Pulmonary pathophysiology Flashcards

(52 cards)

1
Q

What pathology presents: swelling of the bronchi secondary to infection.
Sympt: fever, cough, wheeze

A

Acute Bronchitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

Tx for Acute Bronchitis

A

Antibiotics, Bronchodilators

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What pathology presents: polyps block airways

Symp: low grade fever, dyspnea, cough, decrease FEV1/FVC

A

Bronchiolitis Obliterans

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Tx for bronchiolitis oblitierans

A

antibiotics, bronchodilators, prevent exposure, O2

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

In adults, bronchiolitis obliterans can be a complication:

A

…of infection or due to toxic fumes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What pathology presents as a collapse of part/all lungs secondary to collapse of alveoli

A

Acetilitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

What pathology presents as a collapse of part/all lungs secondary to air/pressure build up around lung

A

Pneumothorax

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

Which way will the trachea shift towards:

A

Toward the lung w/ less pressure & away from the greater pressure.

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

Describe Cor pulmonale

A

R ventricular heart failure tied w/ pulmonary problems

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What pathology presents as swelling of the bronchi & bronchioles secondary to smoking, allergies, air pollution.
Symp: productive cough >3 mo for 2 consecutive yrs, wheeze, polycthemia, R ventric failure

A

Chronic Bronchitis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

Tx for chronic bronchitis

A

IV fluids, antibiotics, bronchodilators, O2, corticosteroids; if R vent failure–> diuretics, digitalis

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

Who is more likely to die:
Both Roan and Kristie Lee are 80 y/o and have chronic bronchitis. Roan stays in bed bc of her frail bones while Lee still plays soccer w/ the grandchildren?

A

Roan- bc 50% pt are 4x more likely to die vs active pt

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

Why is there possibility of having polycthemia as a result from Chronic Bronchitis

A

decreased O2–>Kidney compensates–> increase RBC polycthemia–> increase risk of heart attack

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What pathology presents as abnormal permanent enlargement of airspaces & destruction of terminal bronchioles
Symp: chronic cough, wheeze, SOB, decreased endurance & RR, risk of embolism

A

Emphysema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Tx of Emphysema

A

Mucolytics, non-catecholamines (ephedrine, albuterol) , lung resection or transplant, bronchodilators (Theophylline)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

If an emphysema pt cant get out of bed, would you give them ankle pumps? Why or why not.

A

bc there is increased risk for DVT in pt w/ emphysema–> increased HCT–> embolism risk–> increased risk for DVT

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

What are the 5 common causes of death of pts w/ emphysema

A

CHF, resp failure, pneumonia, bronchiolitis, pulmonary embolism

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Who would most likely have centrilobular emphysema:
Will (who smokes, and has R vent failure and polycythemia)
Nicole (who drinks, and has L atria failure)

A

Will- common in Males and hx w/ chronic bronchitis, rare in non-smokers

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Which is most common Emphysema: Centrilobar or Panlobular

A

Centrilobular is 20x more common than Panlobular

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

What pathology presents as inflammation, edema, thicken bronchiolar walls, destruction of bronchioles. Affects Upper Lobes & Superior Lower Lobes

A

Centrilobular Emphysema

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

What pathology presents as destructive enlargement of alveoli; Bronchioles prone to collapse–> HIGH risk of pneumothorax; Affects Lower Lobes

A

Panlobular Emphysema

22
Q

What pathology presents as narrowing/occluded lumen of the airways by a combination of smooth mm spasm, inflammation of mucosa & overproduction of mucus
Symp: wheeze, SOB, decreased endurance, waking at night, lung sounds gradually decrease

23
Q

Tx for Asthma

A

Non-catecholamines, bronchodilators, steroids, O2

24
Q

Would you suggest that Bronte, who has asthma, to do her HEP in the evening? why or why not

A

Asthma can occur 6-8hrs after submax exercises is stopped.

25
What pathology presents as dilation of medium sized bronchi/bronchioles (usually due to necrotizing infection & something inhaled). Symp: productive cough, recurrent infections emacitation, clubbed fingers; *Bonus Name Symp for chronic
BronchIECTASIS; | *chronic: apnea & R ventricular failure
26
Tx of bronchiectasis
antibiotics, bronchodilators, mucolytics, expectorants
27
What are the types of bronchiectasis & describe them. | Which is the most common*.
- Cylindrical*- causes uniform dilation - Varicose- larger > cylindrical - Sacular- outgrowths like balloons
28
What pathology presents as proliferation of fibrous CT w/in the lungs. Symp: fatigue, SOB w/ exertion, UNproductive cough
Fibrosis
29
How many different types of Fibrosis are there. Name at least 2.
- 130 known types - Diffuse Interstitial Pulmonary Fibrosis - Cystic Fibrosis
30
What pathology is possibly due to poor wound healing at the site of damage to the tissues; Thickening of alveolar wall progressing to scarring.
Diffuse Interstitial Pulmonary Fibrosis
31
What pathology is likely to be genetic. It causes heightened mucus secretions systemically. Life expectancy is now 40 y/o
Cystic Fibrosis
32
Tx for CF
anti-funal, antibiotics, mucolytics, corticosteroids, immunesuppressants
33
What pathology has Symp: fatigue, SOB, unproductive cough.
Eosinophilia
34
Name the 3 Eosinophilia, and out of them which one is self limiting and which one is RARE in Male 30-40's.
1. Simple pulmonary eosinophilia- self limiting 2. Prolonged pulmonary eosinophilia leads to fibrosis. 3. Eosinophilic Granuloma is Rare in Male 30-40's
35
Tx for Eosinophilia
simple P Eo- none prolonged p Ep- corticosteroids, bronchodilators, & antibodiotics Eosinophilic Granuloma- JUST sympt relief =(
36
What pathology presents as alveoli fill w/ lipid rich materials; etiology is unknown. Symp: Male 30-50y/o, SOB, wt loss, hemoptysis, chest pain
Pulmonary Alveolar Proteinosis
37
Tx for pulmonary alveolar proteinosis. | PT Tx.
- whole lung lavage (running saline via the lower lung) | - percussion
38
What pathology presents as affecting multiple organs, lungs most often. Symp: respiratory constriction, hilar adenopathy (enlarged mediastinum lymphnodes), cysts, bullae (bubbles)
Sarcoidosis
39
Tx for Sarcoidosis
corticosteroids
40
What pathology presents as infection that persists in lungs, immune system attempts to fight & fiberous tissue results. Symp: asymptomatic during primary infection--> later develop unproductive cough, fever crackles; * Bonus Xray shows what.
Tuberculosis; Xray shows fluffy shadows
41
What is the mode of transmssion of tuberculosis.
- Incubation is 2-12 wk - blood, kidney, bone, brain, & lymph - Difficult to dx in children due to non-specific symptoms such as failure to thrive, cough, malaise
42
Tx for TB
1st: prevention- keep from airborn pathogens 2nd: riphampin & isoniazid (INH) for 6 mo to suppress infection
43
What pathology presents as Acute Inflammation of the lungs when the airways are not kept clear of infectious agents Symp: productive cough, SOB, fever, crackles, increased WBC count, (+) sputum *Bonus: Whats the prognosis
Pneumonia; *6th leading cause of death.
44
Tx of pneumonia
medicines depend on the cause of the pneumonia; early mobilization; chest PT
45
What pathology presents as- can be from external injury or systemic infection. Symp: rapid onset of respiratory failure unresponsive to oxygen; pulmonary edema, hyaline cartilage formation and hemorrhage, crackles, pink frothy exudate
ARD-Acute Respiratory Distress (ALI- acute lung injury)
46
Factors that predispose pt to ARD's/ALI
mechanical ventilators, pneumonia, sepsis, near drowning, blood transfusions, aspiration
47
Tx for ARD
ventilator, meds if indicated, restrictive breathing patterns
48
3 Phases of ARD/ALI & describe what happens with them
Exudative phase- pulmonary edema, hyaline cartilage formation and hemorrhage. Cellular proliferation- influx of nutraphil--> blocks Finbroproliferation- inj area replaced by fibrin
49
What is Pleurasy
inflammation of pleura (viral/ bacterial); sharp chest pain worsens w. breathing
50
What pulmonary pathology has Symp: SOB, non-productive cough, hemoptosis
Lung CA
51
Tx of Lung CA
Depends on stage of disease, radiation, chemo, sx
52
2 types of CA & describe them
Small cell- tumors are smaller than mature lymphocytes; 25% deaths Non-small cell- tumers are larger; 75% of deaths, usually moderate-advanved by the time it is found