of inflammation depends on pH (more acidic = more inflammation)
26
who is at risk for aspiration pna?
NG tube, decreased LOC, decreased gag reflex, decreased gastric emptying
27
nursing considerations with aspiration pna
aspirate can be subtle or abrupt "silent killer" *dysphagia eval is crucial
28
viral pna
CAP virus alters pulmonary immune defense and makes the lungs vulnerable to another bacterial infection (secondary pna)
29
s/s of viral pna
fever, chills, dyspnea on exertion, cough
30
treatment of viral pna
mild, supportive care NO ATB - unless secondary infection generally improves in 2-3 weeks
31
atypical pneumonia's
pneumocystis carini mycoplasma legionella aspergillus
32
pneumocystis carini
r/t immune suppression yeast-like fungus
33
mycoplasma
"walking pna" mild; pt may complain of persistent cough, headache, earache properties of both bacterial and viral
34
legionella
gram neg spread through water systems --> air conditions, mists sprayed on produce, hot tubs
35
aspergillus
fungal pna released from walls of old buildings, reconstruction, dead leaves, compost affects lung tissues
36
PNA treatment
bacterial --> ATB viral --> symptoms, NO ATB *ventilation/O2 *hydration *pulmonary hygiene/toilet *nebulizer treatments
37
prevention of pneumonia
vaccines: *PCV13: prevents pneumococcal pna caused by 13 strains of strep pna (4 doses) *PPSV23: prevents against an additional 23 types of pna bacteria