Pulmonary Disorders Flashcards

1
Q

Pneumonia

A

Inflamm of lung parenchyma d/t bacterial, viral, fungal infection

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2
Q

Pneumonia risk factors

A
  • Advanced age
  • Long-term care residents
  • Smoking
  • Chronic resp disease
  • Immunocompromised
  • Prolonged immobility
  • Aspiration of stomach content (AMS can cause this)
  • Prolonged NPO status
  • Diminished consciousness, gag reflex, swallow reflex
  • *Hospitalization longer than 48 hrs
  • Recent abx therapy
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3
Q

Pneumonia clinical manifestations

A
  • *Fever
  • Tachypnea
  • *Chills
  • Cough, non-prod/prod
  • *Pleuritic chest pain
  • *Fatigue
  • *Myalgias, arthralgias

Severe: purulent/blood tinged sputum, hypotension, dysrhythmia

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4
Q

Types of pneumonia

A
  • Community-acquired
  • MRSA community-acquired
  • Hospital-acquired (occurs 48 hrs after admission)
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5
Q

Pneumonia labs

A
  • Elevated WBC (>10, 500)
  • Elevated C-reactive Protein (>1)
  • Elevated bands (>5%)
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6
Q

Pneumonia nursing action

A
  • Administer *humidifed O2 as ordered
  • Administer meds: antibiotics
  • *Pulmonary hygiene: incentive spirometer, ambulation, make sure pt coughing up secretions
  • Pt positioning
  • *Monitor intake & output
  • *Ensure adequate nutritional support (most pts tired so don’t wanna eat + difficulty breathing)
  • Activity
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7
Q

Pneumonia pt teaching

A
  • *Hand hygiene
  • Adequate rest, nutrition, fluid intake
  • *Understand S&S of worsening resp status
  • Pneumonia vaccine
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8
Q

Obstructive sleep apnea

A

Muscle tone relaxes during sleep –> can’t overcome increase in resistance –> airway collapse

  • Hypoxia
  • Hypercapnia
  • Apnea
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9
Q

OSA risk factors

A
  • Male
  • *Obesity
  • Smoking
  • Alc use
  • *Age 40-45
  • Craniofacial or upper airway abnormalities

Other:
- Menopause
- Atrial fibrillation
- Nocturnal dysrhythmia
- Type 2 DM
- HF
- Pulm hypertension

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10
Q

OSA clinical manifestations

A
  • Loud snoring
  • Snorting
  • Witnessed apnea
  • Recurrent waking during sleep
  • Choking
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11
Q

OSA risks what?

A

Increased risk of CV disease bc excessive inflamm process d/t decreased blood flow. Also cause scarring.

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12
Q

OSA treatment (supportive)

A
  • CPAP (pressure keeps airway open)
  • Nonsupine sleeping
  • No alc or sedative before bed
  • Oral appliance to forward tongue to keep airway open
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13
Q

OSA treatment (surgery)

A
  • Tonsillectomy
  • Adenoidectomy
  • Uvulopalatopharyngoplasty
  • Septoplasty
  • Nasal polypectomy
  • Tongue reduction
  • Epiglottoplasty
  • Bariatric surgery (reduce weight)
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14
Q

OSA pt teaching

A
  • Disease process
  • Instruct pt on CPAP + meds
  • Instruct pt on weight reduction
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15
Q

Asthma

A
  • Affect bronchial airways, resulting in airway obstruction & bronchial hyperresponsive
  • Cause bronchospasm or contraction of small airways –> inflamm
  • Triggered by exposure to inhaled irritants
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16
Q

Asthma clinical manifestations

A
  • Chest tightness
  • Wheezing (concern when on inspiration & expiration)
  • Dyspnea
  • Coughing
  • Increased sputum
  • *Inability to speak in full sentences bc not getting enough air
  • Tachycardia, increased RR
17
Q

Asthma medications

A
  • Inhaled corticosteroids (Flovent, Pulmicort) = long term
  • Bronchodilator (Albuterol) = rescue
  • Anticholinergics (Sprivia - tiotropium)
  • Leukotriene receptor antagonist (Montelukast)
18
Q

Inhaled corticosteroid action

A

Anti-inflammatory
Decrease mucuous prod & swelling, airways becomes less sensitive to triggers

19
Q

Bronchodilator action

A

Eg. beta-2 adrenergic agonist
Relax bronchial smooth muscle leading to open airways & less obstruct

20
Q

Anticholinergic action

A

Relax muscle around larger airway

21
Q

Leukotriene receptor antagonist action

A

Block leukotriene

22
Q

COPD

A

Results in airflow limitations that are progressive and not fully reversible.

23
Q

Types of COPD

A
  • Emphysema
  • Chronic bronchitis
24
Q

Emphysema

A
  • Alveolar destruction
  • CO2 can’t leave and O2 can’t enter: ineffective gas exchange
  • Loss of lung elasticity causes air trapping and distension in alveolar
25
Q

Chronic bronchitis

A
  • Inflamm of bronchi and bronchioles
  • Small vessels are affected first
  • Increased mucus production, causing vessel wall thickening and airflow obstruction
26
Q

COPD nursing assess

A
  • Vital signs
  • *Lung sounds: crackles or wheezes
  • Pursed lip breathing: keeps airways open for gas exchange
  • *Cough
  • Dyspnea (subjective finding)
  • Weight loss d/t pt fatigue from breathing & not eating
27
Q

COPD nursing action

A
  • Administer meds as ordered: bronchodilators, anticholinergics d/t keeping airway open
  • Provide O2: maintain SpO2 > 90%, continuous O2 for SpO2 below 88% or PaO2 below 60 mmHg
  • Position: semi-fowlers to maximize lung expansion
  • Provide small frequent meals: less energy consumption, no distension of stomach which can prevent maximal lung expansion
28
Q

What to be careful about COPD O2 administration?

A

Don’t give too much O2 becuz used to O2 saturation in the 80s. High O2 will decrease respiration drive.

29
Q

COPD pt teaching

A
  • Breathing technique: pursed lip
  • Pacing of activities
  • Smoking cessation
  • Med regiment: inhaler use, regimen, schedule
  • Vaccine prophylaxis: pneumococcal, influenza, covid
  • Exacerbation recognition
  • Coping: depression, social, isolation, lifestyle changes