Quiz 2 Flashcards

1
Q

probe ear principle

A

AR are absent when there is a conductive pathology in the probe ear

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

poor word recognition score is indicative of

A

retrocochlear loss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

best way to identify conductive loss with reflexes

A

ipsilateral

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

effect of 5 dB ABG on reflexes with CHL in the probe ear

A

reflex has 50/50 chance of being observed

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

effect of a 10dB ABG (or greater) with CHL in the probe ear

A

reflex may be obscured as much as 80% of the time

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

reflexes with a unilateral conductive loss

A

all but ipsi on the side without the conductive component will be affected

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

stimulus ear principle (in other words the stimulus ear is the one with the CHL)

A

conductive loss in the stimulus ear attenuates the stimulus by the amount of the ABG

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

effect of ABG <30dB with CHL in stimulus ear

A

elevated reflexes–50% chance of recorded reflexes at 27dB ABG

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

effect of ABG>30dB with CHL in the stimulus ear

A

absent reflexes

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

what do you need to do to overcome an ABG?

A

raise the stimulus level by the amount of the ABG

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

possible causes of AR in unilateral CHL

A

1) ossicular discontinuity *medial to the stapedial muscle insertion
* or a fribrous tissue that enables reflex contraction to be conveyed to the TM
2) only the crus of the stapes disconnected
3) cholesteatoma that does not impinge on the TM or ossicular chain (localized)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

what is more sensitive to ME changes? AR or behavioral audiometry

A

AR

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

two types of facial paralysis

A

lower motor neuron lesion

upper motor neuron lesion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

lower motor neuron lesion

A

all the muscles of the same side of the face are affected

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

upper motor neuron lesion

A

affects only the muscles on the lower half of the contra side of the face

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

lesion at or proximal to (beyond) the stapedius nerve

A

abnormal AR when the probe is in the affected ear

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

lesion distal to the stapedius nerve

A

normal AR threshold when the probe is in the affected ear

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

VII nerve lesions

A

such as Bell’s Palsy

  • according to the course of the disease
  • *AR may start absent, proceed to elevated and then to normal as the disorder resolves
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

AR for sensorineural hearing loss depends on the __________

A

degree of hearing loss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

if sensorineural loss with thresholds <50-55dB

A

normal ARTs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

if sensorineural loss with thresholds between 50-80dB

A

elevated ARTs with low SLs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

if thresholds are > or = to 80dB

A

absent ARTs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Stimulus ear principle with cochlear SNHL

A

Any time the stimulus is in the ear with the loss, there will be abnormal AR
(This would give a diagonal pattern of AR)
$ depends on the degree of hearing loss

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

Stimulus ear principle with retro cochlear SNHL

A

Abnormal AR when stimulus is in ear with loss

1) usually absent AR regardless of the degree of loss
2) or present AR but elevated with high SLs and
3) abnormal r flex decay (positive decay)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
What constitutes as elevated ART?
An ART above 100 unless there is substantial hearing loss
26
Intraaxial lesion
Internal to pia mater Brain parenchyma Cells:neurons and glial cells Location:involving brain tissue, cells, neurons Cortex, deep white matter, deep gray matter
27
Extra axial lesion
External to the pia mater Meninges, nerve sheath, outside brain stem Schwannoma Locations: outside brain tissue, subarachnoid, subdural, epidural, calvarium (skull base), scalp (soft tissues)
28
Stimulus ear principle with extra-axial lesion
Resembles Ipsi SNHL Consider the degree of loss to interpret absent or elevated AR with high SLs *basically everything you present through the ear with the loss, you will get abnormal AR * pathologies:acoustic neuroma, CPA tumor, extra-axial tumor
29
AR with intraaxial lesions
These lesions are central (brain stem) and lead to bilateral normal Ipsi and bilateral absent contra (crossed pathways) the reflex can't travel from one side to other because problem with communication between sides Examples: demyelination disease, neuromuscular diseases
30
Positive reflex decay
If the response falls to 50% of its initial magnitude during the 10 second stimulus
31
Controversy with reflex decay
5-10 seconds, 500 vs 1000hz (stronger at 5 seconds and 500hz), sensitivity improves if you consider 10 sec and both frequencies * not everyone has decay at 2000 and 4000 so we don't test them
32
Using ART to predict hearing thresholds
ART for pure tone is independent of HL up to 50-60dB HL, then increases as a function of HL ART for BBN increases with increasing SNHL up to 50-60db HL, then plateau In other words it is hard to use ART to predict pure tone thresholds because of SNHL
33
Most proximal auditory center in pathway
Is the highest which is the superior oil art complex
34
Common causes of SNHL
``` Ménière's disease Noise induced hearing loss Infections Trauma Ototoxicity Presbycusis Autoimmune disease Genetic ```
35
Common causes of neural SNHL
Nerve tumors Cochlear neuritis (infection) Other disease processes such as diabetic neuropathy or demylinating disease
36
One test which actually assesses the function
Speech testing | If speech scores are lower than hearing loss then it is retrocochlear
37
Limitations of AR testing for differential diagnosis
AR may be absent in cases of SNHL above 60dB HL Therefore can't differentiate between cochlear and retrocochlear loss With AR decay there is controversy over what is actually abnormal decay
38
Three categories of behavioral tests for seeing if it's a retrocochlear loss
1) word recognition tests such as PI/BI function 2) recruitment tests: Loudness balance which is ABLB or AMLB Short increment sensitivity index (SISI) 3) adaptation tests Tone decay (TD) Bekesy audio
39
Objective tests for retrocochlear
Such as OAE and ERA Maintain high specificity for cochlear disorders Show higher sensitivity to 8th nerve problems
40
Threshold tone decay
A reduction in the ability to hear a barely audible sustained tone Use 500 and 1000hz
41
Supra threshold tone decay
A loss of audibility as a result of presenting a sustained tone at a high presentation level Use high HL such as 70dB HL
42
Carhart tone decay
Present a continuous tone at threshold (or 5dB above) for 1 minute Use 500 and 2000 If they don't hear the tone, raise intensity by 5dB and reset stopwatch, continue until they hear tone for a full minute Measure dB difference start to end and time to Inaudibility
43
Tone decay considered normal
No decay which is 0-10dB from starting level
44
Cochlear pathology amounts of tone decay
Mild pathology would give 15dB in 60 seconds | Moderate pathology would give 20-25dB in 60 seconds
45
Retrocochlear amount of tone decay
Marked or rapid which is greater than 30dB
46
Three types of perceptual phenomenon with tone decay
1) loss of tonality and audibility simultaneously meaning distortion occurs after increase in level due to tone becoming inaudible 2) loss of tonality but remains audible meaning it becomes distorted 3) loss of tonality and audibility but ones tone is withdrawn they know immediately (withdrawing gives neurons break so when it turns back on they can hear it again but it decays quickly)
47
Olsen and noffsinger procedure
Tone decay modification (test of choice) Start at 20dB above threshold Continue until the is heard for one minute Significant decay is above 15dB Disadvantage is that you can't test the rate of decay since you started at 20dB you don't really know when decay began
48
Rosenberg procedure
Shortened tone decay test Tone is presented at threshold and raised 5dB every time it's not heard but only for a total of 1 minute Disadvantage: could miss some retrocochlear due to early test termination
49
Green's procedure
Increase in intensity every time the pt hears change in tonality instead of absence of tone Advantages:increases sensitivity to cochlear vs retrocochlear lesion Reduces missing rate (Distortion is auditory brain stem problem)
50
Owens procedure
Tests for amount and rate of decay Begin at 5dB SL If tone decay before the 60sec period, the stimulus is discontinued for 20 sec before introduction of the tone at 5dB increment Procedure continues until the stimulus is perceived for 60 seconds or a level of 20dB SL is r ached
51
Supra threshold adaptation test (STAT)
Begins at a high level like 100dB HL Total time of 1 minute (cochlear=still heard, retro=decay) Disadvantage: ignores amount of decay, ignores rate of decay, missed cases because of high intensity starting level *this is more of a screening procedure
52
Pros and cons of objective site of lesion testing
Pros: objective so less human error, higher sensitivity than behavioral tests Cons: more expensive, low availability, can determine site but not type of loss (specific etiology)
53
Hyperacusis
Hypersensitivity to normal sounds
54
Phonophobia
Fear of normal sounds resulting in hypersensitivity to them Can occur with normal hearing or with hearing loss
55
Place principle of recruitment
Outer hair cells are more sensitive than inner hair cells | When OHC are damaged, then when intensity is increased the IHC are excited with a concomitant rapid increase in loudness
56
Summation principle of recruitment
The total number of cells excited | As intensity increases, larger areas of the basilar membrane are stimulated
57
Loudness decruitment
A slow growth of loudness aka a summation deficit
58
ABLB
Alternate binaural loudness balance Unilateral HL Compare loudness growth between the same frequencies for the two ears
59
AMLB
Alternate monaural loudness balance Bilateral symmetrical HL with some normal hearing at some frequencies Compare loudness growth between two different frequencies in the same ear
60
What frequencies to test for loudness balancing
500, 1000, 2000, not 4000 because there will be recruitment
61
When might you see recruitment with VIII nerve lesion?
Reduced cochlear blood supply Size of tumors Test contamination: coincidental high frequency hearing loss (cochlear loss)
62
Using the poor ear as reference ear for ABLB
Determines the presence or absence of recruitment Is quick (requires less balances) to make a diagnosis Easy
63
Using the good ear as a reference ear for ABLB
Determine the presence or absence of recruitment and plot loudness growth function More sensitive to changes in intensity =less variable Requires more balances
64
Difference limen for intensity
DLI is smallest change in intensity of a pure tone that can just be detected Normal listeners have problems close to threshold Cochlear loss have smaller DLI for pure tones due to recruitment, this is premise of the SISI test