Postoperative extraction complications Flashcards

1
Q

What are the potential post-operative extraction complications

A
  • pain/swelling/ecchymosis
  • trismus/limited mouth opening
  • haemorrhage
  • prolonged effects of nerve damage
  • dry socket
  • sequestrum
  • infected socket
  • chronic OAF/root in antrum
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2
Q

what are less common post-operative extractions

A
  • osteomyelitis
  • osteoradionecrosis
  • medication induced osteonecrosis
  • actinomycosis
  • bacteraemia/infective endocarditis
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3
Q

what is the most common post extraction complication

A

pain

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4
Q

how do you manage pain

A

make sure patients know it is normal to experience discomfort after an extraction

warn patients and advise/prescribe analgesia

Rough handling causes more pain
- laceration of tissues
exposed bone
-incomplete extraction of tooth

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5
Q

when should you be concerned about post-operative swelling

A

if it only begins about three days after the extraction

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6
Q

what can cause post-operative trismus

A

related to surgery - oedema or muscle spasm

related to giving LA - IDB ( if needle goes into medial pterygoid, it can cause a bleed and a haematoma. Or the muscle may go into a spasm resulting in limited mouth opening

Bleed in muscle - haematoma in medial pterygoid or masseter

damage to TMJ - oedema/join effusion

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7
Q

How do we initially deal with a patient who’s returned with bad post-operative bleeding

A
  • put pressure on immediately
  • calm the patient
  • clean patient, remove bowls of blood etc
  • take a thorough but rapid history
  • rule out bleeding disorder and question anti platelet medication
  • refer urgently if there is a bleeding disorder
  • Remove the large jelly-like clot
  • identify where bleeding is from
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8
Q

what type of haemorrage can occur post operatively

A

immediate post-operative period - within 48 hours, vessels that have been shut down open up and LA vasoconstriction effects wear off. Sutures also loosen or patient traumatises with finger/tongue

Secondary bleeding - often due to infection at 3-7 days. Usually mild ooze of blood

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9
Q

how do we manage/ stop haemorrhage

A
  • pressure with finger or damp gauze packs
  • local anaesthetic with vasoconstrictor
  • haemostatic aids (surgicel - oxidised cellulose acts as a framework for clot formation)
  • bone wax in socket
  • suture the socket

ligation of vessels/diathermy

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10
Q

what post extraction instructions should you give to a patient who has been bleeding

A
  1. Do not rinse for several hours
  2. Avoid trauma - don’t explore socket with tongue or fingers
  3. Avoid hot food
  4. Avoid excessive exercise and alcohol (increases blood pressure)
  5. Give advice on control of bleeding (damp gauze, pressure for 30 minutes, points of contact if bleeding continues)
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11
Q

what is the proper name for dry socket

A

alveolar osteitis

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12
Q

what is the definition of dry socket

A

a condition occurring after tooth extraction which results in a dry appearance of the exposed bone in the socket, due to disintegration or loss of the blood clot

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13
Q

what is the main feature of dry socket

A

intense pain, described as worse than toothache and keeps patient awake at night

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14
Q

what is the aetiology of dry socket

A
  • starts 3-4 days after extraction
  • takes 7-14 days to resolve if you do nothing
  • inflammation affecting the lamina dura (socket wall)
  • some say cot does not form, others say it forms then breaks down
  • ensure you help with pain control
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15
Q

what are the symptoms of dry socket

A
  • dull aching pain (moderate to severe)
  • usually throbs and can radiate to patient’s ear
  • can keep patient awake at night
  • exposed bone is sensitive and source of pain
  • characteristic bad smell
  • patient complains of bad taste in mouth
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16
Q

what are common predisposing factors of dry socket

A
  • molars more common (risk increases from anterior to posterior)
  • mandible more common (less blood supply)
  • smoking (reduced blood supply)
  • female
  • oral contraceptive pill
  • local anaesthetic with vasoconstrictor
17
Q

what are less common predisposing factors of dry socket

A
  • haematogenous bacteria in the socket
  • excessive trauma during extraction
  • excessive mouth rinsing post extraction (clot washes away)
  • family history or previous dry socket
18
Q

how do you manage a dry socket

A
  1. Reassure and give pain relief
  2. provide LA for pain relief
  3. Irrigate socket with warm saline to wash out food and debris
  4. curettage/debridement - encourage bleeding and new clot formation (Dr Bell doesn’t do this)
  5. Antiseptic pack (BIP or Alvogyl)

BIP is a bit of gauze with iodine base chemical in it. it doesn’t dissolve and you need to change them (maybe suture in place) see patient in a few days to remove or change

Alvogyl - take out with tweezers and pack into socket, will disintegrate (mixture of LA and antiseptic)

  1. Advise on analgesia and hot salty mouthwashes
  2. No antibiotics - not an infection
  3. Check that it is a dry socket and that no tooth fragments or bony sequestra remain
19
Q

What is osteomyelitis

A

inflammation of the bone marrow

20
Q

where is osteomyelitis more common

A

the mandible

21
Q

what is the aetiology of osteomyelitis

A
  • invasion of bacteria into cancellous bone causes soft tissue inflammation and oedema in the closed bony marrow spaces
  • oedema in an enclosed space leads to increased tissue hydrostatic pressure
  • the compromised blood supply results in soft tissue necrosis
  • the involved area becomes ischaemic and necrosis
  • the bacteria proliferate because normal blood borne defences do not reach the tissue
  • osteomyelitis spreads until arrested by antibiotics and surgical therapy
22
Q

why does osteomyelitis occur in the mandible more than the maxilla

A

the mandible primary blood supply is via the inferior alveolar artery and dense overlying cortical bone limits the penetration of periosteal vessels

Therefore, the mandible has a poorer blood supply and is more likely to become ischaemic and infected

23
Q

what are predisposing factors for osteomyelitis

A
  • odontogenic infections and fractures of the mandible
  • host defences compromised
    > diabetes
    > alcoholism
    > IV drug use
    > malnutrition
    Mmyeloproliferative disease
24
Q

what bacteria are involved in osteomyelitis

A
  • streptococci
  • anaerobic cocci
  • anaerobic gram negative rods such as fusobacterium and prevotella
25
Q

what is the treatment for osteomyelitis

A

antibiotics - clindamycin/penicillins with longer courses than normal

surgical - drain pus and remove non-vital teeth around infection and any loose pieces of bone

Excise necrotic bone until we reach actively bleeding healthy bone

26
Q

what is osteoradionecrosis

A
  • bone within he radiation beam becomes non-vital after patients have received radiotherapy to head and neck to treat cancer
27
Q

how do you prevent osteoradionecrosis

A
  • scaling/chlorhexidine mouthwash leading uptown an extraction
  • Careful extraction technique
  • antibiotics
  • hyperbaric oxygen
28
Q

how do you treat osteonecrosis

A
  • irrigation of necrotic debris
  • antibiotics not overly helpful unless there is secondary infection
  • loose sequestra removed
29
Q

what is MRONJ

A

a progressive death of the jawbone in a person exposed to a medication known to increase the risk of the disease

30
Q

When can MRONJ occur

A

post extraction

following dental trauma

spontaneously

31
Q

what factors can affect MRONJ

A
  • length of time patient has been on drug
  • diabetics
  • steroids
  • anticancer chemotherapy
  • smoking
32
Q

what drugs can cause MRONJ

A

bisphosphonates

  • alendronate
  • clodronate
  • etidronate
  • ibandronate
  • pamidronate
  • risendronate
  • tiludronate
  • zoledronate

Antiresorptive
- praia
denosumab

Antiangiogenic

33
Q

what are the risk factors for MRONJ

A
  • dental treatment
  • duration of bisphosonate drug therapy
  • dental implants
  • other concurrent medication
  • previous drug history