Pulmonary Artery Catheters 2 Flashcards

(37 cards)

0
Q

What are lung problems?

A
  • non cardiogenic pulmonary edema (aspiration, gram - sepsis, ARDS)
  • excessive PEEP level
  • pulmonary emboli
  • alveolar hypoxia leading to pulmonary artery constriction (COPD)
  • general shunt hypertension
  • shunt
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1
Q

What are right heart problems?

A
  • tricuspid stenosis or regurgitation
  • right sided myocardial infarction
  • cardiac tamponade
  • ruptured septum
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2
Q

What are left heart problems?

A
  • cardiogenic pulmonary edema
  • mitral regurgitation/stenosis
  • aortic stenosis
  • MI to left ventricle
  • constrictive pericarditis
  • patient ductus arteriosus
  • arteriosclerotic heart disease
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3
Q

What should PADP be compared to PCWP so it increases PVR?

A

> 3-5

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4
Q

What causes an increase in PVR?

A
  • acidosis
  • hypercarbia
  • hypoxia
  • less common: pulmonary emboli, alveolar septal defect, surgical removal
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5
Q

What are complications of vein cann?

A
  • local infection
  • bleeding and hematoma
  • pneumo/hemothorax
  • injury to trachea/thoracic duct
  • damage to vein
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6
Q

What are complications during placement?

A
  • cardiac arrhythymias
  • local infections, PA rupture
  • pulmonary emboli
  • balloon rupture and air emboli
  • intercardiac knotting
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7
Q

What are problems that disrupt accuracy in a waveform?

A
  • dampened (decrease in amp due to air or kink)
  • catheter whip (movement of catheter)
  • pressure values (transducer not in correct place)
  • respiratory effect (low press during inhalation)
  • PEEP
  • zones
  • migration
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8
Q

How does peep affect the waveform?

A
  • causes zones to shift
  • compliant lungs transmit more peep
  • 5cmh2o peep = 1cmh2o added to measurement
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9
Q

What determines cardiac output?

A
  • stroke volume

- heart rate

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10
Q

What factors affect stroke volume?

A
  • preload
  • contractility
  • afterload
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11
Q

What is the normal cardiac output?

A

75 x 75 = 5400

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12
Q

What is preload?

A

The stretching of muscle fibers in the ventricle

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13
Q

What is contractility?

A

The inherent ability of the myocardium to contract normally. The greater the stretch the more forceful the contraction

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14
Q

What is afterload?

A

The pressure that the ventricular muscles must generate to overcome the higher pressure in the aorta to get the blood out of the heart

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15
Q

How does heart failure affect preload?

16
Q

How do adrenergic drugs affect left ventricular afterload?

17
Q

How do calcium channel blockers affect contractility?

18
Q

What are vascular factors?

A
  • exercise and venous blood flow
  • peripheral vascular resistance (high pressure and increase in left heart work)
  • blood volume (decreased then low co, increased them high co)
19
Q

How does an increase in contractility affect the heart?

A
  • increase in CO

- decrease pressure

20
Q

How does a decrease in contractility affect the heart?

A
  • decreased CO

- higher pressure

21
Q

How does increase blood volume affect the heart?

A
  • increased CO

- increased pressure

22
Q

How does low blood volume affect the heart?

A
  • decrease CO

- decreased pressure

23
Q

How does uncompensated failure affect the heart?

A
  • decreased pressure

- decreased CO

24
How does compensated failure affect the heart?
- still below normal - increased pressure - increased CO
25
What does hypovolemia cause?
Low pressure, low CO
26
What does sepsis cause?
Low pressure, high CO
27
What does hypervolemia cause?
High pressure, high CO
28
What does CHF cause?
High pressure, low CO
29
How do you know if therapy is effective?
Does intervention change the relationship between intravascular space and blood volume?
30
What are left physical findings?
- dyspnea - crackles - orthopnea - JVD - hepatomegaly - pedal edema
31
What are physical findings of the right heart?
- pedal edema - PCWP will not be as high - no crackles - no pulmonary veins/capillary issues
32
What does an increase in preload cause?
- increased fluid volume | - vasoconstriction
33
What does decreased preload cause?
- hypovolemia | - vasodilation
34
What does increased afterload cause?
- hypovolemia | - vasoconstriction
35
What does decreased afterload cause?
-vasodilation
36
What is the primary goal of all hemodynamic therapeutic intervention?
To optimize cardiovascular function in order to ensure adequate tissue oxygenation