Post parturient problems in the mare Flashcards

1
Q

Post partum reproductive problems (9)

A
  • Uterine artery haemorrhage
  • Uterine perforation
  • Retained placenta
  • Uterine prolapse
  • Septic metritis and/or delayed involution
  • Endometrial haemorrhage
  • Vaginal haematoma
  • Cervical laceration
  • Agalactia/hypogalactia
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2
Q

Post partum GI problems (4)

A
  • Caecal/colonic rupture
  • Colon torsion
  • Impaction
  • Rectal prolapse
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3
Q

Post partum, combine reproductive and GIT problems (2)

A
  • Perineal lacerations
  • Rectovaginal fistula
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4
Q

Miscellaneous post partum problems (4)

A
  • Retroperitoneal abscessation
  • Pelvic fractures
  • Obturator paralysis
  • Post parturient recumbency
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5
Q

Uterine artery haemorrhage

A

Commonly seen in old multiparous mares
Often fatal

Initial signs: acute, moderate to severe colic, sweating, very rapid pulse, progressing to CV shock

Usual site is the (right) middle uterine artery within the broad ligament, not fatal if contained within broad ligament and surrounding tissues

Treatment is controversial, keep mare quiet is agreed on

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6
Q

Uterine perforation

A

Not common
May follow dystocia or normal parturition
Tears can be in the body or uterine horn
Smaller tears may not become apparent until signs of peritonitis develop over the next hours and even days
Prompt surgical repair and antibiotic treatment is usually the key to a successful outcome

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7
Q

Caecal/colonic rupture

A

Can occur at, or soon after, parturition or over the subsequent hours or even days
The mare shows signs of colic, cardiovascular shock and acute peritonitis, with injected mucous membranes
Chronic compression of a focal area of the caecum or colon may follow trapping between the fetus and maternal pelvis
Once the diagnosis has been confirmed, euthanasia should be performed on humane grounds

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8
Q

Colon torsion

A

Typically seen 2-3 days following parturition but may occur up to 2-3 months later
Torsion of the colon follows large intestinal re-positioning within the abdomen, following evacuation of the uterus
The degree of colic is variable depending on the degree of the torsion but, frequently, it is severe and violent, leading to profound cardiovascular shock
Treatment may involve surgery and supportive therapy

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9
Q

Impaction

A

Some become impacted during 24-48 hours after foaling
Lack of exercise and eating straw predisposes to the condition

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10
Q

Retained placenta

A

Third stage labour usually complete within 30 mins-2 hours.

Placental retention may occur spontaneously or due to dystocia, abortion, premature delivery, or caesarian section

Retention of non-pregnant placental horn, especially at its tip, is common but not serious, although endometritis, endotoxaemia, and laminitis are major concerns.

Manual removal of the placenta should be attempted with great care and abandoned if not immediately and easily successful to avoid uterine wall damage, haemorrhage, and/or septic metritis.

Hygiene and safety should be maintained, and exercise is encouraged to stimulate involution of the uterus and to expel fluid.

Oxytocin may be given (20 iu i/m) if necessary, and systemic antibiotics and flunixin are indicated if there are concerns of endotoxaemia. Regularly check for signs of laminitis.

Following the release or removal of a retained placenta, a course of intrauterine antibiotic irrigations may be recommended.

Burn’s method may be used if placental removal is still unsuccessful.

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11
Q

Uterine prolapse

A

Uterine prolapse is uncommon and can occur at normal parturition but is more often associated with dystocia or placental retention.

The mare should be sedated, and an epidural anaesthetic administered to prevent septic metritis.

The uterus should be cleaned with dilute povidone iodine solution and examined carefully for lacerations before it is carefully replaced and re-positioned.

The vulva may be temporarily sutured with heavy-duty suture material.

Exercise will aid uterine involution.

At ‘foal heat’ uterine saline lavages, antibiotics, and oxytocin may be required.

Recovery is usually complete if the uterus is replaced fairly rapidly and without serious damage.

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12
Q

Septic metritis and/or delayed involution

A

Septic metritis and/or delayed involution may be associated with dystocia, retained placenta, uterine infection, or lack of exercise.

Parenteral antibiotic and NSAIDs should be given if there is systemic involvement.

Uterine lavage/antibiotics combined with intravenous oxytocin are also helpful.

Exercise is important both preventively and as part of the treatment for delayed involution or metritis.

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13
Q

Endometrial haemorrhage

A

Diffuse endometrial haemorrhage may occur in mares for reasons that are not altogether clear.

A persistent post-partum haemorrhagic vaginal discharge is seen, but in other cases, the diagnosis is only made at routine foal heat examination when poor involution is detected.

Oxytocin may be useful, and severe cases may require treatment by packing the uterus.

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14
Q

Vaginal haematoma

A

Vaginal haematoma may occur due to trauma during foaling.

It can be treated by drainage and packing with gauze and may require systemic antibiotic therapy if infection occurs.

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15
Q

Cervical laceration

A

Cervical lacerations are commonly associated with dystocia or attempts at fetotomy during foaling.

Mucosal splits may heal with the aid of applications of local antibiotic ointment, but lacerations involving the muscular layer reduce fertility and interfere with closure of the cervical os.

Surgical repair may be attempted after initial healing with the mare sedated and restrained standing in stocks under epidural anaesthesia.

A three-layer closure is made (internal and external mucosae and muscularis) but fertility remains depressed in most cases.

The closure of ventral cervical lacerations in the Trendelenburg position under general anaesthesia may be recommended for mares that had multiple tears.

Foaling rate is lower in mares that had a tear length >75% compared with smaller tears, but pregnancy rate is similar in both groups.

Dilation may be a problem at parturition with more tearing and a worsening of the problem for the next year.

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16
Q

Agalatia/hypogalactia

A

The hypothalamic-pituitary axis, ovaries, and placenta play a role in udder development and initiation of lactation.

Factors involved in lactogenesis include oestrogen, progesterone, prolactin, oxytocin, growth hormone, insulin, and thyroid hormone.

Causes of agalactia include inadequate nutrition, selenium deficiency, stress, systemic illness, surgery, administration of a dopamine agonist such as pergolide or bromocryptine, and ingestion of ergot alkaloid (fescue toxicosis).

Treatment of agalactia focuses on increasing prolactin production by blocking the inhibitory effect of dopamine.

Compounds that block the inhibitory action of dopamine include domperidone, sulpiride, metaclopramide, thyrotropin-releasing hormone, reserpine, phenothiazine tranquilizers, and butyrophenones.

In mares that exhibit idiopathic hypogalactia post-foaling, domperidone alone can be effective in stimulating milk production.

Mares with sick foals that are not nursing should be milked frequently (every two hours) to keep milk production stimulated.

Special attention should be paid to the nutrition of lactating mares that suffer from illness or undergo major surgery.

Mares being treated for PPID should discontinue treatment for approximately 30 days prior to their expected due date to allow for normal udder development to occur.

17
Q

Rectal prolapse

A

Unusual condition

Can occur during or immediately after parturition

Sedate, use epidural, and replace rectum

Intensive somatic and supportive therapy required with broad spec antibiotics and NSAIDs

Often irreparable damage occurs to vasculature leading to necrosis and peritonitis - euthanise on humane grounds

18
Q

Perineal lacerations

A

Often secondary to cases of malposture

First degree: tear of the dorsal commissure and MM of the vestibule, repair with a Caslick’s vulvoplasty

Second degree: Involve vestibular mucosa, submucosa, and perineal body. Require internal repair then Caslick’s vulvoplasty

Third degree: Involve ceiling of vestibule, roof of rectum, perineal septum, musculature, and anal sphincter. Leave to heal, keep clean, antibiotics, NSAIDs, then surgical treatment

19
Q

Rectovaginal fistula

A

Laceration through the rectovaginal shelf
4-6 weeks for second intention healing, may repair spontaneously but often need surgical intervention

20
Q

Retroperitoneal abscessation

A

Following entry of infection through vaginal laceration
Can be very chronic and lead to severe weight loss and general unthriftiness
Discharge and then high dose antibiotics

21
Q

Pelvic fractures

A

Caused by severe traction at foaling or a heavy fall

Iliac shafts or pubic symphyses are likely sites

22
Q

Obturator paralysis

A

Partial obturator paralysis is occasionally seen but is not usually serious

Can cause temporary urinary incontinence