Quiz II study Flashcards

(33 cards)

1
Q

Causes of hyponatremia

A

Medications: amiodarone, angiotensin II receptor blockers, angiotensin* converting enzyme inhibitors, desmopresin

Heart, kidney, and liver problems

SIADH

Chronic, severe vomiting or diarrhea causing dehydration.

Too much fluid intake

hormonal changes

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2
Q

BUN

A

7 to 20 mg/dL

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3
Q

CO2

A

23 to 29 mmol/L

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4
Q

Creatinine

A

0.6 to 1.2 mg/dL

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5
Q

Glucose

A

64 to 100 mg/dL

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6
Q

Chloride

A

95 to 105 mEq/L

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7
Q

Potassium

A

3.5 to 5 mEq/L

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8
Q

Sodium

A

135 to 145 mEq/L

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9
Q

Calcium

A

9 to 11 mg/dL

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10
Q

S/S Hypokalemia

A

Impaired repolarization - flattened T wave, depressed ST segment, and presence of a U wave.

P waves peak and the QRS complex is prolonged

Ventricular dysrhythmias

Basic: constipation, heart palpitations, fatigue, muscle weakness, and tingling and numbness

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11
Q

S/S Hyperkalemia

A

Tall, peaked T waves

depolarization decreases: loss of P waves, a prolonged PR interval, ST segment depression, and widening QRS complex.

Basic: fatigue, confusion, tetany, muscle cramps, paresthesias, and weakness.

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12
Q

Etiology of Hypokalemia

A

Excessive loss of potassium

GI loss: vomiting, diarrhea, or laxative use

Renal loss: diuresis or low magnesium levels (stimulates renin and aldosterone release, resulting in potassium excretion)

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13
Q

Diet - Foods with Potassium

A

Lentils
potatoes
bananas
avocados
spinach/broccoli
dried fruit (raisins, apricots)

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14
Q

Metabolic acidosis impact

A

Renal: kidneys can’t properly filer acids from bloodstream

Raspatory

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15
Q

Metabolic Acidosis

A

Normal
Ph: 7.35-7.45
PaCO2 - 35-45
HCO3 - 22-26

Ph:7.28
PaCO2: 36
HCO3: 19

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16
Q

Lasix indications

A

Edema associated with CHF

Cirrhosis of the liver

Renal disease and nephrotic syndrome

17
Q

Lasix side effects

A

dehydration

electrolyte imbalance

metabolic alkalosis

18
Q

MOA of Lasix

A

Inhibits the reabsorption of sodium and chloride from the loop of Henle and distal renal tubule.

Increases renal excretion of water, sodium, chloride, mag, potassium and calcium.

19
Q

Pathology of Pulmonary Embolism

A

Occurs when a blood clot gets stuck in an artery in the lung, blocking blood flow to part of the lung.

20
Q

Etiology of Pulmonary Embolism

A

Blocked artery in the lungs

21
Q

S/S Pulmonary Embolism

A

dyspnea
hypoxemia
tachypnea
cough
chest pain
hemoptysis
crackles/wheezing
tachycardia
syncope

22
Q

Interventions for PE

A

Supplemental O2, intubation if needed
Monitor labs: hgb, aPTT, INR
Balance activity and rest

23
Q

PE treatment

A

Fibrinolytic agent
Heparin
Warfarin
analgesia

24
Q

Nurse action during intubation

A

Establish O2 support, prepare for a tracheostomy

25
DOPE
displacement or obstruction of the ET, pneumothorax, and ventilator or equipment failure
26
Nurse responsibility if intubated patients sats drop
Disconnect the ventilator and administer high flow 100% oxygen (FiO2) using a bag-valve-mask Assess using MASH approach Movement of chest during ventilation Arterial saturation (SaO2) Skin color Hemodynamic stability
27
What can a UAP do with vented patients
Obtain vitals Measure I/O perform bedside glucose testing
28
Zantac and critically ill patients
Used to prevent stress ulcers
29
Treatment for large PE
heparin bolus - followed by continued intravenous infusion thrombolytics TPA
30
Refractory Hypoxemia
A severe breathing problem that can happen due to mechanical ventilation and acute respiratory failure. Often occurring in the context of acute respiratory distress syndrome (ARDS)
31
Actions to take with PE patients who are anxious
Therapeutic communication and pain medication
32
VAP prevention
VAP bundle: HOB elevation between 30-45 degrees daily sedation vacation (readiness to wean assessment peptic ulcer disease prophylaxis DVT prophylaxis Oral care with chlorhexidine q8hrs Hand hygiene
33
Promoting comfort in ventilated patients
Suction as needed assess pain and sedation needs sedation vacation