Psychology Units 3 & 4 Flashcards
(59 cards)
CNS and PNS (somatic and autonomic-> parasympathetic, sympathetic)
CNS-> brain & spinal cord. Controls body by processing and responding to sensory input from the PNS.
PNS-> all nerves beyond the CNS. Communicates info from the CNS to organs, glands and muscles.
Branches of PNS: Somatic (voluntary movement) and Autonomic (involuntary movement).
Branches of Autonomic: parasympathetic (rest & digest) and sympathetic (fight or flight)
Role of the spinal cord and spinal reflex
The spinal cord carries info from the PNS to the brain. Towards the brain (sensory messages). From the brain (motor messages).
monosynaptic-> one sensory and one motor neuron
eg. hit leg
polysynaptic-> one or more interneurons connecting the
sensory and motor neurons
eg. moving hand from hot surface
Roles & functions of frontal, occipital, parietal and temporal.
Frontal-> In the front & plays key roles in learning, problem-solving, personality and attention.
Occipital-> At the back & almost entirely devoted to vision.
Parietal-> In the upper half of the cerebral cortex & behind the frontal lobe & it’s main role is to receive somatosensory information (body parts).
Temporal-> In the lower, central area & is important for auditory perception and memory.
Role of Broca’s area, Wernicke’s area and Geschwinds territory.
Broca’s-> articulating sounds & coordinates messages to the areas required for speech production. If damaged= trouble speaking (speak in short sentences).
Wernicke’s-> comprehension of speech. If damaged= struggle to speak in a meaningful way.
Geschwind’s-> Connects Broca’s and Wernicke’s. Important in language processing & has a role in integrating information.
Voluntary movement- roles of primary motor cortex, cerebellum and basal ganglia
Primary Motor Cortex-> intiating & controlling voluntary movement.
Cerebellum-> fine muscle movement and skilled set of movements. Balance & posture.
Basal Ganglia-> initiating and inhibiting movement & motor learning.
Emotion occurs within the limbic system, amygdala and
prefrontal cortex
The limbic system is a set of structures involved in emotions and related behaviours. The hypothalamus (controls emotions and motivated behaviours) and hippocampus (regulation & expression of emotion) are considered to be apart of it. Thalamus (perceiving)
Amygdala-> threat detection, fear & anxiety. How we perceive emotion particularly negative.
Prefrontal Cortex-> Connects brain regions that are involved in the processing and production of emotions. Regulates and modifies emotions & deciphers between good & bad actions.
-> communicate neurotransmission using a diagram
- electrical signal is transported along the axon and arrives at the axon terminal.
- the neurotransmitters are released.
- the neurotransmitters diffuse or drift across the synaptic cleft.
- they anneal or attach to receptors on the dendrite.
- these receptors signal for ion channels on the dendrite to open.
- ions flood onto the dendrites of the post synaptic neuron creating an electrical signal.
Excitatory and inhibitory neurotransmitters
Excitatory-> increase the likelihood of the post synaptic neuron firing.
eg. glutamate (is associated with learning & memory)
Inhibitory-> reduce the likelihood of the neuron firing.
eg. GABA (regulating anxiety)
physical and psychological function of acetylcholine, epinephrine,
norepinephrine, dopamine and serotonin
Acetylcholine-> learning, memory & REM sleep
muscle contractions
Epinephrine-> stress responses (fight, flight, freeze) regulating fear, anxeity and emotional arousal.
increase heart rate, breathing & blood pressure.
Norepinephrine-> stress responses, moods & emotional regulation, increase alertness & arousal.
as hormone-> triggers the release of glucose into blood stream, and increases blood pressure
Dopamine-> thoughts, feeling, emotions & behaviours. reinforcer of behaviours (addictive behaviours)
Serotonin-> mood and social behaviour, eating, sleep, memory, arousal & pain. Body’s sleep wake cycle, and digestion system regulation
Parkinson’s disease and Alzheimer’s disease (symptoms and treatments).
Parkinson’s disease-> affects movement and mental health.
symptoms: slow movement, ridgity & involuntary movement. depression & difficulty sleeping.
causes: drop in dopamine (genetics, environmental factors)
Alzheimer’s disease-> destroys memory & important mental functions
symptoms: gradual severe memory loss, confusion.
causes: low levels of acetylcholine
-> explain the process of visual perception
Reception-> light arrives at photoreceptors on the retina (in the eye)
visible light-> between infrared and ultraviolet.
Transduction-> photoreceptors convert light to electrochemical signals
rods- light intensity
cones- distinguish different wavelengths or frequency’s.
Receptive field area of space where the receptor can respond to the stimulus.
Transmission-> signals are passed along nerves (especially optic) to the primary visual cortex.
Selection-> feature detection cells respond to specific elements in visual information.
Organisation & Interpretation-> The brain arranges the info in a meaningful way aided by the use of visual perception principles.
biological influences on visual perception
ageing- lens becomes less flexible & more opaque.
genetics- colour blindness, inherited visual disorders.
explain psychological influences on visual perception
Perceptual set- a predisposition to perceive stimuli in a specific way according to certain preconceptions
eg. past experience, motivation, context, emotional state
Visual Perception Principles:
- gestalt (figure ground organisation, closure, similarity, proximity)
- depth cues
monocular (only one eye)
->pictorial depth cues (illusion of depth on 2D surfaces- texture gradient, interposition, relative size, height in visual field, linear perspective)
->accomodation (lens changes shape to focus on an image)
binocular (use two eyes)
-> convergence (eyes angled towards each-other to converge on the same object)
-> retinal disparity (info from eyes is similar = object is further away)
- visual constancies (objects stay the same but the image they cast on the retina may change eg. shape & size).
the impact of social influences on visual perception,
Hudson’s - images with pictorial depth cues, asked which animal was closer. People from rural community’s did not consider this factor in their response. Study concluded that 3D interpretation of images was a cultural skill.
Strengths: can be used to evaluate the impact of social influences on visual perception
Limitations: ethnocentric and small cultural groups (only 2)
Deregowski’s-> participants from various cultures struggled to interpret perspective drawings and preferred split view one’s. Another test was asking participants to duplicate an image and see if they made it 2D or 3D. Study concluded that pictorial depth perception has cultural variability.
Strengths: cross-cultural research, replication of Hudson’s study.
Limitations: biased and ethnocentric.
Deregowski, Muldrow & Muldrow-> showing participants from remote villages to say what they saw. Study concluded that perception was mostly based on the person’s familiarity with the object, animal or person depicted in the image, can be learned .
Strength: internal validity (researcher had cultural knowledge)
Limitation: participants got stressed (unethical)
-> analyse the fallibility of visual perception, with reference to the Müller-Lyer,
Ames room, and Ponzo visual illusions, as well as ambiguous and impossible
figures.
Muller Lyer- the arrows (closure)
Ames Room (size constancy fails)
Ponzo Visual Illusions- the railway and two same size lines (linear perspective and size constancy)
Ambiguous figures- bunny/duck (equally likely possibilities for image interpretation)
Impossible figures- triangles (in-consist/ impossible to construct)
-> recognise the duration and capacity of sensory memory and short-term and long-term memory
sensory memory (store for incoming, fleeting sensory information):
- iconic (duration: 0.3 seconds, capacity: unlimited)
- echoic (3-4 seconds, unlimited)
Short Term memory: 5-9 items, up to 30 seconds.
Long Term memory: virtually unlimited x2
-> evaluate two models of memory, including
- the working model of memory (Alan Baddeley and Graham Hitch 1974),
including the central executive, phonological loop, visuospatial sketchpad,
and episodic buffer
-the levels of processing (LOP) model of memory, including the role of
encoding in long-term memory
The Working Model of Memory:
phonological loop (auditory)
⬇
episodic buffer (combines info from different systems & LTM to create a unified memory)
⬆
visuospatial sketchpad (visual)
Linked to all is the central executive limited ability to store information & is a supervisor of the other systems.
The Levels of Processing Model of Memory:
- the more deeply a memory is processed, the longer a memory trace is predicted to last.
Shallow processing
- structural (encoding the psychical characteristics of a stimulus)
- phonemic (the sounds of a stimulus)
- maintenance rehearsal (repetition helps hold it in our active attention and working memory)
Deep processing
- semantic processing = elaborative rehearsal (info is linked/associated to the stimulus)
-> explain how information is stored in long-term memory with reference to implicit
(procedural) and explicit (episodic and semantic) memory
Implicit Memories: unconsciously recalled.
- procedural (how to conduct certain procedures)
Explicit: consciously recalled & declarative
- episodic memories (info about events of our lives eg. what you had for dinner last night)
- semantic memories (not episodic eg. info about how many cm’s are in a m).
-> describe the role of the hippocampus in memory formation and storage
- Forms explicit memory.
- consolidating and retrieving LT declarative memories
-> distinguish between recall, recognition and relearning
The more sensitive the method of retrieval = the more likely it is to demonstrate the presence of the memory.
Recall (least sensitive) eg. short answers Q’s
Recognition (moderately sensitive) eg. multiple choice Q’s
Relearning (most sensitive) eg. improving scores after redoing exams
-> consider the role of the cerebellum in forming and storing implicit (procedural) memories
- Encoding, processing and storing of procedural memories
- classically conditioned responses (a form of implicit memory)
- memory for motor skill tasks
-> describe how information is lost from memory through encoding failure, retrieval
failure and interference effects
Encoding failure: memory was never properly encoded eg. disruption to the consolidating process.
Retrieval failure: memory is present however unable to be retrieved
Interference effects:
Proactive-> old memories inhibit new memories eg. can’t remember new classroom bc you only remember your old one.
Retroactive-> new memories inhibit old memories eg. forgetting old passwords cause you keep making new ones.
->discuss strategies to improve memory, including chunking, rehearsal
(maintenance and elaborative) and mnemonics (e.g. the method of loci and
SQ4R method — survey, question, read, recite, relate, and review).
Chunking: improves the capacity of STM by grouping information together.
Rehearsal:
maintenance-> increase duration of STM
elaborative (making connections) -> improve encoding into LTM
Mnemonics: strategies that aids in memories
the method of loci-> associates a particular location on an imaginary journey
SQ4R: survey (scan for key features), question (consider questions for when finished), read (actively read text), recite (speak aloud), relate (relate info) & review (use summaries).
-> compare classical conditioning (Ivan Pavlov 1897/1902), operant conditioning
(BF Skinner 1948) and social learning theory (Albert Bandura 1977)
They are all forms of learning in Psychology. Classical conditioning involves associating two stimuli, operant conditioning focuses on the consequences of behavior, and social learning theory emphasises observational learning.