RCP avanzada Flashcards
(46 cards)
dose, route and uses of adenosine in adults
use: narrow PSVT/SVT, wide QRS tachy
avoid: irregular wide QRS
dose: 6mg IV bolus (followed by flush with 20mL of saline), may repeat with 12 mg in 1-2 mins
notes: there must be continous cardiac monitoring, causes flushing and head heaviness
dose, route and uses of amiodarone in adults
use: VF / pulseless VT, VT with pulse, tachy rate control
dose: 300mg dilute in 20-30ml (may repeat 150mg in 305 mins)
notes: anticipate hipotension, bradicardia, GI toxicity. There must be continouse cardiac monitoring. Has a very long 1/2 life (up to 40 days).
DONT USE IN 2º OR 3º HEART BØ, DONT ADMIN IN ET TUBE
dose, route and uses of atropine in adults
uses:
- symptomatic bradicardia: 0.5mg IV/ET every 3-5mins, max dose 3mg
- specific toxins/overdose (ex: organophosphates): 2 - 4 mg IV/ET
notes: there must be cardiac and BP monitoring, DONT USE IN GLAUCOMA OR TACHIARRYTHMIAS, minimum dose 0.5mg
dose, route and uses of dopamine in adults
uses: shock / CHF
dose: 2 - 20 mcg/min, titrate to desired BP
notes: fluid resuscitation first, there must be cardiac and BP monitoring
dose, route and uses of epinephrine in adults
cardiac arrest:
- initial: 1mg (1:1000) or 2-2.5mg (1:1000) ETT every 3 - 5 min
- maintain: 0.1 - 0.5 mcg/min, titrate to desired BP
anaphylaxis:
- 0.3 - 0.5 IM, repeat every 5 min as needed
symptomatic bradycardia/shock:
- 2 - 10 mcg/min infusion
- titrate to response
notes: there must be continous cardiac monitoring, give via ventral line when possible
dose, route and uses of lidocaine in adults
**recommended when amiodarone isn’t available
cardiac arrest: VF/VT
- initial: 1-1.5mg/kg IV loading
- second: half of first dose in 5 - 10 min
- maintain: 1 - 4 mg/min
wide complex tachicardias with pulse:
- initial: 1 - 1.5mg/kg IV
- second: half of first dose in 5 - 10min
- maintain: 1 - 4mg/min
notes: must have cardiac and BP monitoring, rapid pulse can cause hipotension and bradicardia, use with caution in kidney failure, calcium chloried can reverse hipermagnesemia
dose, route and uses of magnesium sulfate in adults
cardiac arrest / puseless torsades: 1 - 2 gm diluted in 10ml
torsades de pointes with pulse: 1 - 2 gm IV over 5 - 60 min, maintain 0.5 - 1gm/hr IV
notes: must have cardiac and BP monitoring, rapid pulse can cause hipotension and bradicardia, use with caution in kidney failure, calcium chloried can reverse hipermagnesemia
dose, route and uses of procainamide in adults
use: wide QRS tachycardia, preferred for VT with pulse (stable)
dose: 20 - 50mg / min IV until rhythm improves, hypotension occurs, QRS widens by 50% or max dose is given
mas dose: 17mg/kg
drip: 1 - 2 gm in 250 - 500ml ate 1 - 4 mg/min
notes: must have cardiac and BP monitoring, caution with acute MI, may decrease dose with kidney failure, DONT COMBINE WITH AMIODARONE, DONT USE IN PROLONGED QT OR CHF
dose, route and uses of sotalol in adults
use: tachyarrhytmia, nomonorphic VT, 3rd line antiarrithmic
dose: 100mg (1.5mg/kg) IV over 5 min
notes: DONT USE IN PROLONGED QT
parameters that tell you oxygenation is adequate
oxygenated inspired air is best provided via a tight fitting oxygen reservoir face mask with a flow rate of at least 11 L/min
pulse oximetry must be used at all times
- doesn’t measue PaO2
- measured saturation >95% by pulse oximetry is strong coroborating evidence of adequate peripheral arterial oxygenation (PaO2 >70mmHg)
- limited usefulness in pt with severe vasoconstriction & carbon monoxide poisoning because it can’t distinguis oxyhemoglobin from carboxyhemoglobin or methemoglobin
- profound anemia (hb <5g/dl) and hypothermia (<30ºC or <86ºF) decrease the reliability of the technique
how can you tell that the ET tube is in the right place
- hearing equal breathing sounds bilaterally and detecting no borboygmi in epigastrium
the presence of CO2 in exhaled air indicates that airway has been successfully intubated, but doesn’t ensure the correct position of the tube
proper position of the tube is confirmed by chest X-ray
how many joules should a shock be admin. when using a monophasic defibrillator
360 J
must use same energy doe on subsequent shocks
which kind of defibrillator is more effective for terminating a fatal arrythmia
the biphasic defibrillator
- use the manufacturer’s recommended energy dose
what are some AED keypoints
- assure O2 isn’t following across the pts chess when delivering
- don’t stop chest compressions for more than 10 sec. when assessing the rhythm
- STAY CLEAR
- asses pulse after the 1st 2 min of CPR
- if the en tidal CO2 is <10mmHg during CPR, consider adding a vasopressor and improve chest compressions
key components of post cardiac arrest care
therapeutic hypothermia (32 - 36ºC) - recommended for commatose individuals with return of spontaneous ciculation after a cardiac arrest event
optomization of hemodynamias and ventilation
- 100% O2 is acceptable for early intervention but not for extended periods of time
- oxygen should be titrated, so that the pulse oximeter >94%
- don’t over ventilate
- ventilation rates of 10 - 12 breaths x min to achieve ETCO2 at 35 - 40mmHg
- IV fluids and vasoactive meds should be titrated for hemodyamic stability
Percutaneous Coronary Intevention (PCI):
- preferred over thrombolytics
Neurologic Care:
- neurologic assessment is key
- specialty consultation should be obtained to monitor neurologic signs and symptoms throughout the post resuscitation period
life threatening complications of ACS
ventricular fibrillation pulseless ventricular tachycardia bradyarrythmias cardiogenic shock pulmonary edema
what are the 8 D’s of stoke care
Detection: rapid detention of S&S (<10min)
Dispatch: early activation / 911
Delivery: rapid EMS 10, management and transport
Door: transport to stroke center
Data: rapid triage, eval and management
Decision: stroke expertise and therapy selection
Drug: fibrinolytics therapy, intra-arterial strategies
Disposition: rapid admit. to stroke or critical care unit
what are the stoke team alert criteria
threatened airway or labored breathing
altered mental status
bradycardia (<40BPM) or tachycardia (>100BPM)
seizure
hypotension
symptomatic hypertension
sudden and large decrease in urine output
what are the reversible causes of cardiac arrest
Hypovolemia Hypoxia Hypothermia H+ (acidosis) Hypo/Hyperkalemia
Tension Pneumothorax Tamponade Toxins Thrombosis (coronary / pulmonary) Trauma (unrecognized)
what are the most common causes of pulseless electrical activity
Hypovolemia
Hypoxia
1st line treatment of adult bradycardia with pulse (when there’s altered mental status, shock, chest pain, acute heart failure) in an emergency setting
HR <50
Atropine
- IV: 0.5mg bolus, repeat every 3 - 5 min up to 3mg max dose
if ineffective use:
- transcutaneous pacing
- dopamine infusion (2-10mg/kg/min)
- epinephrine (2 - 10mcg/min)
characteristics of a sinus bradycardia
regular
rate generally 40 - <60bpm
PR interval 0.12-0.20 sec, consistent
QRS complex <0.12 sec
characteristics of a 1ºblock
regular
rate depends on underlying rhythm
p wave >0.20sec, consistent
QRS complex <0.12sec
characteristics of a type I 2ºblock (weckenbach)
rhythm isn’t regular but does have a pattern to it, R-R interval gets longer a PR intervals get longer
ventricular rate is usually slightly higher than the atrial rate due to some atrial beats no being conducted
the atrial rate is usually normal
P wave upright and uniform, doesn’t always proceed a QRS complex
PR interval gets progressively longer until there is a dropped QRS complex
QRS complex <0.12sec