PSY3 Eating Behaviour Flashcards

(35 cards)

1
Q

Attitudes to food/eating behaviour

**Social Learning: A01 **Parental Modelling

A
  • Children acquire attitudes by observing behaviour of their parents
  • Parental att. inevitably affect kids
    • parents control foods bought/served in home
  • Brown/Ogden outline no. of ways parents exert influence:
    • basic food pref
    • healthy quality of diets
    • concerns of weight gain/disordered eating behaviour
  • Parents exert direct role through use of reward/punishment (operant)
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2
Q

A02 Parental modelling

A
  • Brown/Ogden - gen. association b/w parents/kids attitudes:
    • correlation b/w parents/kids in terms of snack food intake, eating motivations and body dissatisfaction
  • Birch/Fisher - found best predictors of daughters’ eating behaviour were mums’ dietry restraint/perception of risk of daughters becoming overweight
  • Hall/Brown - mums influence daughter’s satisfaction/dissatisfaction w/ body:
    • mother’s of girls w/ anorexia show greater body dissatisfaction than mums of non-disordered girls
  • Birch - parents directly influence habits through use of praise/reward:
    • praise from adult inc. food pref. for previously disliked food
    • but relationship is complex
      • manipulating pref. may create unitended conseq/counterproductive
        • kids offered juice as a means to be allowed to play showed that using juice as reward reduced pref. for juice (Lowe)
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3
Q

Attitudes to food/eating behaviour:

A01 Peers

A
  • As kids move into wider social world, become inc. influenced by food pref. of friends
  • Gladwell/Harris - best way to get a child to eat something new is not by parental encouragement, but immersing child in room of kids who already like that food
  • Teen years, children may actively differentiate self from parents/parents’ generation
  • Certain foods favoured by teen compared w/ older age groups e.g. pizza, fried foods (Demroy-Luce/Motil)
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4
Q

A02 Peers

A
  • Birch - children will eat veg if friends do:
    • selection/consumption of veg by pre-schoolers influenced by choices of peers
    • children lunched next to kids who pref. different veg to self
      • kids showed shift in veg pref. which persisted at follow-up assessment weeks later!
  • Lowe - impact of observational learning shown in intervention study designed to change eating behav:
    • kids watched series of DVD adventures feauturing ‘fun/cool’ ‘Food Dudes’
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5
Q

A03 Attitudes to food/eating behaviour

A
  • Problems w/ generalisability - studies on clinical/non-clinical population:
    • limits degree generalise from one group to another, thus degree to which studies offer understanding of causal factors
  • Gender bias - conc. on women’s attitudes
    • male homosexuality risk factor in development of d/o (Siever).
      • Findings attributed to male gay subculture - emphasis on lean, muscular body ideal
    • limtd view
  • Need to consider other factors - emphasise imp. of ext. factors, underest. contribution of non-psycho exp.
    • though may exp. specific likes/dislikes/habits, our basic food pref. determined by evolutionary forces
      • humans have strong pref. for nutrient dense foods (meat/fat)
        • pref. set in EEA and only moderated by direct experience
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6
Q

Explanations for success/failure diets:

A01 Restraint theory

A
  • Restraint inc. probability of overeating
  • Herman/Polivy developed Boundary Model to exp. why dieting –> overeating
  • Hunger - when/how much to eat
  • Satiation - occurs when brain judges that eating more not in person’s best interest
  • For ancestors, finding food required effort/time, so when person’s eaten optimal amount, brain signals stop, do something to aid survival!
  • When food plentiful, satiation reached easily
    • don’t need to eat large amounts in single meal as it’s avail in near future
  • But reg. bouts hunger indicate uncertain food supply
    • brain biochem adjusts to satiation less easily achieved - brain judges that food’s scarce
  • When opportunity to comsume arises, org. should consume lots, for it may be while until another opportunity arises
  • Dieting widens gap between hunger/satiation
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7
Q

A02 Restraint theory

A
  • Wardle/Beale - 27 obese women: women in diet cond. ate more than women in exercise & control group; confirmed model’s predications.
    • Strength - assessment under lab cond.
    • Gender bias - research only conc. on success/fail of women’s dieting; studies providing insight only offer limtd exp.
      • Not generalisable to opposite gender? Don’t provide universal exp. for reasons of success/failure.
  • Implications for obesity treatment - theory says restraint –> overeating, yet treatment commonly used as solution to exc. weight gain.
    • But failed attempts to diet leaves obese depressed/feeling failure
      • This is why there may be more emphasis on fitness than diet!
      • Ogden - though obesity not necessarily result of overeating, it may be consequence of obesity if restraint used as treatment.
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8
Q

Explanations for success/failure diets:

A01 Role of denial/Ironic processes

A
  • Research shows trying to suppress/deny thoughts freq. has opposite effect, making it more prominent (Wegner/white bears)
  • A.K.A ‘theory of ironic processes of mental control’ - rep. a paradoxical effect of thought control i.e. denial backfires
  • Denial of food tends to make us think of it more than normal; can become pre-occupation
  • As food’s denied, it simultaneously becomes more attractive
  • Dieter may find only way to deal w/ issue is to succumb to temptation, consume food in question
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9
Q

A02 Role of denial

A
  • Practical application - Wegner suggests dieters think of issues differently; rather than thinking “I must not eat” should reformulate challenge to “I must get fit”.
    • Ppl may be more successful by focusing on fitness, not diet.
  • Soetens - disinhibited restrained eaters used more thought suppression than unrestrained/low disinhibition, showed rebound effect after (thought of more food)
    • Restrained eaters who overeat try to suppress thoughts more often, but when they do, think more of food after
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10
Q

A03 of success/failure of diets

A
  • Culturally biased - some cultural grps find it harder to diet successfully due to natural inclination to obesity
    • Asian adults more prone to obesity than EU (Park); asian kids/teens have a great central fat mass compared to EU (Misra)
  • Due to lifestyle or genes? - issue to degree which lifestyle determines failure/success
    • likely that genetic mechanisms influence weight
      • H. levels enzyme LPL associated w/ greater weight gain.
      • Researchers believe weight loss activates gene prod. LPL, may exp. why it’s easier to regain lost weight than for one who has never been obese to put on weight
        • Determinism plays greater role in success, not free will
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11
Q

Neural mechanisms in eating behaviour:

A01 Homeostasis

A
  • Involves mechanisms which detect state of int. environment/correct situation to restore envrionment to optimal state
  • Hunger inc. as glucose levels dec.
  • Decline in glucose activates lateral hypothalamus, results in hunger, cause person to search/consume food = levels to inc. again
  • Activates ventromedial hypothalamus, leads to feelings of satiation, which inhibits feeding
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12
Q

A02 Homeostasis

A
  • Lashley - evidence for role of hypothalamus:
    • cut out diff areas of rats brain to see effect of lesions on behavior
    • LH identified as ‘hunger centre’, VMH ‘satiety centre’
      • lesions to LH caused animals to stop eating spontaneously
      • reverse occurred after lesions made to VMH
        • ​lesions in VMH caused rats to overeat!
        • Probs w/ animal studies!!!!
  • Limitations - for hunger mechanism to be adaptive, must anticipate/prevent energy deficits, not just react!
    • ​Suggests hunger only triggered when energy falls below desired level
      • Incompatible w/ situations in which systems would’ve evolved!
    • For mechanism to be adaptive, must promote levels of consumption that maintain bodily resources above optimal to act as buffer against future lack food
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13
Q

Neural mechanisms in eating behaviour

A01 Role of leptin

A
  • Fat hormone leptin is an example of a neuropeptide
  • Secreted from fat cells into the blood and signals the brain (via the hypothalamus) that calorie availability is high - leads to satiation
  • When fat reserves are used for energy production, leptin secretion falls
    • hypothalamus detects this, interprets low leptin as lack of calories and generates sensation of hunger
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14
Q

A02 Role of leptin

A
  • Zhang - evidence for role of leptin:
    • some mice received 2 copies of gene related to leptin regulation (ob/ob)
      • have tendency to overeat, esp. foods h. in fat/sugar
    • ob/ob mice have defective genes for leptin regulation
      • injecting ob/ob mice w/ leptin causes them to lose weight dramatically
    • direct evidence of leptin in human eating behav come from rare cases of ppl born w/ leptin deficiency
      • can’t control their eating, freq. become obese - take leptin injections to return to normal weight
    • Problems w/ animal research!!!!!
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15
Q

Neural mechanisms in eating behaviour:

A01 Role of Ghrelin

A
  • Ghrelin, **a **neuropeptide thought to be the ‘hunger hormone’
  • Produced in stomach and hypothalamus
  • Levels inc. before meals as stomach muscle contracts/stretches
    • Dec. after meals when stomach’s full
  • Receptors of ghrelin found in lateral hypothalamus (brain!), h. levels associated w/ hunger and low levels w/ satiation
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16
Q

A02 Role of Ghrelin

A
  • Zigman - explains why low mood leads to comfort eating:
    • ghrelin linked w/ appetite regulation/stress resistance
    • mice genetically engineered to prod. low levels of hormone
    • mice exposed to social stress (sharing enclosure with big mice)
    • normal would would inc. consumption of h. calorie food - special mice didn’t!
      • suggest ghrelin imp. in triggering comfort eating
        • suggests biochem mechanism to block this chemical action may be possible treatment for mood-related eating disorders!
  • Probs w/ animal studies!!!!
17
Q

A03 of neural mechanisms in eating behaviour

A
  • Reductionist - implications of psychological/physiological hunger:
    • there’s diff b/w physical hunger and psychological hunger
      • other learned/cognitive factors:
        • e.g availability of rich foods - ppl tend to gain weight when food plentiful
        • smell - some can’t resist feeling hungry even if they’re not physically hungry
      • ext. stimulu provide other ways to signal LH to make up feel hungary even if not physically (Hara)
    • exp. only focus on LH/bio factors - reductionist!!!
18
Q

Evolutionary exp. for food pref.

A01 Preference for meat

A
  • Human ancestors incl. meat in diet to compensate for decline in quality of plants
  • Meat diet, full of nutrients, provided the catalyst for brain growth
  • We’re hard-wired to consume meat as it was v. difficult to ancestors to obtain this!
  • W/o animals, claims Milton, it’s unlikely that early humans could’ve secured enough nutrition from a vege diet to evolve into the intelligent creatures they became!
19
Q

A02 Preference for meat

A
  • Cultural differences in food pref - vast majority of spec. food likes/dislikes appear not to be predetermined.
    • Though strong affinity of kids for sweets seem universal, innate response don’t account for broad range of likes/dislikes that develop beyond infancy.
    • Evolved factors clearly important, but exp. w/ diff foods, w/ culture partly determining this, modify this!
20
Q

Evolutionary exp. for food pref:

A01 Taste aversion

A
  • Bait shyness: animals that became ill after eating, developed aversion to that food
  • Development of taste aversions aided our ancestors in survival, as surviving from eating poisoned food ensured they would not make same mistake again
  • Once learnt, difficult to shift - adaptive quality designed to keep ancestors alive
21
Q

A02/A03 Taste aversion

A
  • Garcia rats who were made ill through radiation shortly after eating saccharin, developed aversion to it v. quickly
    • ​associated illness w/ saccharin
  • Real-world application - research origins of taste aversion helpful in understanding food avoidance that sometimes occur during treatment of cancer:
    • ​some cancer treatments cause gastrointestinal illness
    • when illness pairs w/ food consumption, taste aversions can result
  • Bernstein/Webster - gave patients novel-tasting ice cream prior to their chemotherapy –> patients acquired aversion to that icecream
    • fled to development of ‘scapegoat technique’ - give patient novel food w/ familiar food prior to treatment
      • patient forms aversion to novel food and not familiar
        • consistent w/ adaptive avoidance of novel foods - neophobia
22
Q

A02/A03 Evolutionary exp. for food preference

A
  • Reductionist - oversimplifies complex human behaviour to evolutionary mechanisms, ignoring psychological/cognitive factors
  • Determinism - overlooks freewill and conscious cognitive and social factors (e.g. dieting, social learning.)
23
Q

Psychological exp. for AN:

Social learning

A01/A02 Cultural ideals/media

A
  • Widely held belief Western standards of attractiveness imp. in AN development
  • Studies show many teens (esp. girls) dissatisfied w/ weight/have distorted body image
  • Media’s source of influence for body image attitudes maintined by West teens
  • Portrayal of thin models on TV/mags signif. in body image concerns/drive for thinness among Western teens girls
  • Media doesn’t influence all in same way e.g. ppl w/ l. self-esteem likelier to compare selves to idealised images portayed in media (Jones/Buckingham)
24
Q

A02/A03 Culture/media

A
  • Hoek - AN not confined to West:
    • tested view that AN rare in non-West:
      • examined records of over 44,000 ppl admitted to hospital over 2 year in Curacao (non-West), where it’s acceptable to be overweight
      • 6 cases of AN, rate claimed to be within range of West
  • Bio factors - don’t exp. individual differences:
    • fail to exp. why some develop AN and others don’t when they’re exposed same cultural influences/experiences/media
      • AN partly influenced by bio/genetic!
  • Real-world application - fashion industry acknowledged damaging infl. of media on body image, by signing charter of good will
    • Charter first step in stopping eating d/o, promoting healthy body image among young women
    • Charter involves a pledge by fashion houses, advertising agencies/mag editors to use diversity of body types, not stereotype of ‘thin ideal’
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**Psychological exp. for AN: ** **Cognitive** **A01 Role of distorted thinking**
* Emphasise role of distorted thinking in AN * **Distorted body image** - person believes self to be overweight, when they are very much underweight * Anorexics tend to show **dichotomous thinking** (seeing everything good/bad, success/fail) * This thinking carries over to issues of weight, anorexics typically agree 'thin is good, but thinnest is best' - normal never agree! * Anorexia exp. w/ **self-serving cognitive biases** * Ppl w/ AN show **reversal of overconfidence bias** (ppl feel self attractive than others would rate them) * may lead to low self-esteem/maladaptive behaviour (**_Jansen_**)
26
**A02/A03 Distorted thinking in AN**
* **Where do these thoughts come from?** * Prob exp. where these thoughts come from * Does thinking lead to change in behaviour or does behaviour change lead to distorted thinking? * Most think they're **dynamically linked**, **dysfunctional thinking leads to disordered eating, and disordered eating in turn, leads to dysfunctional thinking** * May be unhelpful to think of cognitive distortions as a cause of AN, **simply a variable/factor associated w/ AN** * **_Real-world application_ - effective therapies:** * cognitive therapists argue **focus of therapy should be on desire to be thin** * **challenge thought pattern** e.g. by challenging idea that thinnest is best * BUT can be very **ineffective**, as many patients find it **challenging/don't respond well** * **if patient doesn't co-op w/ therapy, can't be successful** * so there's always place for **bio/behavioural therapies too!**
27
**Evolutionary explanation for AN:** **A01 Reproduction suppression hypoethsis**
* **Surbey** suggests adolescent girls’ desire to control weight rep. evolutionary adaptation in which girls delayed offset of sex maturation in response to indication of poor reprod. success * Enabled females to avoid giving birth when conditions aren’t conductive to offspring’s survival * Surbey argues AN is a **‘disordered variant’** adaptive ability of females to alter timing of reproduction when they feel **unable to cope w/ bio/emo/social responsibilities of womanhood**
28
**A02 Reproduction suppression hypoethsis**
* **_Delayed reprod. sometimes makes sense:_** * Anorexica often leads to delayed puberty and suppression of periods in post-pubescent girls * These responses make sense if female becomes pregnant at time when infant is unlikely to survive
29
**A01 The ‘adapted to flee’ hypothesis**
* Proposes symptoms of AN reflect adaptive mechanisms that once caused migration in response to famine conditions * When person begins to lose weight, physiological mechanisms conserve energy/inc. desire for food * adaptations facilitate survival in hard times * But among ancestors, when extreme weight loss due to severe depletion of food resources, **adaptation must be turned off** so person can inc. chances of survival by migrating to more favourable environment
30
**Biological exp. for AN - Neural explanations:** **A01 Genetic factors**
* Twin studies suggest AN has strong genetic component * **Holland**: concordance rates of 25 identical/20 non-identical twins * **56% identical concordant for AN, only 5% non-identical** * Conclude 80% of variability might be exp. by genes
31
**A02 The ‘adapted to flee’ hypothesis**
* **_Why do females stop eating when there's plenty of food?_** * difficult to see what adaptive advantage this behaviour could be * evolutionary exp. of food pref suggest humans find difficulty in not eating when food's available * thought there's a connection b/w starvation and reproductive behaviour, unclear that this extends to anorexia * **_Incomplete explanation:_** * though adaptive advantage under certain conditions, makes sense * **doesn't explain why AN unevenly distributed in human pop'lns** * AN likelier to be diagnosed in **mid-class adolescent girls in developed countries** * **likely that cultural factors imp!**
32
**A02 Genetic**
* **_Limitations of twin studies_ - don't prove AN has bio basis:** * **identical twins share more than identical genetic makeup** * same gender, look same, behave similarly * **environment/experiences shape us!** * likely **social environment for MZ twins more similar than DZ** * exp. patterns w/ reference to genetic other bio factors arguably unnecessary * as concordance rate for twins not 100%,** show other factors important!!!!**
33
**A01 Neurological factors (brain/neurotransmitters)**
* **NT** linked to AN e.g. **serotonin/dopamine** * **Excessive serotonin activity in hypothalamus** has effect of **reducing appetite/inc. anxiety** * Both traits of AN * **Effects may be dynamically linked** * If AN reduces food intake, likely they'll feel less anxious and self-starvation may be reinforced through process of **negative reward (operant)** * **Pre-natal exposure to female hormones linked to AN:** * **​Procopio:** males developed in utero w/ twin sister likelier to develop AN than other males, incl. those who have twin bro * ​suggests pre-natal exposure to something, possibly female sex hormones signif. risk for developing AN
34
**A02 Neurological factors**
* **_Evidence for role of serotonin_** - evidence for NT in AN comes from **action of drugs used to treat AN** * **SSRIs**, which have **direct effect on serotonin regulation effective in treating AN** * **_Pre-natal development imp_** - **_Procopio_** found risk for males w/ female twin inc. risk of AN 10x * suggests pre-natal exp. to female sex hormones changes developing brain to inc. likelihood of AN developing
35
**IDA of biological exp.**
* **_Real-world application_** - in US, treatment for AN restricted under many insurance plans as AN not seen as **'bio based'** * But research creates case for insurance companies to consider AN in same way as other psychiatric cond. that ***are* considered as bio based** * **_Gender bias_** - **most studies conc. on women** * **​**statistics show 25% adults w/ eating d/o are men * Though doesn't est. whether men nowadays suffer fr d/o compared to 10 years ago, or whether males escape attention. * **Still shows AN not exclusively female prob!**