Production EXAM Flashcards

(96 cards)

1
Q

Coccodia Gross & Histopathology:

A

Catarrhal enteritis-congestive, +/-haemorrhagic
Small 1-2 mm white/grey ‘nodules’ in intestinal mucosa
Loss of epithelial cells
Villous atrophy, crypt destruction

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2
Q

Coccidia prepatent period:

A

Can be up to a year

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3
Q

Risk factors for coccidiosis

A
  • Young age
  • low body weight (at weaning)
  • warm wet environment
  • high stock density
  • fecal contamination of feed/water
  • stressors
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4
Q

Describe a drought feedlot:

A

a facility designed to provide a controlled environment for livestock during periods of drought. It plays a crucial role in maintaining animal welfare and ensuring sustainability of livestock farming operations during challenging environmental conditions.

Neucleus flock

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5
Q

Describe a lamb feedlot

A

a facility where lambs are raised for meat production. It comprises pens or enclosures where lambs receive a controlled diet to promote growth and reach market weight efficiently. Proper nutrition and care are essential to ensure the production of high-quality lamb meat.

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6
Q

Describe an export feedlot

A

These feedlots ensure the animals meet specific quality and health standards required for export. They provide a controlled environment with proper nutrition, healthcare, and handling to produce livestock that meet the criteria set by importing countries, allowing for the export of high-quality meat and livestock products to global markets. Export feedlots play a pivotal role in facilitating international trade and meeting stringent import requirements.

Prepare for export

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7
Q

List some feedlot design considerations:

A

Regulatory Compliance
Shade and Shelter
Feed and Water Infrastructure
Environmental Impact
Waste Management
Health and Veterinary Facilities
Security
Accessibility

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8
Q

How many serotypes of salmonella are identified?

A

2500

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9
Q

Enterobacteriaceae

A

a family of bacteria that includes various Gram-negative, rod-shaped organisms. These bacteria are found in various environments, including the gastrointestinal tracts of humans and animals. Many members of the Enterobacteriaceae family are opportunistic pathogens, meaning they can cause infections when the host’s immune system is compromised. Some well-known genera within Enterobacteriaceae include Escherichia, Salmonella, Shigella, and Klebsiella.

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10
Q

How can we identify salmonella?

A

Culture
Serology
PCR
Sensitivity

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11
Q

Salmonella pathogenesis:

A

Fecal oral transmission

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12
Q

Salmonellosis risk factors?

A

Stress
- transport
- husbandry events
- parasites
- other disease
- bad weather
- overcrowding
- poor hygiene

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13
Q

Coccidia ateology:

A

Caused by several species of protozoa from the family Eimeriidae

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14
Q

Coccidia pathogenesis:

A

Ingestion of sporulated oocysts

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15
Q

Salmonella localization:

Where may this occur?

A

Brain - meningo-encephalitis
Tips of ears and tails - dry gangrene
Gastro-intestinal tract - diarrhea
Repro. tract - abortion

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16
Q

Salmonella clinical signs

A
  • Fever
  • DIARRHEA
  • Dehydration
  • Inappentence
  • Weakness
  • Weight loss
  • Pain (abdo.)
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17
Q

Chronic carriers of Salmonella

A

Harboring the bacteria in their intestines for an extended period without displaying symptoms - can potentially shed the bacteria, posing a risk of transmission to others.

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18
Q

Salmonella gross pathology:

A

Varied
congested, red-purple lungs and liver
enlarged fatty liver and thickened and inflamed gall bladder
inflamed abomasum and intestines
enlarged, moist or bleeding intestinal lymph nodes
khaki-coloured diarrhoea, containing mucus or blood flecks
Intestinal fibrinous/diphteric casts

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19
Q

Coccidia diagnosis

A

History (risk factors)
Clinical signs
Gross pathology
Laboratory diagnosis (FEC) is not definitive as little relationship betw. numbers of oocysts and severity of disease.

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20
Q

Acidosis

A

Can manifest as either acute or subacute forms, with acute acidosis often resulting from rapid grain consumption and leading to severe clinical signs. Subacute ruminal acidosis is a chronic condition associated with prolonged exposure to high-carbohydrate diets, leading to subtle performance issues. Preventing acidosis through gradual dietary transitions, careful grain management, and access to forage and clean water is essential for maintaining sheep health.

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21
Q

Urolithiasis

A

Formation of urinary stones in the urinary tract, which can obstruct urine flow. This condition can result from various factors, including diet, water quality, and genetics. Prevention involves maintaining a balanced diet, providing clean and ample water, and managing mineral intake

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22
Q

Vitamin A deficiency

A

Leads to a range of health issues, including poor growth, impaired reproduction, and weakened immune function. This deficiency is often associated with a diet lacking in vitamin A-rich forage or feed sources. To prevent vitamin A deficiency, it’s crucial to provide sheep with a well-balanced diet

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23
Q

Laminitis

A

Inflammation of the laminae, which are the sensitive tissues that connect the hoof wall to the underlying structures. Common causes of laminitis in sheep include overconsumption of high-energy feeds, grain-rich diets, or sudden changes in diet. Clinical signs may include lameness, reluctance to move, heat in the hooves, and a characteristic “sawhorse” stance.

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24
Q

What is BRD?

A

Multifactorial disease
Viral:
- IBR (bovine herpes 1v)
- pestivirus (BVDV)
- parainfluenza
bacterial: (commensals of upper respiratory tract)
- mannheimia haemolytica
- pasteurella multocida
- mycoplasma bovis

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25
What is the usual cause of death for BRD cases?
Bronchopnemonia
26
What is IBR?
Infective Bovine Rinotracitis Caused by Bovine Herpesvirus 1 (BoHV-1) Vaccine available - Bovillis IBR + MH or Rhinogurd BHV 1 (live) ## Footnote Viral respiratory disease that affects cattle, causing symptoms like fever, nasal discharge, and coughing
27
Pestivirus BVDV | Bovine Viral Diarrhea Virus
Respiratory symptoms (including coughing and nasal discharge) However, it's primarily recognized as a reproductive and systemic disease in cattle, but it can cause respiratory issues when transmitted to sheep. Control measures in sheep may involve vaccination and quarantine procedures.
28
Corona virus
An agent of BRD Gastrointestinal and respiratory system
29
Manheimia Haemolytica | Primary BRD pathogen
Pneumonia, high fever, and labored breathing Vaccination = Bovillis IBR + MH or Bovi-shield MH (live) most severe - death <24h
30
Pasturella Multocidia
Pneumonia, nasal discharge, and coughing. Proper management, including vaccination and minimizing stress factors
31
Histophylis somni
BRD pathogen respiratory distress, fever, nasal discharge, coughing, and reduced feed intake
32
What are the factors (other than (viruses + bacteria) of BRD?
Stress Mechanical damage to lungs Immunosupression Transportation Breed (bos torus) Sex (steers) - Disease occurs 2-3 weeks after a stressor
33
Clinical signs of BRD:
Flat Coughing (wet) Mucus from nose Droopy ears Head down Separation from herd Sunken eyes
34
Best chance of recovery is due to:
Being identified early
35
Gross pathology of BRD:
Kills them quickly: Purple discolouration of lung fields 1-2 weeks: emphasematus and fibrous lungs (pasturella) 3-4 weeks: lots of puss, chronic fibrin tags, some adhesions, plutris the more time that goes by the harder it is to identify the causitive bug brisket odemia - heart wont work (histophalus - micro abscesses in heart) lung microabcesses (micoplasma bovis)
36
Treatment of BRD:
Antibiotics NSAIDs = meloxicam (last 3d SQ) Vitamin C Change feed Give a few weeks
37
Antibiotic treatment for BRD:
**Macrolides** (not for milk producing animals) Tulathromycin (draxxin) 7d, WHP 35d **Cephalosporin** = Exceed, Accent & Ceftiofur (last resort) Allomycin
38
Control/prevention of BRD:
Manage concurrent disease Starter diet (reduce acidosis) - high in carbs introduction protocols - lower stress allocate enough bunk space (feeding) **11m2/hd** yard weaning & backgrounding
39
Transportation of poddy calves:
Vehicle and equipment standards, ventilation, temperature control, loading and unloading procedures, maximum travel time, water and feed provisions, stockmanship, and record-keeping.
40
Transportation of late gestation cows:
can't travel in the last 4 weeks >6m preg. needto stop every 12 hours
41
Transportation of cows with horns:
Cows with horns may require more headroom to prevent them from damaging their horns during transport.
42
Transportation of Lactating dairy cows:
stop every 12 hours and milked
43
TEME | Thromboembolic Meningoencephalitis
Causative agent: Histophilus somni Adhers blood vessels in brain Causes convulsions
44
What is backgrounding?
Keeping cattle in paddocks next to the feedlot
45
Yard weaning
Done prior to going to backgrounding getting them used to the feedlot setting/feed
46
Vaccines BVDV BVDV | Mannhymeia haemolytica
2 vaccines - bovillis MHS Bovi-shield Pestigaurd | Backgrounding is better protection than vaccination
47
What should feedlot diets do?
Avoid high non protein nitrogen (NPN) Avoid acidosis in the starter period Support the immune system add vit. E add zinc | add ammonia add vit. B add zinc
48
IBR | part of the BRD complex
Infectious Bovine Rhinotracheitis Bovine Herpes virus 1 (BHV1) severe (death) tracheal lining really sore | one of the most commmon causes of pneumonia ## Footnote Very infectious (nasal and oral droplets) NSAIDs really important Vaccination
49
Malignant Catarrhal Fever | Looks like?
IBR on steroids caused by Alcelaphine Herpes virus 1 No treatment | Sporadic Immediatly cull on humane grounds
50
Shipping Fever
Pasteurella spp. or Mannheimia occurs post transport | having their heads up bacteria enter lungs usually no viral involvment
51
Necrotic laryngitis | "honkers"
Diptheria fusobacterium
52
Shade requirment
2.5m2/head
53
Coccidiosis | Treatment
Ionophores (rumensin, bovitek) work by altering na/k ion channels in bacteria and other organisms
54
Salmonella | treatment
Trisoprim (TMS)
55
Salt toxicity
held off water - dehydrated high Na con. in CSF when regain axcess to water they gourge - water rushes to CSF and causes brain swelling
56
Listeriosis
Listeria monocytogenes comes from silage CNS clinical signs droopy ear/eye facial paralysis
57
PEM | Polioencephalomelacia
Bacteria get "acid shocked" in low pH of acidic rumen, release exoenzymes & thiaminases - breakdown B1 compound caused by increasing ration too quickly or high carbs or sulfur content | Lights on and no ones home
58
Otitis
Pasturella or mycoplasma spread through blood oxitet/eardrops
59
Urolithiasis | water belly
Different types of crystal formation ca:phosp ratio if low - struvite crystals (sand like) if high - calcium carbonate crystals (round stones) Oxalate pastures - oxalate crystal - straining ## Footnote like a blocked cat
60
# Viral Infectious Bronchitis | RNA virus - envelope
61
# Viral Avian Influenza | RNA virus - envelope
zoonotic Exotic animal disease High mortality migratory water fowl carriers horozontal | H7N7 strain
62
# Viral Newcastle Disease envelope | RNA virus - envelope
Exotic animal disease no disease in Aus vaccine avalible neurological signs
63
# Viral Infectious Laryngotracheitis | DNA virus - envelope
64
# Viral Fowl Pox | DNA virus - envelope
65
# Bacterial Fowl Coryza
Avibacterium paragallinarum Sinusitis PCR vaccination
66
# Bacterial Fowl Cholera
Pasteurella multocidia re-emerging animal disease swollen combs/wattles sudden death
67
Infectious Bronchitis (IB)
endemic
68
Infectious Laryngotracheitis (ILT) | Notifiable disease
Comes back when immune system is low histopathology or PCR coughing up blood vaccinate layers
69
# Bacterial Chlamydiosis
70
# Bacterial Chronic Respiratory Disease (CRD)
All respiratory diseases with unknown cause Avian pathogenic E.coli (APEC) peritonitis perihepatitis
71
Antibiotics not registed for any animal in australia:
1. Fluroquinolones 2. Colistin 3. Fourth generation Cephalosporins
72
# Bacterial Mycoplasmosis | pathogenic strains
M. Gallisepticum M. Synoviae M. Meleagridis
73
Mycoplasmosis | Transmission
Both vertical and horizontal Mild in Aus - make vaccines clinical signs may vary difficult to culture - do PCR treatment won't elliminate the bacteria - can researfice
74
# For respiratory disease What antibiotics do we use in chickens?
Amoxicillin Trihydrate Lincomycin | Nil WHP
75
# Fungal Respiratory Mycosis | Aspergillus fumigatus
Occurs in chicks no antifungal registed fungal nodules in lungs
76
Enveloped viruses are more ....... in the environment?
**Fragile** as well as more sensitive to fetergents and disinfectants due to lipidic nature of the envelope
77
Anatomy of respiratory system (chicken)
Conjunctiva Nostrils Nasal cavity Nasal sinuses Pneumatic bones of skull Larynx Trachea Bronchi Lungs Airsacs Pneumatic bones
78
Respiratory illness clinical signs: | Chickens
Clowdy airsacs
79
Chlamtdiosis | (Psittacosis, ornithosis)
chlamydia psittaci notifiable zoonotic | doxicyclin - not registed in chickens
80
Fowl Pox
can vaccinate plaques in mouth (look like scabs)
81
B cell response
Ag components are displayed (with MHCII) T-helper recognise the same ag, binds & produces cytokines Activated B cell forms plasma & memory cells plasma cells produce antibodies and memory cells await another invasion
82
# Primary Lymphoid organs 1. Thymus 2. Bursa of Fabricius
1. T cell precursors 2. B cell precursors
83
# Secondary Secondary Lymphoid orgains
* Spleen * bone marrow * Harderian gland * CALT (conjunctivial-associated lymphoid tissue) * BALT (bronchial-associated lymphoid tissue) * GALT (gut-associated lymphoid tissue ## Footnote Chickens do not have lymph nodes (except water fowls)
84
Chicken immunoglobulins
Ig Y (IgG) Ig M Ig A
85
IgA
Mucosal membranes fades quickly
86
Ig Y (G) Ig M
Systemic
87
Types of vaccines in chickens: | Live vaccines:
* Eye/nose drop * beak dip * spray * wing web * drinking water * in ovo (day 18) * injection (IM)
88
Types of vaccines in chickens: | Inactivated vaccines:
* SC * IM (may cause lesions due to harsh adjuvants)
89
# Neoplastic Marek's Disease (MD)
Most common - may have pathogen without dissease Herpesvirus (1,2,3(HVT) strains) classic paralysis (attacks the siatic nerve (superman)) spread through the feather follicles
90
3 Presentations of Marek's disease (MD)
Classical disease Neurolymphomatosis Acute form Inflammatory form immunosupressive
91
Infectious bursal disease (IBD)
RNA virus non-enveloped serotype 1 = disease (not in Aus) immunosupression 1. inapparant <3 weeks 2. Acute 3-6 weeks 3. Severe (any age)
92
Avian Lymphoid Leucosis | ALV
retrovirus incubation 4-6m horizontal (fecal-oral) reduced weight, weakness, anorexia histopath, PCR, serology no vaccine
93
can we differentiate MDV and ALV | physical signs
sometimes ALV - no nerve, muscle, skin, eye lesions MDV - T cells - herpesvirus (DNA) ALV - B cells - oncovirus (RNA)
94
Chicken infectious anaemia
vertical transmission non-enveloped immunosupression virus replicates in bone marrow CD4 & CD8 T cells in the spleen vaccinate parents
95
Inclusion Body Hepatitis (IBH)
Adenovirus lots of serotypes
96
IBH Fowl Adenovirus (FAV)
gross lesions: * swollen liver, yellow, mottled with petechiae * kidneys and bone marrow pale * bursa and spleen small vertical and horizontal transmission