recession and dentine hypersensitivity Flashcards

To enable the student to understand the aetiology, diagnosis and management of gingival recession and dentine hypersensitivity. Define gingival recession and dentine hypersensitivity Describe prevalence and aetiology Understand the possible consequences of recession including dentine hypersensitivity (64 cards)

1
Q

what is the definition of gingival recession

A

Location of the marginal tissue apical to the cemento - enamel junction with exposure of the root surface with a subtle colour change

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2
Q

what are examples of mucoginigval deformities and conditions around the teeth

A
Gingival phenotype
Gingival / soft tissue recession
Lack of gingiva
Decreased vestibular depth
Aberrant frenum / muscle position
Gingival excess
Abnormal colour
Condition of the exposed root surface
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3
Q

what is the prevalence of gingival recession 1mm or more in people aged 30+

A

58%

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4
Q

what happens to prevalence and extent with age

A

increases

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5
Q

what is the prevalence and extent in 30-39 year olds

A

37.8%

and extent of 8.6%

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6
Q

what is the prevalence and extent in 80-90 year olds

A

90.4

and extent of 56.3%

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7
Q

what is the distribution of recession

A

maxillary 6s

and mandibular central incisors

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8
Q

which teeth can suffer from dentine hypersensitivity

A

upper and lower canine

1st premolar and incisor teeth

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9
Q

where can there be greater gingival recession

A

left side of the jaw
in males vs females
and afrocarribeans rather than caucasians

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10
Q

where is good OHI associated

A

with buccal surfaces

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11
Q

and where is poor OHI associated

A

with lingual surfaces of lower anterior teeth

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12
Q

what is the aetiology of gingival recession

A

periodontal disease OR

normal sulcus and undisposed interdental crystal bone

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13
Q

which teeth are more likely to have gingival recession

A

teeth near the buccal surface

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14
Q

how else can we have crestal bone loss

A

by orthodontic movement-

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15
Q

what can orthodontic tooth movement cause

A

dehiscence

greater risk of recession with XS proclination of lower incisors and arch expansion

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16
Q

what is recession based on

A

the volume of soft tissue surrounding the tooth

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17
Q

when is there a greater risk of recession-regarding orthodontics

A

when XS proclination of lower incisors and arch expansion

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18
Q

give examples of trauma

A
foreign objects- lip/tongue piercing
nail biting
hard tooth brushing 
poorly designed dentures- maintained 
trauma from malocclusion 
chemical trauma- cocaine
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19
Q

what is tissue called when its bound to the bone

A

mucoperiosteum

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20
Q

what features of the mucogingival junction can make it more prone to recession

A

thin and less volume of the tissue can make it more likely to recess

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21
Q

what is the local plaque retention factors

A

high muscle attachment

frenal pull

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22
Q

what procedure can we do to increase the volume of tissue

A

gum graft surgery

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23
Q

what can also occur post treatment

A

recession- need to warn patients sometimes

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24
Q

why does smoking have an effect on recession

A

once the smoke is taken in it pools behind the upper anterior teeth- direct effect on tissues

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25
what are the consequences of recession
``` tooth loss bleeding gums plaque retention root caries aesthetics abrasion dentine hypersensitivity ```
26
what is dentine hypersensitivity characterised by
short sharp-pain arising from exposed dentine in response to certain stimuli-which cannot be attributed to any other dental disease or defect
27
what can dentine hypersensitivity go on to do
a dull ache which means the pulp has also been affected
28
when is the peak incidence of dentine hypersensitivity
20-40 years
29
what is the self reported percentage of hypersensitivity
8-30%
30
what is the % of dentine hypersensitivity in more varied practice population
3.8%
31
which gender is more prone to dentine hypersensitivity
females
32
where does dentine hypersensitivity occur more
buccal, labial,cervical areas of teeth
33
which teeth are most affected in order by DH
``` first premolars canines incisors second premolars molars ```
34
what do you need to have for DH
``` DENTINE EXPOSURE(lesion localisation)+tubules made open(lesion initiation)+ stimulus pulp must be vital*** ```
35
what can the stimuli be
thermal( hot or cold- cold more) spicy acid sweet
36
how does DH occur
hydrodynamic theory due to osmosis and fluid in the dentinal tubules or the microorganisms and their metabolites which can penetrate
37
how can we detect DH
by touching- probing tooth brushing
38
what is the hydrodynamic theory
fluid flow causes a pressure change across the dentine | causes distortion A delta fibres pain
39
which fibres cause pain
A delta fibres
40
who thought of the hydrodynamic theory
Brannstrom
41
what else can affect fluid flow in dentinal tubules
width of the tubules
42
what does rate of fluid flow depend on
4TH power of the radius of the tubules
43
why do older people not suffer from DH
due to the fact secondary dentine is deposited in the dentinal tubules so it blocks fluid flow
44
what does sensitive dental tubules show
disrupted smear layer more dental tubules at the surface and not occluded wider tubules
45
what is root sensitivity
DH from gingival recession due to perio disease and treatment
46
what might cause root sensitivity
potentially microorganisms invading root dental tubules
47
dentine exposure can occur from
enamel of enamel by restorative procedues erosion abrasion attrition
48
does toothbrushing alone has an affect on hard tissues
NO | toothpaste has an effect-abrasive effect
49
what might affect teeth indirectly in regard to teeth
toothbrushing technique
50
what might remove the smear layer-regards to toothpaste
abrasive particles | detergents
51
what might tubules be occluded with
with particulate matter from paste
52
when can TSL increase
by toothpaste abrasion if inter oral environment acidic-eg do not brush straight after breakfast
53
what is erosion influenced by
``` pH type chemical strength exposure times other sources of acid ```
54
what do we look at in history examination and diagnosis
record extent of recession descriptive index-rarely used aetiological factors
55
what are the stages of treatment planning
pain management prevent progression perio screening and treatment
56
how do we manage hypersensitivity
tubule occlusion - promotes formation of new tissue eg new smear layer app of artificial barrier blocking pulpal nerve response
57
what are the ideal qualities of barrier materials
``` retentive insoluble penetrate tubules form mechanical tags into tubules seal the end of tubules ```
58
what can we use for home use of managing HS
``` toothpaste gels contain potassium, strontium oxalate and fluoride salts potassium nitrate novamin ```
59
what does novamin release
Ca and P to form a hydroxyapatite like layer
60
what did west et al (1997) show for a placebo effect
40%
61
what is good about strontium acetate
withstands immersion in acid
62
what can we use in surgery to manage HS
VARNISH- DURAPHAT (5%Naf) reinforced GIC 1-3 LAYERS of adhesive resin bonding system desensitizing polish paste calcium carbonate and arginine
63
WHAT CAN WE ADVISE to prevent DH
toothbrushing technique- modified bass technique roll toothbrush electric- with pressure sensor
64
what advice do we give with smoking cessation
ask advise act